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1                                 By analyzing immunoglobulin V and D gene usages, complementarity-dete
2 ell repertoire is generated by combinatorial immunoglobulin V(D)J gene segment rearrangements that oc
3                  The random recombination of immunoglobulin V(D)J gene segments produces unique IgM a
4 A-breaks induce error-prone DNA synthesis in immunoglobulin V(D)J regions by error-prone DNA polymera
5 tigen receptors through the rearrangement of immunoglobulin V, D, and J gene fragments, whereas jawle
6 l hotspot in the Ser31 codon of a transgenic immunoglobulin V gene allowed us to use PCR to detect tr
7  cell lymphoma leads to a failure to perform immunoglobulin V gene conversion.
8 blated the pattern of diversification of the immunoglobulin V gene in the chicken DT40 B-cell lymphom
9                        Leukemic cells of the immunoglobulin V gene mutation subgroups differ in telom
10 henotypes of B-CLL patients with and without immunoglobulin V gene mutations suggests that the 2 subg
11 thways for processing a common lesion in the immunoglobulin V gene.
12 gnition, VRC01 contacts gp120 mainly through immunoglobulin V-gene regions substantially altered from
13 al cases of CLL cells that expressed mutated immunoglobulin V genes and ZAP-70 also experienced highe
14                    After gene rearrangement, immunoglobulin V genes are further diversified by either
15  lines that constitutively hypermutate their immunoglobulin V genes during culture can be exploited t
16  that major intrinsic mutational hotspots in immunoglobulin V genes in vivo might reflect favoured si
17     We find that although the codon usage in immunoglobulin V genes renders them distinct among trans
18 action, random mutations are introduced into immunoglobulin V genes to increase the affinity of antib
19 of human heavy, kappa and lambda light chain immunoglobulin V-genes are in reasonably good agreement
20                                 Mutations in immunoglobulin V-genes can be due to gene multiplication
21 ytes and that the B-CLL cells that differ in immunoglobulin V genotype may have different antigen-enc
22 reaks specifically in the variable region of immunoglobulin (V(H)) gene locus, leading to hypermutati
23 raction induces hypermutation of heavy-chain immunoglobulin (V(H)) in B cells.
24  shown to express a restricted repertoire of immunoglobulin V(H) and V(L) genes, V(H)1-69 and VkappaA
25                                     Although immunoglobulin V(H) mutation status (IgV(H) MS) is progn
26 hisms in a human autosomal region (i.e., the immunoglobulin V(H) region), through use of single haplo
27 activating function for Pax5 in RAG-mediated immunoglobulin V(H)-to-DJ(H) recombination.
28 mutation (SHM) process that normally targets immunoglobulin V (IgV) genes in B cells appears to misfi
29  immunotherapy with steroids and intravenous immunoglobulins vs. late immunotherapy), and a low white
30 f ligands by the presence of a single set of immunoglobulin-V-like and immunoglobulin-C-like (VC) dom
31 late switch-mediated gene conversions in the immunoglobulin V locus of BRCA1 mutant chicken DT40 cell
32 s that have failed to productively rearrange immunoglobulin V region genes encoding a functional B-ce
33                         Previous analyses of immunoglobulin V region promoters has led to the discove
34 equent DNA repair generates mutations within immunoglobulin V-regions.
35 e the altered alternative splicing of CD33's Immunoglobulin V-set domain 2 and ultimately influence A
36 e, using NMR spectroscopy we showed that the immunoglobulin V-type domain of RAGE is responsible for