ライフサイエンスコーパス: hypothyroidism

1 ) or within the reference range (subclinical hypothyroidism).

2 een for serum TSH concentration >5 mIUL(-1) (hypothyroidism).

3 ated TSH level almost always signals primary hypothyroidism.

4 g data in patients suffering from congenital hypothyroidism.

5 etabolic differences between mild and severe hypothyroidism.

6 icating that Slc30a10 knock-out mice develop hypothyroidism.

7 yroid participants or those with subclinical hypothyroidism.

8 story was significant for hyperlipidemia and hypothyroidism.

9 t benefits in older persons with subclinical hypothyroidism.

10 ssociated with the development of congenital hypothyroidism.

11 s been used for more than a century to treat hypothyroidism.

12 n diet, thiamine deficiency, alcoholism, and hypothyroidism.

13 p toward cell-based regenerative therapy for hypothyroidism.

14 ported a diagnosis of subclinical autoimmune hypothyroidism.

15 ly lost in autoimmune thyroiditis leading to hypothyroidism.

16 adult rodents following prolonged (12 week) hypothyroidism.

17 and 18% in 141 individuals with subclinical hypothyroidism.

18 ith time and dose, likely in compensation to hypothyroidism.

19 ary hypogonadism, adrenal insufficiency, and hypothyroidism.

20 m, proteinuria, reduced kidney function, and hypothyroidism.

21 those affected ultimately develop permanent hypothyroidism.

22 tion, thus creating a state of cell-specific hypothyroidism.

23 n resistance by promoting centrally mediated hypothyroidism.

24 were hearing loss, psychiatric effects, and hypothyroidism.

25 dominantly resulting in blood cytopenias and hypothyroidism.

26 mia, high creatinine, history of stroke, and hypothyroidism.

27 subclinical hyperthyroidism and subclinical hypothyroidism.

28 thyroid hormone levels and of serum PFOA and hypothyroidism.

29 dwh, that rescues hearing despite persistent hypothyroidism.

30 ations in thyroid architecture, and dramatic hypothyroidism.

31 rapies, apart from an increased frequency of hypothyroidism.

32 thyroidism and symptoms consistent with mild hypothyroidism.

33 lts aged 80 years and older with subclinical hypothyroidism.

34 ssibility of preventing progression to overt hypothyroidism.

35 lts aged 80 years and older with subclinical hypothyroidism.

36 isk of progression from subclinical to overt hypothyroidism.

37 ) is a form of thyroid hormone used to treat hypothyroidism.

38 d hormone synthesis, resulting in congenital hypothyroidism.

39 duces the odds of developing thyroid Abs and hypothyroidism.

40 had elevated thyroid manganese and developed hypothyroidism.

41 role for additional signaling modulators in hypothyroidism.

42 estimate hazard ratios (HRs) and 95% CIs for hypothyroidism.

43 mone receptor (TR) during development and in hypothyroidism.

44 Thyroidectomized rats with serum-confirmed hypothyroidism 1 month after surgery were randomized int

45 High-yield tests included thyroid function (hypothyroidism, 10.1%), audiometry (hearing loss, 22.6%)

46 eural hearing loss 76%, miscarriage 33%, and hypothyroidism 14%.

47 nic constipation (prevalence 14.2% vs 3.6%), hypothyroidism (14.7% vs 10.4%), obesity (13.0% vs 7.7%)

48 sease (HR 1.93, 95% CI 1.76-2.12; p<0.0001), hypothyroidism (2.31, 2.05-2.60; p<0.0001), and raised t

49 patients (62%) had comorbidities, including hypothyroidism (20%).

50 t common immune-mediated adverse events were hypothyroidism (25 [16%]) and hyperthyroidism (17 [11%])

51 atigue (29 [55%]), diarrhoea (27 [51%]), and hypothyroidism (25 [47%]).

52 8 [19%]), dysphonia (44 [23%] vs ten [10%]), hypothyroidism (39 [21%] vs seven [7%]), and upper abdom

53 8%), diarrhea (52%), hypertension (47%), and hypothyroidism (42%).

54 th the known risk factors of diabetes and/or hypothyroidism (46% vs 14% for those without).

55 ne events included pneumonitis (6 [13%]) and hypothyroidism (5 [10%]), mostly </= grade 2.

56 ing hormone deficiency (9.2%), and thyroidal hypothyroidism (5.8%).

57 Hypothyroidism, a metabolic disease characterized by low

58 the goal of radioiodine therapy is to induce hypothyroidism, a status that is readily treatable with

59 cellular carcinoma, severe anemia, untreated hypothyroidism, Addison syndrome and major depression be

60 a 21% excess risk of developing subclinical hypothyroidism (adjusted HR 1.21; 95% CI 1.03-1.42).

61 a (adjusted OR = 1.5; 95% CI = 1.4-1.6), and hypothyroidism (adjusted OR = 1.2; 95% CI = 1.1-1.4).

62 These societies argue that subclinical hypothyroidism adversely affects cardiovascular outcomes

63 ecific NEMO knock-out mice gradually develop hypothyroidism after birth, which leads to reduced body

64 ertension, valvular heart disease, diabetes, hypothyroidism, AIDS, lymphoma, solid tumor without meta

65 C>G) previously reported to cause congenital hypothyroidism also developed GDM.

66 he odds of MCI with clinical and subclinical hypothyroidism among men was 1.02 (95% CI, 0.57-1.82) an

67 f interest, thyroid dysfunction (10 cases of hypothyroidism and 1 of Graves disease) developed in 11

68 s of follow-up), 1958 (6.5%) had subclinical hypothyroidism and 2574 individuals (8.6%) developed AF

69 90 participants, 2068 (8.1%) had subclinical hypothyroidism and 648 (2.6%) had subclinical hyperthyro

70 21,025 were euthyroid, 1342 had subclinical hypothyroidism and 671 subclinical hyperthyroidism.

71 Duration of hypothyroidism and a decrease in GFR less than 60 mL/min

72 radioiodine therapy for hyperthyroidism are hypothyroidism and a minimal risk of radiation-induced m

73 ry study involving patients with subclinical hypothyroidism and acute myocardial infarction, treatmen

74 ansaminitis and hypophysitis with documented hypothyroidism and adrenal insufficiency.

75 ng development results in maternal and fetal hypothyroidism and associated serious adverse health eff

76 Maternal hypothyroidism and asthma in the children were defined b

77 al tTGA titers, female sex, and a history of hypothyroidism and autoimmune disease were associated wi

78 t HDAC inhibitors are clearly beneficial for hypothyroidism and could be therapeutics for treatment.

79 horter telomere length increases the risk of hypothyroidism and decreases the risk of thyroid cancer,

80 1/5(dKO)) mice displayed growth retardation, hypothyroidism and defective follicular architecture.

81 t implications for the clinical treatment of hypothyroidism and for susceptibility to other neurologi

82 Hypothyroidism and Hashimoto's thyroiditis are more comm

83 As a consequence, both hypothyroidism and hyperthyroidism associate with clinic

84 eatment of subclinical and undiagnosed overt hypothyroidism and hyperthyroidism in adults without goi

85 Both hypothyroidism and hyperthyroidism lead to increased AF

86 Hypothyroidism and hyperthyroidism produced opposite ele

87 profiles are disrupted in some patients with hypothyroidism and hyperthyroidism.

88 dence suggests that milder forms of maternal hypothyroidism and hypothyroxinemia during pregnancy are

89 Hypothyroidism and its presenting signs and symptoms are

90 evant molecular mechanisms linked to central hypothyroidism and macroorchidism in IGSF1 deficiency ar

91 e investigated a common pituitary origin for hypothyroidism and macroorchidism, and the role of IGSF1

92 in Superfamily 1) gene defects cause central hypothyroidism and macroorchidism.

93 association between clinical or subclinical hypothyroidism and MCI (odds ratio [OR], 0.99 [95% CI, 0

94 onstrated an association between subclinical hypothyroidism and metabolic syndrome and selected compo

95 sm by which GLIS3 deficiency causes neonatal hypothyroidism and prevents goiter development.

96 Thirty percent of HMTs developed hypothyroidism and required long-term T4 supplementation

97 t be indicated for patients with subclinical hypothyroidism and serum thyrotropin levels of 10 mU/L o

98 and middle-aged individuals with subclinical hypothyroidism and symptoms consistent with mild hypothy

99 ll increase in the prevalence of subclinical hypothyroidism and thyroid autoimmunity; whether these i

100 , isolated cleft lip and cleft palate (CLP), hypothyroidism and thyroid cancer all map to the FOXE1 l

101 etermine the mechanisms of manganese-induced hypothyroidism and understand how SLC30A10 and SLC39A14

102 chemotherapy with cediranib, and diarrhoea, hypothyroidism and voice changes were more common during

103 thyroidism, 1.4% primary and 0.1% iatrogenic hypothyroidism, and 1.3% other/unspecified thyroid disea

104 d function, 17% in 313 persons with clinical hypothyroidism, and 18% in 141 individuals with subclini

105 be caused by medications use, renal failure, hypothyroidism, and by prolactinoma - PRL secreting tumo

106 than 50% of women with previously diagnosed hypothyroidism, and can be managed by increasing the lev

107 okers, have a history of hemorrhagic stroke, hypothyroidism, and dementia, and less likely to be trea

108 scabies, chronic mucocutaneous candidiasis, hypothyroidism, and esophageal squamous cell carcinoma.

109 n abnormalities with sensorineural deafness, hypothyroidism, and frequent infections as well as the i

110 n abnormalities with sensorineural deafness, hypothyroidism, and frequent infections comprise the car

111 h intellectual disability, epilepsy, ptosis, hypothyroidism, and genital anomalies, we uncovered bi-a

112 one case each of grade 3 pemphigoid, grade 3 hypothyroidism, and grade 3 peripheral sensory neuropath

113 associated with a decline in renal function, hypothyroidism, and hypercalcaemia.

114 e studied 3 sisters with primary amenorrhea, hypothyroidism, and hypergonadotropic hypogonadism.

115 of CD40 on TECs, development of fibrosis and hypothyroidism, and increased expression of proinflammat

116 hypothyroidism is more common than is overt hypothyroidism, and is usually defined as a serum thyroi

117 oid cancer (n = 25) patients, sex, length of hypothyroidism, and normal versus decreased glomerular f

118 matic mutation carriers at risk of recurrent hypothyroidism, and provides a rationale for adjunct iod

119 ase, obesity, hyperlipidemia, renal disease, hypothyroidism, and the number of outpatient visits for

120 ction within the normal range or subclinical hypothyroidism are also associated with AF.

121 Constipation, obesity, and hypothyroidism are associated comorbidities.

122 of the genomic changes mediated by the TR in hypothyroidism are independent of NCoR1, suggesting a ro

123 Because most of the clinical features of hypothyroidism are nonspecific, the diagnosis requires l

124 mechanism by which GLIS3 dysfunction causes hypothyroidism are unknown.

125 ign thyroid disorders, especially hyper- and hypothyroidism, are the most prevalent endocrine disorde

126 eases, type 1 diabetes, hyperthyroidism, and hypothyroidism, are the result of autoimmunity.

127 -subclinical hyperthyroidism and subclinical hypothyroidism--are common clinical entities that encomp

128 Some authors have defined subclinical hypothyroidism as a serum TSH between 5 and 10 mIU/L, an

129 serum TSH between 5 and 10 mIU/L, and overt hypothyroidism as a serum TSH greater than 10 mIU/L, but

130 ding hypertension and indicate developmental hypothyroidism as an epigenetic risk factor for cardiova

131 ne (TSH) 0.45 to 4.49 mIU/L, and subclinical hypothyroidism as TSH 4.5 to 19.9 mIU/L with free thyrox

132 ed as TSH of 0.45 to 4.49 mIU/L, subclinical hypothyroidism as TSH of 4.5 to 19.9 mIU/L, and subclini

133 Additionally, the first case of hypothyroidism associated with a collodion baby was repo

134 pecific TRbeta1-KO mice, we demonstrate that hypothyroidism-associated changes in gene expression and

135 rium at 9q22 near FOXE1 were associated with hypothyroidism at genome-wide significance, the stronges

136 For subclinical hypothyroidism (based on thyroid-stimulating hormone lev

137 Severe iodine deficiency causes goitre and hypothyroidism because, despite an increase in thyroid a

138 ironmental 131I exposure during childhood on hypothyroidism, but not other thyroid outcomes.

139 Hypothyroidism can have a variety of presentations.

140 Hypothyroidism can lead to depressed breathing.

141 evothyroxine, which is usually used to treat hypothyroidism, can be considered in such situations.

142 Using a mouse model of hypothyroidism caused by a dominant negative TRalpha1PV

143 Overview of congenital hypothyroidism caused by thyroid hormone synthesis defec

144 Central congenital hypothyroidism (CCH) is more prevalent in children born

145 before 20 weeks of gestation for subclinical hypothyroidism, defined as a thyrotropin level of 4.00 m

146 Subclinical hypothyroidism, defined as an elevated serum thyrotropin

147 e-treated patients with residual symptoms of hypothyroidism despite normalization of the serum TSH ha

148 models where autoimmune thyroid disease and hypothyroidism develop in most mice by 4 mo of age.

149 1.02-1.24) for children born to mothers with hypothyroidism diagnosed before and after delivery, comp

150 ren, 3524 children were born to mothers with hypothyroidism diagnosed before delivery and 4664 diagno

151 observed among children born to mothers with hypothyroidism diagnosed before delivery who did not rec

152 his study while accounting for the timing of hypothyroidism diagnosis.

153 12 weeks 3 days) and maternal treatment for hypothyroidism did not result in improved cognitive func

154 hould be treated, but those with subclinical hypothyroidism do not always benefit from treatment, esp

155 adequate evidence that treating subclinical hypothyroidism does not provide clinically meaningful im

156 a non-functional protein lead to congenital hypothyroidism due to I(-) transport defect (ITD).

157 Congenital hypothyroidism due to thyroid dyshormonogenesis is a het

158 Our findings suggest that maternal hypothyroidism, especially when it is untreated, increas

159 and surrounding tissues leading to profound hypothyroidism, findings consistent with our in vitro re

160 Her medical history included hypothyroidism from Hashimoto thyroiditis and pneumonia

161 each had grade 3 rhabdomyolysis and grade 2 hypothyroidism; grade 3 iridocyclitis, grade 1 erythema

162 h in normal thyroid group and in subclinical hypothyroidism group (40.4% and 46.7%, respectively), fo

163 we describe a child with classic features of hypothyroidism (growth retardation, developmental retard

164 However, in some patients, hypothyroidism has been ameliorated by unusually large a

165 Maternal hypothyroidism has previously been shown to increase ris

166 rs7850258 allele (G) associated with CLP and hypothyroidism has significantly greater enhancer activi

167 he effects of antiepileptic drugs in central hypothyroidism have not yet been studied, substantial ev

168 ed with survivors not so exposed for primary hypothyroidism (hazard ratio [HR], 6.6; 95% CI, 5.6 to 7

169 For participants with subclinical hypothyroidism, HRs from higher-quality studies were 1.1

170 ipheral neuropathy were evaluated: diabetes, hypothyroidism, hypercholesterolemia, hypertension, vari

171 sk of reduced urinary concentrating ability, hypothyroidism, hyperparathyroidism, and weight gain.

172 positive associations with antibody-positive hypothyroidism, hyperthyroidism, AIT, or elevated ATPO.

173 sterol, renal impairment, diabetes, obesity, hypothyroidism, hyperthyroidism, anaemia, respiratory di

174 associated with an increased odds ratio for hypothyroidism, hyperthyroidism, and antithyroid antibod

175 nderlies many autoimmune diseases, including hypothyroidism, hyperthyroidism, and rheumatoid arthriti

176 rral, knowledge of the signs and symptoms of hypothyroidism, hyperthyroidism, and thyroid nodules, as

177 We conducted a cross-sectional analysis of hypothyroidism, hyperthyroidism, autoimmune thyroiditis

178 ved for other types of inflammatory disease (hypothyroidism, hyperthyroidism, rheumatoid arthritis) a

179 regression models compared risks of primary hypothyroidism, hyperthyroidism, thyroid neoplasms, hypo

180 n, radiological assessment, and treatment of hypothyroidism, hyperthyroidism, thyroid nodules, and th

181 r radiotherapy were asymptomatic compensated hypothyroidism in 9 patients (10%), osteonecrosis and mo

182 f levothyroxine for treatment of subclinical hypothyroidism in adults aged 80 years and older.

183 We induced hypothyroidism in C57BL/6 and PLA2G2A-overexpressing (II

184 common findings that can be associated with hypothyroidism in children.

185 d mice developed multiple autoantibodies and hypothyroidism in contrast to mice treated with bone mar

186 olymorphism-carrying mouse exhibited UPR and hypothyroidism in distinct brain areas.

187 Hypothyroidism in fetal sheep induced by removal of the

188 Hypothyroidism in humans is typically associated with a

189 1 by TRalpha1 mutants could lead to clinical hypothyroidism in humans.

190 Our findings indicate that hypothyroidism in mice results in a variety of metabolic

191 Genetic disruption of KCNQ1-KCNE2 causes hypothyroidism in mice, resulting in cardiac hypertrophy

192 linical thyroid dysfunction, and subclinical hypothyroidism in particular, is associated with depress

193 ings do not support treatment of subclinical hypothyroidism in patients with acute myocardial infarct

194 Subclinical hypothyroidism in patients with acute myocardial infarct

195 oid hormone receptor alpha gene (THRA) cause hypothyroidism in patients with growth and developmental

196 We tested the hypothesis that the severe hypothyroidism in patients with THRA mutations is due to

197 targets in treating TRalpha1 mutant-mediated hypothyroidism in patients.

198 mation of the true prevalence of subclinical hypothyroidism in persons older than 70 years.

199 Early detection and appropriate treatment of hypothyroidism in pregnant women may be an area for poss

200 The association between 131I dose and hypothyroidism in the Belarusian cohort is consistent wi

201 terest in the possible link between maternal hypothyroidism in the perinatal period and childhood ast

202 Hypothyroidism in the sheep fetus resulted in an asymmet

203 ue the high mortality rate due to congenital hypothyroidism in these mice.

204 Hypothyroidism increased atrial interstitial fibrosis, b

205 genetic complexity of protective effects on hypothyroidism-induced hearing impairment, an F1 intercr

206 Hypothyroidism-induced NAFLD has generally been attribut

207 Our results show that the pathogenesis of hypothyroidism-induced NAFLD is both intra- and extrahep

208 yndrome, hemolytic anemia, thrombocytopenia, hypothyroidism, inflammatory myositis, Raynaud's disease

209 ge (identical to that causing human goitrous hypothyroidism) inhibits this maturation, although the C

210 In contrast, primary hypothyroidism intensified the Ala92-Dio2 phenotype, wit

211 Treatment of hypothyroidism involves the endogenous conversion of thy

212 Hypothyroidism is a common condition in which the thyroi

213 Hypothyroidism is a common condition of thyroid hormone

214 Recent studies suggest that long-term hypothyroidism is associated with human hepatocellular c

215 The definition of hypothyroidism is based on statistical reference ranges

216 Subclinical hypothyroidism is common and most individuals can be obs

217 he use of levothyroxine to treat subclinical hypothyroidism is controversial.

218 Once overt hypothyroidism is diagnosed, treatment with levothyroxin

219 ociated with HLA SNP variation and show that hypothyroidism is genetically correlated with Type I dia

220 Subclinical hypothyroidism is known to be associated with increased

221 The current treatment for patients with hypothyroidism is levothyroxine (L-T4) along with normal

222 Subclinical hypothyroidism is more common than is overt hypothyroidi

223 Subclinical hypothyroidism is most often caused by autoimmune (Hashi

224 ipheral thyrotoxicosis and reverses cerebral hypothyroidism is not yet available.

225 Congenital hypothyroidism is the most common neonatal endocrine dis

226 Hypothyroidism is the most common pregnancy-related thyr

227 Because hypothyroidism leads to growth defects and premature dea

228 cant positive associations of 131I dose with hypothyroidism (mainly subclinical and antibody-negative

229 Subclinical hypothyroidism may be associated with an increased risk

230 n, these findings suggest that developmental hypothyroidism may be the cause of cardiovascular disord

231 iodine deficiency-related maternal and child hypothyroidism may cause severe mental retardation, rece

232 Evidence indicates that cardiac hypothyroidism may contribute to heart failure progressi

233 ition, middle-aged patients with subclinical hypothyroidism may have cognitive impairment, nonspecifi

234 differ between participants with subclinical hypothyroidism (mean difference = 0.29, 95% confidence i

235 enomenon, we used a drug-free mouse model of hypothyroidism: mice lacking the sodium/iodide symporter

236 mic immune-mediated complications, including hypothyroidism (n = 3), colitis (n = 2), and hepatitis (

237 The odds ratio for hypothyroidism (n = 39) was 1.54 [95% confidence interva

238 naemia (n=2), fatigue (n=1), rash (n=1), and hypothyroidism (n=1).

239 We also report novel SNP associations for hypothyroidism near HLA-DQA1/HLA-DQB1 at rs6906021 (comb

240 ther features, including ichthyosis, primary hypothyroidism, neurological symptoms, and cryptorchidis

241 sease was associated with increased rates of hypothyroidism (odds ratio, 2.2; P < .01) and a lower th

242 oidism increases AF incidence, the effect of hypothyroidism on AF is unclear.

243 l will be useful for studying the effects of hypothyroidism on multiple organ systems.

244 e decisive for reaching a status of maternal hypothyroidism or hypothyroxinemia associated with impai

245 Treatment for subclinical hypothyroidism or hypothyroxinemia beginning between 8 a

246 men who are identified as having subclinical hypothyroidism or hypothyroxinemia during pregnancy impr

247 eening of all pregnant women for subclinical hypothyroidism or thyroid autoimmunity is not recommende

248 The strongest association in the PheWAS was hypothyroidism (OR = 0.76, p = 2.7 x 10(-13)), which had

249 vels may be below the reference range (overt hypothyroidism) or within the reference range (subclinic

250 .12; (95% CI: 0.83, 1.52) (n=105 cases); for hypothyroidism, OR=1.35 (95% CI: 0.86, 2.13) (n=49 cases

251 Those results persist after excluding hypothyroidism patients from the statistical analysis.

252 Thus, the hypothyroidism phenotype of Slc30a10 single knockouts is

253 Clinical manifestations of hypothyroidism range from life threatening to no signs o

254 For patients with subclinical hypothyroidism, recommendations for therapy differ betwe

255 perspectives on whether treating subclinical hypothyroidism reduces morbidity and mortality, whether

256 Among the disease conditions studied are Hypothyroidism, (Resistant) Hypertension, Type 1 and 2 D

257 Chronic hypothyroidism resulted in severely blunted basal human

258 Fetal hypothyroidism resulted in significant reductions in mit

259 -specific iPSCs generated from patients with hypothyroidism resulting from NKX2-1 haploinsufficiency.

260 valuate the relationship between subclinical hypothyroidism (SCH) and all-cause and cardiovascular di

261 Subclinical hypothyroidism (SCH) is a common endocrine disorder affe

262 yroid symptoms among adults with subclinical hypothyroidism (SCH).

263 All patients with overt hypothyroidism should be treated, but those with subclin

264 Monotherapy Fails to Restore All Markers of Hypothyroidism" should read "new technology is needed to

265 ss the epidemiology, causes, and symptoms of hypothyroidism; summarise evidence on diagnosis, long-te

266 opmental syndrome with epilepsy, ptosis, and hypothyroidism that differs from JBS.

267 A 45-year-old woman with hypothyroidism that has been treated with a stable dose

268 is a life-threatening form of decompensated hypothyroidism that must be treated with aggressive L-th

269 a genome-wide association study for primary hypothyroidism, the most common thyroid disease.

270 In subclinical hypothyroidism, the presence of depressive symptoms is o

271 rs of age and who had persisting subclinical hypothyroidism (thyrotropin level, 4.60 to 19.99 mIU per

272 fferent thyroid hormone levels, ranging from hypothyroidism to hyperthyroidism on AF inducibility in

273 g levothyroxine in patients with subclinical hypothyroidism to reduce the risk of developing depressi

274 ataplexy, hypocretin deficiency, and central hypothyroidism, together with brainstem encephalitis ref

275 ism did not affect graft function, but acute hypothyroidism transiently impaired human C-peptide secr

276 lts aged 80 years and older with subclinical hypothyroidism, treatment with levothyroxine, compared w

277 In the subclinical hypothyroidism trial, the median IQ score of the childre

278 A total of 677 women with subclinical hypothyroidism underwent randomization at a mean of 16.7

279 The excess odds ratio per 1 Gy for hypothyroidism was 0.34 (95% CI: 0.15, 0.62) and varied

280 ent, the prevalence of overt and subclinical hypothyroidism was 0.82% and 6.06%, respectively.

281 rt study found that treatment of subclinical hypothyroidism was associated with decreased risk for co

282 e analyzed, an increased risk of subclinical hypothyroidism was associated with high blood pressure (

283 rly people, neither clinical nor subclinical hypothyroidism was associated with MCI.

284 Iatrogenic hypothyroidism was associated with sepsis and neonatal a

285 Hypothyroidism was generated in SCID-beige mice using an

286 The prevalence of clinical hypothyroidism was increased in patients taking lithium

287 Hypothyroidism was not observed, and antidrug antibody w

288 Hypothyroidism was reduced in those taking valproate (HR

289 Clinical and subclinical hypothyroidism were ascertained from a medical records l

290 Hyperthyroidism and primary hypothyroidism were associated with sepsis, respiratory

291 Clinical and subclinical hypothyroidism were associated with worse Global gait th

292 .2 years, the incident rates for subclinical hypothyroidism were substantially higher in participants

293 cases distinguishing between NTIS and severe hypothyroidism, which is a rare primary cause for admiss

294 at a greater risk for developing subclinical hypothyroidism, while its mechanisms and temporal conseq

295 can result in autosomal recessive congenital hypothyroidism with deficient thyroglobulin.

296 TBMEHP exposure in the rat produced maternal hypothyroidism with markedly decreased serum T3 (3,3 ,5-

297 Association of clinical and subclinical hypothyroidism with MCI.

298 An association of clinical and subclinical hypothyroidism with mild cognitive impairment (MCI) has

299 t common treatment-related adverse event was hypothyroidism with pembrolizumab (in 33 [13%] patients)

300 The patient showed congenital central hypothyroidism with reduced TSH biopotency, over-secreti