ライフサイエンスコーパス: hyperplasia

1 sed differentiation of adipocyte precursors (hyperplasia).

2 HRL histologic results (eg, atypical ductal hyperplasia).

3 indicative of both adipocyte hypertrophy and hyperplasia.

4 ong-term results of PAE for benign prostatic hyperplasia.

5 pib, prevents vein bypass-induced neointimal hyperplasia.

6 n in enterocytes, catalyzing feedforward ISC hyperplasia.

7 the gastric gland and finally gastric gland hyperplasia.

8 of Klf9 including suppression of melanocytic hyperplasia.

9 cytokine expression, and airway goblet cell hyperplasia.

10 As) to determine their role in smooth muscle hyperplasia.

11 function, and the development of endometrial hyperplasia.

12 ypertrophy, and in men with benign prostatic hyperplasia.

13 tion of prostate cancer and benign prostatic hyperplasia.

14 w that NFATC1 suppression leads to lymphatic hyperplasia.

15 d the results suggested the lesion be benign hyperplasia.

16 logic confirmation of parathyroid adenoma or hyperplasia.

17 ciliated cell state, as well as goblet cell hyperplasia.

18 complex dramatically enhanced Notch-induced hyperplasia.

19 alling results in supernumerary NSCs causing hyperplasia.

20 further show that Hic1 deletion leads to MP hyperplasia.

21 portance of non-coding RNAs in smooth muscle hyperplasia.

22 may mediate oncogene addiction in urothelial hyperplasia.

23 Wnt inhibition suppresses p16(INK4a)-induced hyperplasia.

24 -induced airway inflammation and goblet cell hyperplasia.

25 beta-cell tumours (insulinomas) or beta-cell hyperplasia.

26 ion factor, which contributes to goblet cell hyperplasia.

27 esses that result in dramatic beta-like cell hyperplasia.

28 autoantibody MG have thymic lymphofollicular hyperplasia.

29 nts targeted to inhibit localized neointimal hyperplasia.

30 nfection leading to biliary inflammation and hyperplasia.

31 intense inflammation and prominent epidermal hyperplasia.

32 nesis and results in lumen-occlusive intimal hyperplasia.

33 ed by vasoproliferation and endothelial cell hyperplasia.

34 increased cell proliferation and epithelial hyperplasia.

35 ME bacterial clearance and prolonged mucosal hyperplasia.

36 ink glucagon receptor blockage to alpha-cell hyperplasia.

37 mber of acini without individual acinar cell hyperplasia.

38 g diet prevent tamoxifen-induced endometrial hyperplasia.

39 ials for the treatment of congenital adrenal hyperplasia.

40 derwent ultrasound guided RFA of parathyroid hyperplasia.

41 used antiandrogen drugs for benign prostate hyperplasia.

42 r Apc, respectively, leads to severe colonic hyperplasia.

43 on of late scaffold recoil versus neointimal hyperplasia.

44 d its functional deficiency leads to myeloid hyperplasia.

45 ma, immune cell infiltration, and basal zone hyperplasia.

46 results in hyperglucagonemia and alpha-cell hyperplasia.

47 al therapeutic strategy to reduce neointimal hyperplasia.

48 rized by aberrant inflammation and epidermal hyperplasia.

49 ed expansion of dWAT through hypertrophy and hyperplasia.

50 es to immune cell infiltration and epidermal hyperplasia.

51 ell numbers, and inhibits Wnt activation and hyperplasia.

52 (9 of 40 mice, 22.5%) and bladder urothelial hyperplasia (23 of 40 mice, 57.5%).

53 -derived growth factor) that drive beta-cell hyperplasia (a classic feature of obesity).

54 which are preneoplasia atypical adenomatous hyperplasia (AAH), adenocarcinoma in situ (AIS), minimal

55 ercutaneously diagnosed pure atypical ductal hyperplasia (ADH) is an unresolved clinical issue.

56 ape of normal breast tissue, atypical ductal hyperplasia (ADH), DCIS and invasive breast cancer.

57 d the effects of calcification on neointimal hyperplasia after balloon injury in the rat carotid.

58 n about how lipid signaling regulates intima hyperplasia after vascular injury.

59 Women diagnosed with EC or atypical hyperplasia (AH) were offered LS testing.

60 ol mice, with a greater extent of epithelial hyperplasia and a larger tumor burden.

61 ic compartment, resulting in a pronounced MK hyperplasia and a significantly increased extramedullary

62 evelopment of vascular inflammation, intimal hyperplasia and accelerated atherosclerosis.

63 ed as pre-invasive (P1, atypical adenomatous hyperplasia and adenocarcinoma in situ), minimally invas

64 nomas were included: 31 atypical adenomatous hyperplasia and adenocarcinomas in situ (class H1), 64 m

65 significant (threefold) increase in atypical hyperplasia and adenocarcinomas in the small and large i

66 er understand the contribution of basal cell hyperplasia and associated mucosecretory dysfunction to

67 lar functions in disease, especially intimal hyperplasia and atherosclerosis.

68 omplications resulting from benign prostatic hyperplasia and bladder outlet obstruction continue to b

69 ects in such mechanisms likely contribute to hyperplasia and cancer initiation.

70 ge of pathology occurs, including urothelial hyperplasia and cancer, but associated mechanisms and li

71 ogic alterations involved in organ hypo- and hyperplasia and cancer.

72 pithelial cell proliferation associated with hyperplasia and development of de novo mammary tumors.

73 in the small intestines, which showed crypt hyperplasia and dissociation of villous epithelium from

74 OR inhibitor, rapamycin, prevented lymphatic hyperplasia and dysfunction in mice that expressed an ac

75 mechanistically participate in the beta-cell hyperplasia and dysfunction that characterizes this insu

76 ic p16(INK4a) in the mouse epidermis induces hyperplasia and dysplasia, involving high proliferation

77 otein, increased proliferation, and prostate hyperplasia and dysplasia, whereas Nkx3.1(S186A/-) mouse

78 n of Ptch1 in RP progenitors leads to severe hyperplasia and enlargement of the Sox2(+) stem cell com

79 Basal zone hyperplasia and eosinophil inflammation were greater in

80 promote apoptosis but paradoxically induces hyperplasia and fate specification defects in quiescent

81 pathology as evidenced by attenuated biliary hyperplasia and fibrosis.

82 Trp53 loss was sufficient to drive both ICC hyperplasia and formation of multifocal GIST-like tumors

83 ld-type) and Mdr2KO mice and measure biliary hyperplasia and hepatic fibrosis by quantitative PCR and

84 We investigated Ghr's effects on biliary hyperplasia and hepatic fibrosis in Mdr2-knockout (Mdr2K

85 Benign focal nodular hyperplasia and hepatocellular adenoma could be distingu

86 al TLR9 develop small intestinal Paneth cell hyperplasia and higher Paneth cell IL-17A levels.

87 hibit parathyroid cell proliferation causing hyperplasia and hyperparathyroidism.

88 00A8 and S100A9 in psoriasis-associated skin hyperplasia and immune responses using S100a8(-/-) and S

89 meotic)-like (Drosophila)] develop epidermal hyperplasia and impaired epidermal stratification upon a

90 subepithelial fibrosis, airway smooth muscle hyperplasia and increased angiogenesis.

91 an ILC2-driven, IL-13-dependent goblet cell hyperplasia and increased production of mucins (Muc5b an

92 asthmatic mice exhibited reduced goblet cell hyperplasia and increased TGF-beta production.

93 DR is pathologically recognized as bile duct hyperplasia and is commonly observed in biliary disorder

94 xposure to complete darkness reduces biliary hyperplasia and liver fibrosis in bile-duct-ligated (BDL

95 n biopsy (such as atypical ductal or lobular hyperplasia and lobular carcinoma in situ).

96 the lung immune response, induce goblet cell hyperplasia and metaplasia, and mucus hypersecretion in

97 ammation, fibrosis, and subsequent epidermal hyperplasia and molecularly abolished TGF-beta and NF-ka

98 n the clinical management of benign prostate hyperplasia and prostate cancer by predicting pathologic

99 associated with accelerated arterial intima hyperplasia and restenosis after angioplasty, especially

100 n-autonomous IL-1beta-driven mechanism, both hyperplasia and stem cell lineage defects.

101 yndrome mice with other localized regions of hyperplasia and stromal expansion noted in several addit

102 ut not in endothelial cells, reduced intimal hyperplasia and suppressed the SMC synthetic phenotype c

103 Cellular markers of epidermal hyperplasia and T-cell/dendritic cell infiltration were

104 (particularly clobetasol) restored epidermal hyperplasia and terminal differentiation versus minimal

105 Longitudinal single-cell profiling at the hyperplasia and tumor stages uncovered an altered path o

106 veals severe cortex disruption, spindle cell hyperplasia and X-zone expansion.

107 in aberrant NE differentiation, including NE hyperplasias and cancer.

108 oderate dysplasias and invasive SCCs than in hyperplasias and mild dysplasias, although mutations in

109 develop severe AMF accumulation, AEC and BEC hyperplasia, and adenomas in the lung, leading to early

110 2/T helper 17 cytokine response, mucus cell hyperplasia, and airway hyperresponsiveness in vivo.

111 allergen-induced Th2 cytokines, goblet cell hyperplasia, and airway inflammation.

112 strophy and non classical congenital adrenal hyperplasia, and an essential splice site mutation in a

113 tients and 764 patients with benign prostate hyperplasia, and analyzed their effectiveness to discrim

114 ological indices of eosinophilia, epithelial hyperplasia, and angiogenesis by immunohistochemistry an

115 fectively inhibited joint swelling, synovial hyperplasia, and bone destruction in collagen-induced ar

116 Braf(V600E) causes premalignant melanocytic hyperplasia, and Braf(CA)/Pten(-/-) mice, where Braf(V60

117 ients with PCa compared to those with benign hyperplasia, and correlated with high Gleason score in P

118 attern of organ growth, pancreatic beta cell hyperplasia, and elevated plasma insulin and leptin conc

119 ading to intramural neoangiogenesis, intimal hyperplasia, and luminal occlusion.

120 r injury, focal pancreatitis, adrenocortical hyperplasia, and lymphocyte depletion of spleen and lymp

121 ere protected from eosinophilia, goblet cell hyperplasia, and T(H)2 cell infiltration.

122 SEA-induced airway inflammation, goblet cell hyperplasia, and Th2 cytokine production were attenuated

123 eduction in airway inflammation, goblet cell hyperplasia, and Th2 cytokine production, including IL-4

124 Prostate cancer and benign prostatic hyperplasia are common genitourinary diseases in aging m

125 was no difference with regards to neointimal hyperplasia area (P=0.132).

126 late absolute scaffold recoil and neointimal hyperplasia area as assessed by optical coherence tomogr

127 Neointimal hyperplasia area was smaller among patients with LLL <0.

128 stance in peripheral tissues is secondary to hyperplasia, as well as the activation of multiple cellu

129 roducing adenoma (APA) and bilateral adrenal hyperplasia (BAH), remains a matter of debate.

130 cterized by epithelial shedding, goblet cell hyperplasia, basement membrane thickening, subepithelial

131 thritis the synovial tissue undergoes marked hyperplasia, becomes inflamed and invasive, and destroys

132 TLR4 deletion in TPO(high) mice abrogated Mk hyperplasia, BM fibrosis, IL-6 release, extramedullary h

133 is and prostate diseases of benign prostatic hyperplasia (BPH) and prostatitis is uncertain.

134 ronic prostatitis (CPr) and benign prostatic hyperplasia (BPH) are complex inflammatory conditions fo

135 2 patients with symptoms of benign prostatic hyperplasia (BPH) not candidates for surgery or who expl

136 Benign prostatic hyperplasia (BPH) results in a significant public health

137 Benign prostatic hyperplasia (BPH), a nonmalignant enlargement of the pro

138 d in the initial therapy of benign prostatic hyperplasia (BPH), globally.

139 ct symptoms associated with benign prostatic hyperplasia (BPH), the etiology of which is not well und

140 common male neoplasia, and benign prostatic hyperplasia (BPH), which affects approximately 50% of me

141 continence in patients with benign prostatic hyperplasia (BPH).

142 h normal prostate tissue and benign prostate hyperplasia (BPH).

143 patients with prostatitis or prostate benign hyperplasia (BPH).

144 nevus (492 [61%]), benign reactive lymphoid hyperplasia (BRLH) (38 [5%]), nodular conjunctivitis (31

145 600E) activation was sufficient to drive ICC hyperplasia but not GIST tumorigenesis.

146 Mutant embryos display epidermal hyperplasia, but also apical cell extrusions, during whi

147 by eosinophil-rich inflammation, basal zone hyperplasia (BZH), and dilated intercellular spaces, and

148 mainstay of treatment for congenital adrenal hyperplasia (CAH) but has a narrow therapeutic index and

149 Congenital adrenal hyperplasia (CAH), resulting from mutations in CYP11B1,

150 ely treated patients with congenital adrenal hyperplasia (CAH).

151 cellular stress; and prevented B and T cell hyperplasia caused by Bim haploinsufficiency.

152 Conditional deletion of Lsd1 suppressed GC hyperplasia caused by constitutive expression of BCL6 an

153 though early stent thrombosis and neointimal hyperplasia causing vessel renarrowing were key limitati

154 etic deletion of Sema3d leads to parathyroid hyperplasia, causing PHPT.

155 lung adenocarcinomas and bladder urothelial hyperplasia, combined with our previous findings that EC

156 arteries showed a 20% increase in neointimal hyperplasia compared with similarly injured wild-type co

157 Nlrc5(-/-)) mice exhibit more severe intimal hyperplasia compared with wild-type mice after carotid l

158 Benign prostatic hyperplasia contributes to these mechanical stress field

159 ions show that a history of benign prostatic hyperplasia creates mechanical stress fields in the pros

160 anges from baseline in biomarkers (epidermal hyperplasia/cytokines) at days 29 and 71.

161 t aims to exhibit a case of corneal squamous hyperplasia diagnosed via anterior HR-OCT, prior to surg

162 Atypical ductal hyperplasia diagnosed via excisional biopsy was associat

163 nt complete lesion removal), atypical ductal hyperplasia diagnosed with percutaneous needle biopsy sh

164 ainst aneurysm and injury-induced neointimal hyperplasia, diseases linked to loss of vascular smooth

165 Congenital adrenal hyperplasia due to P450 oxidoreductase deficiency result

166 tion of girls affected by congenital adrenal hyperplasia due to P450 oxidoreductase deficiency.

167 ne treatment alone but reduced the epidermal hyperplasia during 12-O-tetradecanoylphorbol-13-acetate-

168 biopsy tissues at different disease stages, hyperplasia, dysplasia, and cancer, and their subsequent

169 ted by an influx of eosinophils, goblet cell hyperplasia, elevated serum Igs, and induction of Th2 cy

170 TB1 expression was associated with epidermal hyperplasia, eosinophil infiltration, less large-cell tr

171 ture psoriatic plaques by inducing epidermal hyperplasia, epidermal cell proliferation, and recruitme

172 ERbeta(crispr-/-) mice there was epithelial hyperplasia, fibroplasia, inflammation, stromal overgrow

173 MCs promote hyperplasia, fibrosis, and vascular cell activation.

174 Follicular nodular hyperplasia (FNH) is a common benign liver tumor for whi

175 esity-related adipose tissue hypertrophy and hyperplasia for health, critical pathways and mechanisms

176 radiofrequency ablation (RFA) of parathyroid hyperplasia for patients with secondary hyperparathyroid

177 body-deficient mice had decreased neointimal hyperplasia formation in vivo.

178 ng have all been associated with goblet cell hyperplasia (GCH) in small studies.

179 TCPOBOP-induced direct hyperplasia has been considered to be CAR-dependent with

180 ultrastructural mitochondrial abnormalities (hyperplasia, hypertrophy, and crystalline arrays) consis

181 pro-restenotic pathologies including intimal hyperplasia (IH), endothelium impairment, and vessel shr

182 iviral delivery of miR-128 prevented intimal hyperplasia in a mouse model of carotid restenosis witho

183 attenuates imiquimod (IMQ)-induced epidermal hyperplasia in adult mice as well as naturally occurring

184 5HTR)1A/1B receptor agonists inhibit biliary hyperplasia in bile-duct ligated (BDL) rats, whereas 5HT

185 eficiency inhibited premalignant melanocytic hyperplasia in Braf(CA) mice but did not affect formatio

186 Enhanced intima hyperplasia in diabetes is mainly due to insulin signali

187 A-223 inhibits dedifferentiation and intimal hyperplasia in diabetic mice by decreasing PDGFRbeta exp

188 ncover any pituitary adenomas or somatotroph hyperplasia in either group.

189 s are alternate initiating events, fostering hyperplasia in gastrointestinal stromal tumours (GISTs),

190 ity is associated with inhibition of intimal hyperplasia in grafted veins, reduced inflammatory respo

191 (+) fibroblasts before attenuating epidermal hyperplasia in K5.TGFbeta1 skin.

192 ment activity, epithelial proliferation, and hyperplasia in MCF7 cells.

193 elivery of circ_Lrp6 shRNA prevented intimal hyperplasia in mouse carotids.

194 zone significantly rescues excessive intimal hyperplasia in Nlrc5(-/-) mice and attenuates the increa

195 mice displayed mild morphologic changes with hyperplasia in prostates, whereas age-matched Pten litte

196 xposed KKAy mice contained marked epithelial hyperplasia in proximal alveolar ducts and adjacent alve

197 found to reduce psoriasis symptoms and skin hyperplasia in S100a8(-/-) and S100a9(-/-) mice.

198 , bronchial hyperreactivity, and goblet cell hyperplasia in the airways.

199 L-C levels by 93 +/- 26% and reduced intimal hyperplasia in the grafted vein by 38 +/- 6.2%.

200 n human and mouse aortic SMCs and neointimal hyperplasia in the mouse.

201 pment as Hdn-heterozygous adult mice exhibit hyperplasia in the right ventricular wall.

202 pk2-HRAS* oncogene, an inducer of urothelial hyperplasia in transgenic mice.

203 ults indicate that maintenance of urothelial hyperplasia in Upk2-HRAS* mice depends on continuous exp

204 SC proliferation in vitro and influences PBG hyperplasia in vivo in the DDC-mediated mouse biliary in

205 omaffin (EC) and Paneth cells, leading to EC hyperplasia, increased serotonin production, and viscera

206 mice lacking Fancd2 had significant biliary hyperplasia, increased serum bile acid concentration, an

207 of the host cell cycle leading initially to hyperplasia, increasing the number of cells to be invade

208 TSS improvement correlated with changes in hyperplasia, infiltrates, and differentiation markers.

209 ocytes and results in decreases in epidermal hyperplasia, inflammatory cytokine release, immune cell

210 Congenital adrenal hyperplasia is a group of autosomal recessive disorders

211 epithelial goblet cell metaplasia (GCM) with hyperplasia is a prominent feature of asthma, but the ef

212 US-guided RFA of parathyroid hyperplasia is a safe and effective method for treating

213 These data suggest that neointimal hyperplasia is accelerated in calcified arteries and tha

214 Here we report that intima hyperplasia is attenuated in SMIRKO mice, but not in SMI

215 However, onset of hyperplasia is considerably delayed implying there are m

216 n vivo, IL-33-induced intestinal goblet cell hyperplasia is dependent on IL-13.

217 airways of patients with asthma, mucous cell hyperplasia is shown to stem from a novel mucous ciliate

218 Juvenile spongiotic gingival hyperplasia (JSGH) is a distinct clinicopathological ent

219 In addition to neointimal hyperplasia, late scaffold recoil contributed significan

220 cluding 89 premalignant atypical adenomatous hyperplasia lesions, 15 adenocarcinomas in situ, and 55

221 astatic pleural effusion and atypical ductal hyperplasia mammary tumor specimens (21MT-1 and 21PT) en

222 s expression of S100As (S100A8, P < .01) and hyperplasia markers (epidermal thickness, keratin 16, an

223 Hyperplasia measures (thickness/keratin 16/Ki67) showed

224 ischemia/reperfusion model and a neointimal hyperplasia model of the femoral artery.

225 my, but their role in TCPOBOP-induced direct hyperplasia, not yet explored, is investigated in the cu

226 d due to concerns about nodular regenerative hyperplasia (NRH) of the liver.

227 , 0.05, and 0.06 mm, whereas mean neointimal hyperplasia obstruction was 4.5+/-2.4%, 5.2+/-3.4%, and

228 generative changes of mature neutrophils and hyperplasia of bone marrow myeloid cells.

229 istinguish children with neuroendocrine cell hyperplasia of infancy (NEHI), surfactant dysfunction mu

230 dermal junction (DEJ) by RCM correlated with hyperplasia of melanocytes in hematoxylin-eosin sections

231 mice, this TS was hypermuscularized, with a hyperplasia of mural cells expressing more contractile p

232 DDC administration causes hyperplasia of PBGs and periductal fibrosis in EHBT.

233 lymphocytes into the skin and hyperkeratosis/hyperplasia of the nonglandular stomach.

234 h collapse of the glomerular tuft and marked hyperplasia of the parietal epithelial cells (PECs).

235 eeks to months after resection, resulting in hyperplasia of the remnant gut, modification of gut horm

236 control patches developed robust neointimal hyperplasia on the patch luminal surface characterized b

237 To avoid hyperplasia or atrophy, organ turnover demands strict eq

238 re uncoupled, and the organ developed either hyperplasia or atrophy.

239 c bladder dysfunction (from benign prostatic hyperplasia or posterior urethral valves) focuses on sym

240 rtilaginous OA conditions including synovial hyperplasia, osteophyte outgrowth and subchondral sclero

241 exhibited a significantly reduced epidermal hyperplasia, oxidative skin damage, and photocarcinogene

242 H felis, Nlrc5(mo-KO) mice developed gastric hyperplasia (P < .0001), splenomegaly (P < .0001), and i

243 howing myosin II activation and Yki-mediated hyperplasia, paradoxically display decreased cortical te

244 In human, rat, and mouse intimal hyperplasia, PCSK6 was expressed by proliferating SMA+ c

245 temic innate immune response that promotes a hyperplasia phenotype in the midgut.

246 gnaling can partially rescue the parathyroid hyperplasia phenotype.

247 n discovered in primary macronodular adrenal hyperplasia (PMAH), a cause of Cushing syndrome.

248 androgen excess, such as congenital adrenal hyperplasia, premature adrenarche and polycystic ovary s

249 ology similar to pulmonary reactive lymphoid hyperplasia (PRLH), a condition associated with EBV in H

250 H)2 and T(H)17/T(H)22 pathways and epidermal hyperplasia/proliferation.

251 to regions of mucus and alveolar-bronchiolar hyperplasia, proliferations of type 2 epithelial cells,

252 asing atherosclerosis, angiogenesis, intimal hyperplasia, pulmonary arterial hypertension, and cardia

253 ntium infection, manifested by reduced crypt hyperplasia, reduced epithelial expression of IL-6 and T

254 t, transgenic mice display massive beta-cell hyperplasia, reflecting a beneficial mitochondria-induce

255 tate samples, consisting of benign prostatic hyperplasia regions and malignant tumors, from 39 prosta

256 nts with moderate to severe benign prostatic hyperplasia-related symptoms.

257 n cardiovascular surgery, however neointimal hyperplasia remains a significant concern, especially un

258 tery embolization (PAE) for benign prostatic hyperplasia remains limited.

259 c switching or its implication in neointimal hyperplasia remains unclear.

260 Epidermal hyperplasia represents a morphologic hallmark of psoriat

261 Pcsk6(-/-) mice showed reduced intimal hyperplasia response upon carotid ligation in vivo, acco

262 in disease characterized mainly by epidermal hyperplasia, scaling, and erythema; T helper 17 cells ha

263 Histologically, the mean goblet cell hyperplasia score was reduced by a statistically signifi

264 ons were obtained between biomarkers and the hyperplasia score.

265 h interleukin (IL)-13 to promote goblet cell hyperplasia showed increased OPN production in response

266 nsion, apical constriction, and Yki-mediated hyperplasia, spectrin mutant cells, despite showing myos

267 were varying findings: nodular regenerative hyperplasia, steatohepatitis, hemosiderosis, cholestasis

268 nitiation of conditions that promote intimal hyperplasia, suggesting a mechanism by which the IL-2/IL

269 at prior to the development of colonic crypt hyperplasia, T3SS-mediated intimate attachment is not re

270 of ZNRF3, but not RNF43, results in adrenal hyperplasia that depends on Porcupine-mediated Wnt ligan

271 y explain the mechanism underlying adipocyte hyperplasia that occurs much later than adipocyte hypert

272 The median neointimal hyperplasia thickness was 0.04, 0.05, and 0.06 mm, where

273 injury, Gdf5-lineage cells underpin synovial hyperplasia through proliferation, are recruited to a Ne

274 16(INK4a) expression is sufficient to induce hyperplasia through Wnt-mediated paracrine stimulation,

275 damage to skin causes epithelial and dermal hyperplasia, tissue expansion, and proliferation-indepen

276 icantly higher than that in benign prostatic hyperplasia tissues, and PRMT5 expression correlates pos

277 tumors at seven stages, from pre-neoplastic hyperplasia to adenocarcinoma.

278 evious work showed that during colonic crypt hyperplasia, type III secretion system (T3SS)-mediated i

279 capitulates many attributes of "usual ductal hyperplasia" (UDH), a common benign mammary lesion.

280 ith increased activity in congenital adrenal hyperplasia variants associated with 17alpha-hydroxyprog

281 Venous neointimal hyperplasia (VNH) at the outflow vein of hemodialysis AV

282 GC hyperplasia was also evidenced by an increase in numbers

283 However, lymph node hyperplasia was clearly visible postviremia but was asso

284 Biliary hyperplasia was induced in rats via bile duct ligation (

285 After balloon catheter injury, neointimal hyperplasia was significantly increased in rats with med

286 e disease-causing gene in congenital adrenal hyperplasia, we now provide a full structural explanatio

287 l/Forty-one patients with congenital adrenal hyperplasia were evaluated by gray-scale and color Doppl

288 Autoimmunity and peripheral lymphoid hyperplasia were found in 43% of 79 patients with PHTS.

289 Varying levels of mammary hyperplasia were seen in 10 of 16 female Proteus syndrom

290 rt showed mild-appearing epithelial squamous hyperplasia, which confirmed the analysis via anterior H

291 importantly, we observed biliary epithelial hyperplasia, which is an indicator of a high-fat diet.

292 The Stk11(CKO) mice develop prostatic hyperplasia with bladder outlet obstruction, most likely

293 atypical lymphoid infiltrates, Kupffer cell hyperplasia with erythrophagocytosis, and an inconstant

294 and effective procedure for benign prostatic hyperplasia with good long-term results for lower urinar

295 l articular cartilage (CC/TAC), and synovial hyperplasia with increased lining cells was found.

296 equently, loss of ILCs resulted in sebaceous hyperplasia with increased production of antimicrobial l

297 arrow (BM) from the sick mice showed myeloid hyperplasia with predominant mature neutrophils, and dec

298 129S1 knockout mice develop postnatal thymic hyperplasia with thymocyte accumulation.

299 Endometrial hyperplasia worsened with age, and all Smad2/3 cKO mice

300 anism has important implications for intimal hyperplasia, wound healing, and fibrotic diseases.