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1 o those of parent lead structures (i.e., the hypolipidemic 2-hydroxy-2-aryl-(benzo)oxa(or thia)zine a
4 dation via PERPP, and may be relevant to the hypolipidemic actions of dietary PUFAs, the basal regula
5 tory, and squalene synthase (SQS) inhibitory/hypolipidemic activities are combined in simple molecule
6 exhibited substantial antihyperglycemic and hypolipidemic activities when orally administered in thr
8 glycemic efficacy in a db/db mouse model and hypolipidemic activity in hamster and dog models without
10 acid extracted SDF of quince showed in vitro hypolipidemic activity, with cholesterol adsorption capa
13 se have the potential to be a novel class of hypolipidemic agent, which possess combined hypocholeste
14 ses that are highly inducible by fibric acid hypolipidemic agents and other peroxisome proliferators.
15 scular risk independent of changes in use of hypolipidemic agents, smoking, blood pressure, BMI, or l
17 site manners by ritonavir and bezafibrate, a hypolipidemic agonist of the peroxisome proliferator-act
19 or-mediated actions pertain to their role in hypolipidemic and antidiabetic therapies and as potentia
21 Evidently the extract possesses pronounced hypolipidemic and antihyperlipidemic effects which are c
28 cardioprotective, anti-hypercholesterolemic, hypolipidemic, anti-atherosclerosis, anti-obesity agains
29 iated with numerous health benefits, such as hypolipidemic, anti-inflammatory, anti-hypertensive, ant
30 ties compared to flavonoid monomers, such as hypolipidemic, antidiabetic, and neuroprotective activit
31 st has focused on various herbs that possess hypolipidemic, antiplatelet, antitumor, or immune-stimul
33 , without modifying the diet or initiating a hypolipidemic drug, by the transient induction of Cre ex
35 s the primary target of the fibrate class of hypolipidemic drugs and mediates lipid lowering in part
36 ferators are of exogenous origin and include hypolipidemic drugs and other industrial chemicals, seve
38 how here that specific FAs, eicosanoids, and hypolipidemic drugs are ligands for PPARalpha or PPARdel
39 vated by diverse environmental chemicals and hypolipidemic drugs classified as peroxisome proliferato
40 eptor-alpha (PPARalpha), the target for most hypolipidemic drugs in current clinical use, regulates t
41 unds including fatty acids, eicosanoids, and hypolipidemic drugs such as fibrates and thiazolidinedio
42 uctase inhibitors) are an important group of hypolipidemic drugs that are able to modulate inflammati
43 a nuclear receptor activated by fatty acids, hypolipidemic drugs, and peroxisome proliferators (PPs).
44 activators, such as the fibric acid class of hypolipidemic drugs, and PPAR-gamma agonists, including
46 (PPARalpha) mediates the effects of several hypolipidemic drugs, endogenous fatty acids, and peroxis
51 Experiments in humans indicate a profound hypolipidemic effect of fish oil, especially lowering of
52 ult to uncover the biochemical basis for the hypolipidemic effect of fish oils in humans through use
53 that procyanidin A may impart a key role of hypolipidemic effect seen in peanut skin polyphenols.
56 s was conducted to more precisely define the hypolipidemic effects and safety of psyllium when used a
57 ryptophan metabolism are associated with the hypolipidemic effects observed in mice that were given P
58 AR gamma also abolished the hypoglycemic and hypolipidemic effects of rosiglitazone, demonstrating th
65 a from the 1994 PDR for all drugs listed as "hypolipidemics." For comparison, we selected a stratifie
67 all persons who are currently candidates for hypolipidemic medication because they are at high risk f
68 BMI), blood pressure, smoking status, use of hypolipidemic medications, and fasting lipids were deter
72 dely popular because of its hypoglycemic and hypolipidemic properties but various cases of T. polium-
74 of chitin, is a dietary fibre known for its hypolipidemic properties, which are mainly attributed to
79 tablished that PPARs are the targets for the hypolipidemic synthetic compounds known as peroxisome pr