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1 when using their unaffected arm (directional hypokinesia).
2 find the anatomical correlate of directional hypokinesia.
3 on of D2 receptor responses that manifest in hypokinesia.
4  balance disorders but unexpectedly improved hypokinesia.
5           The most common adverse effect was hypokinesia (3 patients in the lorazepam + haloperidol g
6 en turn to the computational lesions causing hypokinesia and deficits of coordination.
7 eration that is associated behaviorally with hypokinesia and rigidity.
8  MPTP: they developed severe DOPA-responsive hypokinesia and tremor together with unresponsive gait a
9 ed that 5% of participants (n=140) had focal hypokinesia, and 1.5% (n=42) had WM abnormalities.
10 d DA tone in direct pathway neurons, whereas hypokinesia at later stages could result from reduced in
11 ts experience loss of normal motor function (hypokinesia), but can develop uncontrollable movements k
12 ) neurons in Parkinson's disease (PD) causes hypokinesia, but DA replacement therapy can elicit exagg
13 induces a dual effect with an improvement of hypokinesia contrasting with a worsening of DOPA-unrespo
14                                             'Hypokinesia', defined as <50% of control group's mean am
15 63%), a mild head tremor (59%), parkinsonism/hypokinesia developing with advancing age (32%) and simp
16 ly, none of the patients without directional hypokinesia had a lesion in the same region.
17      We found that patients with directional hypokinesia had a lesion involving the ventral lateral p
18 tures of PSP, including: decreased survival; hypokinesia; impaired optokinetic responses; neurodegene
19 pacta (SNc), resulting in the characteristic hypokinesia in patients.
20 t over-expressed D1 receptors likely mediate hypokinesia in the D1 transgenic animals.
21            The loss of this ability leads to hypokinesia, known PD motor deficits characterized by a
22                             Occlusion led to hypokinesia of the anterior or anterolateral segments of
23 ade of D2 receptors on D1 agonist-stimulated hypokinesia of the D1 over-expressing animals were inves
24                   Prenatal features included hypokinesia or akinesia and growth restriction.
25 kinsonism was defined as the (1) presence of hypokinesia or bradykinesia plus at least 1 other cardin
26 n which impaired dopamine signaling leads to hypokinesia or impaired initiation of specific voluntary
27 jects, five developed marked RV dilation and hypokinesia, paradoxic septal motion, pulmonary hyperten
28 rotein/activation in adult worms resulted in hypokinesia, reduced vitality and death.
29    (+)JQ1, a bromodomain inhibitor, improved hypokinesia, survival, microgliosis, and brain synapse e
30 ivo, AM404 caused a mild and slow-developing hypokinesia that was significant 60 min after intracereb
31                         The main finding was hypokinesia without decrement in patients with progressi
32 palsy have a specific finger tap pattern of 'hypokinesia without decrement' and they do not have crit