ライフサイエンスコーパス: hypochromic

1 mutant, frascati (frs), that shows profound hypochromic anaemia and erythroid maturation arrest owin

2 Homozygous mk/mk mice have microcytic, hypochromic anaemia due to severe defects in intestinal

3 Here we show that the hypochromic anaemia in shiraz (sir) zebrafish mutants is

4 ing to identify the gene responsible for the hypochromic anaemia of the zebrafish mutant weissherbst.

5 ish mutant sauternes (sau) has a microcytic, hypochromic anaemia, suggesting that haemoglobin product

6 tation develop moderate to severe microcytic hypochromic anaemia.

7 UV-vis spectra displayed pronounced hypochromic and bathochromic shifts upon DNA titration,

8 ean corpuscular volume, as well as increased hypochromic and microcytic RBC fractions.

9 In most cases, the red cells were hypochromic and microcytic, consistent with abnormalitie

10 continued, weh(Tp85c-/-) zebrafish developed hypochromic anemia and impaired erythroid maturation des

11 Belgrade rats exhibit microcytic, hypochromic anemia and systemic iron deficiency due to a

12 use mutant, which suffers from a microcytic, hypochromic anemia apparently due to defective iron tran

13 autosomal recessively inherited, microcytic, hypochromic anemia associated with abnormal reticulocyte

14 ic deficiency of Mfrn1 results in a profound hypochromic anemia in vertebrate species.

15 cation and characterization of the zebrafish hypochromic anemia mutant, gavi, which exhibits transfer

16 afish have resulted in the identification of hypochromic anemia mutants with a range of mutations aff

17 ron deficiency and eventually the microcytic hypochromic anemia or iron deficiency anemia that is the

18 HNF1A(-/-) mice displayed microcytic hypochromic anemia with reticulocytosis that was partial

19 These mice exhibited microcytic, hypochromic anemia, as did lethally irradiated mice that

20 n iron/heme deficiency results in microcytic hypochromic anemia, the most prevalent anemia globally.

21 f3 in Danio rerio resulted in microcytic and hypochromic anemia.

22 distribution width (RDW), the percentage of hypochromic cells (which is an indicator of insufficient

23 Quantification of hypochromic cells is valuable in the differential diagno

24 ast, Fe(III)Blm and HO(2)-Co(III)Blm induced hypochromic effects in the CD spectrum of I and altered

25 DNA base stacking in Pf3, as judged by Raman hypochromic effects, differs significantly from that occ

26 thridinium interactions provide compensatory hypochromic effects.

27 n of DMT1 identified in a female with severe hypochromic microcytic anemia and iron overload.

28 The mutation, zinfandel, has a hypochromic microcytic anemia as an embryo, but later re

29 These animals exhibit hypochromic microcytic anemia due to impaired intestinal

30 d female patient with a history of a 6-month hypochromic microcytic anemia of unknown cause.

31 a low hemoglobin level, i.e. 10.9 g/dL with hypochromic microcytic anemia pattern seen in complete b

32 Homozygous sublytic mice develop hypochromic microcytic anemia with reduced osmotic fragi

33 e zebrafish chianti (cia) mutant manifests a hypochromic, microcytic anemia after the onset of embryo

34 ependent polycythemia in heterozygotes and a hypochromic, microcytic anemia in homozygotes.

35 antation analyses reveal a mild, congenital, hypochromic, microcytic anemia intrinsic to the hematopo

36 ristics and pathogenesis of a new recessive, hypochromic, microcytic anemia mouse mutant, nm1054.

37 A hypochromic, microcytic anemia was present from birth, a

38 of chardonnay (cdy), a zebrafish mutant with hypochromic, microcytic anemia, and positioned the mutan

39 sis reveals a moderately severe, congenital, hypochromic, microcytic anemia, with an elevated red cel

40 to hepcidin induction, iron deficiency and a hypochromic, microcytic anemia.

41 iron acquisition by red blood cells leads to hypochromic, microcytic anemia.

42 Hypochromic, microcytic anemias are typically the result

43 ysplastic syndrome [ATMDS]) characterized by hypochromic, microcytic, anisopoikilocytic red blood cel

44 Examination of the hypochromic mutant, weissherbst, and adult hypoxia-treat

45 /-) adults have mild anemia characterized by hypochromic red blood cells (RBCs), reticulocytosis, and

46 From the hypochromic responses and the induced emission intensiti

47 The hypochromic shift and Tm for triplex formation increases

48 2 and G2A2G5A2G2 are very shallow with small hypochromic shifts denoting negligible binding at physio

49 lectual disability, intractable seizures and hypochromic skin patches, we identified the ribosomal pr