ライフサイエンスコーパス: infant death

1 ate neonatal death, and 50.2 (42.8-59.0) for infant death.

2 th, stillbirth, and neonatal, perinatal, and infant death.

3 gly associated with the risk of neonatal and infant death.

4 a new, potentially treatable cause of sudden infant death.

5 have serious consequences, including sudden infant death.

6 s a significant risk factor for neonatal and infant death.

7 e to thrive, behavioral deficits, and sudden infant death.

8 all live births and are the leading cause of infant death.

9 ence of the effects of MMN on fetal loss and infant death.

10 ge infant (SGA), birth defect, and perinatal infant death.

11 bortion, PTB, macrosomia, SGA, and perinatal infant death.

12 n 50 and is the most common genetic cause of infant death.

13 ociated with a 1.2% reduction in the risk of infant death.

14 trophy (SMA) is the leading genetic cause of infant death.

15 ences in newborn babies and result in sudden infant death.

16 iated with an increased risk of neonatal and infant death.

17 e the burden of fetal death, stillbirth, and infant death.

18 th, stillbirth, and neonatal, perinatal, and infant death.

19 revent the majority of needless maternal and infant deaths.

20 fetal deaths exceeded that of crash-related infant deaths.

21 d deaths and 35.3% (95% CI, 27.8%-42.0%) for infant deaths.

22 nt 107000 (UR, 20000-198000) stillbirths and infant deaths.

23 54% of estimated cases and 65% of all fetal/infant deaths.

24 (1.06; 1.02-1.10; P = 0.007), and perinatal infant death (1.08; 1.03-1.12; P < 0.001); the correspon

25 7-1.23; I2 = 78.5%; n = 12 studies); and for infant death, 1.18 (95% CI, 1.09-1.28; I2 = 79%; n = 4 s

26 y (California) and included 546 infants (112 infant deaths [20.5%] and 434 infants [79.5%] with acute

27 live births were male, and there were 36,485 infant deaths (25,110 neonatal deaths and 11,375 postneo

28 een collected for 369 consecutive unexpected infant deaths (300 SIDS and 69 explained deaths) in Avon

29 sociated with 9208 (95% CI, 8601-9814) fewer infant deaths; 3195 (95% CI, 3017-3372) infant deaths co

30 CI, 19-23), and 24 (95% CI, 22-27); and for infant death, 33, 37 (95% CI, 34-39), and 43 (95% CI, 40

31 e 34 weeks (6.0% vs. 2.6%, P=0.04) and early infant death (4.4% vs. 0.6%, P=0.001), but there were no

32 Among 112 infant deaths (54 girls [48.2%]; 8 [7.1%] African Americ

33 The only high sensitivity was for past infant death (85.4%).

34 ity (0.86 [0.73-1.00], p=0.048), fetal loss (infant death after 16 weeks' gestation and before 7 days

35 The sudden infant death/ALTE families had a greater frequency of tw

36 ntrol) included members with OSA, and all 10 infant death/ALTE families were among these (versus zero

37 Thus, OSA in adults and sudden unexpected infant death/ALTE in their biologic relatives appear to

38 Information on sudden unexpected infant death/ALTE was obtained by questionnaire and was

39 rom families with OSA plus sudden unexpected infant death/ALTE.

40 milies had 10 infants with sudden unexpected infant death/ALTE; two control families had three infant

41 Congenital anomalies are a leading cause of infant death and disability and their incidence varies b

42 Preterm birth (PTB) is the leading cause of infant death and disability worldwide.

43 ontroversies in the evaluation of unexpected infant death and inflicted traumatic brain injury are on

44 had an unexpected and apparently unexplained infant death and is currently available in over 90% of h

45 ve head trauma (AHT) is the leading cause of infant death and long-term morbidity from injury.

46 y associated with the risk of stillbirth and infant death and neonatal morbidity.

47 hyxia at term remains a significant cause of infant death and neurodevelopmental impairment, probably

48 m birth is the leading cause of neonatal and infant death and of disability among survivors.

49 e past, these vaccines were thought to cause infant deaths and brain damage.

50 Data on residual infant deaths and maternal seroprevalence would be valua

51 in the 40 years since 1970, particularly for infant deaths and mortality from non-communicable diseas

52 There were no maternal or infant deaths and no significant between-group differenc

53 fe and has been implicated in ~10% of sudden infant deaths and unexplained stillbirths.

54 11 mo, lost future earnings due to premature infant death, and the costs of purchasing infant formula

55 ity rates for SIDS, other sudden, unexpected infant deaths, and cause unknown/unspecified, and they e

56 uted to averting approximately 60,000-70,000 infant deaths annually.

57 nd 147000 (UR, 47000-273000) stillbirths and infant deaths annually.

58 tudied the relationship of sudden unexpected infant death/apparent life-threatening events (ALTE) to

59 nization was at least 1 million to prevent 1 infant death, approximately 100 000 for ICU admission, a

60 Rates of infant death are highest in children of Pakistani origin

61 s that underlie socioeconomic differences in infant deaths are essential for creating and implementin

62 Repeat unexpected infant deaths are most probably natural.

63 gnancy is a risk factor for sudden fetal and infant death as well as obstructive airway disease in ch

64 to identify those at risk of stillbirth and infant death at term.

65 The number of infant deaths at age 0-6 months was similar in each grou

66 rly 10 000 laboratory-confirmed cases and 14 infant deaths attributed to pertussis.

67 Numbers of early infant deaths (between 24 hours and 6 months of age) cod

68 Low birth weight (LBW) increases the risk of infant death, but little is known about its causes among

69 SV is the second leading infectious cause of infant death, but no vaccine is available.

70 y outcome was the composite of stillbirth or infant death by 1 year of corrected age or moderate or s

71 R) estimates for fetal death, stillbirth, or infant death by at least 3 categories of maternal BMI we

72 Recurrent late-term fetal loss or sudden infant death can result from unsuspected parental mosaic

73 Birth certificate data linked to infant death certificates and to infant discharge abstra

74 ding interhospital transfers), and fetal and infant death certificates to assess neonatal mortality r

75 Among the categories of causes of infant death, congenital anomalies (APC = -7.87%), asphy

76 ewer infant deaths; 3195 (95% CI, 3017-3372) infant deaths could have been avoided had there been no

77 We used national birth cohort linked birth-infant death data (2000-2010) to evaluate the risk of in

78 r for Health Statistics' period linked birth/infant death data set files for 2007-2013 for 26546503 U

79 l and Prevention (CDC)'s Cohort Linked Birth/Infant Death Data Set for births between 2005 and 2010 w

80 or Health Statistics (NCHS) linked birth and infant death data, and Pregnancy Risk Assessment Monitor

81 National Center for Health Statistics birth infant/death data set for 12 256 303 preterm infant birt

82 Infant death due to NEC in preterm babies was identified

83 mass burning in Africa, nearly 75% of global infant deaths due to burning still occur in Africa.

84 Infant deaths due to external causes were most likely to

85 Homicide is the leading cause of infant deaths due to injury.

86 in the United States, equal to the number of infant deaths each year.

87 rd (1980-98) and the Scottish Stillbirth and Infant Death Enquiry (1985-98).

88 d its determinants by using the US birth and infant death files for 1989-1997.

89 Data were derived from linked US birth/infant death files for 1995 and 1996, comprising 7,465,8

90 ed by lack of T and natural killer cells and infant death from infection.

91 ublic between 1986 and 1993, including 3,254 infant deaths from 350,978 first births to married and s

92 Data included all live births and infant deaths from all 50 US states and the District of

93 and PM10 were associated with an increase in infant deaths from congenital malformations of the nervo

94 Early infant deaths from critical congenital heart disease thr

95 Early infant deaths from other/unspecified cardiac causes decl

96 rast, SO2 was associated with an increase in infant deaths from perinatal causes (1.214 [1.156, 1.275

97 d a lethal phenotype characterized by sudden infant death (from cardiac and respiratory arrest) with

98 nown to reduce the risk of sudden unexpected infant death has contributed to a slowing in the decline

99 ses the risk of fetal death, stillbirth, and infant death; however, the optimal body mass index (BMI)

100 Secretory diarrhea is the leading cause of infant death in developing countries and a major cause o

101 gyo virus had a live birth with maternal and infant death in Isiro, the Democratic Republic of the Co

102 th MMN, compared with IFA, on fetal loss and infant death in the setting of routine prenatal care ser

103 s to be the most common cause of unexplained infant death in Western countries.

104 392 neonatal deaths in 18 countries and 8932 infant deaths in 15 countries were avoided over 3 years

105 infant mortality, accounting for 35% of all infant deaths in 2008.

106 natal mortality, and with specific causes of infant deaths in 7,984,366 live births between 2001 and

107 (PFOA) exposure can cause pregnancy loss and infant deaths in animals, but the associations between P

108 er 5 years worldwide, and constituted 65% of infant deaths in India.

109 lation and survival estimates, the number of infant deaths in low-income and middle-income countries

110 The five infant deaths in the group of potential survivors result

111 tation) are approximately equal in number to infant deaths in the United States and are twice as like

112 From 2012 to 2016, infant deaths increased by 63% and maternal mortality mo

113 e on Sudden Infant Death, Sudden Unexplained Infant Death, infant and child death due to maltreatment

114 ical effects on the parents following sudden infant death is discussed and reveals maternal anxiety a

115 nvestigations in cases of sudden unexplained infant death is emphasized.

116 ed to estimate the probability that a second infant death is natural versus unnatural.

117 ostneonatal deaths (first 28 days), and five infant deaths (<1 year).

118 After an event of sudden infant death, many parents moved without a forwarding ad

119 imilarly associated with endocrine causes of infant deaths (NO2, 2.167 [1.539, 3.052; p < 0.001]; PM1

120 Because most infant deaths occur in the first few month of life, mate

121 hat when two or three unexpected unexplained infant deaths occur within a family they are more likely

122 Fetal or neonatal/infant death occurred in 13.3% of those randomized to re

123 Overall, for every 1,000 births, 3.36 more infant deaths occurred among non-Hispanic black women re

124 ted the relative risks (RRs) of neonatal and infant death of neonates with low (0-3) and intermediate

125 The primary outcomes were infant death or CP and severe maternal outcome potential

126 ts CP and reduces the combined risk of fetal/infant death or CP.

127 dicate that whole cell vaccines do not cause infant deaths or neurologic disease.

128 the risk of fetal death (P = 0.99) or early infant death (P = 0.19).

129 bortion, PTB, macrosomia, SGA, and perinatal infant death (P for trend <0.001, <0.001, <0.001, 0.001,

130 5 (95% confidence interval: 2.20, 2.71) more infant deaths per 1,000 births among non-Hispanic black

131 paid maternity was associated with 7.9 fewer infant deaths per 1,000 live births (95% CI 3.7, 12.0),

132 tudied, SIDS rates ranged from a high of 3.0 infant deaths per 1,000 live births for American Indians

133 id sanctions resulted in an additional 129.3 infant deaths per 100 000 livebirths (11.7 to 246.9), an

134 ported cases of disease and from 933 to 5796 infant deaths per year for the diseases under study are

135 as associated with nearly 130,000 additional infant deaths per year globally over our 2004 to 2018 st

136 en food is shared only within kin groups, an infant death permits reallocation of its unneeded food t

137 Additionally, among selective causes of infant death, pneumonia, congenital heart disease, neura

138 tality among American Indians, a group whose infant death rate is consistently above the US national

139 screening policies has been associated with infant death rates is unknown.

140 sible or if it reduces sudden and unexpected infant death rates remains to be studied.

141 es for critical congenital heart disease and infant death rates.

142 obtained from the 2000-2002 US Linked Birth/Infant Death records and included 677,777 black infants

143 in infant mortality when national birth and infant death records began to be linked.

144 identified from the US Linked Livebirth and Infant Death records between 2000 and 2004.

145 emiologic Research expanded linked birth and infant death records database from 2017 to 2020.

146 onal Center for Health Statistics' birth and infant death records for all twin births occurring in th

147 oss-sectional study used cohort-linked birth/infant death records for live births to US residents fro

148 ort study examined maternal-linked birth and infant death records from the National Center for Health

149 Therefore, it seems likely that infant deaths result from sepsis secondary to intestinal

150 Sixty percent of infant deaths resulted from delay in recognition of feta

151 A), one of the most common genetic causes of infant death, results from the selective loss of motor n

152 iking changes in rates of sudden unexplained infant death (SIDS) around 1990, four large case-control

153 ces the resources recaptured by kin after an infant death, so evolved infant mortality is lower.

154 The primary outcomes were infant death, stillbirth, overall mortality (infant and

155 own/unspecified and other sudden, unexpected infant deaths, such as accidental suffocation and strang

156 e most recent published literature on Sudden Infant Death, Sudden Unexplained Infant Death, infant an

157 to-month variation in both sudden unexpected infant death (SUID) and SIDS rates before and during the

158 ased cohort of 221 anonymous cases of sudden infant death syndrome (84 females, 137 males; average ag

159 certificates/autopsy reports included sudden infant death syndrome (n = 544 [44%]), asphyxia (n = 74

160 th of an infant that had succumbed to sudden infant death syndrome (SIDS) (and no other cause of deat

161 The association between sudden infant death syndrome (SIDS) and maternal smoking was co

162 lities in HMs have been implicated in sudden infant death syndrome (SIDS) and obstructive sleep apnoe

163 moking is the highest risk factor for sudden infant death syndrome (SIDS) and prenatal nicotine expos

164 is growth in our understanding of how sudden infant death syndrome (SIDS) and the symptom complex see

165 Many sudden infant death syndrome (SIDS) cases exhibit a partial ( a

166 An estimated 10% to 15% of sudden infant death syndrome (SIDS) cases may stem from channel

167 Sudden infant death syndrome (SIDS) cases often have abnormalit

168 evalence during the past two decades, sudden infant death syndrome (SIDS) continues to be the leading

169 uction campaigns have been conducted, sudden infant death syndrome (SIDS) has become increasingly con

170 ears suggest that the epidemiology of sudden infant death syndrome (SIDS) has changed since the 1991

171 infant sleeping position and risk of sudden infant death syndrome (SIDS) in an ethnically diverse US

172 Sudden infant death syndrome (SIDS) is a leading cause of postn

173 The likelihood of recurrence of sudden infant death syndrome (SIDS) is an issue of biological,

174 Sudden infant death syndrome (SIDS) is postulated to result fro

175 The sudden infant death syndrome (SIDS) is the sudden death of an i

176 The recent US decline in sudden infant death syndrome (SIDS) rates may be explained by a

177 d here support a recent proposal that sudden infant death syndrome (SIDS) results from a developmenta

178 Unexplained stillbirth and the sudden infant death syndrome (SIDS) share some features.

179 o examine recent research relevant to sudden infant death syndrome (SIDS) to determine whether there

180 in the medullae of infants dying from sudden infant death syndrome (SIDS) were identified, suggesting

181 ts under 1year of age are at risk for sudden infant death syndrome (SIDS), and patients with epilepsy

182 associated with an increased risk of sudden infant death syndrome (SIDS), but few studies have asses

183 used in infants at increased risk for sudden infant death syndrome (SIDS), but the efficacy of such d

184 to understanding the aetiology of the sudden infant death syndrome (SIDS), in which there is medullar

185 This has special implications for sudden infant death syndrome (SIDS), insofar as seemingly norma

186 us state in two infants who died from sudden infant death syndrome (SIDS), one with documented prolon

187 edisposes an infant to a high risk of sudden infant death syndrome (SIDS), the authors conducted a po

188 Sudden infant death syndrome (SIDS), the leading cause of postn

189 mortem infection is a risk factor for sudden infant death syndrome (SIDS)-the leading postneonatal ca

190 associated with an increased risk of sudden infant death syndrome (SIDS).

191 Thousands die each year from sudden infant death syndrome (SIDS).

192 is (PGCL), in many infants who die of sudden infant death syndrome (SIDS).

193 sure are the highest risk factors for sudden infant death syndrome (SIDS).

194 ly associated with cardiomyopathy and sudden infant death syndrome (SIDS).

195 may be responsible for some cases of sudden infant death syndrome (SIDS).

196 e sleep position (on the stomach) and sudden infant death syndrome (SIDS).

197 ic exposure (PNE), is responsible for sudden infant death syndrome (SIDS).

198 necrotizing enterocolitis (NEC), and sudden infant death syndrome (SIDS).

199 ties are present in many cases of the sudden infant death syndrome (SIDS).Mice with a targeted disrup

200 ll individuals were identified in 599 sudden infant death syndrome and 258 sudden unexplained death i

201 Exome sequencing was performed on 599 sudden infant death syndrome and 258 sudden unexplained death i

202 for disorders such as sleep apnea or sudden infant death syndrome and for regulating uterine contrac

203 is often a fine line differentiating sudden infant death syndrome and infanticide.

204 erized SCN5A variants associated with sudden infant death syndrome and provide further biophysical co

205 tionately affect males, including the sudden infant death syndrome and sleep apnea.

206 phenotype, the prevalence of TKOS in sudden infant death syndrome and sudden unexplained death in th

207 mend against it to reduce the risk of sudden infant death syndrome and suffocation deaths.

208 genital abnormalities, accidents, and sudden infant death syndrome are predicted to continue increasi

209 We classified sudden infant death syndrome as a separate cause of death consi

210 12 months or younger; most (52%) had sudden infant death syndrome as the reported cause of death.

211 evaluation and prevention of possible Sudden Infant Death Syndrome cases were published this year, wi

212 and an apparent relationship to some sudden infant death syndrome cases.

213 rowning, respiratory arrest, and near sudden infant death syndrome cause significant mortality and mo

214 riants were identified in a Norwegian sudden infant death syndrome cohort (n=201).

215 Sudden infant death syndrome contributed to 5% of deaths in chi

216 Pediatrics in 1992, the incidence of Sudden infant death syndrome has decreased by almost 50%.

217 in a large population-based cohort of sudden infant death syndrome has elucidated mutations in 5-10%

218 t with the observed 50% male bias for Sudden Infant Death Syndrome in humans.

219 here has been a dramatic reduction in sudden infant death syndrome in this country.

220 es were used to locate 230 parents of sudden infant death syndrome infants who died in Southern Calif

221 andardized and systematic approach to sudden infant death syndrome is also reviewed.

222 ns: While its exact cause is unknown, sudden infant death syndrome is believed to be multifactorial,

223 The current literature on sudden infant death syndrome is reviewed in this section by Ste

224 strongly supports the hypothesis that sudden infant death syndrome is the result of dysregulation of

225 rd ratios for total, respiratory, and sudden infant death syndrome mortality per-interquartile-range

226 covariates to total, respiratory, and sudden infant death syndrome mortality.

227 not contribute meaningfully to either sudden infant death syndrome or sudden unexplained death in the

228 iation between infant bed sharing and sudden infant death syndrome or unintentional sleep-related dea

229 Controversy regarding the etiology of sudden infant death syndrome persists, and risk factors are rev

230 of Pediatrics new recommendations on sudden infant death syndrome prevention.

231 al "Back to Sleep" campaign to reduce sudden infant death syndrome provides an opportunity to study w

232 Given the risk of sudden infant death syndrome related to bedsharing, multipronge

233 Sudden infant death syndrome remains the leading cause of death

234 Despite a putative diagnostic shift, sudden infant death syndrome remains the most common cause of d

235 apparent life-threatening events and sudden infant death syndrome remains to be explored further, bu

236 Sudden infant death syndrome reports after IPV were consistent

237 athogenic cause for a small subset of sudden infant death syndrome via a secondary loss-of-function m

238 between meteorologic temperature and sudden infant death syndrome was investigated in the 1982-1983

239 Apgar score at 5 min and the risk of sudden infant death syndrome was noted at any gestational age (

240 dratase subunit A (HADHA), results in sudden infant death syndrome with no cure. To reveal the diseas

241 identified 18 families with two SIDS(sudden infant death syndrome) deaths and two families with prob

242 ion continues to be a risk factor for sudden infant death syndrome, although immunizations may not be

243 ty, apparent life-threatening events, sudden infant death syndrome, and central hypoventilation are r

244 sudden unexpected death in epilepsy, sudden infant death syndrome, and sleep apnea.

245 e sudden unexplained death, including sudden infant death syndrome, can be caused by cardiac channelo

246 ent in a variety of disorders such as sudden infant death syndrome, depression, and anxiety.

247 hy, and now encompasses more cases of sudden infant death syndrome, fulminant hepatic failure, and se

248 thought to underlie the aetiology of sudden infant death syndrome, including: (1) a vulnerable neona

249 ventable-cause mortality in children (sudden infant death syndrome, unintentional injury, and homicid

250 k factor for late fetal death and the sudden infant death syndrome, we investigated cardiorespiratory

251 rovide insight into the mechanisms of sudden infant death syndrome, which has been associated with ab

252 as of prematurity, Rett syndrome, and sudden infant death syndrome.

253 recently long-QT syndrome (LQTS) and sudden infant death syndrome.

254 ciated with maternal smoking, such as sudden infant death syndrome.

255 phenotype in mice, with relevance to Sudden Infant Death Syndrome.

256 ve AR capacity has been implicated in Sudden Infant Death Syndrome.

257 nifestations include fetal hydrops or sudden infant death syndrome.

258 have been identified in some cases of sudden infant death syndrome.

259 ome cases of sudden unexplained death/sudden infant death syndrome.

260 me, familial atrial fibrillation, and sudden infant death syndrome.

261 pathophysiology and risk factors for sudden infant death syndrome.

262 s one of the biggest risk factors for sudden infant death syndrome.

263 rs that may contribute to the risk of Sudden Infant Death Syndrome.

264 c 5-HT brainstem disorders, including sudden infant death syndrome.

265 KCNH2 gain of function mutations and sudden infant death syndrome.

266 associated with an increased risk of sudden infant death syndrome.

267 sturbances culminating in events like Sudden Infant Death Syndrome.

268 mptoms in children and be a factor in sudden infant death syndrome.

269 veness of these campaigns in reducing sudden infant death syndrome.

270 ged QT electrocardiogram interval and sudden infant death syndrome.

271 reatest influence on the reduction of sudden infant death syndrome.

272 find other modifiable risk factors of sudden infant death syndrome.

273 the relationship between smoking and sudden infant death syndrome.

274 , contributing to the pathogenesis of sudden infant death syndrome.

275 ged QT electrocardiogram interval and sudden infant death syndrome.

276 rt rates and cardiac function such as sudden infant death syndrome.

277 nts who are labeled as having died of sudden infant death syndrome.

278 associated with an increased risk of sudden infant death syndrome.

279 normalities thought to be involved in sudden infant death syndrome.

280 sto-pathological findings reported in sudden infant death syndrome.

281 ardiomyopathy or sudden cardiac death/sudden infant death syndrome/idiopathic ventricular fibrillatio

282 accine would reduce the maternal anaemia and infant deaths that are associated with malaria in pregna

283 entation, we estimated the proportion of all infant deaths that were due to RSV.

284 the physiologic mechanisms that cause sudden infant death, the mainstay of risk reduction continues t

285 enetic diseases are a leading cause of early infant death, to our knowledge, the contribution of sing

286 Vaccine effectiveness against infant deaths was estimated at 95% (95% confidence inter

287 d district-level data covering 98% of global infant deaths, we find that exposure to outdoor biomass

288 and social risk factors, and cause-specific infant death were ascertained via linkage between nation

289 Adjusted hazard ratios for infant deaths were 145 (95% CI 141, 149) and 9.8 (95% CI

290 ted an excess of 152,753 (64,120 to 241,386) infant deaths were attributable to living in flood-prone

291 An estimated 15.5 million infant deaths were averted between 2000 and 2019 because

292 tal deaths, 11,294 neonatal deaths, and 4983 infant deaths were included.

293 m 1997 to 2014, 970,583 live births and 6510 infant deaths were registered.

294 Adjusted odds ratios (95% CI; p-value) for infant deaths were significantly increased for NO2, PM10

295 data, developmental outcomes, including four infant deaths, were documented for 888 of (88%) 1010 sin

296 e identified two cases of sudden unexplained infant death where no lung LCAD antigen was detectable.

297 ty loci for a rare monogenic disease (sudden infant death with dysgenesis of the testes syndrome), a

298 ght (m)(2)) of women who had stillbirths and infant deaths with those of their sisters or of populati

299 The possibility of a second sudden infant death within a family is discussed.

300 atrophy (SMA) is a major inherited cause of infant death worldwide.