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1 matory monocytes (Mo)/macrophages (MPhi) and impaired healing.
2 which suffer from neutrophil persistence and impaired healing.
3 een associated with chronic inflammation and impaired healing.
4 is associated with chronic inflammation and impaired healing.
5 f NRF2-activating compounds in patients with impaired healing.
6 agents, is giving new hope to the problem of impaired healing.
7 clinical studies and in our animal models of impaired healing.
8 ammatory monocytes (Mo)/macrophages (MO) and impaired healing.
9 , there is an increased risk of fracture and impaired healing.
10 process and deficiencies in the MMP9 lead to impaired healing.
12 tailored "living bandage" for patients with impaired healing and can serve as prototype for the deve
13 Corneal injury in HO-2 null mice leads to impaired healing and chronic inflammatory complications,
14 urnal rhythm disruption immediately after MI impaired healing and exacerbated maladaptive cardiac rem
16 ic wounds may serve as a molecular marker of impaired healing and may provide future targets for ther
17 istent gaps exist in scientific knowledge of impaired healing and the ability of clinicians to reduce
18 nds is believed to play an important role in impaired healing and the development of infection-relate
22 beta-catenin/c-myc pathway(s) contributes to impaired healing by inhibiting keratinocyte migration an
23 in, thus providing a potential mechanism for impaired healing by local B2AR activation in wound-edge
24 ic foot ulcers and other chronic wounds with impaired healing can be treated with bioengineered skin
28 migration by impairing connexin 43, and that impaired healing during NEC is associated with reduced g
32 actors in chronic wounds and the reversal of impaired healing in animal models, pressure ulcer trials
33 the molecular mechanisms and pathogenesis of impaired healing in chronic ulcers is a serious health i
36 en, to determine the contribution of TSP2 to impaired healing in diabetes, we developed a novel diabe
39 ive shift in wound microbiota coincides with impaired healing in diabetic mice (Lepr(db/db); db/db).
45 aging to compare the course of efficient and impaired healing in wild-type and FXIII-/- mice, respect
46 mice but reduced by 90% in CD73 mutants, (3) impaired healing involves M1-driven immune response with
47 issue formation in healing gingiva, and that impaired healing is associated with factors that decreas
49 und healing complications (wound dehiscence, impaired healing, lymphocele, and incisional hernia) wit
52 peptic ulcer or troublesome dyspepsia led to impaired healing of gastric ulcers and did not affect th
55 ntium Correspondingly, AQP3(-/-) mice showed impaired healing of superficial wounds in the colon and
56 function but is insufficient to correct the impaired healing of the HO-2(-/-) cornea, suggesting tha
57 uring the past 3 decades presented issues of impaired healing or increased risk of cardiac rupture or
58 akage and reduced fibroplasia, indicating an impaired healing response consistent with angiogenesis b
60 d2(-/-) skin microbiome dominated and caused impaired healing, shown in cross-fostering experiments o
62 ith chronic Ly-6C(hi) monocytosis results in impaired healing, underscoring the need for a balanced a
63 Local and systemic factors can contribute to impaired healing, with the potential to prolong function