ライフサイエンスコーパス: hypoalbuminemia

1 35 g/L as a cutoff, 53% of the subjects had hypoalbuminemia.

2 models to identify independent predictors of hypoalbuminemia.

3 t which time they had hyperbilirubinemia and hypoalbuminemia.

4 e edema of malnutrition is not solely due to hypoalbuminemia.

5 serum alkaline phosphatase and amylase, and hypoalbuminemia.

6 , which may prevent ALBU release, leading to hypoalbuminemia.

7 tensive shock, severe hemoconcentration, and hypoalbuminemia.

8 alcium status, especially in the presence of hypoalbuminemia.

9 nts with significant weight loss, edema, and hypoalbuminemia.

10 rgency hematopoiesis, increased D-dimer, and hypoalbuminemia.

11 ht gain, hepatic transaminase elevation, and hypoalbuminemia.

12 A total of 27.3% of the RHD patients had hypoalbuminemia.

13 e predicted by disease severity at onset and hypoalbuminemia.

14 elevated liver transaminases, azotemia, and hypoalbuminemia.

15 d with significant improvement in anemia and hypoalbuminemia.

16 panied by significant weight loss and severe hypoalbuminemia.

17 y toxicity in pregnant women with anemia and hypoalbuminemia.

18 on deficiency along with hypoproteinemia and hypoalbuminemia.

19 ollapse accompanied by hemoconcentration and hypoalbuminemia.

20 k, acute restoration of blood volume, and in hypoalbuminemia.

21 with proteinuria and not as a consequence of hypoalbuminemia.

22 ny cases of hypocalcemia are the artifact of hypoalbuminemia.

23 significant amelioration of proteinuria and hypoalbuminemia.

24 old increase in risk of death independent of hypoalbuminemia.

25 nd alkaline phosphatase (8% and 18%) levels; hypoalbuminemia (10% and 19%); hyperbilirubinemia (10% a

26 l pain (70%), significant weight loss (92%), hypoalbuminemia (100%; 85% lower than 2.0 g/dL), and ane

27 ients (42.4% v 33.2%, P = .023) secondary to hypoalbuminemia (14.1% v 7.9%, P = .023), HIV (3.1% v 0.

28 ight loss (22.0% vs 4.0%, P < 0.001), severe hypoalbuminemia (18.6% vs 5.2%, P < 0.001), and cytopeni

29 sus 0.1 +/- 0.1, mutant versus control), and hypoalbuminemia (2.1 +/- 0.4 versus 2.5 +/- 0.2 G/dl, mu

30 ted prothrombin time (18 +/- 3 seconds), and hypoalbuminemia (2.7 +/- 0.6 g/dL).

31 Those with severe (0.5-2.9 g/dL) and mild hypoalbuminemia (3.0-3.6 g/dL) had posttransplant adjust

32 leukocytosis (2.85 [hazard ratio]; P = .02), hypoalbuminemia (3.41; P = .05), and anti-Saccharomyces

33 RAEs-the most common were pruritus (38%) and hypoalbuminemia (31%); confirmed ORR was 26%, with respo

34 second prolongation; P < .001), more severe hypoalbuminemia (39% vs. 9% with albumin <3 g/dL; P < .0

35 64%) or aspartate (60%) aminotransferase and hypoalbuminemia (51%); most occurred in cycle 1 and were

36 (64%) and aspartate aminotransferase (60%), hypoalbuminemia (55%), peripheral edema (51%), and throm

37 to ER, LR patients were more likely to have hypoalbuminemia (56% versus 80%), hematocrit <28% (33% v

38 21%) than in the Y90 group (0%; P = .031) or hypoalbuminemia (58% in the cTACE group vs 4% in the Y90

39 6%, and it was significantly associated with hypoalbuminemia (76.5%, P = 0.000).

40 Hypoalbuminemia, a biomarker of asparaginase activity, w

41 (adjusted HR, 3.03; 95% CI, 1.05-8.78), and hypoalbuminemia (adjusted HR, 0.89; 95% CI, 0.82-0.97) w

42 Patients with GBS may develop hypoalbuminemia after treatment with IVIG, which is rela

43 rs for poor prognosis after PEG include sex, hypoalbuminemia, age, chronic heart failure, and subtota

44 Blood work revealed hypoalbuminemia (albumin 2 g/dl), and hypogammaglobuline

45 stment formulas was worse in the presence of hypoalbuminemia (albumin level <30 g/L).

46 dL (interquartile range, 3.8-4.5 g/dL), with hypoalbuminemia (albumin, <3.5 g/dL) in 20 (12.8%) of 15

47 obal or liver-specific FcRn deletion exhibit hypoalbuminemia, albumin loss into the bile, and increas

48 oratory abnormalities included anemia (all); hypoalbuminemia (all) and thrombocytopenia (6).

49 luded (in decreasing frequency) grade 1 to 2 hypoalbuminemia, aminotransferase elevations, edema, hea

50 Taken together, this study found that hypoalbuminemia and abnormalities in hepatobiliary labor

51 oing VRS to explore the relationship between hypoalbuminemia and adverse outcomes and to confirm whet

52 ng ART are low and are influenced as much by hypoalbuminemia and age as by CD4 cell status.

53 Common toxic effects included transient hypoalbuminemia and elevated aminotransferase levels.

54 ever, the associations between eGFR and both hypoalbuminemia and hypertension were NS.

55 disease-related factors are associated with hypoalbuminemia and might be valuable items to include o

56 udy demonstrated a substantial prevalence of hypoalbuminemia and obesity among orthopaedic trauma pat

57 estigation was to identify the prevalence of hypoalbuminemia and obesity in orthopaedic trauma patien

58 The association between hypoalbuminemia and posttransplant mortality was stronge

59 entions to slow its progression, predialysis hypoalbuminemia and severe anemia, suboptimal pre-ESRD e

60 In the full mITT population, age, hypoalbuminemia, and cancer were inversely associated wi

61 age, weight loss, anemia, thrombocytopenia, hypoalbuminemia, and excess bone marrow blasts as indepe

62 , malabsorption, protein-losing enteropathy, hypoalbuminemia, and failure to thrive.

63 des, fevers, malaise, anorexia, weight loss, hypoalbuminemia, and gastrointestinal blood loss were co

64 uirement for mechanical circulatory support, hypoalbuminemia, and hepatic dysfunction), intraoperativ

65 is revealed that a nonsurgical intervention, hypoalbuminemia, and higher Acute Physiology and Chronic

66 a lower Karnofsky Performance score, anemia, hypoalbuminemia, and higher C-reactive protein.

67 urvived for at least 12 months, proteinuria, hypoalbuminemia, and hypercholesterolemia improved durin

68 ncluding decreased lymphocyte count, anemia, hypoalbuminemia, and hyponatremia.

69 pain, watery diarrhea, fever, leukocytosis, hypoalbuminemia, and hypovolemia.

70 rum transaminase levels, hyperbilirubinemia, hypoalbuminemia, and prolongation of clotting times.

71 ting with severe protein-losing enteropathy, hypoalbuminemia, and proximal myopathy who had not respo

72 tead, analyses revealed hepatic dysfunction, hypoalbuminemia, and vascular/oxygenation insufficiency.

73 Anemia or hypoalbuminemia are useful clues to NSAID enteropathy.

74 knowledge, the utility of proteinuria and/or hypoalbuminemia as biomarkers of thrombotic risk remains

75 Hypoalbuminemia, ascribed to malnutrition, has been one

76 Our data suggest that hypoalbuminemia at listing reveals a vulnerable populati

77 Hypoalbuminemia at listing was a significant predictor o

78 Anemia and hypoalbuminemia at presentation were independently assoc

79 participants (20%) were seropositive before hypoalbuminemia became apparent, and eight participants

80 t participants (14%) were seropositive after hypoalbuminemia became apparent.

81 antibodies or peptide mimetics, which cause hypoalbuminemia, biliary loss of albumin, and increased

82 ration, and clinical features of AH, such as hypoalbuminemia, bilirubinemia, and splenomegaly.

83 the pathogenesis of edema is usually due to hypoalbuminemia; both extremities are typically involved

84 ffect of albumin treatment (20% solution) on hypoalbuminemia, cardiocirculatory dysfunction, portal h

85 severe but reversible hemoconcentration and hypoalbuminemia caused by leakage of fluids and macromol

86 (in malnourished patients and in those with hypoalbuminemia, cholangitis or long-term jaundice; with

87 Most of them had impaired renal function, hypoalbuminemia, concurrent infection, and/or concomitan

88 The almost ubiquitous hypoalbuminemia confounded the interpretation of acid-ba

89 Our results suggest that proteinuria and/or hypoalbuminemia could be developed as clinically meaning

90 Individuals with hypoalbuminemia (defined as a serum albumin concentratio

91 , and a vascular leak syndrome (hypotension, hypoalbuminemia, edema).

92 ) represents the association of proteinuria, hypoalbuminemia, edema, and hyperlipidemia.

93 Patients typically present with hypoalbuminemia, edema, gastrointestinal symptoms, growt

94 oteinuria in excess of 3.5 g/24 h along with hypoalbuminemia, edema, hyperlipidemia (hypertriglycerid

95 s of nephrotic syndrome include proteinuria, hypoalbuminemia, edema, hyperlipidemia and lipiduria.

96 ated mice exhibited progressive proteinuria, hypoalbuminemia, elevated blood urea nitrogen (BUN) leve

97 Untreated NS rats showed heavy proteinuria; hypoalbuminemia; elevated plasma cholesterol, triglyceri

98 city is vascular leak syndrome manifested by hypoalbuminemia, fluid retention, hypotension and, in on

99 Hypoalbuminemia found at the time of admission to the ho

100 is patient cohort, although strong trends in hypoalbuminemia grade and hyperbilirubinemia grade emerg

101 Stroke patients with hypoalbuminemia had a greater risk of infective complica

102 The high prevalence of pre-ESRD hypoalbuminemia, hematocrit <28%, and lack of EPO use su

103 and geographic factors, the odds ratios for hypoalbuminemia, hematocrit <28%, and lack of EPO use we

104 e trials involving surgery or trauma, burns, hypoalbuminemia, high-risk neonates, ascites, and other

105 arterial pressure, low stroke volume index, hypoalbuminemia, history of cerebral vascular disease, e

106 ling pressures, lower cardiac index, anemia, hypoalbuminemia, hyperbilirubinemia, cognitive impairmen

107 gnificantly correlated with age, alcoholism, hypoalbuminemia, hyperbilirubinemia, renal insufficiency

108 io with anemia, acidosis, hyperphosphatemia, hypoalbuminemia, hyperparathyroidism, and hypertension a

109 ine ratios associated with anemia, acidosis, hypoalbuminemia, hyperparathyroidism, and hypertension b

110 boratory examinations showed the presence of hypoalbuminemia, hypogammaglobulinemia, and an elevated

111 ative health indicators, including diabetes, hypoalbuminemia, hyponatremia, and relative hypocreatini

112 the predominant findings included diarrhea, hypoalbuminemia, hyponatremia, hypokalemia, hypocalcemia

113 860 mg/m2, one of seven patients had grade 3 hypoalbuminemia/hypophosphatemia.

114 lth characteristics that are associated with hypoalbuminemia in community-dwelling older persons, we

115 Hypoalbuminemia in dialysis patients is primarily a cons

116 y volume expansion plays any role in causing hypoalbuminemia in ESRD patients.

117 The profound loss of albumin led to hypoalbuminemia in some diabetic animals.

118 forts to improve nutrition, as it may affect hypoalbuminemia in SPK recipients, may be one strategy f

119 ine the incidence and clinical correlates of hypoalbuminemia in SPK recipients.

120 , are highly correlated with proteinuria and hypoalbuminemia in the puromycin aminonucleoside and adr

121 We defined hypoalbuminemia in two ways: < 35 g/L (1.2% of the sampl

122 suggested by the progressive development of hypoalbuminemia in wild-type mice transplanted with FcRn

123 .2-4.1 g/dL) (P < .001), and the number with hypoalbuminemia increased to 60 (34.5%) of 174 (P < .001

124 Hypoalbuminemia (IRR, 4.35; 95% CI, 2.78-6.81; P < .001)

125 Hypoalbuminemia is an independent risk factor for death

126 Persistent post-SPK hypoalbuminemia is associated with an increased risk for

127 Hypoalbuminemia is associated with mortality in patients

128 Hypoalbuminemia is associated with poorer outcomes in re

129 nosed by the third method; when adjusted for hypoalbuminemia, it reliably detected the hidden abnorma

130 Studies have suggested that hypoalbuminemia itself impairs delivery of effective amo

131 Frailty was associated with anemia, hypoalbuminemia, low creatinine, and the use of suppleme

132 All but one patient showed hypoalbuminemia (<3.5 g/dL); those with HAT also had sig

133 Hypoalbuminemia (<= 30 g/L) was observed in 124 patients

134 Viewed in this manner, hypoalbuminemia may offer an opportunity to improve pati

135 is newly described mechanism, in addition to hypoalbuminemia, may contribute to platelet hyperactivit

136 Hyperbilirubinemia and hypoalbuminemia occurred and survival decreased after tr

137 Hypoalbuminemia occurred in 79%, including 15% with grad

138 Dramatic weight loss and hypoalbuminemia often follow acute hospitalization.

139 The effect of albumin level and hypoalbuminemia on the risk of post-SPK events (cardiac

140 There was no hypoalbuminemia or apparent evidence of lupus nephritis

141 d with LR, and whether LR is associated with hypoalbuminemia or late initiation of dialysis.

142 1.1 to 1.7 per 10 years), those with greater hypoalbuminemia (OR: 0.49, 95% CI: 0.31 to 0.76 per mg/d

143 weakness, cognitive impairment, anemia, and hypoalbuminemia outperformed other frailty scales and is

144 ymphoma, PEL was associated with significant hypoalbuminemia (P < .0027), thrombocytopenia (P = .0045

145 Stepwise multivariate analysis identified hypoalbuminemia (p < 0.001) and the burden of comorbid d

146 were older (p = 0.002), more likely to have hypoalbuminemia (p < 0.001), and more commonly had Child

147 of delirium including older age (P < 0.001), hypoalbuminemia (P < 0.001), impaired functional status

148 Lower performance status, hypoalbuminemia, PDAC diagnosis less than or equal to 18

149 (PFS, 2.95; P = .03; OS, 3.14; P = .03), and hypoalbuminemia (PFS, 2.96; P = .05; OS, 3.64; P = .04).

150 and adverse outcomes and to confirm whether hypoalbuminemia plays a role in risk evaluation.

151 Hypoalbuminemia predicts disability and mortality in pat

152 f 59) of those seropositive for PLA2R-AB had hypoalbuminemia present at the time antibody was initial

153 The nephrotic syndrome is defined by edema, hypoalbuminemia, proteinuria, and hyperlipemia with elev

154 ls: 35-fold; KO: 5400-fold versus baseline), hypoalbuminemia, reduced GFR, and marked glomerular dama

155 Post-SPK hypoalbuminemia resolves over time in many patients.

156 ythmia (RR, 1.51; 95% CI, 1.04 to 2.19), and hypoalbuminemia (RR, 0.56; 95% CI, 0.42 to 0.74 per 1 g/

157 Diabetes can compound hypoalbuminemia's detrimental effects.

158 Hypoalbuminemia (serum albumin level < or =3.5 g/dL) was

159 aundice (total serum bilirubin, 22.2 mg/dL), hypoalbuminemia (serum albumin level, 2.58 g/dL), coagul

160 he prevalence of and factors associated with hypoalbuminemia, severe anemia, and erythropoietin (EPO)

161 me, has been associated with proteinuria and hypoalbuminemia severity.

162 w plasma-cell infiltration greater than 50%, hypoalbuminemia, thrombocytopenia) were predictive of ov

163 h 3 independently significant comorbidities, hypoalbuminemia, thrombocytopenia, and high lactate dehy

164 In era III, hypoalbuminemia, thrombocytopenia, and high primary graf

165 including fever, chills, hypotension, edema, hypoalbuminemia, thrombocytopenia, and transaminasemia w

166 minutes; surgical complexity; liver disease; hypoalbuminemia; thrombocytopenia; mild, moderate, or se

167 The addition of hypoalbuminemia to Euro score enhanced net reclassificat

168 to 50 microg/Kg every other day x 3 included hypoalbuminemia, transaminase elevations, fatigue, and e

169 tory rate, fever, absolute lymphocyte count, hypoalbuminemia, troponin level, and C-reactive protein

170 Hypoalbuminemia was also associated with the presence of

171 Hypoalbuminemia was also independently associated with t

172 Hypoalbuminemia was an independent predictor of in-hospi

173 These findings indicated that hypoalbuminemia was an independent risk factor for in-ho

174 Hypoalbuminemia was associated with an increased chance

175 SPK-related hypoalbuminemia was associated with an increased risk fo

176 Notably, baseline hypoalbuminemia was associated with higher colectomy rat

177 were performed to determine whether post-SPK hypoalbuminemia was associated with pre-SPK variables.

178 Marked hypoalbuminemia was common among the critically ill pati

179 Post-SPK hypoalbuminemia was defined as a serum albumin level < o

180 a (51%), and diarrhea and nausea (both 36%); hypoalbuminemia was reported in 24% of patients.

181 Hypoalbuminemia was significantly associated with persis

182 Significant weight loss, edema, and hypoalbuminemia were independent predictors for the posi

183 Anemia and hypoalbuminemia were not detected in any of the patients

184 1 or 2 fatigue, dryness of skin, anemia, and hypoalbuminemia were the most frequent toxicities report