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1 s), and posterior cerebellum were relatively hypermetabolic.
2 hibit increased fatty acid oxidation and are hypermetabolic.
3 nt to high-fat diet-induced weight gain, and hypermetabolic.
4 ometabolic, whereas E2+/p- mice are lean and hypermetabolic.
5             Mice lacking SCD-1 were lean and hypermetabolic.
6  characteristic curve (AUCs) for determining hypermetabolic (18)F-FDG PET/CT foci that were suspiciou
7  390 enrolled patients, 49% of subjects were hypermetabolic, 30% of subjects were normometabolic, and
8                         PET identified focal hypermetabolic abnormalities in 19 of 22 intracranial me
9                           In particular, the hypermetabolic abnormalities in the brain, evaluated by(
10          In all seven PET scans, the uniform hypermetabolic activity associated with the demarcation
11 that also demonstrates a symmetrical rimlike hypermetabolic activity seen on PET scans.
12 tabolic adrenal masses, gastric ulcer, small hypermetabolic adenopathies, multiple focal bone marrow
13              18F-FDG PET/CT showed bilateral hypermetabolic adrenal masses, gastric ulcer, small hype
14                  Critically ill children are hypermetabolic and in negative NB.
15 is unique population due to their underlying hypermetabolic and inflammatory response following burn
16 glucose levels of 130 mg/dL exert attenuated hypermetabolic and inflammatory responses, as well as si
17 ween 1,5-InsP8 and 5-InsP7 as drivers of the hypermetabolic and p53-elevated phenotypes, we used IP6K
18  biology (immunogenic, benign NF2 wild-type, hypermetabolic and proliferative) that informed therapeu
19 atum, which was heavily weighted (relatively hypermetabolic) and appeared to provide a disease-specif
20                                    The lean, hypermetabolic, and insulin-sensitive E2+/p- phenotype a
21               In contrast, BalphaKO mice are hypermetabolic, and similar to whole body PGC-1alpha nul
22          In SPM single-subject analysis, the hypermetabolic areas were detected only in patients with
23           In SPM single subject analysis the hypermetabolic areas were detected only in patients with
24 ngside (18)F-FDG normalization in previously hypermetabolic areas, was observed after intravenous imm
25                       Thus, instead of being hypermetabolic, as commonly assumed, solid tumours gener
26                                +/p- mice are hypermetabolic at both ambient temperature (21 degrees C
27 peated exposure shifted the hippocampus to a hypermetabolic basal state with concurrent atrophy and p
28 r understand the motor clinical relevance of hypermetabolic brain regions in HD.
29 central in trauma-, burn-, or cancer-induced hypermetabolic catabolism, the mediators are essentially
30 demonstrate that active UC colonoids exhibit hypermetabolic features and cellular stress, specificall
31      The UHR PET image of 1 patient revealed hypermetabolic foci in the cerebellum that were not disc
32 ely shunt oxygenated blood to the imminently hypermetabolic focus or may be due to small local decrea
33                Homozygous mutants are small, hypermetabolic, hyperactive, and infertile, with markedl
34 hermal injury, ameliorates the hyperdynamic, hypermetabolic, hypercatabolic, and osteopenic responses
35                            Further, they are hypermetabolic, hyperphagic and hyperthermic, all consis
36                                      In this hypermetabolic, hypoglycaemic state, propranolol stimula
37 ling of REE suggests that skeletal muscle is hypermetabolic in patients with HIV lipoatrophy.
38               Seriously injured patients are hypermetabolic in the early postinjury period.
39                               At FDG PET, 46 hypermetabolic lesions consistent with tumor were depict
40                                   Fifty-nine hypermetabolic lesions identified on 7 min/bed position
41 eatures on (18)F-FDG PET/CT, such as intense hypermetabolic lesions in lymph nodes and multiple organ
42 y (PET) -computed tomography showed multiple hypermetabolic lesions involving subcutaneous tissue, mu
43  that of PET/CT in allowing the detection of hypermetabolic lesions suspicious for malignancy in pati
44 allows detection and localization of foci of hypermetabolic lesions with high sensitivity because of
45  (7 per patient) were analyzed for number of hypermetabolic lesions.
46 s] had the strongest rhythms, while the most hypermetabolic line had the weakest rhythms.
47 d to predict the presence of vaccine-induced hypermetabolic lymph nodes (v-HLNs) on (18)F-FDG PET/CT
48 ypeptide VGF (nonacronymic) produces a lean, hypermetabolic mouse.
49 many ways mechanistically distinct from both hypermetabolic muscle wasting and denervation-induced mu
50                                              Hypermetabolic, normometabolic, and hypometabolic patien
51                                           In hypermetabolic patients (eg, burns, cancer), the brownin
52                                              Hypermetabolic patients, compared with normometabolic pa
53  Small, non-avid tumors and those within the hypermetabolic, PET-avid brain were falsely negative.
54         The genomic pattern for liver in the hypermetabolic phase after the burn injury involves tran
55            Finally, before hypometabolism, a hypermetabolic phase was identified for some cortical re
56  toward AD involves three adaptive events: a hypermetabolic phase, a prolonged prodromal phase, and a
57 -induced obesity, thereby recapitulating the hypermetabolic phenotype of global SCD1 deficiency.
58 t a disease-associated, cholesterol-related, hypermetabolic phenotype of PMS iNSCs that leads to neur
59 h, endurance, and morphology, attenuates the hypermetabolic phenotype, and enhances insulin sensitivi
60            MEFs lacking Rev-erba exhibited a hypermetabolic phenotype, demonstrating increased levels
61 s metabolism-associated miRNAs, and causes a hypermetabolic phenotype.
62 stored p53 expression but did not affect the hypermetabolic phenotype.
63 SOD1) mouse models of familial ALS possess a hypermetabolic phenotype.
64 uring early adolescence yields a distinctive hypermetabolic prefrontal cortex state that was not obse
65 ly, and the usual interpretation is that the hypermetabolic region represents a seizure focus.
66 re (SPM5) was used to identify all hypo- and hypermetabolic regions in patients with HD relative to c
67                    IGF-I/BP-3 attenuates the hypermetabolic response after thermal injury and may imp
68 rly enteral tube feedings, will decrease the hypermetabolic response and the incidence of sepsis that
69 evere burns are associated with a persistent hypermetabolic response characterized by hyperdynamic ci
70  show that female children exert a decreased hypermetabolic response compared with male children, whi
71 atients exert an attenuated inflammatory and hypermetabolic response compared with males.
72 , early wound closure, and modulation of the hypermetabolic response have decreased morbidity and mor
73                       Severe burns trigger a hypermetabolic response that persists for up to 2 years
74 e, nonsurvivors exhibited a vastly increased hypermetabolic response that was associated with increas
75                                          The hypermetabolic response to severe burn is associated wit
76                   The catecholamine-mediated hypermetabolic response to severe burns causes increased
77              These findings suggest that the hypermetabolic response to thermal injury is maximal in
78 ndrome of skeletal muscle that presents as a hypermetabolic response to volatile anesthetic gases, wh
79  activation during HIV-1 infection induces a hypermetabolic response with increased glucose uptake.
80 nteral nutrition in burn patients blunts the hypermetabolic response.
81  of constitutive proteins, and modulated the hypermetabolic response.
82 tcomes, infection, sepsis, inflammatory, and hypermetabolic responses were determined.
83  with extreme and prolonged inflammatory and hypermetabolic responses, resulting in significant catab
84 ver injury, and that hypoxia subsequent to a hypermetabolic state caused by activated Kupffer cells i
85 ed liver injury, and hypoxia subsequent to a hypermetabolic state caused by activated Kupffer cells p
86 ed, results do not support the notion that a hypermetabolic state contributes to unexplained weight l
87 es the metabolic rate and contributes to the hypermetabolic state in severe illness.
88  putative negative regulator of VDAC1, and a hypermetabolic state that amplified Treg inflammatory re
89                            Thus, we define a hypermetabolic state that incites changes in the epigene
90 pharmacogenetic disorder that manifests as a hypermetabolic state when carriers are exposed to haloge
91 injury and major surgical stress result in a hypermetabolic state with accelerated breakdown of prote
92 he traditional view is that cocaine causes a hypermetabolic state with increased heat production.
93 creased in the septic patients, suggesting a hypermetabolic state with increased protein breakdown.
94 itically ill patients are characterized by a hypermetabolic state, a catabolic response, higher nutri
95            If amyloid were found to induce a hypermetabolic state, this would suggest an alternative
96                     Thermal injury induces a hypermetabolic state, which is reported to increase with
97 oxidation and the detrimental induction of a hypermetabolic state.
98 abolic ceilings vary with respect to whether hypermetabolic states involve relatively short bouts of
99 ue inflammasome and migratory activities and hypermetabolic states.
100        KSHV-MCD activity was associated with hypermetabolic symmetric lymphadenopathy (median maximal
101 an anesthetic-drug-induced, life-threatening hypermetabolic syndrome caused by abnormal calcium regul
102             Approximately half (n = 11) were hypermetabolic; TEE was not different in those with hype
103                   All patients were markedly hypermetabolic throughout acute hospital stay and had si
104                                PET/CT showed hypermetabolic uptake in the skin in all patients, in th
105                                          The hypermetabolic uptake of FDG fell to background levels w
106                             ALS patients are hypermetabolic with increased resting energy expenditure

 
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