ライフサイエンスコーパス: hypopnea

1 pite falling TP EMG and obstructive apnea or hypopnea.

2 esponsible for the development of apneas and hypopneas.

3 uctive, central, and mixed apnea, as well as hypopneas.

4 er, the hourly rate of episodes of apnea and hypopnea (36 +/- 10 versus 47 +/- 21), episodes of arous

5 he mean (+/- SD) hourly episodes of apnea or hypopnea (36 +/- 25 and 20 +/- 27; P = 0.015), the preva

6 re 40 patients without (hourly rate of apnea/hypopnea, 4 +/- 4; group 1) and 41 patients with (51% of

7 h (51% of all patients; hourly rate of apnea/hypopnea, 44 +/- 19; group 2) sleep apnea.

8 iability during sleep was predicted by apnea-hypopnea and body mass indices.

9 ents with upright CSR had the greatest apnea-hypopnea and central apnea index (at daytime and nightti

10 nd fragmentation; standard measures of apnea/hypopnea and periodic leg movement; and results of the m

11 reduced the number of episodes of apnea and hypopnea and the duration of arterial oxyhemoglobin desa

12 of desaturation and/or arousal) to identify hypopneas and apneas.

13 king systems, but the residual events (apnea/hypopnea) and leak data are not as easy to interpret as

14 isturbance index (RDI(Total), sum of apneas, hypopnea, and flow limitation events) of 18 events/h was

15 ereby altering the susceptibility for apnea, hypopnea, and periodic breathing in sleep.

16 a dominant role in the generation of apneas, hypopneas, and snoring in healthy subjects.

17 f upper airway collapsibility, including the hypopnea/apnea + hypopnea ratio and the degree of flow r

18 er design to determine whether apneas and/or hypopneas are temporally associated with episodes of par

19 In contrast, nocturnal rates of apneas and hypopneas, as well as minimal oxygen saturation, did not

20 h percentage of sleep time (>7%) in apnea or hypopnea (both measures of disordered breathing) were as

21 a (OSA) is characterized by repetitive apnea-hypopnea cycles during sleep associated with oxygen desa

22 TI (10 and 15 L/min), obstructive apneas and hypopneas decreased but periodic laryngeal obstructions

23 apnea defined as a cessation of airflow and hypopnea defined as a > or = 30% reduction in airflow or

24 Supplemental O2 significantly reduced hypopnea density, obstructive apnea index, and paradoxic

25 d a mean age SD of 68.9 9.2 years, mean NREM hypopnea duration of 21.73 5.60, and mean NREM apnea dur

26 The number of episodes of apnea and hypopnea during sleep decreased 61 percent.

27 Obstructive apneas and hypopneas during sleep are a well recognized cause of ex

28 management, reduced the number of apnea and hypopnea events and patient-reported sleepiness at 6 mon

29 t Anxiety Scale) and the number of apnea and hypopnea events per hour of sleep (AHI), but were associ

30 was indicated by the frequency of apnea and hypopnea events per hour of sleep, measured by in-labora

31 opause; odds ratios for 15 or more apnea and hypopnea events per hour were 1.1 (0.5, 2.2) with perime

32 confounding factors, for 5 or more apnea and hypopnea events per hour were 1.2 (0.7, 2.2) with perime

33 -hypopnea index (AHI; the number of apnea or hypopnea events per hour, with a score of >/=15 indicati

34 or having 5 or more and 15 or more apnea and hypopnea events per hour.

35 pnea index (AHI; ie, the number of apnea and hypopnea events/h; 15-30 indicates moderate and >30 indi

36 index, and the fraction of events that were hypopneas (Fhypopneas) were independent predictors of th

37 ived from combinations of conventional apnea/hypopnea, flow limitation events (transient elevated upp

38 Men developed more severe hypopnea in response to identical applied external loads

39 However, apnea/hypopnea incidence was similarly increased in both MI-CH

40 icipants with obstructive sleep apnea (apnea-hypopnea index > 5 events/hr) were administered an antio

41 t, for clinically defined sleep apnea (apnea/hypopnea index > or = 10 and daytime symptoms), men had

42 lity of life in 122 patients with SDB (apnea-hypopnea index > or = 5 events/hour), this study found t

43 polysomnography between 1992 and 2004 (apnea-hypopnea index > or =15) who subsequently underwent a PC

44 eight/obese matched patients with OSA (apnea-hypopnea index >/= 15 events per hour) and 11 normal-wei

45 OSA (apnea-hypopnea index >/= 5 events/h) was assessed using home-b

46 SDB was characterized by apnea-hypopnea index >/=15 events per hour (polysomnography).

47 -to-severe sleep-disordered breathing (apnea-hypopnea index >/=15%) was significantly higher in parti

48 with newly revascularized CAD and OSA (apnea-hypopnea index >/=15/h) without daytime sleepiness (Epwo

49 aroxysmal AF (43 with >/=moderate OSA [apnea-hypopnea index >/=15] and 43 without OSA [apnea-hypopnea

50 predicted by age >60 years (HR: 5.53), apnea-hypopnea index >20 (HR: 1.60), mean nocturnal O2sat <93%

51 To evaluate the presence of OSA (apnea-hypopnea index >= 15 events . h(-1)), all patients under

52 ds: Data from 1,207 patients with OSA (apnea-hypopnea index >= 15 events/h) were used to evaluate the

53 ticipants with moderate-to-severe OSA (apnea-hypopnea index >=15 events per hour) were randomized to

54 The presence of SDB (defined as apnea-hypopnea index >=15/h) was assessed with a portable SDB

55 erweight/obese subjects without apnea (apnea-hypopnea index < 15 events per hour) with 25 overweight/

56 normal subjects (n = 15) without SDB (apnea-hypopnea index < 5 events/hour) recruited from the gener

57 compared with individuals without OSA (apnea-hypopnea index < 5), significantly increased risk for pr

58 erate degrees of sleep apnea (5 < or = apnea-hypopnea index < or = 30).

59 ith snoring and mild oSDB (Obstructive Apnea-Hypopnea Index <3 with tonsillar hypertrophy [Brodsky sc

60 y for mSDB (snoring and an obstructive apnea-hypopnea index <3) between June 29, 2016, and February 1

61 opnea index >/=15] and 43 without OSA [apnea-hypopnea index <5]), right atrial and left atrial voltag

62 ragmentation, all participants with an apnea-hypopnea index (AHI) > or = 1 were analyzed separately.

63 ive sleep apnea was assessed using the apnea-hypopnea index (AHI) (>=4% oxygen saturation criterion a

64 Nasal occlusion increased the apnea hypopnea index (AHI) (occlusion mean = 6.6 +/- 8.0 versu

65 OSA-associated quantitative phenotypes apnea-hypopnea index (AHI) and body mass index (BMI).

66 Between-group differences on the apnea-hypopnea index (AHI) and Epworth sleepiness scale (ESS).

67 table monitors, or association between apnea-hypopnea index (AHI) and health outcomes among community

68 A polysomnographically derived apnea-hypopnea index (AHI) and hypoxemia index (percent of sle

69 SDB was quantified with the apnea hypopnea index (AHI) and oxygen desaturation measures.

70 Apnea-hypopnea index (AHI) and percent nighttime with oxygen s

71 ificant dose-relationships between REM apnea-hypopnea index (AHI) and prevalent hypertension.

72 SDB was categorized using the apnea-hypopnea index (AHI) and the hypoxemia index (percent sl

73 for all-cause mortality, with FEV1 and apnea-hypopnea index (AHI) as the primary exposure indicators

74 disordered breathing, depending on the apnea-hypopnea index (AHI) cutoff, ranged from 40 to 60%.

75 te to severe SDB, defined as having an Apnea-Hypopnea Index (AHI) greater than 15 as assessed by poly

76 pnea-predominant OSA was defined as an apnea hypopnea index (AHI) greater than 2 with more than 50% o

77 n fully adjusted models, patients with apnea-hypopnea index (AHI) greater than 30 had a 30% higher ha

78 OSAS was defined by an apnea/hypopnea index (AHI) greater than or equal to 1 event/h,

79 on a nondialysis day, subjects with an apnea-hypopnea index (AHI) greater than or equal to 20 had flu

80 nce of periodontal disease between the apnea-hypopnea index (AHI) groups, with a negligible Spearman

81 ation of head and facial form with the apnea hypopnea index (AHI) in 364 white individuals and 165 Af

82 on analyses in independent cohorts for apnea-hypopnea index (AHI) in a total of 7,708 individuals of

83 Women with an apnea-hypopnea index (AHI) less than 10 comprised the control

84 Women with an apnea-hypopnea index (AHI) less than 10 were the control group

85 Patients with an apnea-hypopnea index (AHI) less than 15 were the control group

86 years with snoring and an obstructive apnea-hypopnea index (AHI) less than 3 enrolled at 7 US academ

87 a significant correlation between the apnea hypopnea index (AHI) measured by polysomnography and res

88 Nonetheless, an obstructive apnea-hypopnea index (AHI) of 1 or greater is often used to de

89 nts with resistant hypertension and an apnea-hypopnea index (AHI) of 15 or higher.

90 r ejection fraction of 45% or less, an apnea-hypopnea index (AHI) of 15 or more events (occurrences o

91 A total of 14.7% of subjects with an apnea-hypopnea index (AHI) of 15 or more had a diagnosis of di

92 ently diagnosed (<6 months) OSA and an apnea-hypopnea index (AHI) of 20 events/hour or more.

93 ts with moderate to severe OSA with an apnea-hypopnea index (AHI) of 20 to 65 events per hour and bod

94 OSA was defined as a nightly mean apnea-hypopnea index (AHI) of more than 15 events/h.

95 The prevalence varies based on the apnea-hypopnea index (AHI) threshold used for the evaluation (

96 olution of symptoms and a reduction in Apnea/Hypopnea Index (AHI) to < 5/h, and partial response (PR)

97 Mean apnea-hypopnea index (AHI) was 52.6 +/- 28.2 (SD) events/h wit

98 The apnea-hypopnea index (AHI) was derived from standardized sleep

99 A greater reduction in apnea hypopnea index (AHI) was strongly associated with a grea

100 h a tomograph and corneal topographer, apnea-hypopnea index (AHI) with polysomnography, and serum HIF

101 OSA, facial shape correlated with the apnea-hypopnea index (AHI), and DS individuals with severe OSA

102 OSA, physiological data including the apnea-hypopnea index (AHI), and nocturnal oxygen saturation (O

103 have variable success at reducing the apnea-hypopnea index (AHI), and predicting responders is chall

104 ith hsCRP and MRP 8/14 levels and with apnea-hypopnea index (AHI), BMI z score, and apolipoprotein B

105 ur main exposures were the obstructive apnea-hypopnea index (AHI), central apnea index (CAI >/= 5), a

106 o four severity groups on the basis of apnea-hypopnea index (AHI), followed by comparisons of cogniti

107 In combination with apnea-hypopnea index (AHI), identification of "respiratory eve

108 pping on three primary OSA traits [the apnea hypopnea index (AHI), overnight average oxyhemoglobin sa

109 hy study that obtained measurements of apnea-hypopnea index (AHI), peripheral oxygen saturation level

110 luded oxygen desaturation index (ODI), apnea-hypopnea index (AHI), subjective sleepiness (Epworth Sle

111 associated with a 55% reduction in the apnea-hypopnea index (AHI), which decreased from a preflight v

112 Logistic regression assessed apnea-hypopnea index (AHI)-adjusted associations between HGNS

113 rdered breathing was quantified by the apnea-hypopnea index (AHI)-the average number of apneas and hy

114 hours of use, mask leak, and residual apnea-hypopnea index (AHI).

115 p apnea (OSA) have been defined by the apnea-hypopnea index (AHI).

116 e diagnosis of OSAS and calculation of Apnea-Hypopnea Index (AHI).

117 ges would correlate with reductions in apnea-hypopnea index (AHI).Methods: A total of 67 individuals

118 divided into 3 sub-groups based on the apnea/hypopnea index (AHI): mild, moderate, or severe OSA.

119 OSA severity was defined by the apnea-hypopnea index (AHI): severe >30, moderate >15-30, mild

120 Apnea-hypopnea index (AHI, the average number of apneas plus h

121 Outcome measures included apnea-hypopnea index (AHI; average number of apneas plus hypop

122 Primary outcome measures were the apnea-hypopnea index (AHI; ie, the number of apnea and hypopne

123 The primary outcome measures were the apnea-hypopnea index (AHI; the number of apnea or hypopnea eve

124 channel home polysomnography, were the apnea-hypopnea index (average number of apneas/hypopneas per h

125 We used the apnea-hypopnea index (events per hour) to define obstructive s

126 factors (mean age, 63 yr; 52% female), apnea-hypopnea index (mean, 13.8; SD, 15.0), smoking, and prev

127 tween 2 to 4 years with an Obstructive Apnea-Hypopnea Index (OAHI) score of 2 or greater and less tha

128 ea (OR, 6.31; 95% CI, 1.94-20.51), and apnea-hypopnea index (OR, 1.22; 95% CI, 1.08-1.39 [per 5-unit

129 iated with significant improvements in apnea-hypopnea index (P < 0.001); microarousal index (P = 0.00

130 arbon dioxide were determinants of the apnea-hypopnea index (P value range = 0.04-0.001).

131 y with DeltaNC (r=-0.755, P<0.001) and apnea-hypopnea index (r=-0.765, P<0.001), it did not in women.

132 t positive correlation between SAA and apnea-hypopnea index (r=0.40, P=0.03).

133 n diffusivity correlated with a higher apnea hypopnea index (Spearman's r = -0.50, p = 0.008) and a g

134 isordered breathing, as defined by the apnea-hypopnea index (the number of episodes of apnea and hypo

135 coronary artery disease [CAD] and OSA [apnea-hypopnea index 15 events/h] with Epworth Sleepiness Scal

136 le) with Cheyne-Stokes breathing (mean apnea-hypopnea index 19.8 [SD 2.6] and stable symptomatic chro

137 leep laboratory and clinical criteria (apnea/hypopnea index [AHI] > or = 10 and the presence of dayti

138 ersomnolence in 741 patients with SDB (apnea-hypopnea index [AHI] >/= 10 events/h).

139 he 24-h prevalence of predominant CAs (apnea/hypopnea index [AHI] >/=5 events/h, with CA of >50%) was

140 Mild to severe OSA (apnea-hypopnea index [AHI] >=5) was associated with poorer glo

141 e new outpatients diagnosed with SAHS (apnea/hypopnea index [AHI] >or= 5/hour, and >or= 2 symptoms in

142 1997, and had to have normal results (apnea hypopnea index [AHI] <5).

143 cal responses (>/=50% reduction in the apnea-hypopnea index [AHI] and <10 events/hour) in patients wi

144 ipants were identified as free of OSA (apnea-hypopnea index [AHI] of <5 events/h and not treated) by

145 1.00 to 1.21] per 10-unit increase in apnea-hypopnea index [AHI]) but not in older men or in women o

146 126 patients with moderate-severe OSA (apnea hypopnea index [AHI], 25.6 [SD 12.3]) were randomly assi

147 patients (55 males, 13 females; median apnea-hypopnea index [AHI], 35) not receiving hypotensive medi

148 We examined 55 sleep apnea probands (apnea-hypopnea index [AHI]: 43.2 +/- 26.3 events/h), 55 proban

149 scarinic (oxybutynin) on OSA severity (apnea-hypopnea index [AHI]; primary outcome) and genioglossus

150 tal of 25 adults with positional OSAS (apnea-hypopnea index [AHI]supine:AHInon-supine >/= 2) were pro

151 bserved between either the obstructive apnea-hypopnea index and any aggregation parameter, but parent

152 used to model the association between apnea-hypopnea index and echocardiographic measures while acco

153 ts, which directly correlated with the apnea-hypopnea index and hypoxemic indices.

154 each interval, we assessed the median apnea-hypopnea index and the relative risk of sudden death fro

155 -year follow-up study according to the apnea-hypopnea index at base line were estimated after adjustm

156 nt CVD experienced larger increases in apnea-hypopnea index between polysomnograms.

157 periencing at least a 50% reduction in apnea-hypopnea index between sham and active treatment.

158 relation of incident CVD to change in apnea-hypopnea index between the 2 polysomnograms was tested w

159 The difference in adjusted mean apnea-hypopnea index change between subjects with and without

160 causes during other intervals, and the apnea-hypopnea index correlated directly with the relative ris

161 Mean number of events measured by the apnea-hypopnea index decreased from 35.7/h to 2.1/h at 6 month

162 The apnea-hypopnea index does not capture the range of physiologic

163 pothesis, we correlated loop gain with apnea-hypopnea index during supine, nonrapid eye movement slee

164 ation of all children with obstructive apnea-hypopnea index greater than 5/hour total sleep time in a

165 non-REM sleep was quantified using the apnea-hypopnea index in REM (AHIREM) and non-REM sleep (AHINRE

166 lation was found between loop gain and apnea-hypopnea index in the atmospheric group only (r = 0.88,

167 At baseline, the mean apnea-hypopnea index in the patients with the syndrome was 35,

168 aphy, and severe SDB was defined as an apnea-hypopnea index of >30 per hour of sleep.

169 lative to the reference category of an apnea-hypopnea index of 0 events per hour at base line, the od

170 idence interval, 1.13 to 1.78) with an apnea-hypopnea index of 0.1 to 4.9 events per hour at base lin

171 sted rate ratio [caffeine/placebo] for apnea-hypopnea index of 0.89 [95% CI = 0.55-1.43]; P = 0.63).

172 disordered breathing was defined as an apnea-hypopnea index of 15 or more events per hour of sleep.

173 valence of sleep-disordered breathing (apnea-hypopnea index of 15 or more) among hormone users (61 of

174 Participants with an apnea-hypopnea index of 15 to 50 events per hour were randomly

175 idence interval, 1.46 to 5.64) with an apnea-hypopnea index of 15.0 or more events per hour.

176 ndrome was 35, as compared with a mean apnea-hypopnea index of 2 in the comparison group.

177 tive patients were enrolled who had an apnea-hypopnea index of 20 h(-1) or greater and an Epworth Sle

178 e sleep apnea syndrome was based on an apnea-hypopnea index of 5 or higher (five or more events per h

179 rs.Measurements and Main Results: SDB (apnea-hypopnea index of 5 or more) and insomnia (Women's Healt

180 idence interval, 1.29 to 3.17) with an apnea-hypopnea index of 5.0 to 14.9 events per hour, and 2.89

181 ore events per hour); patients with an apnea-hypopnea index of less than 5 served as the comparison g

182 An apnea-hypopnea index of more than 10 per hour was significantl

183 ere total sleep time on actigraphy and apnea-hypopnea index on polysomnography.

184 isordered breathing was ascertained by apnea-hypopnea index or clinical diagnosis.

185 t to 6 a.m. had a significantly higher apnea-hypopnea index than those with sudden death from cardiac

186 ified based on conventionally accepted apnea/hypopnea index thresholds: >=5.0/h (OSA(>=5)), >=15.0/h

187 cardiovascular events with increasing apnea-hypopnea index values.

188 90 m than placebo and autoCPAP: median apnea/hypopnea index was 5.8 events per hour (5.8/h) (IQR, 3.0

189 Median apnea hypopnea index was 6.0 (IQR, 3.1-10.3) events per hour w

190 Mean (SEM) apnea-hypopnea index was also significantly higher in particip

191 Each 10-unit increase in apnea-hypopnea index was associated with 0.2 (95% CI, 0.1-0.3)

192 Initially, the apnea-hypopnea index was determined by polysomnography followe

193 betes mellitus, and creatinine levels, apnea-hypopnea index was independently associated with increas

194 The mean (+/-SD) of apnea-hypopnea index was significantly higher in patients with

195 (1,839 in fully adjusted models), the apnea-hypopnea index was used to classify OSA as none (0-4.9/h

196 rth Sleepiness Scale score >10) and an apnea-hypopnea index with 3% desaturation and from 5 to 30 eve

197 Apnea-hypopnea index with a 4% desaturation criterion (primary

198 Apnea-hypopnea index with a 4% desaturation criterion decrease

199 er hour or <15/h, respectively, on the apnea-hypopnea index) and the median PWAD index.

200 pneas and hypopneas per hour of sleep (apnea-hypopnea index) was determined by unattended, single-nig

201 ath, sleep study measures (such as the Apnea-Hypopnea Index), measures of cardiovascular status (such

202 to determine SDB severity (obstructive apnea-hypopnea index).

203 diagnosed with moderate to severe OSA (apnea-hypopnea index, >/=15) in 19 Spanish sleep units.

204 ss: severe sleep-disordered breathing (apnea-hypopnea index, >30 episodes/hr), self-report of poor sl

205 to each criterion that was satisfied: (apnea-hypopnea index, <30 events per hour) + (nadir oxygen sat

206 Seventy-one subjects (ages, 55-76 yr; apnea-hypopnea index, 0.2-96.6 events/h) were evaluated by mag

207 sue structures in 48 control subjects (apnea-hypopnea index, 2.0 +/- 1.6 events/hour) and 48 patients

208 [SD] age, 61.2 [8.7] years; mean [SD] apnea-hypopnea index, 31.2 [17] events per hour; 71% with hype

209 e recruited 30 obese control subjects (apnea-hypopnea index, 4.7 +/- 3.1 events per hour) and 72 obes

210 [10] yr; median [interquartile range] apnea-hypopnea index, 41 [35-53]; mean [SD] Epworth sleepiness

211 Seventeen subjects (apnea-hypopnea index, 42.6 +/- 6.2 [SEM]) were studied during

212 nd 72 obese patients with sleep apnea (apnea-hypopnea index, 43.5 +/- 28.0 events per hour).

213 our) and 48 patients with sleep apnea (apnea-hypopnea index, 43.8 +/- 25.4 events/hour).

214 leep apnea (75% male; mean age, 52 yr; apnea-hypopnea index, 49/h; baseline sleepiness score, 15.7) w

215 year-old male patient with severe OSA (Apnea-Hypopnea Index, 54 events/h) underwent implantation of a

216 OSA prevalence was 65% (median apnea-hypopnea index, 7.2; range, 0-93), 40% of which were mod

217 sleep testing was used to measure the apnea-hypopnea index, a measure of SDB severity.

218 isodes of apnea and hypopnea per hour (apnea-hypopnea index, AHI).

219 pneas and hypopneas per hour of sleep (apnea-hypopnea index, AHI).

220 Body mass index, apnea-hypopnea index, and cognitive abilities were modeled as

221 omarker expression, functional status, apnea-hypopnea index, and overnight hypoxemia.

222 eep time with oxygen saturation < 90%, apnea-hypopnea index, and oxygen desaturation index-did not sh

223 Cheyne-Stokes respiration, obstructive apnea-hypopnea index, and percentage of sleep time with less t

224 1765, GPR83, P = 1.90 x 10(-8) for the apnea-hypopnea index, and rs35424364; C6ORF183/CCDC162P, P = 4

225 uration as measured by pulse oximetry, apnea-hypopnea index, and the fraction of events that were hyp

226 The effects of severe apnea (apnea-hypopnea index, at least 30 episodes/h), which occurred

227 metaanalyzed for association with the apnea-hypopnea index, average oxygen saturation during sleep,

228 s assessed for baseline sleep indices: apnea-hypopnea index, central sleep apnea (central apnea index

229 Apnea hypopnea index, defined as number of apneas and hypopnea

230 to severe sleep-disordered breathing (apnea-hypopnea index, measured as events/hour, >/=15) are 10%

231 age, race and ethnicity, obesity, and apnea-hypopnea index, were not associated with improvement of

232 es focused on traits defined using the apnea-hypopnea index, which contains limited information on po

233 rtality, but its diagnostic metric-the apnea-hypopnea index-is a poor risk predictor.

234 nt gradations of OSA severity based on apnea-hypopnea index.

235 ent in nocturnal oxygen saturation and apnea/hypopnea index.

236 leg fluid volume and either DeltaNC or apnea-hypopnea index.

237 uctive sleep apnea was assessed by the apnea-hypopnea index.

238 groups and correlated negatively with apnea-hypopnea index.

239 Nocturnal polysomnography to evaluate apnea-hypopnea index.

240 matched for age, body mass index, and apnea/hypopnea index.

241 overnight sleep study to determine an apnea-hypopnea index.

242 ii) is most efficacious (reduction in apnoea-hypopnea index; AHI) in patients with a posteriorly-loca

243 tion (SaO2); right putamen tCho/Cr and apnea hypopnea index; right putamen GABA/Cr and baseline SaO2;

244 re nocturnal oxygen saturation and the apnea/hypopnea index; secondary outcomes were sleep structure,

245 oxygen desaturation indices (ODI) and apnea-hypopnea indices (AHI) according to two different defini

246 sleep apnea characterized by high apnea and hypopnea indices during rapid eye movement (REM) sleep.

247 Similar high apnea and hypopnea indices were also noted in prehypertensive spon

248 ent diagnostic sleep studies to obtain apnea-hypopnea indices.

249 During the hypopnea, laryngeal adductor activity was prominent, acc

250 Repeated episodes of apnea and hypopnea may result in desaturation and arousals, which

251 te ventilation initially increased, and then hypopnea occurred.

252 defined as an increased number of apneas and hypopneas on overnight monitoring.

253 d by periodic breathing that alternates with hypopnea or apnea.

254 Apnea occurred more than hypopnea (p < 0.0001).

255 01; 1.7 +/- 0.7 versus 4.1 +/- 0.5 mm Hg for hypopnea, p < 0.05).

256 more than 15 episodes per hour of apnea and hypopnea participated in the study.

257 eases in the number of episodes of apnea and hypopnea per hour (18 +/- 17, vs. 37 +/- 23 with placebo

258 rams showed 15 or more episodes of apnea and hypopnea per hour (apnea-hypopnea index, AHI).

259 onse and the number of episodes of apnea and hypopnea per hour during sleep (r=0.6, P=0.01).

260 a index (the number of episodes of apnea and hypopnea per hour of sleep).

261 ing, with more than 10 episodes of apnea and hypopnea per hour.

262 our (25.1 versus 17.1; P < 0.0001) and apnea-hypopneas per hour (27.2 versus 15.2; P < 0.0001) and gr

263 The frequency of apneas and hypopneas per hour of sleep (apnea-hypopnea index) was d

264 graphy to assess the frequency of apneas and hypopneas per hour of sleep (apnea-hypopnea index, AHI).

265 ratory disturbance indices (number of apneas/hypopneas per hour of sleep) corrected for normal increa

266 ea index (AHI; average number of apneas plus hypopneas per hour of sleep), systolic/diastolic hyperte

267 -disordered breathing (more than 5 apneas or hypopneas per hour of sleep).

268 (RDI), defined as the number of apneas plus hypopneas per hour of sleep, measured during in-home pol

269 (RDI), defined as the number of apneas plus hypopneas per hour of sleep, was measured during in-home

270 ndex (AHI, the average number of apneas plus hypopneas per hour of sleep, with apnea defined as a ces

271 opnea index, defined as number of apneas and hypopneas per hour of sleep.

272 index (AHI)-the average number of apneas and hypopneas per hour of sleep.

273 nea-hypopnea index (average number of apneas/hypopneas per hour) and the hypoxemia index (percentage

274 f 15 or more events (occurrences of apnea or hypopnea) per hour, and a predominance of central events

275 trations, abnormal breath conditions (apnea, hypopnea, polypnea) and the asymmetric breath conditions

276 re OSA (AHI >10) compared with children with hypopnea predominance (OR, 2.30; 95% CI, 1.03-5.03); bas

277 s between children with apnea-predominant vs hypopnea-predominant OSA.

278 llapsibility, including the hypopnea/apnea + hypopnea ratio and the degree of flow reduction that acc

279 uctive airways disease (OAD) and sleep apnea-hypopnea (SAH) nor the sleep consequences of each disord

280 However, the effect of apnea-hypopnea suppression on DKD progression is unclear.

281 interactions between obstructive sleep apnea-hypopnea syndrome (OSAHS) and cardiovascular diseases, t

282 Obstructive sleep apnea hypopnea syndrome (OSAHS) is a prevalent disorder, for w

283 ated health data, an obstructive sleep apnea-hypopnea syndrome (OSAHS) monitoring and intervention sy

284 ice in patients with obstructive sleep apnea-hypopnea syndrome (OSAHS) who require higher CPAP (10 cm

285 tes in patients with obstructive sleep apnea hypopnea syndrome (OSAHS).

286 Around 50% of patients with the sleep apnea/hypopnea syndrome (SAHS) are not obese: body mass index

287 is increasing evidence that the sleep apnea-hypopnea syndrome (SAHS) is associated with daytime hype

288 as performed with 46 adults with sleep apnea-hypopnea syndrome (SAHS) to determine the effect of ther

289 ted for treatment of snoring and sleep apnea/hypopnea syndrome (SAHS), but often it does not effect a

290 ure (CPAP) are used to treat the sleep apnea/hypopnea syndrome (SAHS).

291 Patients with obstructive sleep apnea/hypopnea syndrome can experience residual daytime sleepi

292 ess in patients with obstructive sleep apnea/hypopnea syndrome who are regular users of nasal continu

293 ronic hypercapnia in obstructive sleep apnea/hypopnea syndrome.

294 O2)) during spontaneous breathing, the apnea-hypopnea threshold for CO2, and then calculated the diff

295 leep nor proportionally decrease their apnea-hypopnea threshold.

296 ons were among RDIs that required apneas and hypopneas to be associated with some level of desaturati

297 starting bronchoscopy when hypoventilation (hypopnea, two successive breaths of at least 50% reducti

298 defined by various definitions of apneas and hypopneas were assessed in 5,046 participants in the Sle

299 ET(CO2) thereby protecting against apnea and hypopnea, whereas reduced ventilatory drive and hypovent

300 ne learning is used to autodetect apneas and hypopneas with 100% sensitivity and 95% precision in pre