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1 cells treated with the antioxidant butylated hydroxyanisole.
2 ntioxidant standards quercetin and butylated hydroxyanisole.
3 mopreventive agents ethoxyquin and butylated hydroxyanisole.
4 oxygen species and was blocked by butylated hydroxyanisole.
7 ROS accumulation by ROS scavengers butylated hydroxyanisole, and N-acetyl-L-cysteine prevented the lu
8 the antioxidants N-acetylcysteine, butylated hydroxyanisole, and pyrrolidine dithiocarbamate and was
10 was very high, similar to that of butylated hydroxyanisole (BHA) and higher than that of other synth
11 Butylated hydroxytoluene (BHT) and butylated hydroxyanisole (BHA) are two commonly used antioxidants
12 al sensor for the determination of butylated hydroxyanisole (BHA) by electropolymerization of O-creso
14 Inhibition of ATO-induced ROS with butylated hydroxyanisole (BHA) does not affect Nrf2 activation or
15 Administration of 0.75% 2(3)-tert-butyl-4-hydroxyanisole (BHA) in AIN-76A diet to female CD-1 mice
20 iet that contained the antioxidant butylated hydroxyanisole (BHA), and others were given daily intrap
21 e synthetic phenolic antioxidants, butylated hydroxyanisole (BHA), butylated hydroxytoluene (BHT) and
23 s of 4 preservatives [acetic acid, butylated hydroxyanisole (BHA), ethylenediaminetetraacetic acid (E
24 rolidinedithiocarbamate (PDTC) and butylated hydroxyanisole (BHA), indicating that the generation of
26 ocopherol, catechin, lycopene, and butylated hydroxyanisole (BHA)] for conjugated dienes (during a 90
27 ct synthetic phenolic antioxidant (butylated hydroxyanisole: BHA), a common ingredient in food additi
28 crylhydrazyl radical (DPPH) assay: butylated-hydroxyanisole, butylated-hydroxytoluene, tert-butylhydr
29 -2 (Nrf2) activators (oltipraz and butylated hydroxyanisole) can induce hepatic Mrp transporters and
30 d Nrf2-null mice with oltipraz and butylated hydroxyanisole demonstrated that the induction of Mrp2,
31 e of cell death, the ROS scavenger butylated hydroxyanisole efficiently blocked TNF-induced necrotic
33 tructurally unrelated antioxidants butylated hydroxyanisole, N-acetyl-L-cysteine, and pyrrolidine dit
34 of CHO cells with the antioxidants butylated hydroxyanisole or butylated hydroxytoluene blocked the t
35 tioxidants N-acetyl-l-cysteine and butylated hydroxyanisole, or an inhibitor of NADPH oxidase, diphen
36 ncluding butylated hydroxytoluene, butylated hydroxyanisole, TEMPOL (4-hydroxy-2,2,6, 6-tetramethylpi
37 a pancaspase inhibitor but not by butylated hydroxyanisole, which inhibits reactive oxygen species.