コーパス検索結果 (left1)
通し番号をクリックするとPubMedの該当ページを表示します
1 nal cortisol infusion (1.2 mg kg(-1) day(-1) i.v. for 5 days, n = 20) on fetal glucose, lactate and o
2 ty was rejected as CL316,243 (0.15 mg kg(-1) i.v.) evoked similar rises in oxygen consumption (VO2) i
3 rial lipopolysaccharide (LPS, 500 mug kg(-1) i.v.) in rats at an ambient temperature of 22 degrees C.
4 (-1) i.v. dopamine, (3) 10 ug kg(-1) min(-1) i.v. dobutamine, and (4) following creation of an intra-
5 ns: (1) at baseline, (2) 2 ug kg(-1) min(-1) i.v. dopamine, (3) 10 ug kg(-1) min(-1) i.v. dobutamine,
8 ium-based contrast agent can be administered i.v. to the animal to detect mural inflammation or tumor
9 .c. HT-1080 tumors in nude mice administered i.v. docetaxel-containing nanoparticles was more effecti
10 However, the majority of MSCs administered i.v. encounter a pulmonary impasse and soon disappear fr
11 at a dose of 9.0 x 10(5) PfSPZ administered i.v. three times at 8-wk intervals to 15 malaria-naive a
17 s were experimentally seeded via an adoptive i.v. transfer of luciferase-expressing CT26 CRC cells th
21 nism, but the antinociceptive activity after i.v. administration could not be directly correlated to
24 acquisitions were performed before and after i.v. or intrapulmonary administration of the nanoparticl
25 on, the amount of accumulated antibody after i.v. application was calculated relative to its apoptosi
26 um concentration obtained in the blood after i.v. administration correlates with body weight across a
27 The mean t1/2 of KRN23 was 8-12 days after i.v. administration and 13-19 days after s.c. administra
31 levels of effector cytokines in hosts after i.v. AML induction, consistent with abortive T cell acti
32 4.7 T before, during and up to 60 min after i.v. injection of EP-3533, or of its non-binding isomer
33 ssue rapidly and homogeneously; 30 min after i.v. injection, siRNA-L2 achieved uptake in 99% of tumor
34 Bacteria were captured within minutes after i.v. injection and were associated with Mphis in both li
35 schemia reperfusion injury mouse model after i.v. injection confirms the ability of injected neutroph
36 MRI and optical imaging were performed after i.v. injection of the liposomal nanoprobes into mice bea
37 s spectrometry (ICP-MS), respectively, after i.v. injection of the payload loaded HA NPs in tumor bea
38 nal centers (GCs), and the Ab response after i.v. administration of IgG3 anti-trinitrophenyl (TNP) in
40 e antigen to erythrocyte cell surfaces after i.v. injection, one using a conjugate with an erythrocyt
41 nized mice were completely protected against i.v. Stx2 challenge, and weaned mice receiving an oral c
42 ere we report significant protection against i.v. acquisition of SIVmac239 using a long-lasting appro
43 netics and immune responses induced after an i.v. infection with a Brazilian ZIKV clinical isolate (H
45 n the legs of mice followed 3 wk later by an i.v. exposure to Staphylococcus aureus This procedure re
46 ability to protect pigtail macaques from an i.v. high-dose cell-associated SHIVSF162P3 challenge.
48 time each active lever press resulted in an i.v. cocaine infusion paired with one of two cues that a
49 ich time active lever presses resulted in an i.v. infusion of cocaine that was paired with a light/to
51 unized and five control subjects received an i.v. challenge with P. falciparum-infected erythrocytes.
52 w paradigm in the treatment of PAH, using an i.v./sublingual tissue-penetrating homing peptide to sel
53 ective role, we studied both aerosolized and i.v. models of mycobacterial infection and observed MIF-
54 everal volatile (isoflurane, desflurane) and i.v. (propofol) general anesthetics excite peripheral se
56 ortant finding is that both intratumoral and i.v. administration demonstrated a significant enhanceme
57 mmed death ligand 1 binding site just before i.v. injection of IgM/LPS-pretreated BMDC or using IL-10
58 cer cell lines with anti-CD73 AD2 mAb before i.v. injection into mice inhibited extravasation/coloniz
60 erum, and up to 70% were protected from both i.v. and mosquito bite challenge with transgenic Plasmod
62 ntation of in vivo-formed Ag-Ab complexes by i.v. injecting mice with Ag-specific Abs followed by the
65 levels of corticosterone were normalized by i.v. leptin infusion at a dose that raises low plasma le
70 us aureus implant infections were treated by i.v. administration of activated or non-activated MSC, w
71 NV infection, we administered exogenous CCL7 i.v. to WNV-infected Ccl7(-/-) mice and observed a signi
74 Macrophage depletion was achieved by daily i.v. clodronate liposomes (-1 day to +3 days) during Ang
75 t up to 0.20% to 0.97% of antibody delivered i.v. reached the brain tumor, but that apoptosis inducti
80 MyTrCa(-/-)) mice that succumbed to low-dose i.v. VSV infection with similar kinetics as IFN-I recept
83 ared both a three-dose i.m. and a three-dose i.v. parenteral ARS regimen with the standard five-dose
84 = 331; three-dose i.m.: n = 338; three-dose i.v.: n = 333); 139 participants were lost to follow-up.
86 with ischemic AKI, administration of ECFCs (i.v.) at the time of reperfusion significantly attenuate
88 rug memory was later reactivated with either i.v. or i.p. cocaine presentation in the absence of any
89 subjects underwent experimental endotoxemia (i.v. administration of 2 ng/kg Escherichia coli endotoxi
93 y superior compared with free drug following i.v. delivery, exploiting the "enhanced permeability and
95 a fluorescent dye, illuminated MB following i.v. injection and was able to distribute throughout the
96 t to the spleen, were all observed following i.v. but not foodborne transmission of L. monocytogenes.
97 ntrauterine infection was observed following i.v. rhCMV inoculation during the early second trimester
98 half-life was 46.1 h in mini-pigs following i.v. administration, and semaglutide has an MRT of 63.6
99 ecific regulatory function in vivo following i.v. injection of non-T cells derived from the spleen of
101 available in vivo gene delivery formulation, i.v. delivery of the core/PEGylated shell (CPS) nanopart
102 lated with their in vivo Ag acquisition from i.v. injected Ag-loaded splenic non-T cells, and in vivo
103 ly protected mice with humanized livers from i.v.- and mosquito bite-delivered P. falciparum sporozoi
105 n with Abraxane(R) (12mg paclitaxel/kg given i.v.), ACT induced a strong increase in the therapeutic
106 when submitted to hyperglycemic (40% glucose i.v.) and hypoglycemic (5 U/kg insulin i.v.) challenges.
107 (10 and 20 mg/kg, 10 and 20 mg kg/h for 1 h, i.v.), pazopanib (30 and100 mg/kg, i.p.), or vandetanib
108 nib (3 and 6 mg/kg, 3 and 6 mg/kg/h for 1 h, i.v.), sorafenib (10 and 20 mg/kg, 10 and 20 mg kg/h for
109 f blast trauma with multiorgan hemorrhaging, i.v. administration of the hemostatic nanoparticles led
112 Splenocytes from wild-type mice infected i.v. produced significantly more IFN-beta than did those
115 ter 5 days of recovery, mothers were infused i.v. for 30 min with either vehicle (n = 11), low dose (
117 y charged microdroplet clusters are injected i.v., activated within the target pathology by diagnosti
118 on biased data collected with cells injected i.v., a route in which most transferred cells enter via
121 e radiolabelled imaging probes were injected i.v. into wild-type, Sglt1(-/-) , Sglt2(-/-) and Glut2(-
122 scapular and suprarenal region were injected i.v. with 1 mg of TF12 and, after 16 hours of tumor accu
124 tered methamphetamine (0.03 mg/kg/injection, i.v.) under a fixed-ratio 20 schedule of reinforcement (
128 himeric H77/JFH1 virus (HJ3-5), intravenous (i.v.) challenge with 10(6) FFU H77S.2 virus resulted in
129 body localization of NPs after intravenous (i.v.) injection into live mice bearing human lung tumors
130 s in the retina and brain after intravenous (i.v.) injection of infected monocytes or dendritic cells
132 n CSF following intrathecal and intravenous (i.v.) administration of ziconotide was investigated.
133 transmission efficiency between intravenous (i.v.) and i.c. routes to estimate dose-dependent TTvCJD
137 ycoside moiety was required for intravenous (i.v.) but not for intracerebroventricular (i.c.v.) activ
139 ction also extended to a lethal intravenous (i.v.) C. albicans challenge but had no effect in the C.
141 al efficacy and tolerability of intravenous (i.v.) and intraperitoneal (i.p.) paclitaxel combined wit
143 d investigate the efficiency of intravenous (i.v.), intraperitoneal (i.p.), and intramuscular (i.m.)
144 1:1 to treatment with serelaxin intravenous (i.v.) infusion (for 60 min at 80 mug/kg/d and then 60 mi
146 ied Carba NP test that utilized intravenous (i.v.) imipenem-cilastatin, which is less expensive than
150 cked C. albicans mutant given intravenously (i.v.) and S. aureus given intraperitoneally (i.p.) faile
152 into two groups and injected intravenously (i.v.) 6 hours post-MCAO with either 1 mg/kg PNU-120596 (
153 that underlie the ability of intravenously (i.v.) administered STING-NPs to induce STING activation
154 enged intratracheally (i.t.), intravenously (i.v.), or intraperitoneally (i.p.) with Escherichia coli
157 Current treatment of these attacks involves i.v. administration of hemin, but a faster-acting, more
158 ., NIR light exposure; group 4=cet-tra-IR700 i.v. and additional cet-tra-IR700 i.v. at 24h but no lig
159 -tra-IR700 i.v. and additional cet-tra-IR700 i.v. at 24h but no light exposure; group 5=cet-tra-IR700
160 light exposure and additional cet-tra-IR700 i.v. immediately after NIR but no additional NIR light e
161 but no light exposure; group 5=cet-tra-IR700 i.v., NIR light exposure and additional cet-tra-IR700 i.
162 v., no light exposure; group 3=cet-tra-IR700 i.v., NIR light exposure; group 4=cet-tra-IR700 i.v. and
163 group 1=no treatment; group 2=cet-tra-IR700 i.v., no light exposure; group 3=cet-tra-IR700 i.v., NIR
168 sepsis, the administration of LXA4 (7 mug/kg i.v.) 1 h after surgery increased neutrophil phagocytic
169 apomorphine (100-200 ug/kg i.p. or 20 ug/kg i.v.) administration, suggesting that it was not due to
170 days, received an insulin bolus (10 units/kg i.v.) plus insulin infusion (50 mU/kg/min i.v.) until hy
171 (KOR) agonist salvinorin A (0.01-1.8 mg/kg, i.v.) before administration of the KOR selective radiotr
172 i.m.) attenuated cocaine-induced (1.0 mg/kg, i.v.) dopamine overflow in the caudate nucleus but not i
173 istering subanesthetic ketamine (1-30 mg/kg, i.v.) or vehicle to conscious rats (n=12) and testing th
174 at brain demonstrated that 4-MEC (1-3 mg/kg, i.v.) produced large increases in extracellular 5-HT, sm
176 single daily cocaine-reinforced (1.0 mg/kg, i.v.) runway trials 10 min after intracranial injection
180 pha2-AR agonists dexmedetomidine (25 mug/kg, i.v.) and clonidine (100 mug/kg, i.v.) inhibited shiveri
181 The effects of CP 55 940 (1.0-10 mug/kg, i.v.) and THC (3.0-300 mug/kg, i.v.) on food-maintained
182 (25 mug/kg, i.v.) and clonidine (100 mug/kg, i.v.) inhibited shivering EMGs, BAT SNA, and BAT thermog
183 .0-10 mug/kg, i.v.) and THC (3.0-300 mug/kg, i.v.) on food-maintained responding and body temperature
184 olidine methiodide (nicotine(PM), 30 mug/kg, i.v.) resulted in habituation (tolerance) of the same ph
187 gh, shock-inducing dose of LPS (5000 mug/kg, i.v.); this attenuation was due to a blockade of cold-se
191 at 30 mug/kg/d) or terlipressin (single 2-mg i.v. bolus), and the regional hemodynamic effects were q
193 kg i.v.) plus insulin infusion (50 mU/kg/min i.v.) until hypoglycemia ensued (</=3.9 mmol/L) (experim
194 n = 7) or SSTR2a infusion (3,000 nmol/kg/min i.v., n = 12) 60 min prior to the same insulin regimen.
197 s in contrast to single IA (sIA) or multiple i.v. (mIV) injections of relaxin-2 with which the ROM re
200 f the profound tolerance-inducing ability of i.v. administered Ag-coupled splenocytes (Ag-SP) in mice
202 ly enhance the pulmonary vascular effects of i.v. vasodilators in Sugen5416/hypoxia/normoxia-exposed
206 was moderately correlated with the number of i.v. sedations personally performed in periodontal pract
209 te an increasing use of high-dose therapy of i.v. gammaglobulin (IVIg) in the treatment of various T
211 ivided into 3 groups: control (group 1), one i.v. bolus DNase I before CPB start (group 2) and a seco
212 at 0, 24, and 48 h) either i.m. (n = 348) or i.v. (n = 351), both of which were the intervention arms
213 ed whether tumor cells were injected s.c. or i.v. and independently of the injection route of the hUC
217 y assigned to receive either 50 mg pethidine i.v. (n = 48), or an equal volume of 0.9% normal saline
223 n Pd1-/- mice were recapitulated by repeated i.v. applications of nivolumab in WT mice, even though n
224 acteria and wound healing following repeated i.v. administration of activated allogeneic canine MSC.
226 in data obtained during a frequently sampled i.v. glucose tolerance test showed that the antidiabetic
227 relapsed HL were evaluated with up to seven i.v. infusions of the radiolabeled anti-CD25 antibody (9
229 ee groups of mice were treated with a single i.v. injection of rAvPAL-RBCs at three different doses t
230 re examined a model without injury; a single i.v. injection of unfractionated BMCs in healthy mice.
232 ynamic stress via sodium nitroprusside (SNP) i.v.] on stimulus evoked responses of sensory processing
233 urnover under post-absorptive and fed state (i.v. Glamin to double amino acids, dextrose to sustain g
236 detailed account is provided for successful i.v. administration of SERRS nanoparticles such that del
237 o experiments were carried out upon systemic i.v. injections with anti-CD137 mAbs and showed CD137 in
238 munized human MUC1 transgenic mice (MUC1.Tg) i.v. with a MUC1 peptide vaccine against which they gene
243 ialysis in rat nucleus accumbens showed that i.v. administration of 0.3 and 1.0 mg/kg of mephedrone o
244 the tumor Ag MUC1, we previously showed that i.v. immunization of MUC1 transgenic mice, but not wild-
253 table role in mediating host survival by the i.v. route was not recapitulated following a mucosal inf
256 n this phase 1b trial, we concluded that the i.v. administration of peptide-loaded dendritic cells is
257 Administration of T4(+) T cells using the i.v. or intratumoral routes achieves partial tumor regre
258 s a higher bioavailability compared with the i.v. administration of the commercial docetaxel solution
259 We added stable isotope-labeled threonine i.v. to mice and combined fluorescence in situ hybridiza
260 with the addition of aerosolized colistin to i.v. treatment, whereas the addition of aerosolized coli
262 the incidence of AKI in patients exposed to i.v. contrast medium was directly compared with the inci
265 erosolized colistin as adjunctive therapy to i.v. antimicrobials or as monotherapy in the treatment o
266 ne (4get) or Cd45.1 allele was used to track i.v. transferred eosinophils into the airway following a
267 Accordingly, when infected mice were treated i.v. with an MHC-II-restricted M. tuberculosis epitope p
268 a consequence of STING activation in tumors, i.v. administered STING-NPs reprogram the TME towards a
276 he largest percentage of periodontists using i.v. sedation (74.0%) was reported from American Academy
277 were comparing adjunctive aerosolized versus i.v. colistin (seven observational cohort or case-contro
278 be delivered into the blood circulation via i.v. injection, recharged by 400-nm photoexcitation ligh
281 ncing in xenograft and orthotopic tumors via i.v. injection or aerosol inhalation, respectively.
283 tions, we compared the efficacy of 12 weekly i.v. infusions of PerT-GUS versus native GUS on (i) deli
287 ve rats received three weekly sessions where i.v. injected Lipo-DOX was combined with FUS-BBBD; an ad
290 y almost an order of magnitude compared with i.v. delivery with substantially lower plasma concentrat
293 e improved anticancer activity compared with i.v. Taxotere, observed in the metastatic lung cancer mo
295 sculature, achieved by pretreating mice with i.v. liposome-encapsulated clodronate, significantly att