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1 d to 49% after 3 cycles (P < .001 vs cycle 1 hypericin).
2 the administration of emodin, a precursor of hypericin.
3 hemotherapeutic agents 8-methoxypsoralen and hypericin.
4 the dark to minimize photoactivation of the hypericin.
5 e of pseudohypericin (0.135-3.280 mug/g) and hypericin (0.277-6.634 mug/g) in all the oils, whereas c
9 intravenously injected with (64)Cu-bis-DOTA-hypericin (24 h after laser treatment in treated mice).
14 cence agents (hexylester aminolevulinate and hypericin) and their application to flexible cystoscopy.
15 cycles of hypericin PDT (P < .001 vs cycle 1 hypericin) and to 49% after 3 cycles (P < .001 vs cycle
16 l cultures revealed the gene responsible for hypericin biosynthesis after the administration of emodi
20 plasmon resonance study showed that bis-DOTA-hypericin had higher binding affinity to phosphatidylser
26 all-molecular-weight (64)Cu-labeled bis-DOTA-hypericin in the noninvasive assessment of response to p
32 300 mg of H. perforatum standardized to 0.3% hypericin (n = 27) or a matched placebo (n = 27) 3 times
34 ions were incubated with serial dilutions of hypericin or 8-methoxypsoralen and then stimulated with
35 e 1, patients were randomized 2:1 to receive hypericin or placebo to 3 index lesions twice weekly for
36 to 40% in patients who received 2 cycles of hypericin PDT (P < .001 vs cycle 1 hypericin) and to 49%
37 mized clinical trial indicate that synthetic hypericin PDT is effective in early-stage patch and plaq
44 eration is dependent on the concentration of hypericin used and the dose of light required to photoac
47 hat bioactive polycyclic quinones related to hypericin were, and still are, globally widespread in po