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1 afish larvae, whereas nmu mutant animals are hypoactive.
2  The deep tendon reflexes were symmetrically hypoactive.
3  lacking rostral brainstem acetylcholine are hypoactive.
4           Most cases of delirium were of the hypoactive (46%) and mixed (45%) subtypes; only 8% of de
5 of highly edited mRNA variants (which encode hypoactive 5-HT2CR receptors) in the brains of suicide v
6 ccumbal neuronal ensembles become profoundly hypoactive across the development of heroin seeking and
7 y suggest that midbrain dopamine neurons are hypoactive after prolonged cocaine exposure, a state tha
8 ctive aggression in youths with CU traits to hypoactive amygdala responses to emotional distress cues
9                             Mice displayed a hypoactive and anxious phenotype during behavioral testi
10 oxylase (TH) in dopaminergic neurons, become hypoactive and aphagic and die by 4 weeks of age.
11 wever, these mice developed seizures, became hypoactive and approximately 30% of them died by 1 year
12   We allowed for interaction between days of hypoactive and hyperactive delirium and adjusted for bas
13  to examine the associations between days of hypoactive and hyperactive delirium with cognition outco
14                          Interestingly, both hypoactive and hyperactive ZAP70 can lead to the develop
15                            These mice become hypoactive and hypophagic and die of starvation by 4 wee
16 eurons, are born normal but gradually become hypoactive and hypophagic, and die at 3 weeks of age.
17               Moreover, BLA-BNST neurons are hypoactive and less excitable in females.
18       The mutant animals do not feed and are hypoactive and markedly hypotonic.
19  (44%), mixed (57%), or equal percentages of hypoactive and mixed delirium (43%) as most prevalent.
20 n (PV) INs and their immature precursors are hypoactive and transiently decoupled from excitatory neu
21 , motoric subtypes of delirium (hyperactive, hypoactive), and the association of delirium with dement
22 ive and, interestingly, the S147P protein is hypoactive as compared to the DUSP5 WT protein in two di
23 es and precocious neurodevelopment, yielding hypoactive, asynchronous neural networks.
24  opioid, remifentanil, causes a long-lasting hypoactive basal state evidenced by a decrease in ex viv
25                              In females, the hypoactive basal state is driven by a reduction in excit
26                                     Abnormal hypoactive behavior was manifested by 4.5-mo-old Naglu-/
27 bladder afferent pathways, thereby improving hypoactive bladder function in diabetes.
28                           The emergence of a hypoactive, but not hyperactive basal state following re
29 gically enhance calcium signaling within the hypoactive capillary endfeet while reducing the hyperact
30                        We defined a day with hypoactive delirium as a day with positive Confusion Ass
31 f 11 studies reporting on subtype identified hypoactive delirium as most prevalent (46-81%) with each
32 equally distributed between the groups, with hypoactive delirium most frequent (61%), followed by mix
33                               Survivors of a hypoactive delirium subtype performed significantly bett
34 mixed, 10 (11.5%) hyperactive and 26 (29.9%) hypoactive delirium subtypes.
35                       Each additional day of hypoactive delirium was associated with higher instrumen
36                           Longer duration of hypoactive delirium was associated with worse global cog
37                           Longer duration of hypoactive delirium was independently associated with wo
38                                              Hypoactive delirium was more common and persistent than
39 of 556 adults with a median age of 62 years, hypoactive delirium was more common than hyperactive (68
40                           Longer duration of hypoactive delirium, but not hyperactive, was associated
41  26-52%, with a significant percentage being hypoactive delirium.
42                                        Other hypoactive DeltaVD mutants formed stable and characteris
43                   The brain is assumed to be hypoactive during cardiac arrest.
44 onically hyperactive in mutants, but becomes hypoactive during social behaviour.
45 g striatal projection neurons (D2-SPNs) were hypoactive during synchronous cortical slow-wave activit
46 quire a combination therapeutic strategy for hypoactive endfeet and astrocytic hyperactivity.
47                          The pivotal role of hypoactive endogenous fibrinolysis in the occurrence of
48                         Transgenic mice with hypoactive EPO receptor (EPOR) signaling (hWtEPOR) were
49                                         This hypoactive form of ANTP, but not the alanine-substituted
50   Delirium is a common event, especially the hypoactive forms in the elderly.
51  the agitated (hyperactive) and nonagitated (hypoactive) forms of delirium are harbingers of impendin
52 lementation assay to measure both hyper- and hypoactive GCK variation, capturing 97% of all possible
53 s using elastic net regression and found the hypoactive group exhibited higher reflection, lower in-s
54  a largely hyperactive compared to a largely hypoactive group.
55 t (i) A3B expressed in human cells exists in hypoactive high-molecular-weight (HMW) complexes, which
56      Pediatric delirium can be classified as hypoactive, hyperactive, and mixed.
57     Mice without CB1 receptors are extremely hypoactive in a test for exploratory behavior (open-fiel
58 ge-sensitive superior temporal cortices were hypoactive in ASD toddlers with poor language outcome.
59 nt findings that the default mode network is hypoactive in autism, our data raise the possibility tha
60 re hyperactive in proximal CA3, but possibly hypoactive in distal CA3, compared with young (Y) rats.
61 egulates fear memories and is reported to be hypoactive in PTSD.
62 he cardiac surgery ICU and was predominately hypoactive in subtype.
63 endfeet were as active as fine processes but hypoactive in the presence of amyloid plaques, while the
64                   Depletion of OLA1 caused a hypoactive ISR and greater survival in stressed cells.
65        The mice with the small deletion were hypoactive like the large deletion mice and they also sh
66                                Specifically, hypoactive medial OFC and hyperactive right hippocampus
67                                        Under hypoactive neuronal conditions, microglia also exhibited
68 adherin positive tumor cells by recruiting a hypoactive NKG2D(-ve) NK population, phenotypically remi
69 % of patients near the end of life develop a hypoactive, nonagitated delirium.
70 ations in mice and humans with a spectrum of hypoactive or hyperactive activities, we have gained ins
71 e engineered deletion or the duplication are hypoactive or hyperactive, respectively.
72 dentified 21% with delirium, 88% of whom had hypoactive or normal psychomotor features.
73                                    A similar hypoactive pattern was found during early conditioning (
74                     Both the hyperactive and hypoactive phases of motor dysfunction preceded the dete
75                        These DCs exhibited a hypoactive phenotype with low CD40, MHC II, CD80/CD86 ex
76 in increased SFK activity, but paradoxically hypoactive platelets resulting from negative feedback me
77 estored coagulation ex vivo in patients with hypoactive platelets.
78 yperactivation in the neutral condition, but hypoactive prefrontal (ventromedial and lateral prefront
79 he combination of a hyperactive amygdala and hypoactive prefrontal regions is associated with diminis
80                Furthermore, compensating for hypoactive prelimbic cortex neurons with in vivo optogen
81  salinities, directly exposed F0 larvae were hypoactive relative to the F0 controls; however, the ind
82 ntaneous locomotor activity were found to be hypoactive relative to young animals.
83                    We classified delirium as hypoactive (Richmond Agitation and Sedation Scale 0) or
84 n receptors for the treatment of generalized hypoactive sexual desire disorder (bremelanotide) and er
85 int worldwide, current treatment options for hypoactive sexual desire disorder (HSDD) are limited in
86        Distressing low sexual desire, termed Hypoactive Sexual Desire Disorder (HSDD), affects approx
87 re; the most prevalent psychological form is hypoactive sexual desire disorder (HSDD), which affects
88 for FSD are available, specifically to treat hypoactive sexual desire disorder, female sexual arousal
89                                Additionally, hypoactive shifts in neuronal activity (isoflurane anest
90 tosensory and auditory cortex shifted from a hypoactive state in P45 to hyperactivity in P120.
91           Chemogenetic compensation for this hypoactive state prior to testing restored cognitive fle
92 esting restored cognitive flexibility, basal hypoactive state, and remifentanil-induced plasticity.
93 wing 10-16 days of self-administration, with hypoactive states arising only following 25-30 days of s
94                    Alternatively, hyper- and hypoactive states in males align selectively with decrea
95 ulated by phosphorylation in both hyper- and hypoactive striatal DA neurons; in the latter case, acti
96               Nearly all (92%) exhibited the hypoactive subtype of delirium.
97 ereas SLC10A4 null mutant mice were slightly hypoactive, they displayed hypersensitivity to administr
98 ermined activity scores, we find that 43% of hypoactive variants also decrease cellular protein abund
99                                              Hypoactive variants are concentrated at buried positions
100         Delirium symptoms were nearly always hypoactive, were detected mean 6 days after intracerebra
101                                              Hypoactive ZAP70 function compromises key developmental

 
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