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1 All patients recovered after intravenous immunoglobulin therapy.
2 eresis (PPH) in conjunction with intravenous immunoglobulin therapy.
3 uble-blind, placebo-controlled trials of RSV immunoglobulin therapy.
4 e illness may be ameliorated by passive oral immunoglobulin therapy.
5 the anti-inflammatory activity of human i.v. immunoglobulin therapy.
6 taminated, open wounds with immunization and immunoglobulin therapy.
7 f response to corticosteroid and intravenous immunoglobulin therapy, a 3-day course of plasmapheresis
8 Group 1 and 2 patients received no hepatitis immunoglobulin therapy after transplantation and receive
10 d on novel therapeutic interventions such as immunoglobulin therapy and immunosuppressive therapy in
11 analysis of anti-Hbs elimination can improve immunoglobulin therapy and prevent recurrence of clinica
12 f juvenile rheumatoid arthritis, intravenous immunoglobulin therapy appeared ineffective in the disea
13 imen using plasmapheresis and/or intravenous immunoglobulin therapy, but underlying mechanisms that c
14 must be in some doubt, although intravenous immunoglobulin therapy has been shown to be beneficial i
16 November 2002 to November 2004, intravenous immunoglobulin therapy (IVIG) was administered to patien
18 gy in 80 patients established (>6 months) on immunoglobulin therapy; prospective analysis of HBV sero
19 atory properties associated with intravenous immunoglobulin therapy require the sialic acid modificat
22 roid pulse therapy and high-dose intravenous immunoglobulin therapy was successful in complete re-epi
23 anaged effectively with monthly prophylactic immunoglobulin therapy whereas SSCLS frequently does not
24 nd controversial indications for intravenous immunoglobulin therapy, with special emphasis on its mec