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1 ondrial intermembrane protein AIF (apoptosis-inducing factor).
2 of mitochondrial cytochrome c and apoptosis-inducing factor.
3 tress and mitochondrial release of apoptosis-inducing factor.
4 of Toll-like receptor-associated interferon-inducing factor.
5 , to mature caspase-8, which is an apoptosis-inducing factor.
6 the ability to respond to the male soporific-inducing factor.
7 ic apoptosis through caspase-3 and apoptosis-inducing factor.
8 lled and released cytochrome c and apoptosis-inducing factor.
9 n, controls production of a diffusible heart-inducing factor.
10 ic protein with some similarity to apoptosis-inducing factor.
11 hondrial proteins cytochrome c and apoptosis-inducing factor.
12 mac/DIABLO, but not the release of apoptosis-inducing factor.
13 tein was termed S. mansoni-derived apoptosis-inducing factor.
14 riosteal cells release a soluble cholinergic inducing factor.
15 /TRX, originally described as an IL-2R alpha-inducing factor.
16 ulation of the proapoptotic factor apoptosis inducing factor.
17 eceptor-interacting protein 1, and apoptosis-inducing factor.
18 ptotic pathway associated with the apoptosis-inducing factor.
19 e it also coincided with increased apoptosis-inducing factor.
20 fect of HDLs on beta-cells against ER stress-inducing factors.
21 for the production of endoderm-derived heart-inducing factors.
22 rasites and reversed the induction of anergy-inducing factors.
23 y, suggesting that bone may provide dormancy-inducing factors.
24 rounds of stimulation in the presence of Th1-inducing factors.
25 termediates (ROI), and release of cell death-inducing factors.
26 on requires cooperation with differentiation-inducing factors.
27 ntified in studies to reveal cell senescence-inducing factors.
28 expression of miR-200 in the absence of EMT-inducing factors.
30 to the prestalk cell inducer Differentiation inducing factor 1 (DIF-1), or are subjected to hyper-osm
31 ette-inducing small molecule, dubbed rosette-inducing factor 1 (RIF-1), produced by the Gram-negative
34 DNA arrays were used to identify the hypoxia-inducing factor-1 (HIF-1) as a downstream target of TGF-
35 looxygenase-1 (COX-1) and COX-2, and hypoxia-inducing factor-1 alpha (HIF-1 alpha) and vascular endot
36 of several stress pathways, such as hypoxia-inducing factor-1-alpha, receptor-interacting protein 1,
41 rocess involving secretion of the cell death-inducing factor 24p3 by mouse leukemia cells, raising th
42 nsity, release of cytochrome C and apoptosis inducing factor, (4) chromatin condensation, nuclear lam
43 poxia, (3) nanoparticles suppressing hypoxia-inducing factor, (4) nanoparticles that relieve tumor hy
44 derived activator of caspases, and apoptosis-inducing factor, accompanied by a proteolytic cascade wi
45 survival factors or overexpression of death-inducing factors accounted for the phenotypes observed.
46 al transition (EMT) in that a variety of EMT-inducing factors activate signaling pathways that conver
47 sed on our initial finding that the mesoderm-inducing factor activin A is suppressed by DeltaNp63 in
50 ecursor, mature, and apoptotic) of apoptosis-inducing factor (AIF) and co-localizes with apoptotic AI
51 ted Bcl-2, resulting in release of apoptosis-inducing factor (AIF) and cytochrome c from mitochondria
53 dependent mitochondrial release of apoptosis-inducing factor (AIF) and cytochrome complex (Cyt c) is
54 ax and Bak were enhanced, and both apoptosis-inducing factor (AIF) and endonuclease G (Endo G) were r
55 ation of the proapoptotic factors, apoptosis-inducing factor (AIF) and endonuclease G (EndoG), throug
56 bserved a nuclear translocation of apoptosis-inducing factor (AIF) and endonuclease G in CNGA3(-/-)/N
57 h involve release of mitochondrial apoptosis-inducing factor (AIF) and its translocation to the nucle
60 nylate kinase-2, cytochrome c, and apoptosis-inducing factor (AIF) during apoptosis and compared the
61 lease and nuclear translocation of apoptotis-inducing factor (AIF) followed by irreversible caspase-i
64 ctivation and the translocation of apoptosis-inducing factor (AIF) from the mitochondria to the nucle
65 n is required for translocation of apoptosis-inducing factor (AIF) from the mitochondria to the nucle
66 ion as a proviral insertion in the apoptosis-inducing factor (Aif) gene, causing about an 80% reducti
67 duced the nuclear translocation of apoptosis-inducing factor (AIF) in A2058 and SKMEL5 cells, and the
68 on in the mitochondrial release of apoptosis-inducing factor (AIF) in cisplatin-treated renal tubular
71 ndrial release of cytochrome c and apoptosis-inducing factor (AIF) in the penumbra region were reduce
80 tivation, and cleavage, as well as apoptosis-inducing factor (AIF) nuclear translocation and executio
82 haracterize the mechanism by which apoptosis-inducing factor (AIF) regulates CI biogenesis by trackin
83 dependent cell death, triggered by apoptosis-inducing factor (AIF) release from mitochondria and its
85 sm of cisplatin-induced apoptosis, apoptosis-inducing factor (AIF) release into the cytosol was obser
87 nsity and nuclear translocation of apoptosis-inducing factor (AIF) suggesting caspase-independent cel
88 release of cytochrome c (CytC) and apoptosis-inducing factor (AIF) through upregulation of Bax expres
89 hat the intramitochondrial protein apoptosis-inducing factor (AIF) translocates to the nucleus and pr
90 ndent neuronal death that involves apoptosis-inducing factor (AIF) translocation from mitochondria to
91 through a unique pathway involving apoptosis-inducing factor (AIF) translocation into the nucleus.
92 d with the mitochondrial localized apoptosis inducing factor (AIF) under both normal and oxidant stre
95 Cytosolic cytochrome c and nuclear apoptosis-inducing factor (AIF) were increased 3 h after OGD, and
96 uction increased the activation of apoptosis-inducing factor (AIF), a caspase-independent cell death
100 sitive for nuclear localization of apoptosis-inducing factor (AIF), an early event in apoptosis.
101 d AIFM1 gene encodes mitochondrial apoptosis-inducing factor (AIF), an FAD-containing and NADH-specif
102 rylation, as well as cytochrome c, apoptosis-inducing factor (AIF), and endonuclease G (EndoG) releas
103 tivation, nuclear translocation of apoptosis-inducing factor (AIF), and induction of p53, and prevent
104 nd modified caspase-3, Bcl-2, Bad, apoptosis-inducing factor (AIF), and PARP were quantified by immun
105 apoptogenic factors cytochrome c, apoptosis-inducing factor (AIF), and proinflammatory high-mobility
107 etion and mitochondrial release of apoptosis-inducing factor (AIF), but the causal relationships betw
108 ction in the mitochondrial protein apoptosis-inducing factor (AIF), exhibited signs of oxidative stre
109 of caspase-independent apoptosis, apoptosis-inducing factor (AIF), from mitochondria is induced by H
110 ear translocation of mitochondrial apoptosis-inducing factor (AIF), known to trigger both apoptotic m
111 of mitochondrial cytochrome C and apoptosis inducing factor (AIF), LC3B-positive neurons, and expres
112 ial release of cytochrome c, Smac, apoptosis-inducing factor (AIF), or loss of mitochondrial membrane
113 iators, including cytochrome c and apoptosis-inducing factor (AIF), was studied in the absence and pr
114 zed a human gene homologous to the apoptosis-inducing factor (AIF), which is named AIF-like (AIFL).
115 X, and release of cytochrome c and apoptosis-inducing factor (AIF), which was translocated to the nuc
129 Liberibacter solanacearum": the apoptosis-inducing factor AIF3 was downregulated in LsoA-infected
130 dentified mitochondrion-associated apoptosis inducing factor (AIFM1) have roles in the induction of a
131 loss was linked to upregulation of apoptosis-inducing factor, although only a minute fraction of cell
133 expression of apoptotic proteins (apoptosis-inducing factor and cleaved caspase-3) and autophagy pro
134 rleukin (IL)-18 is an interferon (IFN)-gamma-inducing factor and contributes to the Th1 immune respon
135 rial depolarization and release of apoptosis-inducing factor and cytochrome c Furthermore, this prote
136 in both cell types, but release of apoptosis-inducing factor and endonuclease G was detected only in
137 ed from the mitochondrion, such as apoptosis-inducing factor and endonuclease G, may induce caspase-9
139 ribofuranosylbenzimidazole (DRB) sensitivity-inducing factor and found that the IC(50) determined was
140 ented intranuclear localization of apoptosis-inducing factor and protected neurons from excitotoxic i
142 further examined the association of Th1/Th2 inducing factors and allergic disease and intestinal inf
143 under inflammatory conditions; however, the inducing factors and underlying mechanisms remain unknow
144 mitochondrial caspase-independent (apoptosis-inducing factor) and caspase-dependent (Smac/Diablo and
145 in (IL)-1beta, IL-18 (interferon (IFN)-gamma inducing factor) and IFN-gamma, but not tumour-necrosis
147 irectly the release of Cyt c, AIF (apoptosis-inducing factor), and Smac (second mitochondria-derived
148 elongation factor) and DSIF (DRB sensitivity-inducing factor)--and P-TEFb (positive elongation factor
149 ndria, release of cytochrome c and apoptosis-inducing factor, and activation of caspase-9 and caspase
150 translocation of cytochrome c and apoptosis inducing factor, and active caspases 3 and 7, consistent
153 osolic release of cytochrome c and apoptosis-inducing factor, and mitochondrial membrane potential ch
154 t stress, mitochondrial release of apoptosis inducing factor, and nuclear DNA fragmentation resulting
155 respiration, prevented release of apoptosis-inducing factor, and reduced neuronal cell death trigger
156 e leakage of both cytochrome c and apoptosis-inducing factor, and significantly improved cell surviva
157 ded MHC class II molecules, interferon-gamma inducing factor, apolipoprotein E, and cathepsin S.
160 and the pausing factor DSIF (DRB sensitivity-inducing factor) are still present at the hsp70 loci in
161 CCK-induced caspase 3 activation, apoptosis-inducing factor, as well as X-linked inhibitor of apopto
162 d that in this nondividing oocyte, the nerve-inducing factor Ascl1 can remain bound to a specific chr
163 one copy of the binding site for the neural-inducing factor Ascl1 is injected directly into a Xenopu
166 otic cells and expression of total apoptosis-inducing factor, Bcl-2, Bak, and Bax in the pre-CP/Rep a
169 varian cancer, we show that "helper" NK cell-inducing factors can be used to enhance local production
170 d key apoptotic mediators, such as apoptosis-inducing factor, caspase 3, caspase 8, caspase 9, poly(a
171 Bcl-2 and increased expression of apoptosis-inducing factor, caspase-3, and cleavage of BID, c-IAP-1
172 mediated by the enhanced induction of anergy-inducing factors cbl-b, c-cbl (cbl is abbreviation for C
173 FLP represses the expression of the mitosis-inducing factor CDKB1;1, which, along with CDKB1;2, is s
174 ial meningitis and induces a novel apoptosis-inducing factor-dependent (AIF-dependent) form of brain
175 ath proceeded predominately via an apoptosis-inducing factor-dependent pathway in XY neurons versus a
176 sion is induced by the stalk differentiation-inducing factor DIF-1 and is restricted to a subset of p
177 osed to the prestalk inducer differentiation inducing factor (DIF-1), a chlorinated hexaphenone.
179 eta-d-ribofuranosylbenzimidazole sensitivity-inducing factor (DSIF) and negative elongation factor (N
180 eta-D-ribofuranosylbenzimidazole sensitivity-inducing factor (DSIF) are involved in pausing RNA Polym
181 elongation factor (NELF) and DRB sensitivity inducing factor (DSIF) contribute in the establishment o
187 itochondria, and cytochrome c, and apoptosis-inducing factor escaped from mitochondria to the cytopla
188 or Toll-like receptor-associated interferon-inducing factor expression does not affect the LPS-trigg
189 rental cells, whereas knockdown of apoptosis-inducing factor expression suppressed lapatinib toxicity
192 brane permeability, and release of apoptosis-inducing factor from mitochondria are partially blocked
193 AD(+) blocked translocation of the apoptosis-inducing factor from mitochondria to nuclei, a step prev
194 phate levels, and translocation of apoptosis-inducing factor from mitochondria to the nucleus, result
196 of cytochrome c, Smac/DIABLO, and apoptosis inducing factor from mitochondria, and reduced mitochond
199 hondrial mu-calpain and release of apoptosis-inducing factor from the mitochondrial intermembrane spa
200 sary for the complete discharge of apoptosis-inducing factor from the mitochondrial intermembrane spa
202 sion of caspase 4, cathepsin B, or apoptosis-inducing factor function significantly suppressed cell k
203 ced immunotherapeutic efficacy; (c) the MDSC-inducing factors G-CSF and GM-CSF facilitated IRF-8 down
204 No new classes of extracellular mesoderm-inducing factors have been identified in more than two d
207 inase (PDK1) and EGFR along with the hypoxia-inducing factor (HIF)-1alpha in human glioblastoma multi
208 are also induced by the exudate (hormogonium-inducing factor [HIF]) of a symbiotic plant partner.
209 es of T. versicolor roots to these haustoria inducing factors (HIFs) included localized swelling and
211 The equine herpesvirus 1 (EHV-1) alpha-trans-inducing factor homologue (ETIF; VP16-E) is a 60-kDa vir
213 dependent nuclear translocation of apoptosis-inducing factor in NMDA-treated neurons and reduced tPA-
215 is the major cyclooxygenase-2 mRNA and PGE2-inducing factor in pulmonary edema fluid and accounts fo
216 failed to release cytochrome c or apoptosis-inducing factor in response to recombinant Bax or trunca
217 t an additional function of EHD1 as a tubule-inducing factor in the Arf6 pathway for recycling of pla
218 ble protein 3alpha was the most active mucin-inducing factor in the RSV-infected human small airway e
219 One candidate for an endogenous mesoderm-inducing factor in Xenopus is derriere, a member of the
220 growth by down-regulating other angiogenesis-inducing factors in addition to VEGF and that the centra
222 management and enable identification of NET-inducing factors in individual patients for targeted tre
224 Exposure of hAd-PSCs to differentiation-inducing factors in vitro upregulated steroidogenic gene
225 ce and reveal a role for Id4 as a quiescence-inducing factor, in contrast with its role of promoting
227 1) peptide has been reported as an autophagy-inducing factor inhibiting the replication of pathogens
229 transfer experiments reveal the PS30-derived inducing factor is soluble and promotes mitogenic ERK an
230 and Toll-like receptor-associated interferon-inducing factor, is blocked by both FADD and phosphatidy
232 ormation on whether Smac/Diablo or apoptosis-inducing factor might play a role in chronic neurodegene
233 We found that the flavoprotein apoptosis-inducing factor mitochondria-associated 2 (AIFM2) is a p
234 tein 1 (FSP1) (previously known as apoptosis-inducing factor mitochondrial 2 (AIFM2)) as a potent fer
235 ndrial release of cytochrome c and apoptosis-inducing factor, mitochondrial membrane depolarization,
236 ) for the phosphorylation of DRB sensitivity-inducing factor, negative elongation factor, and C-termi
237 Accordingly, ablation of the primary pause-inducing factor NELF does not increase expression of lin
238 ss of pausing through knockdown of the pause-inducing factor NELF leads to broadly attenuated immune
239 e earliest identified BMP antagonists/neural-inducing factors, noggin and chordin, were expressed in
242 and efflux rate modulation as the strongest inducing factor of chronic inflammation for a wide range
243 e the activity of Nodal, a secreted mesoderm-inducing factor of the transforming growth factor-beta (
245 be maintained by continued expression of the inducing factor or by a mechanism that confers a stable
247 induction of autophagy, release of apoptosis-inducing factor, or opening of the mitochondrial permeab
248 the elongation factors DSIF (DRB Sensitivity-Inducing Factor), P-TEFb (Positive Transcription Elongat
249 ng RNA to reduce the expression of apoptosis-inducing factor partially inhibited CDDO-induced apoptos
251 f biphenyl dioxygenase (BphA4) and apoptosis-inducing factor, Pdr lacks one of the arginine residues
252 ep, the amount of apoptotic cells, apoptosis-inducing factor, phospho-Bad, phospho-PKC-alpha, phospho
253 ukin (IL) -1beta, TNF-alpha, and proteolysis-inducing factor (PIF) by cancer cells used in this study
256 ollagen XVIII (NC1) functioned as a motility-inducing factor regulating the extracellular matrix-depe
257 y transition, and cytochrome C and apoptosis-inducing factor release from isolated mitochondria.
258 x translocation, cytochrome c, and apoptosis-inducing factor release) and apoptosis by imatinib mesyl
259 tivation) and caspase-independent (apoptosis-inducing factor release) pathways, and limited neuronal
260 g., cytochrome c, Smac/DIABLO, and apoptosis-inducing factor release), caspase activation, and apopto
261 P binding protein with low pI, and apoptosis-inducing factor release), caspase activation, poly(ADP-r
262 rial dysfunction (cytochrome c and apoptosis-inducing factor release), caspase-3 and -8 activation, a
263 rial dysfunction (cytochrome c and apoptosis-inducing factor release), caspase-3 and -8 activation, a
264 ificantly reduced cytochrome c and apoptosis-inducing factor release, loss of mitochondrial membrane
265 ed by the mitochondrial release of apoptosis-inducing factor, resulting in caspase-independent cell d
267 in some chicken embryos and named resistance-inducing factor (RIF) because it interferes with RSV.
269 ergizes with activating sulfonolipid rosette-inducing factors (RIFs) to recapitulate the full bioacti
271 These exosomes increased chemoresistance-inducing factor, Snail, in recipient epithelial cells an
272 cells were hypersensitive to various stress-inducing factors, such as salts, SDS, and H(2)O(2), espe
276 melanocortin peptide agonists act as satiety-inducing factors that mediate their action through the m
277 ial-derived signal in the intestinal stroma, inducing factors that restrict epithelial proliferation
279 ly delivering a specific combination of Treg inducing factors through degradable polymer microspheres
280 ces BMP4, a well-established HFSC quiescence-inducing factor, thus contributing to a proliferation-in
282 lease and nuclear translocation of apoptosis-inducing factor to initiate chromatinolysis and cell dea
283 s activation, the translocation of apoptosis-inducing factor to the nucleus, and DNA fragmentation in
285 ted, at least in part, by specific fugetaxis-inducing factors to which only mature cells respond.
287 lycolysis in response to the differentiation-inducing factor transforming growth factor beta1 (TGF-be
288 ough mitochondrial dysfunction and apoptosis-inducing factor translocation from the mitochondria to t
289 induced mitochondrial dysfunction, apoptosis-inducing factor translocation, and subsequent cell death
290 yD88 adaptor-like protein, Toll receptor IFN-inducing factor (Trif), and Trif-related adaptor molecul
293 eta-synuclein can act as a neurodegeneration-inducing factor, we demonstrated that wild-type beta-syn
294 d release from mitochondria of the apoptosis-inducing factor were selectively abrogated in iNOS(-/-)
296 polarization and relocalization of apoptosis-inducing factor, whereas the BRAF-V600E-mutated melanoma
298 ation of caspases-1, -3, and -8 or apoptosis-inducing factor within MNs, with a blockade of apoptosis
300 ndothelial cells by BADrUL131 and the fusion-inducing factor X clinical human cytomegalovirus isolate