コーパス検索結果 (１語後でソート)
通し番号をクリックするとPubMedの該当ページを表示します
1 essenger produced during the type III CRISPR immune response.
2 ty of transmission, vectorial competence and immune response.
3 ay be at play during the earliest AD-related immune response.
4 lopment of one arm of the mammalian adaptive immune response.
5 , leukocyte adherence, and activation of the immune response.
6 ary vascular bed is crucial for an effective immune response.
7 ture-enabled selection for enhanced adaptive immune response.
8 t their binding domains elicits a protective immune response.
9 ry (TBI) inadequately recapitulate the human immune response.
10 oglia that are not associated with an innate immune response.
11 replication and/or by stimulating the innate immune response.
12 tent with either an appropriate or excessive immune response.
13 CT in the monitoring of tumoral and systemic immune response.
14 closely associated with the induction of an immune response.
15 ntly initiating a well-orchestrated adaptive immune response.
16 pathogen products and initialize the innate immune response.
17 redetermination and dynamics of the adaptive immune response.
18 ignaling through a genuine tissue-autonomous immune response.
19 significant impact on modulation of the host immune response.
20 etween mast cell tumors, microbiota and host immune response.
21 tance of protein synthesis during the larval immune response.
22 gesting a role for TLR sensing in the innate immune response.
23 epitopes and the formation of a hierarchical immune response.
24 tance by developing a more reactive cellular immune response.
25 of Dot1L in T cells resulted in an impaired immune response.
26 affects several pathways of the host innate immune response.
27 AC/JENVAC combination exhibiting the highest immune response.
28 fore may increase the efficacy of the innate immune response.
29 in their affinity for antigen throughout the immune response.
30 bacterial effector functions and the humoral immune response.
31 invasion and hence are important targets of immune responses.
32 d non-coding regions could elicit anti-tumor immune responses.
33 d to the control of host innate and adaptive immune responses.
34 robiome, which is known to influence mucosal immune responses.
35 of immune cells with functions in different immune responses.
36 regimen does not up-regulate inflammatory or immune responses.
37 th limited understanding of lncRNAs in human immune responses.
38 d with TSLP and antigen to evaluate cellular immune responses.
39 onists to CD40 have shown to induce acquired immune responses.
40 or (NLR) genes as an important mechanism for immune responses.
41 it is protected from host cytoplasmic innate immune responses.
42 ular links between innate cells and adaptive immune responses.
43 to navigate the B-cell compartment and evade immune responses.
44 e ALVAC vector could increase the protective immune responses.
45 tial adjuvants for the induction of adaptive immune responses.
46 e in the gills and the suppression of type 1 immune responses.
47 n recognition receptors that initiate innate immune responses.
48 do not typically generate robust anti-tumour immune responses.
49 obiota has a leading role in shaping (early) immune responses.
50 roperties, leading to detrimental effects on immune responses.
51 cellularly to trigger the modulation of host immune responses.
52 ved virulence proteins (effectors) to induce immune responses.
53 , can result in deleterious pro-inflammatory immune responses.
54 s on the role of CoV receptors in modulating immune responses.
55 1-expressing exosomes can inhibit antitumour immune responses.
56 vity of lysozyme, virulence, and host innate immune responses.
57 ung (especially ILC2s), and decreased type 2 immune responses.
58 t may contribute to phenotypic variations in immune responses.
59 often fail to elicit potent and long-lasting immune responses.
60 ete of the host population, and muted innate immune responses.
61 logy of the intestinal epithelium and innate immune responses.
62 ls, by enhancing the quality and survival of immune responses.
63 potentiate ionizing radiation-induced innate immune responses.
64 for infection, gastroenteritis symptoms, and immune responses.
65 ve been described in normal and dysregulated immune responses.
66 of an intracellular niche, and modulation of immune responses.
67 icacy and potency to help Env trimer humoral immune responses.
68 rase 1 (PRMT1) is a key regulator of hepatic immune responses.
69 ire and highlight their potential effects on immune responses.
70 y for the generation of adaptive anti-tumour immune responses.
72 n drive protective humoral and cell-mediated immune responses(2) and might reduce the potential for d
73 kin cell biology, including activation of an immune response, a switch in cell metabolism and process
75 nous Myd88 can effectively modulate the host immune response against AAV-mediated gene therapy and in
76 administered to mice and generated a humoral immune response against both peptide antigen, and the pa
78 induced a remarkable increase of functional immune responses against GBS compared to the simple co-a
80 Metabolic fitness of T cells is crucial for immune responses against infections and tumorigenesis.
83 vaccine elicits strong humoral and cellular immune responses against pathogenic Ebola viruses and su
88 r cells have the potential to amplify innate immune response and activate antitumor adaptive response
90 rine xenogeneic model, activating the innate immune response and increasing tumor infiltrating macrop
91 interactions between the virus and the host immune response and of the pathogenesis of infection is
93 monstrate the emergence of pathogen-specific immune responses and a concomitant rise in plasma inflam
94 lamydia must evade both intracellular innate immune responses and adaptive cytotoxic T cell responses
95 ersity of Mycobacterium tuberculosis affects immune responses and clinical outcomes of tuberculosis (
97 MHC II-presented antigens, are essential for immune responses and develop from CD4(+)CD8(+) thymocyte
99 ulation of NEMO is essential for host innate immune responses and for maintenance of homeostasis.
100 ccine adjuvants capable of inducing cellular immune responses and protective efficacy against intrace
101 es robust systemic humoral and cell-mediated immune responses and protects against lung infection, in
102 the protease MALT1 is required for adaptive immune responses and regulatory T (Treg)-cell developmen
103 inhaled allergens and microbes, which alter immune responses and subsequent risk for diseases, such
104 a transcription factor (AhR) that regulates immune responses and the biological responses to xenobio
105 tion, ranging from cellular communication to immune responses and the protein-driven mineralization o
106 In this study, we assess the anatomy of immune responses and the relationship with bacterial loc
107 ct on the quality of the HIV-specific T cell immune responses and transcriptional profiles of PBMC, r
108 hat said, S1P plays pleiotropic roles in the immune response, and it will be important to build an in
109 licted by scratching, a T(H)2 cell-dominated immune response, and susceptibility to viral skin infect
110 s B (CHB) is associated with a dysfunctional immune response, and therefore a selective TLR8 agonist
111 veral mechanisms to modulate the host innate immune response, and these likely contribute to the seve
112 ion of genes involved in cell proliferation, immune responses, and cholesterol biosynthesis, increase
114 Triplex elicited and amplified CMV-specific immune responses, and fewer Triplex-vaccinated patients
116 ge and thromboinflammation, dysregulation of immune responses, and maladaptation of ACE2-related path
117 ower respiratory tract, humoral and cellular immune responses, and pathologic evidence of viral pneum
118 ystemically affect homeostasis in poststroke immune responses, and pinpointed multiple affected pathw
119 r of subtype C pox-protein vaccines restored immune responses, and slowed response decay compared to
120 way hyperresponsiveness, innate and adaptive immune responses, and type 2 cytokine production in a mo
126 phages, an important component of the innate immune response, are a key regulator of intestinal micro
128 ed as modulators of both adaptive and innate immune responses, as well as players in an important rol
131 nisms underlying the heterogeneity of type 2 immune responses between individuals and between differe
132 gh levels of NS1-specfic antibody and T cell immune responses but provided only partial protection ag
133 diverse proinflammatory conditions promotes immune responses, but certain signals also induce tolero
134 flanking the CD4bs, and 2) minimize the nND immune response by engineering fusion proteins consistin
136 ically, we show that acetate enhances innate immune responses by acting on both neutrophils and ILC3s
138 the NF-kappaB family play a crucial role for immune responses by activating the expression of chemoki
139 alterations that blunt productive anti-tumor immune responses by directly or indirectly excluding eff
140 ombinations could synergistically potentiate immune responses by elevating cytokine and chemokine pro
141 ave shown that resetting the host intestinal immune responses by treatment with either a healthy feca
142 f the capsid and transgene to avoid the host immune response (by genetically modifying these componen
144 cells, and tumor cells to determine how the immune response changes due to three macrophage-based im
146 reatment in high altitude reduced the type 2 immune response, corrected the increased CRTH2 expressio
147 host genes related to translation and innate immune response could contribute to differential resista
150 rial-based AML vaccination can induce potent immune responses, deplete AML cells and prevent disease
151 canine heartworm disease, mounts a stronger immune response during infection than does a susceptible
152 ous dsRNA formation and a deleterious innate immune response during mammalian hematopoietic developme
153 cacy offer a unique opportunity to study the immune response during Plasmodium falciparum infection.
155 populations that have provided insight into immune responses during acute infection, including addit
158 Consistent with the known roles of ILF3 in immune response, epithelial-mesenchymal differential edi
159 ies of SARS-CoV-2 are necessary to determine immune responses, evidence from other coronaviruses can
161 ytokine production to promote an altered Th2 immune response following RSV infection that leads to mo
163 intly infer infection histories and describe immune responses generated by these infections using ant
164 ntly increased, whereas expression of innate immune response genes are significantly decreased, in hu
165 oad, with a greater effect in patients whose immune response had not yet been initiated or who had a
166 mplex interplay between tumoral and systemic immune response has been provided through tissue and liq
167 egulating not only mucosal but also systemic immune responses has led to investigations into the impa
169 ding of the interplay between tumors and the immune response holds the promise for increasing the res
170 nfection, mice lacking TAGAP mount defective immune responses, impaired Th17 cell differentiation, an
171 ation that lack rapid testing of the patient immune response, impeding clinicians from providing appr
172 he signals leading to a T(H)2 cell-dominated immune response in AD are not completely understood.
173 and to verify the development of an adaptive immune response in infected individuals.METHODSWe studie
176 ghly nitrated proteins induces a tolerogenic immune response in the food allergy model and in human i
177 acrophages fit into the context of an innate immune response in whole animals with multiple cell type
179 ened group 3 regimen induced comparable peak immune responses in 30 rhesus monkeys as in humans and r
181 extremely useful for the in vivo analyses of immune responses in a variety of models, including organ
182 accine-induced activating versus suppressive immune responses in affording protection from HIV.IMPORT
183 he mRNA-1273 vaccine induced anti-SARS-CoV-2 immune responses in all participants, and no trial-limit
185 ranscriptional gene expression profiles, and immune responses in HIV-infected adult solid organ trans
187 ave characterized the T cell recognition and immune responses in mice to two naturally presented infl
191 istration on post myocardial infarction (MI) immune responses in vivo and paracrine-mediated immune c
192 -CoV-2 antigen-specific humoral and cellular immune responses, including potent neutralizing antibodi
195 s have provided evidence that the anti-tumor immune response is reduced in both conditions, while res
196 within dystrophic muscle, or that an innate immune response is required for effective donor cell eng
198 ed regulation of cell death and inflammatory immune responses is essential to promote protection agai
199 indicate that the modulation of heterologous immune responses is induced by vaccination with Tdap and
201 smitted infection, is potentially cleared by immune response, is not focused in the liver, and possib
202 lecular pattern on host cells that may guide immune responses leading to the concomitant damage assoc
208 -CoV-2 in rhesus macaques, and that cellular immune responses may contribute to protection if antibod
210 two clinical isolates, we identify distinct immune response networks for each strain, leading to pre
211 nction in antibiotic drug resistance and the immune response of cells against viruses, by stabilizing
213 mechanisms contributing to the dysfunctional immune response of the elderly to the vaccine against in
215 oxidase (PO) and the cytokine Spatzle during immune responses of insects is mediated by a network of
216 ral nervous system regulate the inflammatory immune responses of macrophages during clearance of bloo
217 es of elevated CO(2) (eCO(2) ) on endogenous immune responses of monarch larvae to infection by O. el
219 pathogenesis, innate viral control, adaptive immune responses or the balance of inflammation and tiss
220 rating the likely critical role these innate immune responses play in early mucosal HIV replication i
224 r pharmacologically without compromising the immune response, providing a new approach to treat disea
229 viral reservoir along with an HIV-1-specific immune response seems to be key for the spontaneous func
230 dentified several inflammatory responses and immune response signaling pathways, including NF-kB sign
231 Overexpression of OsWAKL21.2 in rice induces immune responses similar to those activated by LipA trea
235 ed kidney damage without augmenting adaptive immune responses, suggesting it might offer a novel adju
236 tablished dominance of STAT1 function in the immune response suggests an obligate link between inflam
237 protective immunity and amplifies the type 2 immune response that may favor the development of crypto
238 nds we postulated to achieve heightened Th-1 immune response that would yield pronounced protective v
239 dicate that C6(-/-) mice have reduced innate immune responses that result in less organ injury and im
241 ted with exaggerated allergen-induced type 2 immune responses, these responses are strongly influence
242 CD4(+) T cells control and orchestrated most immune responses, this shortcoming severely hampers the
243 ts demonstrated that stromal Lama5 regulated immune responses through altering LN structures and T ce
244 ems have provided evidence that balancing of immune responses through inhibitory receptors is an impo
245 for orchestrating diverse, pathogen-specific immune responses through their differentiation into a nu
247 cs (including variation in HLA genes) in the immune response to coronaviruses, as well as the clinica
249 to explain alternate B cell recruitment into immune response to foreign antigens vs. induction of tol
250 cal first step in understanding the earliest immune response to HIV-1 and suggest that changes in blo
254 ient sera to facilitate investigation of the immune response to NoV.IMPORTANCE NoV infections are a l
256 hronic disease, based on a persistent type 2 immune response to respiratory infection with a natural
258 on what we do and do not know regarding our immune response to SARS-CoV-2, and provide a number of s
259 COVID-19) requires understanding the natural immune response to severe acute respiratory syndrome cor
261 s suggest that assessing the proinflammatory immune response to trauma exposure immediately after tra
262 earch is needed to understand how to monitor immune response to vaccines in immunosuppressed patients
267 inding, neutralizing antibodies and cellular immune responses to Ebolavirus (EBOV) glycoprotein.
274 e, Takahashi et al. found sex differences in immune responses to SARS-CoV-2 and the predictors of dis
275 f CD4(+) T cells to protective or pathogenic immune responses to SARS-CoV-2 infection remains unknown
277 ming immunization provides a method to focus immune responses to the desired target region, in the ab
279 ory function, CD40L has been used to enhance immune responses to vaccines, including candidate vaccin
280 e proteins that regulate innate and adaptive immune responses to viral infection by engaging with rec
281 ome, an escalation of the cytotoxic adaptive immune response triggered upon the binding of pathogenic
283 ycle, cell death, cell motility, DNA repair, immune response, two phosphorylation pathways, and a ran
284 Cancer immunotherapies enhance anti-tumor immune responses using checkpoint inhibitors, such as PD
285 s in antiviral defense, influencing adaptive immune responses via interactions with dendritic cells (
287 m the thymus to the periphery and during the immune response, we discuss in broad terms developmental
288 bset differentially influences the nature of immune responses, we sought factors that would allow the
289 on lung function, tissue remodeling, and the immune response were analyzed by using wild-type and B-c
292 s (JNKs) and p38s that are required for host immune response, whereas extracellular signal-regulated
293 d by the same drug (5-azacytidine) elicit an immune response, which may be important for patient's re
294 , Salmonella Typhi, generates cross-reactive immune responses, which provide far greater resistance a
295 tissues show a massively upregulated innate immune response with signatures of nuclear factor-kappaB
297 oplasma components that stimulate the innate immune response with the same basic regulatory mechanism
298 n dictates pathogenesis or counters the host immune response would provide targets for the developmen
299 hat IgE production was not limited to type 2 immune responses yet was generally constrained in vivo.
300 Tumors subvert immune cell function to evade immune responses, yet the complex mechanisms driving imm