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1 t occurs in Brazil (where VL is caused by L. infantum).
2  and moderately effective against Leishmania infantum.
3  LdCen(-/-)group following challenge with L. infantum.
4 be considered as secondary reservoirs for L. infantum.
5 ntified in a phenotypic screen of Leishmania infantum.
6  tropica, and 10 have been diagnosed with L. infantum.
7 pica infection, and 16 were infected with L. infantum.
8  against T. brucei and amastigote Leishmania infantum.
9 es in the host immune response to Leishmania infantum.
10 immunity mediated by prior infection with L. infantum.
11 ipalpis for Leishmania species other than L. infantum.
12 n against visceral infection with Leishmania infantum.
13 irst linked to the clinical syndrome roseola infantum.
14 tment murine model for acute infection by L. infantum.
15 parasites Leishmania donovani and Leishmania infantum.
16  14alpha-demethylase (CYP51) from Leishmania infantum.
17  5 showed potent in vivo activity against L. infantum.
18 eral leishmaniasis (VL) caused by Leishmania infantum.
19 tabase of the parasitic protozoan Leishmania infantum.
20  murine infection model employing Leishmania infantum.
21 positive patients coinfected with Leishmania infantum.
22  (IC(50) between 4 and 36 nM) and Leishmania infantum (1.27 and 1.11 uM), placing it as a unique comp
23 pesvirus 6 is the causative agent of roseola infantum, a generally benign rash illness of infants.
24 iasis is caused by infection with Leishmania infantum, a Protist parasite transmitted by blood-feedin
25                                           L. infantum amastigotes were detected in BM direct smear af
26 omastigotes as well as L. amazonensis and L. infantum amastigotes.
27 ed tandem repeat (TR) proteins of Leishmania infantum and an evaluation of VL patient antibody respon
28                        Vector-transmitted L. infantum and L. donovani caused >/=5-fold increase in sp
29                   These data suggest that L. infantum and L. major differentially activate keratinocy
30 mes of two species of Leishmania: Leishmania infantum and Leishmania braziliensis.
31 l disease caused by the parasites Leishmania infantum and Leishmania donovani The gold standard diagn
32 ies of Leishmania, L. tarentolae, Leishmania infantum, and L. major, produced hypersensitivity to bot
33 s, including Leishmania donovani, Leishmania infantum, and Leishmania braziliensis, a capacity to gen
34 icity against Trypanosoma brucei, Leishmania Infantum, and Trypanosoma cruzi.
35 ful development of an SPR sensor for anti-L. infantum antibodies detection in short time, showing a g
36 f anti-Trypanosoma cruzi and anti-Leishmania infantum antibodies in human and dog serum samples.
37 ensor shows good specificity against anti-L. infantum antibodies.
38 or chip for the detection of anti-Leishmania infantum antibodies.
39 or W2 were obtained for T. cruzi (W1) and L. infantum antigen (W2) samples in three different electro
40                           We used Leishmania infantum as an in vitro model of intracellular infection
41 gly indicate that hamsters infected with Le. infantum become significantly more attractive to a great
42 rimary reservoirs of Leishmania infantum (L. infantum), but Leishmania tropica (L. tropica) infection
43 hmania donovani complex - L. donovani and L. infantum - cause the fatal disease visceral leishmaniasi
44                              Leishmania (L.) infantum causes visceral, cutaneous, and mucosal leishma
45 ntly, this analysis suggests that Leishmania infantum chagasi alters the expression profile of certai
46        The vector-borne protozoan Leishmania infantum chagasi causes minimal inflammation after inocu
47 si-containing dermal leukocytes and total L. infantum chagasi parasites in draining lymph nodes were
48 noculation site, we introduced metacyclic L. infantum chagasi promastigotes intradermally into BALB/c
49                        Metacyclic Leishmania infantum chagasi promastigotes were treated with methyl-
50 n abundance during development of Leishmania infantum chagasi to a virulent metacyclic stage, as did
51 e dermis even late after inoculation, and L. infantum chagasi trafficked through neutrophils in both
52  role of HO-1 in the infection by Leishmania infantum chagasi, the causative agent of VL cases in Bra
53                 From 3 days onward, total L. infantum chagasi-containing dermal leukocytes and total
54             Nonetheless, a second wave of L. infantum chagasi-containing neutrophils occurred 7 days
55 caused by Leishmania donovani and Leishmania infantum chagasi.
56  tracked with fluorescent mCherry-labeled L. infantum chagasi.
57 th in the presence and absence of Leishmania infantum chagasi.
58 lpis sand flies, known vectors of Leishmania infantum/chagasi parasites, in a Brazilian city endemic
59 n (CA), derived from an Iranian strain of L. infantum, compared to direct agglutination test (DAT) fo
60 oplasma gondii, Neospora caninum, Leishmania infantum, Cryptosporidium parvum, or canine DNA under an
61                                     Thus, L. infantum CYP51 is the first example of a plant-like ster
62                                  Although L. infantum CYP51 prefers C4-monomethylated sterol substrat
63 ination experiments employing the Leishmania infantum D-13 (p80) antigen, significantly higher levels
64 L. major and a visceral strain of Leishmania infantum, each bearing a different drug-resistant marker
65 intracellular amastigotes of Leishmania (L.) infantum evaluated in vitro.
66 roM semipermeable membrane suggested that L. infantum-exposed keratinocytes release soluble factors t
67                           By screening an L. infantum expression library with sera from human VL pati
68 pffer cells (resKCs) drives early Leishmania infantum growth in the liver, leading to granuloma forma
69 2.5 mg/kg (b.i.d., orally) in the Leishmania infantum hamster model.
70  a protein of unknown function in Leishmania infantum (hypothetical C1 protein) and specific antibodi
71 g-lasting parasitemia and the presence of L. infantum in bone marrow, revealed that cats could be con
72 mmune system function and pathogenesis of L. infantum in cats.
73 ortance and the primary vector of Leishmania infantum in the Americas.
74 ethods for molecular detection of Leishmania infantum in the canine reservoir host.
75 apeutic strategies for control of Leishmania infantum in this important reservoir species.
76 (9) promastigotes of leishmania infantum (L. infantum) in the stationary phase intravenously and ster
77  agent of zoonotic leishmaniasis, Leishmania infantum, in France and Iberia, and provides a rare case
78 ne-resistant clinical isolates of Leishmania infantum, indicating its potential as antileishmanial le
79      Therefore, our results indicate that L. infantum induces IL-17A production, which promotes the c
80                                   Leishmania infantum-infected dogs are a naturally occurring model o
81 imental vector transmissions with Leishmania infantum-infected Lutzomyia longipalpis.
82                         The prevalence of L. infantum infection in the females fell from 85 to 45%.
83                        Control of Leishmania infantum infection is dependent upon Th1 CD4(+) T cells
84 SE induced significant protection against L. infantum infection, with reductions in parasite loads of
85 were not detected in the initial phase of L. infantum infection.
86 related to the protection against Leishmania infantum infection.
87                     Only vector-initiated L. infantum infections caused cutaneous lesions at transmis
88  more sensitive than an IFA for detecting L. infantum infections in patients with AIDS.
89 razilian State of Minas Gerais where L. (L.) infantum is also endemic.
90 sceral leishmaniasis (VL), due to Leishmania infantum, is a persistent intracellular parasitic infect
91 Visceral leishmaniasis, caused by Leishmania infantum, is a zoonosis, and culling seropositive dogs h
92 parasites Leishmania donovani and Leishmania infantum, is one of the major parasitic diseases worldwi
93 ge of clinically relevant L. donovani and L. infantum isolates.
94  10(8) and 10(9) promastigotes of leishmania infantum (L. infantum) in the stationary phase intraveno
95 ogs are the primary reservoirs of Leishmania infantum (L. infantum), but Leishmania tropica (L. tropi
96 es datasets from Leishmania braziliensis, L. infantum, L. major, L. tarentolae, Trypanosoma brucei an
97 15 were screened in vitro against Leishmania infantum , Leishmania braziliensis , Leishmania guyanens
98 ite species, exhibiting a silent (Leishmania infantum, Li) and a fully operational (Leishmania major,
99 -CoA synthetase (ACS) enzyme from Leishmania infantum (LiAcs1), which, unlike many organisms, also ex
100               Our work focuses on Leishmania infantum mitochondrial 2-Cys-Prx, whose reduced, decamer
101 e vector-borne protozoan parasite Leishmania infantum, mitochondrial peroxiredoxin (Prx) exerts intri
102  5), Leishmania mexicana (n = 1), Leishmania infantum (n = 3), Leishmania aethiopica (n = 4), Leishma
103 s sorted on contact with visceral Leishmania infantum on a susceptible mice model evaluating the subs
104 ave been recently developed using Leishmania infantum or Chikungunya virus (CHIKV).
105 39 in HIV-negative patients infected with L. infantum or L. chagasi declined during treatment with me
106 Lutzomyia longipalpis to transmit Leishmania infantum or Leishmania donovani to hamsters.
107 rtalized human keratinocytes with Leishmania infantum or Leishmania major, which cause visceral or cu
108 s are the major natural vector of Leishmania infantum parasites, responsible for transmission of visc
109 y confocal microscopy analysis applied to L. infantum promastigotes.
110 copy, we have previously identified three L. infantum protein biomarkers (Li-isd1, Li-txn1, and Li-nt
111 ts suggest that TR regions from the novel L. infantum proteins identified in this study are immunodom
112 enhance monocyte control of intracellular L. infantum replication (P < 0.01).
113              In contrast, the majority of L. infantum samples fell into one globally-distributed grou
114 n prepared from Iranian strain of Leishmania infantum showed high accuracy for the serodiagnosis of V
115                        MDMs infected with L. infantum showed significantly downregulated expression o
116              Keratinocytes incubated with L. infantum significantly increased expression of proinflam
117 rypanosoma brucei, Trypanosoma cruzi, and L. infantum) suggests that substrate preferences of plant-
118 ations of the medically important Leishmania infantum (syn.
119 ondrial chaperone reservoir, which allows L. infantum to deal successfully with protein unfolding con
120         A trend of improvement in Leishmania infantum TR inhibition was detected along the optimizati
121                     Inhibition of Leishmania infantum trypanothione disulfide reductase (LiTryR) by d
122 up to a dilution of 1:10,240 and for anti-L. infantum up to 1:5120 in canine serum samples.
123  if the odour of hamsters, infected with Le. infantum, was more attractive than the odour of the same
124 n L. mexicana, L. major, L. donovani, and L. infantum, we demonstrate how this tool can efficiently g
125 ruzi, Leishmania braziliensis and Leishmania infantum were not clinically relevant.
126                   The soluble antigens of L. infantum were securely immobilized on an SPR gold disk b
127 y gut populations of both L. mexicana and L. infantum were significantly reduced in caspar-depleted f
128  activity against the amastigote stage of L. infantum while no activity was observed against promasti
129                   Transfection of Leishmania infantum with LmACR2 augmented Pentostam sensitivity in

 
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