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1 ors were heterogeneous and depicted areas of hypervascularity.
2 poxia reversed the polycythemia and cerebral hypervascularity.
3 o the etiology of Peters' anomaly and ocular hypervascularity.
4 eatures of the adenomas included evidence of hypervascularity (63%), intratumoral fat (50% of patient
5 atment of ovarian disorders characterized by hypervascularity and hyperplasia.
6 duration, allowing for detection of arterial hypervascularity and portal venous washout.
7 y blocked the cellular infiltrative pattern, hypervascularity, and cell proliferation in vivo, and de
8 en, and led to mesenchymal cell hyperplasia, hypervascularity, and inflammatory cell infiltration.
9 T2-hypointense bands, uterine bulge, serosal hypervascularity, and signs of extrauterine placental sp
10                    Color Doppler US-depicted hypervascularity correlated with biologically aggressive
11 c lesions around the mediastinum testis with hypervascularity dispersing in ten patients and hypovasc
12                  Despite marked induction of hypervascularity, HIF-1alpha did not induce edema, infla
13 creased rCBV in lymphomas is probably due to hypervascularity in foci of active tumor growth; and dec
14 ger than 2 cm shows typical features of HCC (hypervascularity in the arterial phase and washout in th
15 essential role in extensive infiltration and hypervascularity in the xenograft model.
16                                              Hypervascularity induced by HIF-1alpha could improve the
17                                         This hypervascularity is evident by both gross and microscopi
18 tumors), propensity for lung metastases, and hypervascularity may make these tumors valuable models f
19                              Hyperplasia and hypervascularity of the ovarian theca interna and stroma
20 us autofluorescence, middle to outer retinal hypervascularity on OCT angiography, early hyperfluoresc
21 n in vascularity was the measure of success, hypervascularity was a strong indicator of success (P <.
22 The synovial lining layer thickness, but not hypervascularity, was significantly reduced.
23                  Methotrexate did not reduce hypervascularity, which is a prominent differentiating f
24 lay a role in the ability of HIF-1 to induce hypervascularity without excessive permeability.