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1 c nodules with subfoci of HCC, hyperintense, hypointense.
2 high-grade dysplastic nodules, hyperintense, hypointense.
3 c low-grade dysplastic nodules, hypointense, hypointense.
4 low-grade dysplastic nodules, hyperintense, hypointense.
5 version recovery hyperintense (18 of 18), T1-hypointense (17 of 18), and diffusion-hyperintense (15 o
6 /- 12, and 238 msec +/- 17, respectively) or hypointense (296 msec +/- 27, 163 msec +/- 12, and 199 m
7 isointense (8.5/s vs 9.5/s, p=0.02) and T(1) hypointense (7.7/s vs 9.5/s, p=0.003) compared with NAWM
8 66 from lesions on T2-weighted MR images (43 hypointense and 23 isointense on T1-weighted MR images),
13 inversion recovery (FLAIR) hyperintense, T1-hypointense, and appeared as perivascular demyelinated l
17 and at the external surface of the cortex, a hypointense band deeper in the cortex, and a hyperintens
21 (eg, myometrial thinning, intraplacental T2-hypointense bands, uterine bulge, serosal hypervasculari
24 r MR imaging parameters, including number of hypointense brain lesions on T1-weighted MR images, pres
27 tion by effectively separating responding T2-hypointense-collagenized-mature components from potentia
32 "Bagel Sign" pattern: a central lesion with hypointense core and hyperintense rim with or without co
40 MRI, owing to its ability to demonstrate hypointense endocyst, can act as a useful adjunct to cor
41 contrast than adjacent normal prostate, and hypointense features on T2-weighted imaging; these findi
42 Cerebral microbleeds (CMBs) are defined as hypointense foci visible on T2*-weighted and susceptible
43 e defined as discrete, well-defined markedly hypointense foci within the adnexal lesion on T2-weighte
44 mall HCC, hyperintense, hypointense (n = 7); hypointense, hyperintense (n = 2); hyperintense, hyperin
46 n T2-weighted images that showed significant hypointense in striatum region which close to the germin
47 in vascular tissue, and they are found to be hypointense in T1 sequences and hyperintense in T2 seque
49 ocardial hemorrhage was taken to represent a hypointense infarct core with a T2* value of <20 ms.
51 ganglion cell, and inner nuclear layer; the hypointense layer 2, the outer nuclear layer and the inn
53 on volume (0.45% vs 13.00%, p<0.0001) and T1 hypointense lesion volume (36.68% vs 60.93%, p<0.0001).
54 tense lesion volume [T2LV]), the ratio of T1 hypointense lesion volume [T1LV] to T2LV [T1:T2]), brain
55 asures of chronic lesion activity such as T1 hypointense lesion volume accumulation and mean normaliz
56 lesion activity measured by longitudinal T1 hypointense lesion volume accumulation in new focal T2 l
57 lesion activity measured by longitudinal T1 hypointense lesion volume accumulation in slowly expandi
58 on that most of total brain non-enhancing T1 hypointense lesion volume accumulation was derived from
59 d tiw versus DT (nominal p<0.001); T2 and T1 hypointense lesion volume change was lower for sc IFN be
62 ional areas, T2 hyperintense lesions, and T1 hypointense lesions (38.1% +/- 2.6%, 45.0% +/- 2.6%, 51.
63 in T(1) isointense (44.6 +/- 7.2 mM) and T1 hypointense lesions (46.8 +/- 8.3 mM) compared with norm
65 (66.0%, p<0.001), annualised rate of new T1 hypointense lesions (62.8%, p<0.001) and CUA lesions per
66 of lateral ventricles, Dawson's fingers, T1 hypointense lesions (multiple sclerosis), fluffy lesions
67 lesions (all P<0.001) and new T(1)-weighted hypointense lesions (P<0.001, P<0.001, and P=0.002, resp
68 ns (both BG-12 doses), and new T(1)-weighted hypointense lesions (thrice-daily BG-12) (nominal P<0.05
69 adolinium-enhancing (Gd+) T1 lesions, new T1 hypointense lesions and combined unique active (CUA) les
71 er sodium concentration was observed in T(1) hypointense lesions in secondary-progressive (49.0 +/- 7
72 characterize the spatial distribution of T1-hypointense lesions in the spinal cord at MRI, its assoc
73 ection of blood-brain barrier break down and hypointense lesions on T1-weighted images, magnetization
74 1 mL, p=0.024) and a higher number of new T1-hypointense lesions over 0-12 months (p=0.005) as well a
75 mbers of new T2, gadolinium-enhancing and T1 hypointense lesions were lower with sc IFN beta-1a qw (n
76 imaging with ultra-highfield MRI that phase hypointense lesions were significantly more prevalent in
77 radient-recalled-echo MR images demonstrated hypointense lesions with variable contrast material enha
83 rast-to-noise ratio between hyperintense and hypointense liver regions, coefficient of variation, and
84 ic analysis; at MR imaging, PTLD appeared as hypointense masses on T1-and T2-weighted images with min
85 For both diseases, T1-weighted images showed hypointense masses with progressive enhancement (differe
87 intense rim or detached internal T2-weighted hypointense membrane, a correct diagnosis of hydatid cys
88 ted images, respectively: HCC, hyperintense, hypointense (n = 3); hyperintense, hyperintense (n = 1);
90 nosis of acute RVOD and alternating bands of hypointense OCTA flow signal on en face projections.
91 hyperintense areas on T1-weighted images and hypointense on fat-suppressed T1-weighted images, compat
92 ntrifugal DCE lesions appeared isointense or hypointense on phase images, whereas centripetal DCE les
94 intrathoracic and subcutan masses as mainly hypointense on T1-weighted images and hyperintense on T2
95 te depiction of IRE ablation zones that were hypointense on T1-weighted images and hyperintense on T2
96 eatures were the following: all lesions were hypointense on T2- and hyperintense (n=12) and isointens
98 lesions that shrink least and become more T1 hypointense over time suggests that the rim might mark f
100 whereas centripetal DCE lesions showed thin, hypointense phase rims that clearly colocalized with the
101 ic lesions also selected for the presence of hypointense phase rims, the findings were stable over ti
105 oxide nanoparticles are detected in vivo as hypointense regions in the liver up to two weeks post in
106 ADC threshold, (ii) visual determination of hypointense regions on ADC maps, and (iii) visual determ
107 al microbleeds, observed as small, spherical hypointense regions on gradient echo (GRE) or susceptibi
108 ice that received labeled cells demonstrated hypointense regions within the tumor that evolved over t
109 Fibroids were classified as hyperintense or hypointense relative to skeletal muscle on pretreatment
110 APAs (mean size, 20 x 16 mm) were iso- or hypointense relative to the liver on T1-weighted images
112 nse pulmonary cystic lesion with T2-weighted hypointense rim or detached internal T2-weighted hypoint
113 nal, well-defined margin, lobulation, and/or hypointense rim, together with restricted diffusion and
114 signal, well-defined margin, lobulation, and hypointense rim, were detected in a higher proportion of
116 , fluid-fluid or air-fluid level, incomplete hypointense ring due to hemosiderin deposition, pseudotu
118 elected for treatment had no hyperintense or hypointense signal intensity changes on the DW images or
119 sence of fluid collection with isointense or hypointense signal on T1-weighted images, fluid-equivale
120 ed by subcortical linear or arc lesions with hypointense signal on T1-weighted imaging and hyperinten
123 categorized into four subgrades: subgrade A, hypointense; subgrade B, inhomogeneous; subgrade C, hype
124 antified on a three-point ordinal scale (0 = hypointense to brain parenchyma, 1 = isointense to brain
126 ced more than metastases, they also remained hypointense to liver on T1-weighted images (from -4.87 +
135 sity and greater relative accumulation of T1 hypointense volume in slowly expanding/evolving lesions
136 -hyperintense) and rarefied or cystic (FLAIR-hypointense) white matter, and ventricular and extracere