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1 neuroendocrine tumors, which are known to be hypervascular.
2 ls), as was adhesion severity (17% thick and hypervascular adhesions vs 46%).
3 es typically were irregular, hypoechoic, and hypervascular and demonstrated indistinct margins or an
4                                    FNHs were hypervascular and hyperattenuating to liver on 106 of 10
5 ts with advanced hepatocellular carcinoma, a hypervascular and VEGF-rich tumor type, were treated wit
6 ncreatitis showed in the majority of cases a hypervascular appearance in the early arterial phase of
7 in the prostate and may improve depiction of hypervascular cancer.
8                          Current therapy for hypervascular cancers, e.g., hepatocellular carcinoma, i
9 Clear cell RCCs and oncocytomas tended to be hypervascular, chromophobe lesions and angiomyolipomas t
10 hat recapitulates the extensive invasive and hypervascular features of glioblastoma (GBM) is a major
11                                              Hypervascular focal lesions with high maximal enhancemen
12 sociated architectural distortion) and has a hypervascular, hemorrhagic, and heterogeneous appearance
13 endothelial growth factor (VEGF)-transfected hypervascular human melanoma xenografts and their nontra
14                     The mass was prominently hypervascular in color Doppler ultrasonography scan.
15    CT scan of the abdomen confirmed multiple hypervascular lesions and central areas of necrosis with
16 ry endolymphatic sac tumors are destructive, hypervascular lesions that arise from the temporal bone
17                         The CNR of simulated hypervascular liver lesions can be substantially increas
18 ely evaluate, for the depiction of simulated hypervascular liver lesions in a phantom, the effect of
19                     Twenty-nine patients had hypervascular liver lesions smaller than 2 cm that could
20 with various iodinated solutions to simulate hypervascular liver lesions was scanned with a 64-sectio
21 etection of statistically significantly more hypervascular liver metastases than do HAP plus PVP imag
22  for detecting hepatocellular carcinomas and hypervascular liver metastases.
23 n; age range, 35-77 years) with 60 malignant hypervascular liver tumors (mean diameter, 20.1 mm +/- 1
24 hnique improves the conspicuity of malignant hypervascular liver tumors during the late hepatic arter
25 nge, 51-77 years) known or suspected to have hypervascular liver tumors underwent dual-energy 64-sect
26 ificity for differentiating hemangiomas from hypervascular malignant tumors.
27                             In patients with hypervascular metastases, double-echo conventional SE im
28 f conventional SE imaging for evaluation of "hypervascular" metastases (n = 9).
29                        Abnormalities such as hypervascular nodules are often observed; in the presenc
30 imaging cirrhosis is the characterization of hypervascular nodules smaller than 2 cm, which often hav
31 athway is therefore believed to underlie the hypervascular phenotypes of the VHL tumors.
32                      Glioblastoma (GBM) is a hypervascular primary brain tumor with poor prognosis.
33 nts with biopsy-proved liver metastases from hypervascular primary tumors other than hepatocellular c
34         At color Doppler US, six masses were hypervascular, seven were isovascular, and five were hyp
35 ted with a mixed enhancement pattern of both hypervascular soft-tissue components and low-attenuation
36                 Childhood neuroblastoma is a hypervascular tumor of neural origin, for which antiangi
37                         Invasion of IJV with hypervascular tumor thrombosis is an extremely rare cond
38 ary thyroid carcinoma which invades IJV with hypervascular tumor thrombus.
39 and invading the left jugular vein wall with hypervascular tumor thrombus.
40  in renal clear cell carcinoma, a clinically hypervascular tumor unlikely to be constrained by nutrie
41                    Two patients had solitary hypervascular tumors and liver metastases.
42 and 1.7-fold higher oxygen saturation in the hypervascular tumors as compared with the control tumor
43 -Lindau (VHL) develop renal cell cancers and hypervascular tumors of the brain, adrenal glands, and p
44 nt for inoperable hepatic tumors, especially hypervascular tumors such as hepatocellular carcinoma.
45                           The failure of the hypervascular VEGF188-expressing tumors to grow may be d