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1 100 mmol/L glucose, or no additional solute (hypotonic solution).
2 nd blocked cell swelling normally induced by hypotonic solution.
3 ed ciliary epithelial (NPCE) cells by a 23 % hypotonic solution.
4 onic solution and was decreased 38% by a 33% hypotonic solution.
5 howed impaired regulatory volume decrease in hypotonic solution.
6 volume decrease during sustained exposure to hypotonic solution.
7 esponses to the TRPV4 agonists 4alphaPDD and hypotonic solutions.
8 rtonic media but with a steeper slope in the hypotonic solutions.
9 e filament, and varied it with hypertonic or hypotonic solutions.
10  cells secreted insulin when challenged with hypotonic solutions.
11 in cRNA by measuring the rate of swelling in hypotonic solutions.
12 t sympathetic neurones during application of hypotonic solutions.
13 ved by transferring the DEs to hypertonic or hypotonic solutions.
14 also observed in cells following exposure to hypotonic solutions.
15 esicles followed by washes with isotonic and hypotonic solutions.
16 ha-phorbol 12,13-didecanoate (4alphaPDD) and hypotonic solutions.
17                    Exposure of beta-cells to hypotonic solutions (10 and 33% hypotonicity) caused an
18 creatic islets was stimulated by exposure to hypotonic solutions (10-33% hypotonicity).
19         Cell swelling induced by exposure to hypotonic solutions (203 mOsm) caused a rapid increase i
20 o single-fiber recordings in rat showed that hypotonic solution activated 54% of C-fibers, an effect
21                                  Exposure to hypotonic solution activated a current that was blocked
22                     Exposure to a hyposmotic/hypotonic solution also led to a significant increase in
23       HIT cell volume initially increased in hypotonic solution and was followed by a regulatory volu
24 al membrane is permeable when replicating in hypotonic solution and within macrophages, resulting in
25 anges in relative cell volume in response to hypotonic solutions and glucose were studied in single i
26                                              Hypotonic solutions ( approximately 230 mOsm) activated
27            For CBCEPCs challenged with a 10% hypotonic solution at 37 degrees C, the kinetic constant
28 could be repeatedly activated by exposure to hypotonic solution but when ATP was omitted the current
29 cell and perforated patch recording methods, hypotonic solution caused cell swelling and the activati
30                                Exposure to a hypotonic solution caused the activation of a large, out
31                                      Whereas hypotonic solutions caused a transient stimulation of in
32 and water content were combined to show that hypotonic solutions caused water to move from the extrac
33 pting membrane properties, such as hyper- or hypotonic solutions, cholesterol removal and nonmuscle m
34 icantly attenuated cell volume regulation in hypotonic solutions, consistent with VRAC inhibition.
35                  Here, we describe a gelling hypotonic solution containing a low concentration of a t
36                                              Hypotonic solution did not cause a rise in intracellular
37                                              Hypotonic solutions evoked outwardly rectifying, non-ina
38                                              Hypotonic solutions first elicited a rapid increase in f
39                                              Hypotonic solutions increased and hypertonic solutions d
40                                  Exposure to hypotonic solutions increased relative CSAs of normal BD
41                                              Hypotonic solution induced transient Cl- channel activit
42 ngs, cell swelling induced by 1) exposure to hypotonic solution or 2) intracellular perfusion with hy
43 owing cell swelling induced by exposure to a hypotonic solution or in response to calcium-mobilizing
44 ntly placed in a high ionic strength buffer, hypotonic solution, or detergent.
45                   Longer (>1 h) exposures to hypotonic solutions permitted a subsequent slow decrease
46 ic acid and, to a lesser extent, DIDS to the hypotonic solution potentiated swelling and blocked the
47  sodium absorption greater than control with hypotonic solution represented sodium absorption stimula
48 by glucose, greater than that noted with the hypotonic solution, represented sodium absorption stimul
49              Exposure of beta-cells to a 15% hypotonic solution resulted in a transient depolarizatio
50                                        A 15% hypotonic solution stimulated insulin release to a peak
51                                4alphaPDD and hypotonic solutions stimulated SP and CGRP release from
52 ed with isomolar amounts of sucrose, whereas hypotonic solution was the same as isotonic solution wit
53                                      For the hypotonic solution, we found that hypotonicity inhibited
54 ls, we show that Ca2+ influx is triggered by hypotonic solutions, which can be partially blocked by G
55  currents inactivated during the exposure to hypotonic solution with time constants of 59 and 304 s f
56                                           In hypotonic solutions with ionic strengths ranging from <