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1 ulin less than 2 ng/ml (95%, euthyroid; 96%, hypothyroid).
2 unction (127 were hyperthyroid, and 168 were hypothyroid).
3 d dysfunction: 19 became hyperthyroid and 14 hypothyroid.
4 eveloped secondary leukemia and three became hypothyroid.
5  indicating that the lungs were functionally hypothyroid.
6                  Peripheral tissues are also hypothyroid.
7 tal thyroidectomy and then allowed to become hypothyroid.
8 he most common persistent chronic irAEs were hypothyroid (38 [70.4%]), arthritis (18 [33.3%]), dermat
9                      AF incidence was 78% in hypothyroid, 67% in hyperthyroid, and the duration of in
10                                           In hypothyroid adult ventricular myocardium, there was a se
11  and approximately 12-fold, respectively, in hypothyroid and euthyroid rats.
12 en linked to delayed diastolic relaxation in hypothyroid and failing hearts.
13 4, 12 and 24 months of age during euthyroid, hypothyroid and hyperthyroid states.
14 eptor, and affected animals are congenitally hypothyroid and profoundly deaf as a consequence when th
15                                  Five became hypothyroid and required long-term thyroxine.
16             The behavior of six congenitally hypothyroid and six normal control rats was assessed und
17 nts (14.9%) were euthyroid, 477 (11.6%) were hypothyroid, and 3018 (73.5%) were hyperthyroid.
18                 Mice with severe TEC H/P are hypothyroid, and normalization of serum thyroxine levels
19 ological rat cardiac hypertrophy models with hypothyroid- and hyperthyroid-like changes in the TH tar
20 ver, deletion of SMRT in either euthyroid or hypothyroid animals had little effect on TH signaling.
21  increased SREBP-2 nuclear protein levels in hypothyroid animals results in thyroid hormone-independe
22                                              Hypothyroid animals showed cardiac atrophy and reduced c
23 n was not derepressed in liver or kidneys of hypothyroid animals, our results indicated that the thyr
24  of direct cardiac effects in the genesis of hypothyroid cardiac dysfunction, the cardiac myocyte was
25              The half-life of D2 activity in hypothyroid cells was 47 min after cycloheximide and 60
26  were studied at euthyroid, hyperthyroid and hypothyroid conditions.
27 tified in the hypothyroid, mutant progeny of hypothyroid dams by tracking developmental changes in th
28                                  Subclinical hypothyroid disease in pregnancy is a special case and a
29              We retrospectively analyzed 535 hypothyroid dosimetry studies performed as part of routi
30                                Unexpectedly, hypothyroid double KO mice also failed to mount a signif
31            These mice also become profoundly hypothyroid due to deregulation of genes involved in thy
32              At last, we have confirmed that hypothyroid environment attenuated ovarian cancer growth
33                     At the end of treatment, hypothyroid, euthyroid, and hyperthyroid status was conf
34                     Transgenic mice rendered hypothyroid exhibited a TSH response that was only 30% o
35 tination in normal extracts toward levels in hypothyroid extracts, which showed little E3alpha-depend
36 ion, TED duration and clinical signs between hypothyroid eye disease (Ho-TED) and hyperthyroid eye di
37 eatic islets, and that growth retardation in hypothyroid fetal sheep is associated with reductions in
38  prepartum development of cerebral OXPHOS in hypothyroid fetal sheep.
39                      Mice were then rendered hypothyroid, followed by graded T(3) replacement.
40                                  A 12-yr-old hypothyroid girl was diagnosed at birth as athyreotic be
41 rocytes of animals suffering from congenital hypothyroid goiter with defective thyroglobulin, GRP94 a
42 hormone) is an important cause of congenital hypothyroid goiter; further, homozygous mice expressing
43 ratio was 1.3:1 in hyperthyroid and 4.5:1 in hypothyroid group (P = 0.005).
44  CAL and PISA were also higher in autoimmune hypothyroid group as compared with the systemically heal
45  were significantly higher in the autoimmune hypothyroid group as compared with the systemically heal
46                      Most of the patients in hypothyroid group developed eye involvement after thyroi
47                  Whole-body half-life in the hypothyroid group was significantly longer at 19.3 +/- 7
48  severity of periodontitis in the autoimmune hypothyroid group were significantly higher compared wit
49 t whole-body half-life becomes longer in the hypothyroid group.
50 reased whole-body half-life occurring in the hypothyroid group.
51 e and ending at the second poly(A) signal in hypothyroid hearts were 0.26 euthyroid levels (P < 0.05)
52      Energy restriction produces a transient hypothyroid-hypometabolic state that normalizes on retur
53                    The energy expenditure of hypothyroid IIA+ mice did not increase; however, the ene
54  implications for the health of pre-term and hypothyroid infants.
55                                        These hypothyroid-like abnormalities are completely reversed b
56                  Mct8-deficient mice display hypothyroid-like cone gene expression and compromised el
57 sults in similar shifts in cone identity and hypothyroid-like gene expression whereas reexpression of
58 downregulated in 2 hypertrophy models with a hypothyroid-like mRNA phenotype, phenylephrine in cultur
59 28.1% and 21.7%, respectively); while in the hypothyroid, low T3, and low T3T4 groups minimal change
60 ydrate intake, kidney function, arrhythmias, hypothyroid, lung disease, osteoarthritis, and rheumatoi
61 tant TSHR (Pro --> Leu at 556) in congenital hypothyroid mice activates osteoclast differentiation, c
62 etion of thyroid hormones in vivo and rescue hypothyroid mice after transplantation.
63 riglyceride esterification protects severely hypothyroid mice against NAFLD.
64 deleted NCoR1 in the livers of euthyroid and hypothyroid mice and examined the effects on gene expres
65 oid stimulating hormone receptor (Tshr(-/-)) hypothyroid mice by X-ray, histology, transcriptional pr
66                          Grafts from chronic hypothyroid mice contained fewer beta-cells, heterogenou
67                   Here, we found that mildly hypothyroid mice develop NAFLD without down-regulation o
68                                        These hypothyroid mice displayed oxidative stress in the thyro
69                                              Hypothyroid mice exhibited reduced Luc expression across
70               Brown adipocytes isolated from hypothyroid mice replaced with T3, but not from those re
71                        In contrast, severely hypothyroid mice show down-regulation of TH signaling in
72  owing in part to reduced food intake, these hypothyroid mice show signs of compensatory up-regulatio
73                                              Hypothyroid mice were treated for 10 days with varying d
74 tablished by demonstrating that treatment of hypothyroid mice with thyroxine resulted in a specific i
75                                           In hypothyroid mice, hepatic expression of Spot 14, Bcl-3,
76  in a sequential manner in euthyroid but not hypothyroid mice, thus providing evidence that chondrocy
77  the cell cycle was significantly reduced in hypothyroid mice.
78 is and thymopoiesis were normal in dwarf and hypothyroid mice.
79 tive T3 (3,3',5-triiodothyronine) targets in hypothyroid mice.
80 observed for malic enzyme mRNA expression in hypothyroid mice.
81 f Dio3 in the fetus and produced a permanent hypothyroid milieu in the adult.
82 tios (IRRs) of asthma among children born to hypothyroid mothers versus children born to mothers with
83 was markedly reduced compared with all other hypothyroid mouse genotypes.
84                             Using a systemic hypothyroid mouse model, we found that intra-amygdala in
85                                     However, hypothyroid mouse models have been reported to have a le
86 ed developmental stage was identified in the hypothyroid, mutant progeny of hypothyroid dams by track
87 m 1 month after surgery were randomized into hypothyroid (N=9), euthyroid (N=9), and hyperthyroid (N=
88 s of term infants that weighed <2,500 g, and hypothyroid non-Hispanic white women had higher odds of
89 d that in the CA1 region of the hippocampus, hypothyroid or stress partially blocked E-LTP.
90             When these animals were rendered hypothyroid or thyrotoxic, mRNA expression of MHC isofor
91 , and C-reactive protein (CRP) in autoimmune hypothyroid patients and systemically healthy subjects.
92                                          Two hypothyroid patients evaluated with thyroid ultrasonogra
93                              Six of 15 (40%) hypothyroid patients had suppressed TSH concentrations b
94 s-sectional study comprised of 65 autoimmune hypothyroid patients under treatment and 75 systemically
95 thyroid uptake was 4.2% +/- 1.8% for the 300 hypothyroid patients versus 3.8% +/- 1.6% (P = 0.12) for
96 Research Database (NHIRD) of Taiwan of which hypothyroid patients who received a diagnosis between 20
97 hole body scans (84% of euthyroid and 94% of hypothyroid patients) and stimulated thyroglobulin less
98 nts and 84 ng/100 ml in 15 untreated primary hypothyroid patients.
99                     However, patients with a hypothyroid phase of more than 4 wk, and in particular t
100 s and the results are comparable to those of hypothyroid protocols in the majority of patients.
101                                              Hypothyroid R429Q KI mice displayed elevated TSH subunit
102  TSH levels in the euthyroid and subclinical hypothyroid range with incident AF was examined by using
103  high serum thyrotropin (TSH) levels (in the hypothyroid range) while in therapy with L-T4 in tablet.
104 levels worsened again reaching levels in the hypothyroid range.
105 riptional enhancement obtained in T3-treated hypothyroid rat liver (1.8-fold increase) but not in T3-
106 s to the urinary concentrating defect in the hypothyroid rat.
107 ride and urea transporters and aquaporins in hypothyroid rats (HT) with diminished urinary concentrat
108                                           In hypothyroid rats (induced by addition of propyl-thiourac
109  muscle UCP3 levels were decreased 3-fold in hypothyroid rats and increased 6-fold in hyperthyroid ra
110 le large dose of L-triiodothyronine given to hypothyroid rats caused a 4.7-fold increase in myocardia
111                    Administration of T(3) to hypothyroid rats elevated the abundance of PGC-1 alpha m
112 us were identical in both euthyroid rats and hypothyroid rats induced by 6-n-propyl-2-thiouracil in d
113 s compared with euthyroid controls (CTL) and hypothyroid rats replaced with L-thyroxine (HT+T).
114            Thyroid hormone administration to hypothyroid rats resulted in a doubling of the HCN2/beta
115 T3 and T4 levels were significantly lower in hypothyroid rats than control.
116  pituitary glands were significantly less in hypothyroid rats than the corresponding normal littermat
117 of MDA-bound proteins, hyperthyroid rats and hypothyroid rats were compared to euthyroid controls.
118 d septic rats, and more slowly in those from hypothyroid rats, than in controls.
119  the other hand, in extracts of muscles from hypothyroid rats, where overall proteolysis is reduced b
120 d in the liver of hyperthyroid compared with hypothyroid rats.
121 significantly influence EEG-defined sleep in hypothyroid rats.
122 region, the preoptic region, was examined in hypothyroid rats.
123 leasing Hormone (TRH) in the hypothalamus of hypothyroid rats.
124 and spine alterative changes in the brain of hypothyroid rats.
125                                          The hypothyroid responsiveness to LPS, however, was restored
126 m that exploited paired euthyroid and severe hypothyroid serum samples from human subjects.
127  cell proliferation and mass observed in the hypothyroid sheep fetus and may have consequences for pa
128 t rats were treated chemically to induce the hypothyroid state and cause a transition in the ventricu
129 body radiation exposure as compared with the hypothyroid state in withdrawal patients.
130 cts with corepressor proteins and mimics the hypothyroid state, regardless of the circulating thyroid
131                               Hence, for the hypothyroid state, transcriptional and post-transcriptio
132 cellular carcinoma (HCCs), suggesting that a hypothyroid status favors the onset and progression of p
133              The results also suggest that a hypothyroid status of preneoplastic lesions may contribu
134 pre-B, and B cells in the bone marrow of the hypothyroid strain of mice are significantly reduced com
135 ls is significantly reduced in the dwarf and hypothyroid strains of mice, which have defects in the p
136                                   Autoimmune hypothyroid subjects exhibited a higher prevalence and s
137                 L-thyroxine does not improve hypothyroid symptoms among adults with subclinical hypot
138 PRO) questionnaire scores for the domains of hypothyroid symptoms and tiredness at 1 year (range, 0-1
139                             1-year change in Hypothyroid Symptoms and Tiredness scores (range, 0 to 1
140  changes in body weight, serum lipid levels, hypothyroid symptoms as measured by a HRQL questionnaire
141 significantly associated with improvement in hypothyroid symptoms or fatigue.
142 den at baseline, L-thyroxine did not improve hypothyroid symptoms or tiredness compared with placebo.
143 ticipants with high symptom burden (baseline Hypothyroid Symptoms score >30 or Tiredness score >40) v
144 ferences in the mean change at 1 year in the Hypothyroid Symptoms score (0.2+/-15.3 in the placebo gr
145  two primary outcomes were the change in the Hypothyroid Symptoms score and Tiredness score on a thyr
146                                          The hypothyroid symptoms score decreased from 21.7 at baseli
147 mong the group with high symptom burden, the Hypothyroid Symptoms score improved similarly between th
148          There was no evidence that baseline Hypothyroid Symptoms score or Tiredness score modified t
149                         132 participants had Hypothyroid Symptoms scores greater than 30, and 133 had
150 in clinical presentation and the presence of hypothyroid symptoms, especially in pregnancy and in chi
151                                              Hypothyroid tadpoles did not exhibit the decline in lary
152                      Laryngeal cartilages of hypothyroid tadpoles exhibited low density and minimal p
153 ce appear grossly normal, however, when made hypothyroid the repression of many positively regulated
154               Animals that had been rendered hypothyroid through removal of the thyroid gland showed
155 dogenous SMRT and NCoR mRNA were observed in hypothyroid transgenic mice.
156                          When mice were made hypothyroid, TSH levels increased, obliterating the diff
157 o compare clinical characteristics of TED in hypothyroid vs. hyperthyroid patients.
158 ased in TR-beta2-null mice to levels seen in hypothyroid wild-type mice and did not change significan
159       Prepro-TRH expression was increased in hypothyroid wild-type mice and markedly suppressed after
160 mental and growth delays and were profoundly hypothyroid, with no detectable thyroid hormone and elev
161 increased inflammatory markers were found in hypothyroid WT mice exposed to VILI compared with euthyr

 
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