戻る
「早戻しボタン」を押すと検索画面に戻ります。 [閉じる]

コーパス検索結果 (１語後でソート)

通し番号をクリックするとPubMedの該当ページを表示します
1  with frequent germinal center reactions and hypergammaglobulinemia.
2 ice with AdTACI prevented the development of hypergammaglobulinemia.
3 rentiation, compromised B cell function, and hypergammaglobulinemia.
4 including splenomegaly, lymphadenopathy, and hypergammaglobulinemia.
5  burden, cachexia, massive splenomegaly, and hypergammaglobulinemia.
6 d with B cell activation and exhaustion, and hypergammaglobulinemia.
7 ph nodes; hepatosplenomegaly; and polyclonal hypergammaglobulinemia.
8 hadenopathy, hepatomegaly, splenomegaly, and hypergammaglobulinemia.
9 ntibody-secreting cell (ASC) frequencies and hypergammaglobulinemia.
10 ggerated in aging mice that normally develop hypergammaglobulinemia.
11                                      All had hypergammaglobulinemia (3.0 +/- 1.0 g/dL) and autoantibo
12 ariants have recently been reported to cause hypergammaglobulinemia, abnormal plasma cell differentia
13 with remodeling of lymphoid architecture and hypergammaglobulinemia, although the mechanisms leading
14  on chromosome 1 (peak at 106 cM), linked to hypergammaglobulinemia; an NZB locus on chromosome 5 (Nb
15 eregulated B lineage system, as evidenced by hypergammaglobulinemia and autoantibodies, remains elusi
16         These AND alpha beta T cells induced hypergammaglobulinemia and autoantibody production, as s
17  T cell-intact mice, consisting of augmented hypergammaglobulinemia and autoantibody production, more
18 roles in the development of lupus-associated hypergammaglobulinemia and autoantibody production; howe
19 ous form of this disease is characterized by hypergammaglobulinemia and defective cell-mediated immun
20 be an important factor in driving polyclonal hypergammaglobulinemia and elevated autoantibody titers
21                Immunologic analysis revealed hypergammaglobulinemia and high frequencies of certain l
22  linked (or showed a trend for linkage) with hypergammaglobulinemia and IgG1, IgG2a, and/or IgG3 leve
23 h novel heterozygous CD95 mutations revealed hypergammaglobulinemia and immune-mediated cytopenias in
24 h known hallmarks of HIV-1 infection, namely hypergammaglobulinemia and increased frequencies of peri
25 osomal P autoantibodies and normalization of hypergammaglobulinemia and low C3/C4 levels.
26                       Babies of mothers with hypergammaglobulinemia and normal IgG levels had similar
27 evelopment of lupus nephritis by suppressing hypergammaglobulinemia and renal disease.
28 temic granulomatous disorder associated with hypergammaglobulinemia and the presence of autoantibodie
29 rther, these autoreactive CD4+ T cells cause hypergammaglobulinemia and the production of autoantibod
30 vivo T cell cycling and activation, moderate hypergammaglobulinemia and, at low penetrance, anti-chro
31 minates in lymphadenopathy and splenomegaly, hypergammaglobulinemia, and autoantibodies.
32 or/memory T cells and IgG+ B cells, profound hypergammaglobulinemia, and autoantibody production.
33 f B cells resulting in lymphoid hyperplasia, hypergammaglobulinemia, and autoimmunity in neonatal pig
34 ells in peripheral lymphoid organs, moderate hypergammaglobulinemia, and deposited complexes in the k
35 low, progressive systemic immune activation, hypergammaglobulinemia, and enhanced germinal center for
36 ng immune complexes, immune complex disease, hypergammaglobulinemia, and high levels of antiviral ant
37 stations, including autoantibody production, hypergammaglobulinemia, and immune-complex renal disease
38 /-) mice developed more severe splenomegaly, hypergammaglobulinemia, and immunodeficiency than infect
39  mice develop splenomegaly, lymphadenopathy, hypergammaglobulinemia, and immunodeficiency; thus, this
40 ressive destruction of infected CD4 T cells, hypergammaglobulinemia, and loss of memory B cells.
41 gic sequelae, including lymphoproliferation, hypergammaglobulinemia, and overt systemic autoimmunity.
42 ng splenomegaly, multifocal lymphadenopathy, hypergammaglobulinemia, and plasmacytosis.
43 stream induction of germinal center B cells, hypergammaglobulinemia, and production of antilipid anti
44 hepatitis (based on histologic examination), hypergammaglobulinemia, and production of autoantibodies
45 terized by low sensitivity to bacterial LPS, hypergammaglobulinemia, and reduced secretion of proinfl
46 nuclear cell infiltration, hepatic fibrosis, hypergammaglobulinemia, anti-nuclear and anti-smooth mus
47 se that included leukocytosis, splenomegaly, hypergammaglobulinemia, antinuclear autoantibody and pro
48      Terminal differentiation of B cells and hypergammaglobulinemia are hallmarks of B-cell hyperacti
49 fested by the presence of autoantibodies and hypergammaglobulinemia, as well as clinical/serological
50 n to induce polyclonal B cell activation and hypergammaglobulinemia, as well as increased production
51 but they develop over time plasmacytosis and hypergammaglobulinemia, as well as systemic inflammation
52 mpaired and which may be responsible for the hypergammaglobulinemia associated with HIV disease.
53 uman systemic lupus erythematosus, including hypergammaglobulinemia, autoantibodies against DNA and s
54 model of lupus, including the development of hypergammaglobulinemia, autoantibodies including anti-do
55 sus-like autoimmune disease characterized by hypergammaglobulinemia, autoantibodies, lymphadenopathy,
56 oimmune disease indicator analyzed including hypergammaglobulinemia; autoantibodies including anti-DN
57 habeta T cells in conjunction with unaltered hypergammaglobulinemia, autoantibody production, and imm
58 ted autoimmunity, characterized by increased hypergammaglobulinemia, autoantibody production, and imm
59            All disease parameters, including hypergammaglobulinemia, autoantibody production, glomeru
60 MRL/lpr) mice consists of severe pan-isotype hypergammaglobulinemia, autoantibody production, lymphad
61  SLE in IL-21R-competent Yaa mice, including hypergammaglobulinemia, autoantibody production, reduced
62 to massive splenomegaly and lymphadenopathy, hypergammaglobulinemia, autoimmunity, B-cell lymphocytos
63  all patients presented with autoantibodies, hypergammaglobulinemia, bowel inflammation, dermatologic
64                                              Hypergammaglobulinemia, but not PM, was associated with
65 lele of Roquin develop tumors accompanied by hypergammaglobulinemia by 6 months of age.
66                              The generalized hypergammaglobulinemia characteristic of MRL-Fas(lpr) mi
67             Protein electrophoresis revealed hypergammaglobulinemia consistent with chronic inflammat
68                 The transgenic mice also had hypergammaglobulinemia contributed by elevations of seru
69 which manifest excitotoxic brain lesions and hypergammaglobulinemia, for the presence of AMPA-recepto
70 on, accumulation of double-negative T cells, hypergammaglobulinemia G and A, and autoimmune cytopenia
71  As a result, MAIDS-associated splenomegaly, hypergammaglobulinemia, germinal center formation, and t
72 auses a persistent infection associated with hypergammaglobulinemia, high levels of antiviral antibod
73 ntal IgG transfer efficiency due to maternal hypergammaglobulinemia; however, infants with and withou
74 ediated by elevated maternal IgG levels (ie, hypergammaglobulinemia) in HCMV-transmitting women.
75  syndrome, characterized by isotype-specific hypergammaglobulinemia, incompletely penetrant autoantib
76 features such as, non-necrotizing granuloma, hypergammaglobulinemia, increased local and circulating
77 piglets (PIPs), explaining why PRRSV-induced hypergammaglobulinemia is seen in all major isotypes.
78 racterized by splenomegaly, lymphadenopathy, hypergammaglobulinemia, loss of T and B cell function, a
79 nase-elevating virus of mice, develop severe hypergammaglobulinemia, lymph node adenopathy, and autoi
80 ne retroviruses cause profound splenomegaly, hypergammaglobulinemia, lymphadenopathy, and an immunode
81 ansplacental transfer of IgG3, adjusting for hypergammaglobulinemia, maternal malaria, and infant mal
82 ons, but placental malaria (PM) and maternal hypergammaglobulinemia might interfere with transplacent
83            Some B cell abnormalities include hypergammaglobulinemia, nonspecific B cell activation, c
84  to numerous B cell abnormalities, including hypergammaglobulinemia, nonspecific B cell activation, n
85                                       PM and hypergammaglobulinemia occurred in 60% and 54% of Alexis
86 and chronically infected people, whereas the hypergammaglobulinemia often present during chronic infe
87 cell phenotypic changes, hyperproliferation, hypergammaglobulinemia, or splenomegaly.
88 f a significant increase in splenic IL-4 and hypergammaglobulinemia primarily of the IgG1 isotype, wi
89         Immune defects in these mice include hypergammaglobulinemia, production of multiple autoantib
90 roviral isolate LP-BM5 induces splenomegaly, hypergammaglobulinemia, profound B- and T-cell immunodef
91 0 mutations, including recurrent infections, hypergammaglobulinemia, profoundly reduced lymphocyte ch
92 r Abs and IgG anti-chromatin autoantibodies, hypergammaglobulinemia, renal Ig deposition, and protein
93 l as extensive B cell hyperproliferation and hypergammaglobulinemia, resulting in splenomegaly and ly
94                          We believe that the hypergammaglobulinemia results from the products of thes
95                  Autoimmune features such as hypergammaglobulinemia, rheumatoid factor, and circulati
96 neous infections, plasma cell proliferation, hypergammaglobulinemia, severe deficiencies of leukocyte
97 er characterized by parenchymal destruction, hypergammaglobulinemia, specific autoantibody production
98                     Moreover, the pattern of hypergammaglobulinemia suggests that the requirement for
99 elop profound splenomegaly, lymphadenopathy, hypergammaglobulinemia, terminal B-cell lymphomas, and a
100 netic background, TSAd deficiency results in hypergammaglobulinemia that affects all immunoglobulin (
101                                     Maternal hypergammaglobulinemia was associated with lower cord-to
102                                              Hypergammaglobulinemia was associated with severe immune
103                                              Hypergammaglobulinemia was maintained with a switch from
104 uninfected women, maternal HIV infection and hypergammaglobulinemia were independently associated wit
105 ralized Ag-independent B cell activation and hypergammaglobulinemia with biased expansion of a subpop
106 ally deficient in CD152 (CTLA-4), polyclonal hypergammaglobulinemia with increased levels of systemic

 
Page Top