戻る
「早戻しボタン」を押すと検索画面に戻ります。 [閉じる]

コーパス検索結果 (１語後でソート)

通し番号をクリックするとPubMedの該当ページを表示します
1 ay cause bowel obstruction, chronic pain, or infertility.
2 n homeostasis were dysregulated in secondary infertility.
3 mation and scarring, ultimately resulting in infertility.
4 eatment option for women with uterine factor infertility.
5 HCT, which carries an extremely high risk of infertility.
6 n ZFP628-deficient mice that results in male infertility.
7 se of specific procedures, or the underlying infertility.
8  recurrent pregnancy loss, subfertility, and infertility.
9 el therapeutic opportunity for treating male infertility.
10 iosis can cause severe abdominal pain and/or infertility.
11 otropin-releasing hormone (GnRH) release and infertility.
12  unapproved use for ovulation induction with infertility.
13 n's upper genital tracts, resulting in tubal infertility.
14 CaMKII activation for clinical use to manage infertility.
15 inary birth defects and their impact on male infertility.
16 ad to various urogenital symptoms, including infertility.
17 ng genome evolution and Prdm9-related hybrid infertility.
18 st-meiotic defects, abnormal sperm, and male infertility.
19  with endometriosis are at increased risk of infertility.
20 s the most common identifiable cause of male infertility.
21 bolism, reduced interleukin-22 secretion and infertility.
22  worldwide and has been associated with male infertility.
23 e antibodies and a history of miscarriage or infertility.
24 of candidate genes associated with moss male infertility.
25 ounts for a significant percentage of female infertility.
26 atibility of recombination hotspots and male infertility.
27 ection is an important global cause of human infertility.
28  few survivors display accelerated aging and infertility.
29 sisted reproductive technology used to treat infertility.
30  ectopic lesions is associated with pain and infertility.
31 terine bleeding, pelvic pain or pressure, or infertility.
32  a leading cause of pregnancy loss and human infertility.
33  the human population and is associated with infertility.
34 ude fallopian tube fibrosis and tubal factor infertility.
35 rs of endometrial receptivity, fertility and infertility.
36 d apply to treatment of disease such as male infertility.
37 ovarian reserve, leading to subfertility and infertility.
38 ere differentiation defects, leading to male infertility.
39 ssive in mice and results in female-specific infertility.
40 h chronic airway diseases, hydrocephalus and infertility.
41 s loss results in neurodegeneration and male infertility.
42 s on bone and the cardiovascular system, and infertility.
43 ses a number of side effects, including male infertility.
44 nd have been implicated in leptin-associated infertility.
45 ll growth can lead to endometrial cancer and infertility.
46 tion to 9 women with absolute uterine factor infertility.
47 gain insight into the underlying reasons for infertility.
48 causes a severe temperature-sensitive female infertility.
49 he first meiotic division, resulting in male infertility.
50 t compromise gametogenesis and contribute to infertility.
51 for the treatment of absolute uterine factor infertility.
52  offer unique perspectives on sex chromosome infertility.
53 a2 develop hypogonadotropic hypogonadism and infertility.
54 doxins (PRDXs) is associated with human male infertility.
55 al and nuclear genomes contributes to female infertility.
56 and safety of procedures used to treat human infertility.
57 n of the PF reserve leads to subfertility or infertility.
58 disease, ectopic pregnancy, and tubal factor infertility.
59 s attenuate gonadotropin release, leading to infertility.
60 reased motility, resulting in selective male infertility.
61              CCP5 deficiency does cause male infertility.
62 amination are essential in diagnosis of male infertility.
63 nerative medicine solution to uterine factor infertility.
64 the gametes, and their loss results in adult infertility.
65 ic defects are directly responsible for male infertility.
66   Mitochondrial dysfunction can cause female infertility.
67 olism and who had not received treatment for infertility.
68 n assisted reproduction for the treatment of infertility.
69 t for infertility or to prompt treatment for infertility.
70 y high fidelity to prevent birth defects and infertility.
71 e gene, are an important cause of human male infertility.
72 llele in PRL2(-/-) male mice causes complete infertility.
73 ure to PFOS might induce BTB dysfunction and infertility.
74 , loss of the GAS2 homolog, Pigs, results in infertility.
75 ase progression and link SMN to general male infertility.
76 acute and chronic prostatitis linked to male infertility.
77 lation in mice caused complete male-specific infertility.
78 reasing the risk of reproductive disease and infertility.
79 lpingography (HSG) findings and all types of infertility.
80 oup 31-35 years had the highest frequency of infertility.
81         There were 299 patients referred for infertility.
82 aracterized by delayed/absent puberty and/or infertility.
83  progress to pelvic inflammatory disease and infertility.
84 hich have been previously implicated in male infertility.
85 BTB) of contraceptive drugs or to treat male infertility.
86 ssion of ANXA2 and APP proteins in secondary infertility.
87 blished solution for absolute uterine-factor infertility.
88 matory disease (PID), ectopic pregnancy, and infertility.
89 agnostic biomarker for primary and secondary infertility.
90 nal analysis and molecular diagnosis of male infertility.
91  can result in chromosome missegregation and infertility.
92 icant risk factor in developing all types of infertility.
93 ntation by male partners in the treatment of infertility.
94 including cancer, psychiatric disorders, and infertility.
95 icle loss, endocrine hormone deficiency, and infertility.
96  remains the cornerstone for evaluating male infertility, advanced diagnostic tests to investigate sp
97 ing positive feedback.SIGNIFICANCE STATEMENT Infertility affects 15%-20% of couples; failure to ovula
98  for species survival.SIGNIFICANCE STATEMENT Infertility affects 15%-20% of couples; failure to ovula
99                                  Unexplained infertility affects 2%-3% of reproductive-aged couples.
100                         It is estimated that infertility affects 8-12% of couples globally, with a ma
101                                         Male infertility affects ~7% of men, but its causes remain po
102 regnancy (aHR, 1.87; 95% CI, 1.38-2.54), and infertility (aHR, 1.85; 95% CI, 1.27-2.68).
103 of the assessment and management of men with infertility, along with current controversies and future
104         Our aim in this study was to examine infertility among male firefighters in Denmark.
105 expressed on sperm and testis occur in human infertility and after vasectomy.
106 ether thyroid antibodies are associated with infertility and assisted reproductive technology outcome
107 l plasma from men with primary, or secondary infertility and compare it with proven fertile men.
108 ed during female meiosis, a leading cause of infertility and congenital disorders.
109 hereas 523 peptides were common to secondary infertility and control group.
110 ed a total of 515 peptides common to primary infertility and control group; whereas 523 peptides were
111 te defects lie at the heart of some forms of infertility and could potentially be addressed therapeut
112  in flagella/cilia are often associated with infertility and disease.
113  was a 27-year-old female who presented with infertility and dysmenorrhea.
114 atory disease (PID) is an important cause of infertility and ectopic pregnancy, and Chlamydia trachom
115 rvicitis, pelvic inflammatory disease (PID), infertility and ectopic pregnancy.
116 of the etiology, diagnosis, and treatment of infertility and gonadal cancers, and in efforts to augme
117 cy of the micronutrient copper (Cu) leads to infertility and grain/seed yield reduction in plants.
118 wever, TRT is linked to side effects such as infertility and increased risk of prostate cancer and ca
119 pollution has been linked to higher risks of infertility and miscarriage.
120  important broader relevance to human female infertility and mitochondrial replacement therapy.
121                   Hydrosalpinx is a cause of infertility and negatively impacts in vitro fertilizatio
122    The findings have relevance to overcoming infertility and other trisomic phenotypes.
123 nce intervals were estimated for male-factor infertility and overall infertility through Cox regressi
124 ered in the International Network on Cancer, Infertility and Pregnancy (INCIP) database.
125 e and cell proliferation, resulting in pain, infertility and pregnancy loss.
126 , and limited data suggest associations with infertility and preterm birth, yet the attributable risk
127 te to negative reproductive outcomes such as infertility and preterm birth.
128 al disease globally and the leading cause of infertility and preventable infectious blindness (tracho
129 nalyses of gene variants that underlie human infertility and research into fertility-related molecule
130  findings identify Piwi as a factor in human infertility and reveal its role in regulating the histon
131 r that is responsible for 80% of anovulatory infertility and that is associated with hyperandrogenemi
132 per modeling of the process is important for infertility and toxicology research.
133 rivers directly and indirectly contribute to infertility, and argue that a complete picture of the ge
134 ditions such as pelvic inflammatory disease, infertility, and blindness.
135  leading genetic abnormality contributing to infertility, and chromosome segregation errors are commo
136 se, and possibly preterm birth, tubal factor infertility, and ectopic pregnancy in women.
137 cations such as pelvic inflammatory disease, infertility, and ectopic pregnancy.
138 underexpression of SEMG2 proteins in primary infertility; and overexpression of ANXA2 and APP protein
139                     Though endometriosis and infertility are clearly associated, the pathophysiologic
140  of any offspring conceived, and always view infertility as a possible symptom of a more general or c
141 rous genetic conditions associated with male infertility as well as emerging translational evidence o
142 onsequently, mice lacking miR-34/449 display infertility as well as severe chronic airway disease lea
143  health evaluation of men who present for an infertility assessment.
144 B in spermatocytes resulted in complete male infertility, associated with not only meiotic pachytene
145                Here we report that the human infertility-associated missense mutation (N64I) in MEIOB
146             We identified 5,370 men seen for infertility at Frederiksberg Hospital, Denmark, during 1
147 re and Rosa26CreERT2 lines) resulted in male infertility, atrophic testes with vacuolation, azoosperm
148  new insights into the mechanisms that cause infertility attributable to malnourishment.
149  and Bardet-Biedl syndrome, also suffer from infertility because cilia and sperm flagella share sever
150 ked to CF carriers (e.g., pancreatitis, male infertility, bronchiectasis), as well as some conditions
151 s of Spocd1 in mice results in male-specific infertility but does not affect either piRNA biogenesis
152 eric variation has been linked to cancer and infertility, but centromere sequences contain multiple t
153 pelvic inflammatory disease and tubal factor infertility, but it is unclear why some women are more s
154 rian Insufficiency (POI) is a major cause of infertility, but its etiology remains poorly understood.
155       Defects in Sertoli cells often lead to infertility, but replacement of defective cells has been
156 al of cumulin to improve treatment of female infertility, but, as a noncovalent heterodimer, cumulin
157 ic deficiency, AgRP signaling contributes to infertility by inhibiting Kiss1 neurons.
158 further reproductive research and treat male infertility by using natural plant extracts.
159 of STIs include pelvic inflammatory disease, infertility, cancer, and AIDS.
160  associated with many pathologies, including infertility, cardiovascular diseases, metabolic syndrome
161 olled at 4 US reproductive endocrinology and infertility care study centers between June 2013 and Dec
162 otect mice against infection, pathology, and infertility caused by different C. trachomatis serovars.
163 ormation for developing strategies to reduce infertility-causing pathology induced by infections.
164                           High prevalence in infertility clinic attendees and in women with miscarria
165 ) in female sex workers, 11.3% (9.0-13.7) in infertility clinic attendees, 12.4% (7.9-17.7) in women
166 d, placebo-controlled trial was conducted in infertility clinics in Tehran, Iran.
167               Out of 717 men referred to the infertility clinics, 180 asthenozoospermic men were rand
168 opulation-based registry data and the Danish Infertility Cohort (individual record linkage) that incl
169 eractions amongst VRS genes control spikelet infertility, determinacy and outgrowth, and novel routes
170 , body mass index, race, smoking, education, infertility diagnosis, and ART cycle year.
171 em among Arab women and is the main cause of infertility due to anovulation.
172 ondition characterized by absent puberty and infertility due to gonadotropin releasing hormone (GnRH)
173 lation of Raptor in the male germline causes infertility due to meiotic arrest and impaired inactivat
174 ed maternal-conceptus interactions can cause infertility due to pregnancy loss or later pregnancy com
175 ween oxidative stress and varicocele-related infertility due to testicular hyperthermia, which can re
176 liopathy-associated disorders including male infertility, early growth retardation, excessive weight
177  of these genomic parasites on evolution and infertility, few meiotic drive loci have been identified
178 disease, ectopic pregnancy, and tubal factor infertility) following chlamydia infection and repeat in
179 oor sperm motility is a common cause of male infertility for which there are no empirical therapies.
180              Information on vital status and infertility from the Danish Civil Registration System, t
181 , the mechanisms of temperature-induced male infertility have not been fully elucidated [5].
182 revalent zoonosis that leads to abortion and infertility in cattle, and undulant fever, debilitating
183 nt, c.1166C>T (p.Pro389Leu), segregated with infertility in five men from a consanguineous Turkish fa
184 induced leptin resistance is associated with infertility in humans and rodents, and treatments for hu
185 n globozoospermia, one of the causes of male infertility in humans.
186 and provides evidence of SYCP2-mediated male infertility in humans.
187 rmatozoa in the cauda epididymis, leading to infertility in male mice.
188 ications include neurotoxicity, memory loss, infertility in males, and development of a neurologic ps
189                                              Infertility in men and women is a complex genetic trait
190 mmune reaction response as the main cause of infertility in men with primary and secondary infertilit
191 ations in the PLCZ1 gene are associated with infertility in men.
192 nd loss of libido, erectile dysfunction, and infertility in men; they are generally treated with the
193 homatis serovar D (strain UW-3/Cx) to induce infertility in mice whose major histocompatibility compl
194 NAs and inactivation of Alkbh5 leads to male infertility in mice.
195  proliferation, and subsequently led to male infertility in mice.
196 ter risk of being diagnosed with male-factor infertility in our cohort.
197  physicians across the world when evaluating infertility in patients of similar background to our pat
198  to elevated temperatures are linked to male infertility in several organisms, the mechanisms of temp
199  highlights the conditional manifestation of infertility in specific genotypic combinations.
200 sr2+ cells, uterine hypoplasia, and complete infertility in the adult female.
201 ial to lead to a therapy for idiopathic male infertility in the clinic, and could open the door to pr
202 lls in the BT-IgSF-KO mice, we conclude that infertility in these mice is most likely caused by a fun
203 ct development is the dominant cause of male infertility in these mouse models, and this likely exten
204 on resulted in severe growth retardation and infertility in two Arabidopsis transfer DNA insertion li
205 menorrhea, loss of libido, galactorrhea, and infertility in women and loss of libido, erectile dysfun
206 cted is consistent with an increased risk of infertility in women from everyday exposures to our chem
207 matory disease (PID), ectopic pregnancy, and infertility in women with a previous Chlamydia trachomat
208 is is the leading cause of infection-induced infertility in women.
209 homatis, a leading infectious cause of tubal infertility, induces upper genital tract pathology, such
210                                              Infertility is a challenging phenomenon in cattle that r
211                                  Male factor infertility is a common problem.
212                                              Infertility is a widespread problem and a male contribut
213 ely 50% of patients have situs inversus, and infertility is common.
214                                  Male factor infertility is increasing and recognized as playing a ke
215    A promising therapeutic treatment of male infertility is the use of follicle stimulating hormone (
216 ne approach to identifying genes involved in infertility is to study subjects with this clinical phen
217 se of sexually transmitted infection-induced infertility, is frequently detected in the gastrointesti
218  pain, abdominal mass, obstructive uropathy, infertility, menstrual irregularities and abnormal renal
219                                         Male infertility might be caused by genetic and/or environmen
220               Defective crossing over causes infertility, miscarriage and congenital disease.
221 ity status: proven fertile (n = 39); primary infertility (n = 11) and secondary infertility (n = 9).
222 ; primary infertility (n = 11) and secondary infertility (n = 9).
223 ghts on some of the potential causes of male infertility, new underlining molecular mechanisms still
224 as an easily accessible system to study male infertility of humans and animals in terms of flagellar
225 ead, it is phenotypically reminiscent of the infertility of olt mice.
226 ay correctly consider themselves at risk for infertility on the basis of their previous treatments, s
227 ame infertile after 4 months of HCD feeding, infertility onset in knock-out females was delayed by 4
228 letion of CETN1 or CETN2 in mice causes male infertility or dysosmia, respectively, without affecting
229 t high risk for neurodegenerative disorders, infertility or having children with a disability as a re
230 l of development leads hybrid individuals to infertility or inviability, the world awoken to the dawn
231    No increase in risk of either male-factor infertility or overall infertility was seen among the pa
232 ) on pregnancy rates in women with confirmed infertility or the need for semen donation who were elig
233 festyle intervention preceding treatment for infertility or to prompt treatment for infertility.
234 , dysregulation of which could contribute to infertility or weight gain.
235 diagnosed because of recurrent miscarriages, infertility, or aneuploid offspring.
236 for 10 years for ectopic pregnancy and tubal infertility; our findings suggest both infections confer
237 and facilitate therapeutic treatment of male infertility patients.
238  numbers and motility in a sub-population of infertility patients.
239 tified to be associated with male idiopathic infertility patients.
240  spermatozoa motility and moderately rescued infertility phenotype.
241                                          The infertility phenotypes of females with a Zp3-Cre-driven
242 lized LRX dimer interface fail to rescue lrx infertility phenotypes.
243 ction that has direct implications for human infertility, premature reproductive aging due to oxidati
244 th Lrrc8a(-/-) mice that include curly hair, infertility, reduced longevity, and kidney abnormalities
245 treatment for patients with certain forms of infertility refractory to current clinical strategies.
246 ans, ALOX15 inhibitors might counteract male infertility related to GPX4 deficiency.
247           Although in most men the origin of infertility remains unexplained, genetic causes are incr
248 ther Piwi is an actual disease gene in human infertility remains unknown.
249 nfertility in men with primary and secondary infertility, respectively.
250  disease, ectopic pregnancy and tubal factor infertility resulting from genital chlamydial infection.
251                     Further, the increase in infertility seemed restricted to duration of time employ
252 humans and rodents, and treatments for human infertility show a decreased success rate with increasin
253                         Treatments for human infertility show a decreased success rate with increasin
254  An analysis of RBMY1 copy numbers among 376 infertility subjects failed to replicate a previously re
255 an Estonian cohort of idiopathic male factor infertility subjects.
256 mined whether the proportion of tubal factor infertility (TFI) that is attributable to Chlamydia trac
257  in the male germline, which may explain the infertility that has been associated with such inhibitor
258 ng women affected by absolute uterine-factor infertility the possibility of carrying their own pregna
259 B N64I substitution is associated with human infertility, the point mutant mice are fertile despite m
260 n multiparous (p = 0.0167), had a history of infertility therapies (p = 0.0004), and had pre-eclampsi
261 ated for male-factor infertility and overall infertility through Cox regression analyses comparing th
262 ggest that NCOA5 deficiency could cause male infertility through increased IL-6 expression in epididy
263 ntially improved the ability of couples with infertility to have biological children.
264 creases livebirth rate among couples seeking infertility treatment (2013-2017).
265 pplementation Trial studying couples seeking infertility treatment (2016-2019).
266 mization was stratified by site and intended infertility treatment (in vitro fertilization (IVF), non
267 block randomized by study center and planned infertility treatment (in vitro fertilization, other tre
268 ion is critical for new developments in both infertility treatment and contraception.
269 r both the development of contraceptives and infertility treatment drugs.
270  cytoplasm may offer a source of oocytes for infertility treatment or mitochondrial replacement thera
271 g associations between lifestyle factors and infertility treatment outcomes.
272 8 years and women aged 18-45 years) planning infertility treatment were enrolled at 4 US reproductive
273 le women, a lifestyle intervention preceding infertility treatment, as compared with prompt infertili
274 fertility treatment, as compared with prompt infertility treatment, did not result in higher rates of
275 mong a general population of couples seeking infertility treatment, the use of folic acid and zinc su
276 th female partners aged 18-45 years, seeking infertility treatment.
277 t importance not only for the development of infertility treatments but also to assess the overall he
278                      By contrast with female infertility treatments-especially hormonal manipulations
279 lecular analysis to identify male idiopathic infertility using genome wide alterations in sperm DNA m
280 y provides a paradigm for triaging candidate infertility variants in the population for in vivo funct
281 l-time firefighters, the risk of male-factor infertility was increased in comparison with the sample
282 of either male-factor infertility or overall infertility was seen among the part-time/volunteer firef
283                                    Secondary infertility was seen in 211 patients (46.9%), primary in
284 ty was seen in 211 patients (46.9%), primary infertility was seen in 79 patients (17.6%), and subfert
285    Risk of PID, ectopic pregnancy, or female infertility were evaluated using of Cox proportional haz
286 on and vesicle mediated transport in primary infertility, whereas immune system response, regulation
287 lting accumulation of GlcCer results in male infertility, whereas mutations in the GBA1 gene and loss
288  substantial reduction in sperm motility and infertility, whereas those carrying missense changes wer
289 otential therapeutic value for treating male infertility, which afflicts >100 million men world-wide.
290 ) perceived themselves at increased risk for infertility, which was significantly associated with soc
291 81) aged 30 to 44 years without a history of infertility who had been trying to conceive for 3 months
292 men aged 30 to 44 years without a history of infertility who had been trying to conceive for 3 months
293 the HSG patterns and their correlations with infertility will help physicians across the world when e
294 e that a complete picture of the genetics of infertility will require focusing on both the standard a
295      Loss of oocyte Ube2i resulted in female infertility with major defects in stability of the primo
296 Csmd1-knockout males show increased rates of infertility with significantly increased complement C3 p
297 essive severe spermatogenic failure and male infertility with strong clinical validity.
298      Oocyte Kat8 deletion resulted in female infertility, with follicle development failure in the se
299           Genetic deletion of mir-7a2 causes infertility, with low levels of gonadotropic and sex ste
300 pproximately 7% of men worldwide suffer from infertility, with sperm abnormalities being the most com

 
Page Top