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1 aumatic brain injury, and eight patients had hypoxic ischemic encephalopathy.
2 ter cardiac arrest could aggravate prolonged hypoxic ischemic encephalopathy.
3 One patient died as a result of hypoxic ischemic encephalopathy.
4 r disability at 18 months among infants with hypoxic-ischemic encephalopathy.
5 g/L who died had a cause of death other than hypoxic-ischemic encephalopathy.
6 disability at age 18 months in infants with hypoxic-ischemic encephalopathy.
7 ity during the critical period for perinatal hypoxic-ischemic encephalopathy.
8 early neonatal death and moderate-to-severe hypoxic-ischemic encephalopathy.
9 in young children who did not have neonatal hypoxic-ischemic encephalopathy.
10 sing therapeutic target in infants suffering hypoxic-ischemic encephalopathy.
11 isability in infants with moderate or severe hypoxic-ischemic encephalopathy.
12 s a window of therapeutic opportunity during hypoxic-ischemic encephalopathy.
13 izures, and received a clinical diagnosis of hypoxic-ischemic encephalopathy.
14 hemorrhage (IPH), extraaxial hemorrhage, and hypoxic-ischemic encephalopathy.
15 d in human brains in Alzheimer's disease and hypoxic-ischemic encephalopathy.
17 oke (7.0%), intracerebral hemorrhage (3.4%), hypoxic ischemic encephalopathy (3.6%), and spinal cord
18 confidence interval) were calculated: adult hypoxic-ischemic encephalopathy: absent 0% (0%-1%), abno
19 stem (adjusted HR, 46.4; 95% CI, 42.2-51.0), hypoxic ischemic encephalopathy (adjusted HR, 23.6; 95%
20 ere NDI, and cerebral palsy in neonates with hypoxic ischemic encephalopathy and correlated well with
22 0 days) were compared with severity of acute hypoxic-ischemic encephalopathy and long-term clinical o
23 atal cerebral sinovenous thrombosis, 11 with hypoxic ischemic encephalopathy, and 5 with neonatal art
24 onatal nontraumatic intracranial hemorrhage, hypoxic-ischemic encephalopathy, and neonatal mortality.
25 ces delivery room cardiorespiratory support, hypoxic-ischemic encephalopathy, and therapeutic hypothe
26 al in neonates (>/=36 weeks' gestation) with hypoxic-ischemic encephalopathy at 18 US centers in the
27 reduces death or disability for infants with hypoxic-ischemic encephalopathy at 36 weeks' or later ge
28 dmission to the NICU with moderate-to-severe hypoxic-ischemic encephalopathy at day 1 to 5 during hos
29 ive care unit (NICU) with moderate-to-severe hypoxic-ischemic encephalopathy at day 1 to 5 during hos
30 35 weeks' gestation with moderate or severe hypoxic-ischemic encephalopathy at less than 6 hours aft
32 wn to reduce death or disability in neonatal hypoxic ischemic encephalopathy but data on seizures dur
33 have neuroprotective effects in infants with hypoxic-ischemic encephalopathy, but its effects on neur
34 Among term neonates with moderate or severe hypoxic-ischemic encephalopathy, cooling for longer than
35 sonance imaging following moderate or severe hypoxic-ischemic encephalopathy developed by the Nationa
36 borns undergoing therapeutic hypothermia for hypoxic-ischemic encephalopathy did not result in a lowe
37 ' or later gestation with moderate or severe hypoxic-ischemic encephalopathy enrolled at 6 to 24 hour
39 Epo treatment for term infants with moderate hypoxic-ischemic encephalopathy found reduced disability
43 impairment (NDI) for infants diagnosed with hypoxic ischemic encephalopathy (HIE) is important for p
44 osis (MS) and newborn brain injuries such as hypoxic ischemic encephalopathy (HIE) that cause cerebra
45 s, unexpected neonatal unit (NNU) admission, hypoxic ischemic encephalopathy (HIE), and perinatal mor
46 th therapeutic hypothermia (TH) for neonatal hypoxic-ischemic encephalopathy (HIE) (cases) compared t
47 were male; 152 (72%) had moderate to severe hypoxic-ischemic encephalopathy (HIE) and 147 (84%) had
48 NFIA expression patterns in human neonatal hypoxic-ischemic encephalopathy (HIE) and MS as well as
50 uced hypothermia, the standard treatment for hypoxic-ischemic encephalopathy (HIE) in high-income cou
51 birth and hypothermic neuroprotection after hypoxic-ischemic encephalopathy (HIE) in low- and middle
60 ic hypothermia is the standard treatment for hypoxic-ischemic encephalopathy (HIE), but despite its w
61 pothermia is widely used after mild neonatal hypoxic-ischemic encephalopathy (HIE), safety and effica
66 Among infants 33 to 35 weeks' gestation with hypoxic-ischemic encephalopathy, hypothermia at less tha
69 less than or equal to 17 mug/L argue against hypoxic-ischemic encephalopathy incompatible with reawak
71 s who were full-term with moderate or severe hypoxic ischemic encephalopathy, longer cooling, deeper
72 is at birth (umbilical cord artery pH <7.0), hypoxic ischemic encephalopathy, neonatal seizures, neon
74 ational guidelines (adult cardiac arrest and hypoxic-ischemic encephalopathy of newborns) or intraope
76 f mechanical ventilation, and risk of death, hypoxic ischemic encephalopathy or respiratory arrest.
78 separately analyze the hypoxic component of hypoxic-ischemic encephalopathy, rats were prepared such
79 at 33.5 degrees C for 72 hours for neonatal hypoxic ischemic encephalopathy reduces death or disabil
80 for 72 hours at 33.5 degrees C for neonatal hypoxic-ischemic encephalopathy reduces death or disabil
81 rophylactic barbiturate therapy for neonatal hypoxic-ischemic encephalopathy showed no significant ef
82 trial of whole-body hypothermia for neonatal hypoxic-ischemic encephalopathy showing a significant re
83 or more of gestation with moderate or severe hypoxic-ischemic encephalopathy to receive erythropoieti
84 neurodevelopmental outcomes in neonates with hypoxic-ischemic encephalopathy using MRI and basic clin
86 18 girls; age range, 2-12 days) with severe hypoxic-ischemic encephalopathy were examined during the
88 efined as stroke, traumatic brain injury, or hypoxic ischemic encephalopathy with a Glasgow Coma Scal