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1 opathy, seizures, autonomic instability, and hyperkinetic movements.
2 ment disorder was observed in 3/21 patients: hyperkinetic movements (1), truncal ataxia (1) and dysto
3 : altered mental status (all), seizures (4), hyperkinetic movements (4), psychiatric features (3), me
4 orphism, progressive psychomotor disability, hyperkinetic movements, and axial hypotonia with variabl
5 variants of CAMK4 found in individuals with hyperkinetic movement disorder and comorbid neurological
7 lonus dystonia syndrome is a childhood onset hyperkinetic movement disorder characterized by predomin
8 individuals from two families affected by a hyperkinetic movement disorder due to homozygous mutatio
10 isorder progresses, a complex hypertonic and hyperkinetic movement disorder is a common phenotype.
13 et torsion dystonia is an autosomal dominant hyperkinetic movement disorder that has recently been li
15 sion dystonia (ITD) is an autosomal dominant hyperkinetic movement disorder with incomplete penetranc
16 se in the first years of life to late-onset, hyperkinetic movement disorder with poor fine motor skil
17 atures, including central visual impairment, hyperkinetic movement disorder, and epilepsy or electroe
18 mental delay and/or intellectual disability, hyperkinetic movement disorder, transient elevation of t
26 ment (49%) to exercise intolerance (25%) and hyperkinetic movement disorders (41%), including dystoni
28 at endogenous mu agonists may play a role in hyperkinetic movement disorders by inducing sustained ac
29 The dystonias are a heterogeneous group of hyperkinetic movement disorders characterised by involun
30 s are still lacking, effective management of hyperkinetic movement disorders demands that physicians
31 therapy for the treatment of hypokinetic and hyperkinetic movement disorders has, however, led to an
33 manifest in parkinsonism or a wide range of hyperkinetic movement disorders including chorea, ballis
34 man mutations in the PDE10A gene manifest in hyperkinetic movement disorders that phenocopy many feat
36 nt for medication-refractory hypokinetic and hyperkinetic movement disorders, and it is being explore
37 azine, show therapeutic efficacy in managing hyperkinetic movement disorders, including Huntington's
38 on studies in animal models and humans with hyperkinetic movement disorders, it is postulated that d
39 elated differences in PD and the most common hyperkinetic movement disorders, namely, essential tremo
40 treat medically intractable hypokinetic and hyperkinetic movement disorders, the course of the palli
51 n the subthalamic nucleus (STN) results in a hyperkinetic movement syndrome, similar to the HD phenot