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1 d cells may thus be central to the described immunodeficiency.
2 fection can be seen in cases with underlying immunodeficiency.
3 defects in POLD1 as a novel cause of T-cell immunodeficiency.
4 s of the nuclear factor-kappaB pathway cause immunodeficiency.
5 thus characterizing the disorder as primary immunodeficiency.
6 first-line tool for the diagnosis of primary immunodeficiency.
7 k, particularly in individuals with previous immunodeficiency.
8 e for a severe, clinically relevant systemic immunodeficiency.
9 o a severe clinical presentation of combined immunodeficiency.
10 toinflammatory disorder, 14 (15%) a combined immunodeficiency, 3 (3%) an innate immune defect, and 2
11 ed in an adult with ectodermal dysplasia and immunodeficiency a germline, gain-of-function mutation,
13 ut hematopoietic cell transplant, congenital immunodeficiency (adjusted odds ratio, 1.90; 95% CI, 1.2
14 ratio, 3.15; 95% CI, 2.09-4.74), congenital immunodeficiency (adjusted odds ratio, 6.94; 95% CI, 3.8
17 associated with food insecurity can lead to immunodeficiencies and shifts in the microbiome that dri
18 cy in humans, causing clinically significant immunodeficiency and an autoimmune lymphoproliferative s
20 reported to be pathogenic in common variable immunodeficiency and glomerulocystic kidney disease.
23 mophagocytic lymphohistiocytosis, congenital immunodeficiency, and hematopoietic cell transplant are
24 s in the LFA-1-C3-axis contribute to primary immunodeficiency, and identifies intracellular C3 as bio
26 NF-kappaB1-related phenotype, which includes immunodeficiency, autoimmunity, autoinflammation, and ca
27 erlying EBV-induced disease in these primary immunodeficiencies but also identified molecules and pat
28 lokathexis (WHIM) syndrome is a rare primary immunodeficiency caused by gain-of-function mutations in
29 within the last 20 yr to correct the T cell immunodeficiency caused by gammac-deficiency (SCID X1) a
32 scribe five patients from four families with immunodeficiency coupled with atopy, lymphoproliferation
33 RSF13B [n = 3]) or cause a CVID-like primary immunodeficiency (CTLA4 [n = 2], KMT2D [n = 2], or BIRC4
35 of immune deficiency in 235 common variable immunodeficiency (CVID) patients seen in the United Stat
36 omplication in patients with common variable immunodeficiency (CVID), but the optimal treatment is un
37 demy of Allergy, Asthma & Immunology Primary Immunodeficiency Diseases Committee established a work g
40 -Aldrich syndrome (WAS), an X-linked primary immunodeficiency disorder (PID) resulting from the defic
41 conditions have been clustered with primary immunodeficiencies in the latest practice parameters; ho
42 eath downstream of FAS and FASL-cause severe immunodeficiency in addition to autoimmune lymphoprolife
43 der cases with severe intestinal atresia and immunodeficiency in cases with complete loss of protein.
46 in this field provided insights into primary immunodeficiencies, inherited autoimmune and autoinflamm
47 velopment of diabetes in NOD severe combined immunodeficiency mice receiving diabetogenic splenocytes
50 d after establishment of the Italian Primary Immunodeficiency Network registry (84 months before vs 2
51 lticenter study based on the Italian Primary Immunodeficiency Network registry was established in 200
52 The precise mechanism leading to profound immunodeficiency of HIV-infected patients is still only
54 lones associated with cell cycle and primary immunodeficiency pathways identifies patients with signi
56 nic granulomatous disease (CGD) is a primary immunodeficiency resulting in life-threatening infection
57 mined in LNCaP tumor-bearing severe combined immunodeficiency (SCID) mice after sacrifice at defined
60 ional Maternal Pediatric Adolescent Acquired Immunodeficiency Syndrome Clinical Trials [IMPAACT] P106
63 d-Specific)/Lsh in human DNA causes a severe immunodeficiency syndrome, but the nature of the defect
64 HIV-2, a human pathogen that causes acquired immunodeficiency syndrome, is distinct from the more pre
68 hrough newborn screening for severe combined immunodeficiency using the T-cell receptor excision circ
69 ect on LDL-C Lowering in SubJEcts with Human Immunodeficiency VirRus and INcreased Cardiovascular Ris
71 those of humans with cytomegalovirus, human immunodeficiency virus (even when under antiretroviral t
74 ne is an effective means of decreasing human immunodeficiency virus (HIV) acquisition among women.
75 e been implicated in increased risk of human immunodeficiency virus (HIV) acquisition, but whether th
79 rate the central nervous system during human immunodeficiency virus (HIV) and cryptococcal meningitis
80 rapy has transformed the management of human immunodeficiency virus (HIV) and hepatitis C (HCV) coinf
81 We assessed prevalence of testing for human immunodeficiency virus (HIV) and hepatitis C virus (HCV)
82 ch associated with the pathogenesis of human immunodeficiency virus (HIV) and hepatitis C virus .
83 e is frequent among people living with human immunodeficiency virus (HIV) and is associated with redu
84 nflammation in adults living with both human immunodeficiency virus (HIV) and tuberculosis (TB), but
86 d with hepatitis B virus (HBV) and the human immunodeficiency virus (HIV) are at risk of developing h
87 ed fibrosis in hepatitis B virus (HBV)-human immunodeficiency virus (HIV) co-infected patients receiv
89 itical barrier to the development of a human immunodeficiency virus (HIV) cure is the lack of a scala
91 idelines recommend genotype testing at human immunodeficiency virus (HIV) diagnosis ("baseline genoty
92 ltimorbidity (MM) according to year of human immunodeficiency virus (HIV) diagnosis in elderly people
99 ons may be an effective way to prevent human immunodeficiency virus (HIV) in adolescent girls and you
100 th and renal impairment in people with human immunodeficiency virus (HIV) in resource-limited setting
101 V) drugs are highly active against the human immunodeficiency virus (HIV) in the body compartment, th
102 s of immune activation in persons with human immunodeficiency virus (HIV) in whom a sustained virolog
103 cause of morbidity and mortality among Human Immunodeficiency virus (HIV) infected patients; however
106 ontributes to immune activation during human immunodeficiency virus (HIV) infection and is usually as
107 in sub-Saharan Africa are at risk for human immunodeficiency virus (HIV) infection and unintended pr
108 the incidence of rectal gonorrhea and human immunodeficiency virus (HIV) infection in men who have s
109 at among people living with late-stage human immunodeficiency virus (HIV) infection initiating antire
110 rimoxazole preventive therapy (CPT) in human immunodeficiency virus (HIV) infection is a World Health
111 revious studies suggest that untreated human immunodeficiency virus (HIV) infection is associated wit
114 Identification of nonviral markers of human immunodeficiency virus (HIV) infection that increase bef
115 h as, diabetes, organ transplantation, Human immunodeficiency virus (HIV) infection, and elevated ser
116 ng global infectious diseases, such as human immunodeficiency virus (HIV) infection, malaria and infl
120 adelphia identified an outbreak of new human immunodeficiency virus (HIV) infections among persons wh
121 se management during the past decades, human immunodeficiency virus (HIV) infections are still respon
125 asing injection drug use (IDU)-related human immunodeficiency virus (HIV) outbreaks, particularly in
127 ed 2007-2017 medical records data from Human Immunodeficiency Virus (HIV) Outpatient Study (HOPS) par
130 naX mRNA and the gag-pol transcript of Human Immunodeficiency Virus (HIV) perturb translation elongat
131 troviral therapy (ART), CD4+ count and human immunodeficiency virus (HIV) plasma viral load (PVL) on
133 the feasibility of daily and nondaily human immunodeficiency virus (HIV) preexposure prophylaxis (Pr
137 old, in Montreal, we compared observed human immunodeficiency virus (HIV) seroconcordance in previous
139 <1 year, 1-4 years, adults), site, and human immunodeficiency virus (HIV) status (adults only) were c
140 cted controls and investigated whether human immunodeficiency virus (HIV) status is independently ass
144 ic to the VS.IMPORTANCE The lentivirus human immunodeficiency virus (HIV) targets and destroys CD4(+)
146 have made it possible for persons with human immunodeficiency virus (HIV) to live a near expected lif
149 iral load testing within 6 months when human immunodeficiency virus (HIV) viral loads exceed 1,000 co
150 ive inflammation in people living with human immunodeficiency virus (HIV) who have pulmonary tubercul
151 ses, occurring in patients living with human immunodeficiency virus (HIV) who were on antiretroviral
152 y for the syndemic of opioid overdose, human immunodeficiency virus (HIV), and hepatitis C virus (HCV
153 ation, violence, alcohol and drug use, human immunodeficiency virus (HIV), and stigma and discriminat
154 tiation rates among people living with human immunodeficiency virus (HIV), and then examined who was
155 sted for antiviral effects against the human immunodeficiency virus (HIV), followed by a comprehensiv
156 ully protective vaccines, particularly human immunodeficiency virus (HIV), malaria and tuberculosis.
159 HAART), lung disease remains common in human immunodeficiency virus (HIV)-infected (HIV+) adolescents
160 al therapy (cART)-treated, perinatally human immunodeficiency virus (HIV)-infected adolescents (PHIV+
164 data with new data from a prospective human immunodeficiency virus (HIV)-negative UK cohort of 333 t
166 rates of liver injury among cotreated human immunodeficiency virus (HIV)-TB coinfected patients were
167 ing 30 elite controllers (ECs), and 30 human immunodeficiency virus (HIV)-uninfected controls were as
182 ct on clinical endpoints among treated human immunodeficiency virus (HIV)/HCV coinfected persons is u
185 sful in controlling the replication of Human Immunodeficiency Virus (HIV-1) in many patients, current
186 r weight gain among people living with human immunodeficiency virus (HIV; PLWH) starting efavirenz-ba
187 c variability among people living with human immunodeficiency virus (HIV; PLWH), but remains unexplor
188 ment (NCI) in aging people living with human immunodeficiency virus (PLHIV) and people not living wit
190 syndrome (IRIS) in people living with human immunodeficiency virus (PLHIV) is rarely reported.This s
191 with enrichment for people living with human immunodeficiency virus (PLHIV), within 72 hours of intak
194 gically suppressed persons living with human immunodeficiency virus (PLWH) switched from older antire
199 an observational study of persons with human immunodeficiency virus (PWH) and uninfected controls, IL
200 ribing trends among people living with human immunodeficiency virus (PWH) are not well described.
205 e and one pivotal experiment in simian-human immunodeficiency virus (SHIV)-infected rhesus macaques,
209 nodeficiency virus type 1 (HIV-1) and simian immunodeficiency virus (SIV) acquisition in humans and m
210 m macaques infected with a mixture of simian immunodeficiency virus (SIV) and simian-human immunodefi
211 drophobic residues typically found in simian immunodeficiency virus (SIV) Env to improve rhesus CD4 b
212 mployed a barcoded synthetic swarm of simian immunodeficiency virus (SIV) in rhesus macaques to inves
213 rded as the main source of latent HIV/simian immunodeficiency virus (SIV) infection of adult humans a
215 ur approach was to deliver the entire simian immunodeficiency virus (SIV) proteome by serial vaccinat
217 we performed a comprehensive study of simian immunodeficiency virus (SIV)/SHIV-infected infant rhesus
219 ations in hair among women living with human immunodeficiency virus (WLHIV) in the United States.
222 s ac4C to RNAs, have been subverted by human immunodeficiency virus 1 (HIV-1) to increase viral gene
225 The majority of cells with latent human immunodeficiency virus 1 infection are located in lympho
226 cute respiratory syndrome coronavirus, human immunodeficiency virus 1, hepatitis C virus, and Japanes
228 hinder efforts to end the intertwined human immunodeficiency virus and opioid overdose epidemics.
229 cohort study, one-half of adults with human immunodeficiency virus and pulmonary tuberculosis experi
231 Using partially validated data from a human immunodeficiency virus cohort, we illustrate the calcula
233 epatocellular carcinoma [HCC]), non-acquired immunodeficiency virus defining malignancy (NADM; exclud
235 in injury induced by acute simian (or human) immunodeficiency virus infection may persist or spontane
237 Is) were the first drugs used to treat human immunodeficiency virus infection, and their use can caus
238 hronic inflammatory diseases including human immunodeficiency virus infection, psoriasis, rheumatoid
241 ith diabetes at baseline, those with a human immunodeficiency virus or hepatitis B virus coinfection,
243 stingly, primer extension assays using human immunodeficiency virus reverse transcriptase (HIV-RT) an
244 Ms) (n = 13) were infected with simian/human immunodeficiency virus SHIV.C.CH505.375H.dCT, and triple
246 50 to 8000 Hz included weekly AG dose, human immunodeficiency virus status with CD4 count, age, serum
248 oviral therapy (ART) during home-based human immunodeficiency virus testing improved engagement in ca
250 rst innate immune cells that encounter human immunodeficiency virus type 1 (HIV-1) after sexual conta
251 accine provided modest protection from human immunodeficiency virus type 1 (HIV-1) and simian immunod
252 the suitability of EBOV GP pseudotyped human immunodeficiency virus type 1 (HIV-1) and vesicular stom
260 tion for patients with newly diagnosed human immunodeficiency virus type 1 (HIV-1) infection, but pro
265 icipants: baseline CD4 count, baseline human immunodeficiency virus type 1 (HIV-1) RNA, and CYP2B6 me
266 is an important preclinical model for human immunodeficiency virus type 1 (HIV-1) vaccines, therapeu
268 ment of therapeutic strategies against human immunodeficiency virus type 1 (HIV-1), but their assembl
271 cardiovascular diseases in people with human immunodeficiency virus who were exposed to darunavir (DR
274 s, syphilis, gonorrhea, chlamydia, and human immunodeficiency virus, as well as elevated blood lead l
276 with an immunocompromising condition (human immunodeficiency virus, diabetes, or malnutrition) were
277 hly treatment-experienced persons with human immunodeficiency virus, extensive antiretroviral drug re
278 eles from divergent clades of HIV and simian immunodeficiency virus, including from primary patient i
279 al human-pathogenic viruses, including human immunodeficiency virus, influenza A virus, and yellow fe
280 ted viruses including cytomegalovirus, human immunodeficiency virus, influenza virus, and dengue viru
281 pear to retain modest activity against human immunodeficiency virus-1 with these mutations possibly a
283 We used Mycobacterium tuberculosis/simian immunodeficiency virus-coinfected (M. tuberculosis/SIV-c
286 antiretroviral therapy (ART)-treated simian immunodeficiency virus-infected rhesus macaques (RMs) un
287 i sarcoma is the most common cancer in human immunodeficiency virus-positive individuals and is cause
288 d treatment, of whom 77% (n = 23) were human immunodeficiency virus-positive, 47% (n = 14) had ever i
294 gnant and postpartum women living with human immunodeficiency virus; however the relative contributio
297 spectively studied 502 children with primary immunodeficiency who were transplanted at our center bet
298 ause reticular dysgenesis, a severe combined immunodeficiency with agranulocytosis, lymphopenia, and
299 n patients with a recessive form of combined immunodeficiency with defects in T, B, and NK cell activ
300 erpes zoster (HZ) include increasing age and immunodeficiency, yet estimates of HZ risk by immunocomp