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1 inhibitor of hedgehog ligands, which induced hypochlorhydria.
2 mbrane of gastric parietal cells and induces hypochlorhydria.
3 in gastric parietal cells and thereby causes hypochlorhydria.
4 of gastric inflammation and did not develop hypochlorhydria.
5 event disease caused by hyperchlorhydria and hypochlorhydria.
6 increase in serum gastrin levels and severe hypochlorhydria.
7 ressed to decreased parietal cell number and hypochlorhydria.
8 Kalpha), which could contribute to transient hypochlorhydria.
9 Thus, genetic or pharmacologic induction of hypochlorhydria and decreased gut Bacteroidales species
10 uals with extensive corpus gastritis develop hypochlorhydria and gastric atrophy, which are presumpti
13 ric hyperplasia in Kcne2(-/-) mice, and both hypochlorhydria and hyperplasia in Kcne2(+/-) mice, sugg
15 ation; corpus-predominant gastritis leads to hypochlorhydria and predisposes to gastric ulceration an
16 foveolar hyperplasia, hypergastrinemia, and hypochlorhydria associated with endocrine dysfunction.
17 h usually arises from atrophic gastritis and hypochlorhydria but other mechanisms seem to be involved
18 with calcium absorption through induction of hypochlorhydria but they also may reduce bone resorption
21 l mice, HKCre/Shh(KO) mice developed gastric hypochlorhydria, hypergastrinemia, and a phenotype that
24 othesis that chronic atrophic gastritis from hypochlorhydria in the gastrin-deficient mouse predispos
26 istance, demonstrating that parasite-induced hypochlorhydria increases gastric survival of S. Typhimu
27 re associated with an increased risk of both hypochlorhydria induced by H. pylori and gastric cancer.
28 d, and 6-month-old mice, indicating that the hypochlorhydria phenotype did not progress over time, as
29 protein-1 had lower levels of gastric acid (hypochlorhydria), reduced production of somatostatin, an
30 tions, revealing the H. pylori infection and hypochlorhydria status by measuring ammonium concentrati
31 known that H. pylori infection can result in hypochlorhydria, the molecular mechanisms underlying thi
32 on, the frequency of achlorhydria (or severe hypochlorhydria) was 44% in patients with idiopathic IDA
33 nfection with Helicobacter pylori results in hypochlorhydria, whereas chronic infection can cause eit
34 Acute infection is associated with transient hypochlorhydria, whereas chronic infection is associated
35 nfection with Helicobacter pylori results in hypochlorhydria, whereas chronic infection may be associ
36 ctose-maldigesting subjects with and without hypochlorhydria with Lactobacillus acidophilus BG2FO4 fo