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1 motic stabilizer and compatible solute under hypertonic stress.
2 ase in AQP1 luciferase-driven activity under hypertonic stress.
3 lationship also occurs in kidney cells under hypertonic stress.
4 et unknown to be up-regulated in response to hypertonic stress.
5 response shared by all eukaryotes exposed to hypertonic stress.
6 ooperate to induce COX2 expression following hypertonic stress.
7  and NFkappaB was greatly enhanced following hypertonic stress.
8  osmolytes restored their ability to survive hypertonic stress.
9  only PGI2 enhanced RMIC viability following hypertonic stress.
10 died the recovery of human kidney cells from hypertonic stress.
11 r channel protein expression is increased by hypertonic stress.
12 n, but it blocked NRG release in response to hypertonic stress.
13 o increased levels of AQP1 expression during hypertonic stress.
14 etween medullary COX1 and COX2 expression in hypertonic stress.
15 G) pathway functions to sense and respond to hypertonic stress.
16 ctors, phorbol ester, calcium ionophore, and hypertonic stress.
17 egies to prevent internal dehydration during hypertonic stress.
18 P A1 subcellular localization in response to hypertonic stress.
19 onsequently delays caspase activation during hypertonic stress.
20 +)/2Cl(-) cotransporter (NKCC) and RVI under hypertonic stress.
21 egulates its subcellular localization during hypertonic stress.
22 s that regulate hnRNP A1 localization during hypertonic stress.
23 found for the zinc-finger protein ZAC1 under hypertonic stress (219-fold, p < 0.001).
24 8-10 h following exposure to acute sublethal hypertonic stress (550 mosmol/kg H(2)O).
25                                 We find that hypertonic stress allows MEN mutants to exit from mitosi
26  activation of TNF gene transcription during hypertonic stress alone.
27                                              Hypertonic stress also increases system A activity as a
28 st that Nup88 is up-regulated in response to hypertonic stress and acts to retain TonEBP in the nucle
29                       p38 mRNA is induced by hypertonic stress and is attenuated with p38 kinase inhi
30 ngly suggest that ZAC1 is up-regulated under hypertonic stress and negatively regulates expression of
31 s study demonstrates that AQP5 is induced by hypertonic stress and that induction requires activation
32 ssive accumulation of betaine may counteract hypertonic stress and thus attenuate hypertonicity-induc
33 was selectively activated in MLE-15 cells by hypertonic stress, and inhibition of ERK activation with
34 inhibitors increased RMIC survival following hypertonic stress, and transduction of RMICs with a cons
35 mal growth conditions, but when subjected to hypertonic stress, become spheroidal in shape and growth
36 ter (P(gpdh-1)::GFP) is detected only during hypertonic stress but is not induced by other stressors.
37 at coordinates the intracellular response to hypertonic stress but was not previously implicated in t
38 HOG1 does prevent Clb2p-Cdc28p inhibition by hypertonic stress, but does not block Cdc28p phosphoryla
39 pithelium in the medulla of the kidney under hypertonic stress by correctly localizing Cldn4 to the t
40 urthermore, lens epithelial cells respond to hypertonic stress by raising taurine transport activity.
41 ys a key role in protecting renal cells from hypertonic stress by stimulating transcription of specif
42 -GlcNAc activity and function, we found that hypertonic stress can upregulate the expression of O-lin
43                                              Hypertonic stress caused mRNA levels, and primarily that
44                                              Hypertonic stress causes a decrease in CLB2 mRNA, phosph
45                  A novel technique involving hypertonic stress causes membrane 'blebbing' of the Xeno
46 issociation of mRNA stress granules (SGs) in hypertonic-stressed cells and the role of compatible osm
47                               In response to hypertonic stress, cells need rapid electrolyte influx t
48 t induction of active p53 in mIMCD3 cells by hypertonic stress contributes to cell survival.
49 ion of the G(2) delay in IME cells after the hypertonic stress created by adding NaCl.
50                 In contrast, the response to hypertonic stress does not involve eukaryotic initiation
51                                   A 450 mOsm hypertonic stress elicited 2-fold Ca2+ transients that w
52                                              Hypertonic stress-elicited TRPV1 channel stimulation med
53 at a stress cycle consisting of deflationary hypertonic stress followed by an inflationary hypotonic
54 ia rapidly induced c-fos expression, whereas hypertonic stress had no effect.
55 ical if cells are to combat shrinkage during hypertonic stress; however, how phosphorylation accelera
56                                              Hypertonic stress (HS) can alter the function of mammali
57                               In this study, hypertonic stress, hyperthermia and hydrogen peroxide al
58 ntribute to proper nuclear segregation after hypertonic stress in cells that lack Swe1p.
59 responsive enhancer, which was refractory to hypertonic stress in fibroblasts lacking NFAT5, establis
60          We showed that WNK1 is activated by hypertonic stress in kidney epithelial cells and in brea
61 in vivo and reduce their ability to tolerate hypertonic stress in vitro.
62 tion was higher during oxidative stress than hypertonic stress, in agreement with a dramatic decrease
63                                              Hypertonic stress increased PGI2 synthesis 330% above ba
64                     We previously found that hypertonic stress increases PIP(2) by selectively activa
65            Exposure of C2C12 muscle cells to hypertonic stress induced an increase in cell content of
66                                              Hypertonic stress induced in inner medullary (IMCD3) cel
67                                              Hypertonic stress induced the eIF2alpha phosphorylation-
68                      These results suggest a hypertonic stress-induced cell cycle delay in G2 phase t
69 cal PKC was required for TPA-induced but not hypertonic stress-induced cleavage of all EGF family lig
70                                              Hypertonic stress-induced dephosphorylation is blocked b
71  Thus, deletion of SWE1 does not prevent the hypertonic stress-induced inhibition of Clb2p-Cdc28p kin
72 WNK1 inhibition or Piezo1 knockdown, whereas hypertonic stress-induced phosphorylation is not affecte
73 isotonic conditions involves CRM-1 but under hypertonic stress is CRM1-independent.
74 ay, in addition to its role in responding to hypertonic stress, is required at a basal level for the
75                                 For example, hypertonic stress led to strong cleavage of HB-EGF and N
76  mimic of diacylglycerol and PKC activator), hypertonic stress, lysophosphatidic acid (LPA)-induced G
77 under hypertonic conditions, suggesting that hypertonic stress may act through the Thr866 site.
78              Upon exploring the mechanism of hypertonic stress on eNOS O-GlcNAc activity and function
79     Under conditions of nutrient limitation, hypertonic stress or elevated temperature, spm1 delta ce
80 ompare tRNA cleavage patterns in response to hypertonic stress, oxidative stress (arsenite), and trea
81 hysiological stimuli of ectodomain cleavage--hypertonic stress, phorbol ester, or activation of G-pro
82 ropose that eIF2alpha phosphorylation during hypertonic stress promotes apoptosis by sequestration of
83 ys a key role in the mechanism through which hypertonic stress regulates the function of T cells.
84 ralized signaling principle in the mammalian hypertonic stress response relevant to aquaporin express
85 em the chondroitin synthesis pathway and the hypertonic stress response.
86 stent with calcineurin-mediated induction of hypertonic stress-response genes, and they suggest that
87 ddition of bacterial sphingomyelinase, or by hypertonic stress, S358 is rapidly dephosphorylated.
88 rbitol from glucose during hyperglycemic and hypertonic stress, the aldose-reducing property of AR ha
89     Consequently, cells actively counter the hypertonic stress through regulatory volume increase (RV
90  recruited to membranes and was activated by hypertonic stress through Ser/Thr dephosphorylation.
91                                              Hypertonic stress triggers a cell cycle delay in G2 phas
92 that GSK 3beta inhibitors protect RMICs from hypertonic stress via induction of NFkappaB-COX 2-depend
93 the transcriptional activation of aqp1 under hypertonic stress, we examined the role of the transcrip
94 igating the relationship between insulin and hypertonic stress, we have discovered that osmotic shock
95 f system A activity and SNAT2 mRNA levels by hypertonic stress were independent of eIF2alpha phosphor
96  increased O-GlcNAc modifications induced by hypertonic stress were reversed.
97 tes were important for infection we utilized hypertonic stress, which prevents formation of CPSF6 con