ライフサイエンスコーパス: hypersensitivity

1 ficantly reduced both thermal and mechanical hypersensitivity.

2 n reduces calcineurin inhibitor-induced pain hypersensitivity.

3 ttenuates calcineurin inhibitor-induced pain hypersensitivity.

4 the dorsal root ganglia to promote visceral hypersensitivity.

5 ecific function of beta-1,4-galactan in salt hypersensitivity.

6 osphorylation in DRG neurons to promote pain hypersensitivity.

7 ropathic pain such as mechanical and thermal hypersensitivity.

8 pport a more complex pathobiology of aspirin hypersensitivity.

9 E(2) (PGE(2)) contribute to aspirin-induced hypersensitivity.

10 ed spinal cord microglia activation and pain hypersensitivity.

11 s, which correlated with the aggravated salt hypersensitivity.

12 ation influences susceptibility towards drug hypersensitivity.

13 st cell-related diseases, including allergic hypersensitivity.

14 potential for diagnostic purposes in (drug) hypersensitivity.

15 ntibiotics are the most common cause of drug hypersensitivity.

16 orn interneurons facilitate neuropathic pain hypersensitivity.

17 ferents and reduced incidence of post-injury hypersensitivity.

18 st frequent clinical entity induced by NSAID hypersensitivity.

19 tivity reactions and determine their role in hypersensitivity.

20 mplified DNA damage signaling and checkpoint hypersensitivity.

21 ortant target for treating pathological cold hypersensitivity.

22 immune complexes (ICs) could directly induce hypersensitivity.

23 stic workups for evaluating subjects with BL hypersensitivity.

24 8 deaths, 4 among persons with no history of hypersensitivity.

25 represent an endogenous biomarker for their hypersensitivity.

26 y activation of TRPM3, and inflammatory heat hypersensitivity.

27 as well as inflammation-mediated behavioural hypersensitivity.

28 ted analgesic effects on MO-induced visceral hypersensitivity.

29 iological changes with an endotype of immune hypersensitivity.

30 icantly reduced inflammatory and neuropathic hypersensitivity.

31 er a CD8(+) T-cell response underpinning the hypersensitivity.

32 lpha3 subunit is a pivotal mechanism of pain hypersensitivity.

33 ion of TRPA1, as well as BK- and CFA-induced hypersensitivity.

34 prevent or inhibit allergic and nonallergic hypersensitivity.

35 enuated FK506-induced thermal and mechanical hypersensitivity.

36 of unconventional T cells and their role in hypersensitivities.

37 potential role of unconventional T cells in hypersensitivities.

38 that male and female mice develop mechanical hypersensitivity 24 h after oxaliplatin treatment.

39 idelines provide a risk stratification in BL hypersensitivity according to index reaction(s), as well

40 MDARs) in primary afferents leads to tactile hypersensitivity across skin types, and loss of GABA(A)R

41 n 8 in sensory neurons and causes mechanical hypersensitivity after peripheral injection in vivo Thes

42 or the development of mechanical and thermal hypersensitivity after peripheral nerve injury.

43 racteristic feature of allergies and mediate hypersensitivity against allergens through activation of

44 included "penicillin allergy" or "penicillin hypersensitivity" alone or in combination with "adverse

45 a descriptive analysis, including history of hypersensitivity (anaphylaxis, respiratory allergies, an

46 ons for the molecular basis of metal-induced hypersensitivities and allergies.

47 chanism, and recover correlation between DNA hypersensitivities and TF/Chromatin marks.

48 studies, in patients with a suspicion of BL hypersensitivity and an immediate need for antibiotic th

49 Mast cells are typically linked to immediate hypersensitivity and anaphylaxis.

50 erents, that are responsible for alleviating hypersensitivity and chronic pain.

51 We analyzed abdominal mechanical hypersensitivity and collected tumors and analyzed them

52 ages reduces nerve injury-induced mechanical hypersensitivity and expansion of DRG macrophages in bot

53 yelin degradation product induces mechanical hypersensitivity and global transcriptomics changes in a

54 lin-related key adverse events, and rates of hypersensitivity and injection-site reactions were low.

55 pic tumors significantly reduced nociceptive hypersensitivity and nerve fiber hypertrophy and improve

56 Visceral hypersensitivity and pain result, at least in part, from

57 ents with THC (2 mg/kg) alleviate mechanical hypersensitivity and pain unpleasantness, modify uterine

58 tivated MAP kinase signaling, conferred IL-3 hypersensitivity and sensitized the cells to various MEK

59 ly, neurofibromin depletion causes estradiol hypersensitivity and tamoxifen agonism, explaining the p

60 covery cohort) with a diagnosis of immediate hypersensitivity and the main finding was replicated in

61 ategories based on chromatin features, DNase hypersensitivity and transcription factor localization,

62 t, altered mucosal immune function, visceral hypersensitivity, and abnormal gastrointestinal motility

63 , leading to motility disturbances, visceral hypersensitivity, and alterations in gastrointestinal mi

64 , leading to motility disturbances, visceral hypersensitivity, and altered CNS processing.

65 l body for peripheral nerve injury generated hypersensitivity, and monitored ex vivo reporter biolumi

66 probing (BoP), gingival margin level, dentin hypersensitivity, and percent of pockets converted to si

67 e annotations including histone marks, DNase hypersensitivity, and transcription factor binding.

68 Multiple forms of drug hypersensitivity are strongly linked to expression of a

69 A-DRB1*10:01 increased the risk of immediate hypersensitivity at a genome-wide level (odds ratio, 2.9

70 some 6: 99593111) in the deoxyribonuclease 1 hypersensitivity binding site on chromosome 6q16 causing

71 nsequently, WaTx elicits acute pain and pain hypersensitivity but fails to trigger efferent release o

72 n, NPY-saporin decreased mechanical and cold hypersensitivity, but disrupted neither normal mechanica

73 ine (macrocyclic contrast agent) led to pain hypersensitivity, but neither affected spatial working m

74 The latest classification of NSAIDs hypersensitivity by the European Academy of Allergy and

75 pecific therapeutic strategies for ASD noise hypersensitivity can be challenging.

76 /-) mice demonstrated an exaggerated contact hypersensitivity (CHS) response to oxazolone with increa

77 Neutrophils are essential during contact hypersensitivity (CHS), a common skin allergic disease.

78 tion between Blastocystis infection, colonic hypersensitivity (CHS), behavioral disturbances and gut

79 immunotolerant program in a model of contact hypersensitivity (CHS).

80 Dentin hypersensitivity commonly occurs due to opened dentinal

81 lergy specialists deal with beta-lactam (BL) hypersensitivity demonstrated a significant heterogeneit

82 ffects of toothpaste formulations for dentin hypersensitivity (DH), tested in randomized controlled t

83 from patients with AX-Clav-induced immediate hypersensitivity diagnosed by positive skin test.

84 graphic search on large studies regarding BL hypersensitivity diagnosis was performed by an EAACI tas

85 ommon and important immunologically mediated hypersensitivity disease.

86 IgE causes anaphylaxis in type I hypersensitivity diseases by activating degranulation of

87 G DNMT1 alleviated nerve injury-induced pain hypersensitivities, DRG DNMT1 contributes to neuropathic

88 o and in vivo in an Ag-specific delayed-type hypersensitivity (DTH) model in the cynomolgus macaque (

89 newly developed mouse model of delayed-type hypersensitivity (DTH) to sMRBC.

90 The guanine-rich nuclease hypersensitivity element III(1) present in the upstream

91 n patients with food-dependent NSAID-induced hypersensitivity (FDNIH).

92 d motifs mapped to the genome within DNase I hypersensitivity footprints to characterize regions with

93 Similarly, somatosensory hypersensitivity has also been described in mice lacking

94 However, moisture hypersensitivity has made their manufacture complicated

95 Two mechanisms of ASNase hypersensitivity have been identified in mice.

96 vely, displayed distinct patterns of DNase I hypersensitivity, histone acetylation and NFAT1 transcri

97 portant role in mechanosensation and thermal hypersensitivity; however, little is known about their i

98 red between horses affected with insect bite hypersensitivity (IBH) and control horses.

99 Insect bite hypersensitivity (IBH) is the most common seasonal pruri

100 Hypersensitivity Immunoglobulin E mediated syndromes are

101 2 depletion causes cellular cisplatin and UV hypersensitivity in an epistatic manner with DDB1-DDB2.

102 Chronic constriction injury led to sensorial hypersensitivity in both the short term and long term.

103 -HT(3) antagonist, reversed bladder afferent hypersensitivity in CVH mice.

104 nsory processing in health and mechanisms of hypersensitivity in disease.SIGNIFICANCE STATEMENT Jugul

105 neurial injection produces robust mechanical hypersensitivity in female, but not in male, mice.

106 1Tat peptide reversed thermal and mechanical hypersensitivity in FK506-treated mice.

107 ersensitivity.SIGNIFICANCE STATEMENT Sensory hypersensitivity in fragile X syndrome (FXS) and autism

108 ble cellular targets for alleviating sensory hypersensitivity in FXS and other forms of autism.

109 out-and may be considered a model of sensory hypersensitivity in FXS.

110 f these chemokines or their receptors reduce hypersensitivity in mice with orthotopic tumors, and pat

111 instability (LSI), or aging, induces spinal hypersensitivity in mice.

112 ral and functional mechanisms of nociceptive hypersensitivity in models of chronic pain in vivo SIGNI

113 njection was sufficient to induce mechanical hypersensitivity in naive mice.

114 These mice demonstrate hypersensitivity in pain processing in the absence of ne

115 olonic mast cells appears to induce visceral hypersensitivity in patients with IBS-D.

116 splayed dehydration stress tolerance and ABA hypersensitivity in terms of stomatal closure.

117 n this model, female mice develop mechanical hypersensitivity in the caudal abdomen, mild anxiety-lik

118 staltic motion, fluid secretion and visceral hypersensitivity in the GI tract, and has been implicate

119 pain manifested with thermal and mechanical hypersensitivity in the ipsilateral hind paw.

120 imulation in SCD are indicative of autonomic hypersensitivity in the microvasculature.

121 dence demonstrates circadian rhythms of pain hypersensitivity in various chronic disorders.

122 al Wnt signaling for alleviating nociceptive hypersensitivity in vivo.

123 n factors, enhancer histone marks, and DNase hypersensitivity) in mouse embryonic stem cells (mESCs).

124 tributes to neurogenic inflammation and pain hypersensitivity, in part because of its potentiation do

125 estospongin C attenuated the female-specific hypersensitivity induced by MBP(84-104).

126 odeling and significantly reduced mechanical hypersensitivity induced by peripheral injury as well as

127 n network dysfunction that underlies sensory hypersensitivities is unknown.

128 Visceral hypersensitivity is common in patients with irritable bo

129 demonstrate that the duration of mechanical hypersensitivity is dependent on circulating sex hormone

130 Diagnosis of NSAIDs hypersensitivity is hampered by different factors, inclu

131 Type I hypersensitivity is mediated by allergen-specific IgE, w

132 tion of FODMAPs can induce IBS-like visceral hypersensitivity mediated by fermentation products of in

133 sponses were also observed in a delayed-type hypersensitivity model in which mTORC2 was absent in cut

134 In the delayed-type hypersensitivity (model 1), the HLA-DQ8 transgenic (mode

135 any first asparaginase-associated toxicity (hypersensitivity [n = 13]; osteonecrosis [n = 29]; pancr

136 spected non-steroidal anti-inflammatory drug hypersensitivity (NSAIDH), challenge tests with the culp

137 We show that hypersensitivity of ATM-mutant cells to topotecan or the

138 Hypersensitivity of autophagy gene-deficient cells to IF

139 atalase (katG) overexpression suppresses the hypersensitivity of both strains to H(2)O(2) These resul

140 how that ssDNA replication gaps underlie the hypersensitivity of BRCA-deficient cancer and that defec

141 nset autoimmune diseases related to cytokine hypersensitivity of immune cells.

142 The hypersensitivity of PUT3OE is inhibited by the sos1 and

143 Hypersensitivity of the carotid chemoreceptor leading to

144 ortholog or overexpression of G3BP1 confers hypersensitivity of the male germline to heat stress in

145 Overexpression of PUT3 (PUT3OE) results in hypersensitivity of the transgenic plants to polyamine a

146 that thermal exposure would induce autonomic hypersensitivity of their microvasculature with an incre

147 but not the sensory-discriminative features (hypersensitivity) of the pain experience.

148 hibitors to factor VIII were detected and no hypersensitivity or anaphylaxis events were reported up

149 ug exposure will result in a health benefit, hypersensitivity or both.

150 (ASD) are strongly associated with auditory hypersensitivity or hyperacusis (difficulty tolerating s

151 atients with IBS-D failed to induce visceral hypersensitivity or increase the level of PGE2 in W/W(V)

152 lergic in nature, are low risk for immediate hypersensitivity, or are a potential true allergy that h

153 und's adjuvant-induced thermal or mechanical hypersensitivity, or in partial sciatic nerve injury-ind

154 tor gating deficits, psychostimulant-induced hypersensitivity, or motor impairment.

155 Hypersensitivity pneumonitis (HP) is a complex syndrome

156 Hypersensitivity pneumonitis (HP) is a T(H)1 lymphocyte-

157 Identifying early stages of hypersensitivity pneumonitis (HP) is hampered by variabl

158 d: This guideline addresses the diagnosis of hypersensitivity pneumonitis (HP).

159 ); idiopathic pulmonary fibrosis (PAF, 26%); hypersensitivity pneumonitis (occupational burden, 19%);

160 She was diagnosed as having summer-type hypersensitivity pneumonitis (SHP).

161 with sarcoidosis, beryllium sensitivity, or hypersensitivity pneumonitis and healthy subjects were a

162 No clear effects of gender on rates of hypersensitivity pneumonitis have been shown.

163 A CT appearance resembling hypersensitivity pneumonitis, reflecting the exquisitely

164 nts with sarcoidosis, beryllium disease, and hypersensitivity pneumonitis.

165 ponse evoked by TRPM3 agonists, and the heat hypersensitivity produced by Freund's Complete Adjuvant

166 the mechanisms underlying inflammatory heat hypersensitivity provide a rationale for developing TRPM

167 MDH) is used to describe persons with a drug hypersensitivity reaction (DHR) to at least two chemical

168 83 expression resulted in aggravated contact hypersensitivity reaction accompanied by enhanced CD4(+)

169 A) is a common, potentially life-threatening hypersensitivity reaction associated with (1) a specific

170 ospitals who had experienced a perioperative hypersensitivity reaction clinically suggesting anaphyla

171 The CSD is hypersensitivity reaction due to exposure to complex mot

172 Hypersensitivity reaction rate after allergy skin testin

173 Hypersensitivity reaction rate after switching GBCAs was

174 Anaphylaxis is a severe, systemic hypersensitivity reaction that can be potentially life-t

175 Conclusion Patients with a prior hypersensitivity reaction to a gadolinium-based contrast

176 nd potentially fatal IgE-dependent immediate hypersensitivity reaction to apparently harmless antigen

177 We describe a delayed hypersensitivity reaction to Jorveza(R), a newly develop

178 etween 1997 and 2017 for a suspicion of drug hypersensitivity reaction to NSAIDs.

179 Ten percent developed a neutralizing hypersensitivity reaction to PEGasparaginase, of which 4

180 case report describes a patient with severe hypersensitivity reaction to vancomycin who successfully

181 dered when treating patients with a previous hypersensitivity reaction within the same class of antib

182 xis, a rare and potentially life-threatening hypersensitivity reaction, can occur after vaccination.

183 After a neutralizing hypersensitivity reaction, patients were started with 20

184 ng the effector phase of the classic type IV hypersensitivity reaction.

185 e treating physician to the possibility of a hypersensitivity reaction.

186 unology (EAACI) differentiates between cross-hypersensitivity reactions (CRs), associated with COX-1

187 Drug hypersensitivity reactions (DHRs) are associated with hi

188 Drug hypersensitivity reactions (DHRs) are common, and the sk

189 A correct diagnosis of drug hypersensitivity reactions (DHRs) is very important for

190 adverse reactions-collectively known as drug hypersensitivity reactions (DHRs).

191 e in the number of associated occurrences of hypersensitivity reactions (HSRs).

192 bout the diagnostic approaches for immediate hypersensitivity reactions (IHRs) due to 5-nitroimidazol

193 en SARS or MERS vaccines resulted in vaccine hypersensitivity reactions (VAH), similar to those in hu

194 lobulin E (IgE) and its key role in allergic hypersensitivity reactions against normally harmless sub

195 study was to detect ASNase ICs in mice after hypersensitivity reactions and determine their role in h

196 nd control mice developed comparable contact hypersensitivity reactions and imiquimod-triggered psori

197 drawbacks, including delayed healing, local hypersensitivity reactions and neoatherosclerosis, which

198 group 2 gadolinium-based contrast agents for hypersensitivity reactions and nephrogenic systemic fibr

199 nvestigation of immediate-type perioperative hypersensitivity reactions and to provide practical info

200 Delayed-type, T cell-mediated, drug hypersensitivity reactions are a serious unwanted manife

201 n about the diagnosis and management of drug hypersensitivity reactions due to current and candidate

202 Abacavir is associated with hypersensitivity reactions in individuals positive for t

203 also termed delayed-type or T cell-mediated) hypersensitivity reactions in response to protein-derive

204 lish a better differential diagnosis of drug hypersensitivity reactions in the course of the disease.

205 syndrome (AGS) describes a wide spectrum of hypersensitivity reactions mediated by specific IgE to t

206 17 studies reporting 14 850 administrations, hypersensitivity reactions occurred in 0.3% (31 of 14 85

207 iagnosis and clinical management of the drug hypersensitivity reactions that can potentially occur du

208 was identified in 268 patients with delayed hypersensitivity reactions to beta-lactams.

209 Hypersensitivity reactions to drugs are often unpredicta

210 atients with a clinical history of immediate hypersensitivity reactions to either penicillins or ceph

211 Background Hypersensitivity reactions to gadolinium-based contrast

212 HLA-DRB1*10:01 predisposed to immediate hypersensitivity reactions to penicillins.

213 Hypersensitivity reactions with allopurinol and febuxost

214 (myocarditis, myocardial rupture, neoplasm, hypersensitivity reactions, and immune sensitization).

215 These toxicities include infusion or hypersensitivity reactions, cytokine release syndrome, p

216 ociated with a variety of immune-mediated or hypersensitivity reactions, including immediate (type I)

217 The percentages of patients with hypersensitivity reactions, infusion-related reactions,

218 tic acid adverse events, including immediate hypersensitivity reactions, NSF, and intracranial gadoli

219 Three percent (n=18) had severe hypersensitivity reactions.

220 ccessfully reduced the incidence of abacavir hypersensitivity reactions.

221 inflammation, are the main triggers of drug hypersensitivity reactions.

222 s, naive mice receiving ASNase ICs developed hypersensitivity reactions.

223 lar in the two trial groups, with no notable hypersensitivity reactions.

224 n the occurrence of self-reported penicillin hypersensitivity reactions.

225 IgE antibodies that lead to (at times fatal) hypersensitivity response.

226 t cells in W/W(V) mice restored the visceral hypersensitivity response.

227 specific to rats as similar female-specific hypersensitivity responses were seen in mice, where incr

228 ly on functional regions delineated by MNase hypersensitivity reveals new SNP-trait associations in k

229 p-chlorophenylalanine attenuated mechanical hypersensitivity seen after mTBI.

230 We develop a technique, named MNase hypersensitivity sequencing (MH-seq), to identify genomi

231 ut in pathologic conditions, leading to pain hypersensitivity.SIGNIFICANCE STATEMENT Noxious stimuli

232 a therapeutic strategy to alleviate sensory hypersensitivity.SIGNIFICANCE STATEMENT Sensory hypersen

233 le for this ion channel in inflammatory heat hypersensitivity.SIGNIFICANCE STATEMENT The ion channel

234 Drug hypersensitivity such as severe cutaneous adverse reacti

235 Multiple drug hypersensitivity syndrome (MDH) is used to describe pers

236 Multiple drug hypersensitivity syndrome almost exclusively appeared as

237 al necrolysis, Stevens-Johnson syndrome, and hypersensitivity syndrome.

238 Drug-induced hypersensitivity syndrome/drug reaction with eosinophili

239 initiation and maintenance of the mechanical hypersensitivity that characterizes the neuropathic pain

240 RNA mimetic poly(I:C) likewise produces pain hypersensitivity that is blunted in mice lacking MNK-eIF

241 trum disorder (ASD) is associated with noise hypersensitivity, the suboptimal extraction of meaningfu

242 s exogenous intrathecal CARTp evoked painful hypersensitivity through GPR160-dependent ERK and cAMP r

243 ed an experimental model of ACD (ie, contact hypersensitivity to 2,4-dinitrofluorobenzene) that is me

244 on on macrophages, and restored delayed-type hypersensitivity to a model hapten, consistent with reco

245 oduction data and demonstrates selection for hypersensitivity to a weather cue, high temporal variabi

246 an autoinflammatory disease characterized by hypersensitivity to apoptosis and necroptosis and increa

247 ls are detectable in patients with immediate hypersensitivity to AX-Clav, to assess whether these T-c

248 thus alleviated myeloproliferation and mice hypersensitivity to bacterial challenge.

249 tion through this pathway exposes coincident hypersensitivity to BCL-2 inhibition.

250 Amplification of MET was associated with hypersensitivity to cabozantinib and the combination of

251 sickle cell disease (SCD) exhibit subjective hypersensitivity to cold and heat perception in experime

252 Contact hypersensitivity to dinitrofluorobenzene was examined.

253 inhibited the T(H)1 cell response in contact hypersensitivity to dinitrofluorobenzene.

254 he expression profiles established predicted hypersensitivity to DNA-damaging agents and were associa

255 ions in the ATM tumor suppressor gene confer hypersensitivity to DNA-damaging chemotherapeutic agents

256 vity to ABA, impaired stomatal apertures and hypersensitivity to drought stress.

257 No AEs of hypersensitivity to exebacase were reported.

258 downregulation of striatal D2Rs triggers D1R hypersensitivity to facilitate cocaine locomotor sensiti

259 peroxidation, abnormal iron metabolism, and hypersensitivity to free iron.

260 a defining genetic feature of SCLC, leads to hypersensitivity to Hh ligand in vitro, and during neura

261 w elevated levels of basal IFN signaling and hypersensitivity to IFN-alpha stimulation.

262 ine caused an increase in ongoing firing and hypersensitivity to intracranial-dural and extracranial-

263 This implies a hypersensitivity to low luminance contrast and abnormal

264 (OM) layer of these organisms, resulting in hypersensitivity to many antibiotics.

265 iorally divergent mouse strain that displays hypersensitivity to mechanical stimuli.

266 We show that ATAD5 deficiency leads to hypersensitivity to methyl methanesulphonate (MMS), camp

267 Hypersensitivity to nonsteroidal anti-inflammatory drugs

268 nthesis, which lead to growth inhibition and hypersensitivity to osmotic stress.

269 insufficiency, including premature aging and hypersensitivity to PARPi.

270 s, in vitro reduction of BDNF induced axonal hypersensitivity to PlexinA4 signaling.

271 e aversion after nerve injury despite marked hypersensitivity to punctate mechanical stimuli.

272 xcessive ssDNA, chromosomal instability, and hypersensitivity to replication stress inducers.

273 In this study we investigated whether hypersensitivity to reward might contribute to these pat

274 The findings suggest that hypersensitivity to rewards might be a contributing fact

275 -1, the C. elegans ortholog of ATF4, causing hypersensitivity to rotenone, which was reversible with

276 amage of the mutant and partially rescue its hypersensitivity to salt stress, but not affect NAD conc

277 tents in the mutant were responsible for its hypersensitivity to salt stress.

278 20 and the rice homologous genes resulted in hypersensitivity to salt stress.

279 ghtened susceptibility to peer influence and hypersensitivity to social rejection in adolescence incr

280 ts in severely attenuated growth and confers hypersensitivity to the compounds.

281 Using hypersensitivity to the DNA crosslinking agents, mitomyc

282 Loss of GCN5 also causes hypersensitivity to the fungicidal drug caspofungin.

283 thogenic Mycobacterium smegmatis, results in hypersensitivity to the macrolide-lincosamide class of a

284 A VIG1-deleted mutant shows hypersensitivity to the translation elongation inhibitor

285 Pain hypersensitivity to thermal and mechanical stimuli (P <

286 escribed in neurodevelopmental disorders and hypersensitivity to viral infection.

287 uced ISC function and Lgr5 positivity, drove hypersensitivity to Wnt inhibition, and impaired tissue

288 We hypothesize that the D1R hypersensitivity underlies abuse vulnerability by facili

289 A total of 95 asthma patients with aspirin hypersensitivity underwent sputum induction.

290 uced by mast cells, in mediation of visceral hypersensitivity using mice with the Y385F substitution

291 Electroacupuncture (EA) relieves visceral hypersensitivity (VH) with underlying inflammatory bowel

292 males, while both paw and intrathecal PGE(2) hypersensitivity was more persistent in females.

293 In males, the duration of hypersensitivity was regulated by testosterone.

294 Intraplantar IL-6 induced hypersensitivity was similar in magnitude and duration i

295 induced growth inhibition and osmotic stress hypersensitivity were rescued by treatments with ABA, a

296 s expressing HLA risk alleles do not develop hypersensitivity when exposed to culprit drugs suggestin

297 Loss of functional HLP1 causes HS hypersensitivity, whereas HLP1 overexpressors display in

298 with atypical sensory processing and sensory hypersensitivity, which can lead to maladaptive behavior

299 odulation, significantly reversed mechanical hypersensitivity, with stochastic rate modulation achiev

300 h responses, and the expected frequencies of hypersensitivities within the human population.