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1 ogenic shock (CS), 7% as distributive, 3% as hypovolemic, 20% as mixed, and 4% as unknown.
2  the renin-angiotensin-aldosterone system to hypovolemic and hypotensive episodes.
3 cts a widespread clinical assumption that in hypovolemic cardiac arrest, the ventricles are collapsed
4 rcine models of ventricular fibrillation and hypovolemic cardiac arrest.
5 asopressors given during resuscitation after hypovolemic cardiac arrest.
6 ntilation and various shock states including hypovolemic, cardiogenic, obstructive, and septic shock.
7 model was used to induce progression towards hypovolemic cardiovascular instability.
8  or salt appetite in response to an isotonic hypovolemic challenge.
9 d the hypothesis that patients with POTS are hypovolemic compared with healthy controls and explored
10 e changes are of particular importance under hypovolemic conditions, especially when central venous b
11  were performed randomly in normovolemic and hypovolemic conditions.
12 42 (11.3%), and 34 (9.1%) were classified as hypovolemic, euvolemic, and hypervolemic at Time zero .
13 g disease and according to whether they have hypovolemic, euvolemic, or hypervolemic hyponatremia.
14                            All patients were hypovolemic (global end-diastolic volume index<680 mL/m)
15 them according to their fluid volume status (hypovolemic hyponatremia, euvolemic hyponatremia, or hyp
16 hen intravenously during ischemia induced by hypovolemic hypotension and bilateral common carotid art
17 set of cardiovascular collapse during severe hypovolemic hypotension in spontaneously breathing human
18 nvasive hemodynamic support in patients with hypovolemic hypotension once the blood loss has been con
19 nd limitations of this new approach to treat hypovolemic hypotension.
20 the prevention of ischemic tissue damage and hypovolemic (low blood volume) shock.
21 ontrol (n = 3), control plus L-NAME (n = 5), hypovolemic (n = 4), and hypovolemic plus L-NAME (n = 5)
22 s with pulmonary edema during the study were hypovolemic or euvolemic at the time pulmonary edema dev
23 igh plasma aldosterone levels detected under hypovolemic or hyperkalemic challenge can lead to increa
24  investigators as cardiogenic, distributive, hypovolemic, or mixed.
25                  Plasma lactate was lower in hypovolemic patients before (rs=0.38; p=0.05) and after
26 e minimal; however, hypotension can occur in hypovolemic patients.
27 g initial studies with both normovolemic and hypovolemic pigs, sequential increases in inspiratory im
28 lus L-NAME (n = 5), hypovolemic (n = 4), and hypovolemic plus L-NAME (n = 5).
29                                              Hypovolemic rabbits were bled of 10% of their circulatin
30 ean (p = .009) blood pressures compared with hypovolemic rabbits who received saline placebo.
31 al conditions other than hypoxia, such as in hypovolemic, septic, or cardiogenic shock.
32     The 30-day mortality rate was 18.4%, and hypovolemic shock (odds ratio 4.75; 95% confidence inter
33 tcomes Consortium in patients with traumatic hypovolemic shock (shock) or traumatic brain injury (TBI
34  injured patients, age 15 years or more with hypovolemic shock [systolic blood pressure (SBP) </= 70
35                                              Hypovolemic shock and hemorrhage continue to shape healt
36 ude that L-NAME may blunt hypotension during hypovolemic shock by inhibiting nitric oxide synthase an
37                                              Hypovolemic shock causes a whole body ischemia/reperfusi
38 c activity and mean arterial pressure during hypovolemic shock compared with control rabbits.
39 olecular research applies more to protracted hypovolemic shock followed by the systemic inflammatory
40  induce plasma loss from the circulation and hypovolemic shock in animals.
41       IL-4 exacerbation of histamine-induced hypovolemic shock in mice was dependent on VE expression
42                           Hypotension during hypovolemic shock may be attributable, in part, to the f
43                                              Hypovolemic shock of marked severity and duration may pr
44 with shock or TBI occur within 24 hours from hypovolemic shock or TBI.
45 during a prearrest control period and during hypovolemic shock produced by rapid hemorrhage of 35% of
46 d vs crystalloid solutions for management of hypovolemic shock remains unclear.
47                                       During hypovolemic shock there was a decrease in log low-freque
48  and left ventricular diameter compared with hypovolemic shock values by 34 +/- 2.5% and 20 +/- 2.5%,
49                                              Hypovolemic shock was maintained for 50 min.
50                                              Hypovolemic shock was present in 21.8% of patients, and
51 l stratified by case mix (sepsis, trauma, or hypovolemic shock without sepsis or trauma).
52 ied Model for End-Stage Liver Disease score, hypovolemic shock, and bacterial infection at inclusion
53 ortality resulting from intestinal bleeding, hypovolemic shock, and sepsis, even at a very low DSS co
54 cy, Model for End-Stage Liver Disease score, hypovolemic shock, hepatocellular carcinoma, and active
55                                              Hypovolemic shock, high-density lipoprotein, platelet co
56  randomized, double-blind trial of traumatic hypovolemic shock, HSD (250 mL) versus lactated Ringer's
57        Among patients with severe TBI not in hypovolemic shock, initial resuscitation with either hyp
58 y to damage human cells in vitro and prevent hypovolemic shock, organ necrosis and death in mice with
59 a variety of serious complications including hypovolemic shock, thrombocytopenia, and bleeding.
60 ons of hemorrhage, plasma extravasation, and hypovolemic shock, which leads to death.
61 ere largely protected from histamine-induced hypovolemic shock, which was associated with protection
62 e of 8 or less who did not meet criteria for hypovolemic shock.
63 ew therapeutic approach for the treatment of hypovolemic shock.
64 ension and acute renal failure, secondary to hypovolemic shock.
65 eased vascular permeability that may lead to hypovolemic shock.
66 k syndrome is a very rare cause of recurrent hypovolemic shock.
67 (ED) because of recurrent abdominal pain and hypovolemic shock.
68 h in vivo model systems of histamine-induced hypovolemic shock.
69 k: 3998 (31.5%), septic shock; 1457 (11.5%), hypovolemic shock; and 3625 (28.6%), other causes of sho
70 uces a potent vasoconstrictive effect during hypovolemic states.
71  by the collecting duct, particularly during hypovolemic states.
72 ntegrated baroreflex response to progressive hypovolemic stimuli.
73 oxone would augment tolerance to hypotensive hypovolemic stress (lower body negative pressure [LBNP])