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1 on, mesoappendiceal invasion, lymphovascular infiltration).
2 otein repression along with decreased T-cell infiltration.
3 use alveolar damage with perivascular T-cell infiltration.
4 ells, which resulted in decreased macrophage infiltration.
5 ited autoimmune phenotypes, including T cell infiltration.
6 e correlated with intratumoral CD8(+) T-cell infiltration.
7 tural sediment and compared to fine sediment infiltration.
8 ld serve as a viable basis for combating GBM infiltration.
9 with patient survival and tumor immune cell infiltration.
10 ocytes with moderate perivascular lymphocyte infiltration.
11 f tumor-residing cDC1s overcomes poor T-cell infiltration.
12 mouse cancer model boosted cytotoxic T cell infiltration.
13 cells, inducing immune and inflammatory cell infiltration.
14 aining microthrombi with neutrophil-platelet infiltration.
15 lly dependent on local EpAT accumulation and infiltration.
16 d intestinal damage and increased neutrophil infiltration.
17 sis, reactive oxygen species, and neutrophil infiltration.
18 patially selective delignification and epoxy infiltration.
19 mage, LPS-induced weight loss, and leukocyte infiltration.
20 enefit and positively correlated with T cell infiltration.
21 mmatory cytokine expression and myeloid cell infiltration.
22 patterns is an effective way to inhibit the infiltration.
23 disease mediated by pulmonary CD8(+) T cell infiltration.
24 rties, and dysfunctional intratumoral immune infiltration.
25 hrough of B cells and their aggressive graft infiltration.
26 ing step to improve intratumoral immune cell infiltration.
27 lowed with much delay by reduction in T cell infiltration.
28 a subgroup characterized by distinct stromal infiltration.
29 cating ubiquitous variability in immune cell infiltration.
33 nd 1 (CXCL1), a key chemokine for neutrophil infiltration (a hallmark of NASH), is highly elevated in
36 recruited in vivo) induces local immune-cell infiltration and activated dendritic cells, evoking a po
37 rrelation between Arf1 expression and T-cell infiltration and activation along with patient survival
39 sensor pathway, which results in macrophage infiltration and activation during Kras-driven PDAC in m
40 ciated with better immune control: increased infiltration and activation of cytotoxic T lymphocytes (
41 PD-1/PD-L1 T cell checkpoint induces T cell infiltration and anticancer responses in murine and huma
44 atory responses in the lungs with neutrophil infiltration and edema, further confirmed as consolidati
50 erize the microstructural effects of amyloid infiltration and is a contrast-free method to identify t
51 ry response is associated with higher immune infiltration and leads to enrichment of pre-existing ICB
52 ic, and, in the VP, there was massive immune infiltration and massive desquamation of the luminal epi
53 ificantly reduced lymphocytic and neutrophil infiltration and mast cells degranulation (p < 0.05).
54 ing NACT, increased natural killer (NK) cell infiltration and oligoclonal expansion of T cells were d
58 and as a negative regulator of T-cell tumor infiltration and patient survival in diverse human cance
59 lar subtype and share a high level of immune infiltration and PD-L1 expression, similar to basal tumo
61 ivKO mice showed increased inflammatory cell infiltration and proinflammatory gene expression in the
62 tic combination partners that augment T-cell infiltration and proliferation in so-called immune cold
63 e of NTS in C57BL/6 mice by decreasing renal infiltration and proliferation of T cells, which resulte
64 emic EP2 antagonism prevented monocyte brain infiltration and provided broader rescue of SE-induced e
65 helial barrier permeability, lower leukocyte infiltration and reduced activation of the endothelial b
70 nforced expression of CIITA increased T cell infiltration and sensitized tumors to anti-PD-1 therapy.
71 Gal-3 markedly attenuated F4/80+ macrophage infiltration and significantly increased the expression
72 we find that Cxcr4 promotes initial monocyte infiltration and subsequent territorial restriction of m
73 d by significant weight loss and immune cell infiltration and the expression of early inflammatory ma
74 lls displayed reduced cerebral CD4(+) T-cell infiltration and thrombotic activity following experimen
76 ix metalloproteinases and reduction in MDSCs infiltration, and all these contributed to inhibit pulmo
77 a, tubular injury, neutrophil and macrophage infiltration, and complement activation (plasma C5a) and
80 ifested by increased weight loss, macrophage infiltration, and inflammatory cytokines in the colon ti
81 ial chemotactic ability is a major player in infiltration, and plant stomatal defense in closing the
82 of sterile kidney injury, reduced neutrophil infiltration, and serum creatinine levels were apparent.
83 id biosynthesis, neutrophils and macrophages infiltration, and STAT3 and MAPK signaling in the liver.
84 asured via CT, alveolar wall thickness, cell infiltration, and surfactant protein A concentration in
88 ng disrupted tissue structures, inflammatory infiltrations, and oral microbiome, whereby several mast
89 ommon types (n = 16 each), followed by local infiltration around the lids and facial nerve (n = 6), t
91 e collagen content and attenuated muscle fat infiltration as well as pathogenetic molecular pathways
93 ovel MRI-based techniques to assess cellular infiltration beyond the macroscopic tumor margins and to
94 ranasal delivery of CXCL10 siRNA blocked Th1 infiltration but did not fully rescue microglial activat
95 is debate: rocks that experience significant infiltration by a water-bearing fluid may release orders
97 nor T cells, corresponding with robust tumor infiltration by donor effector over regulatory cells for
100 ow-grade inflammation and progressive tissue infiltration by immune cells and increased expression of
102 changes in intestinal microbiota, diminished infiltration by myeloid cells, and an accumulation of in
104 lex coacervate treatment displayed increased infiltration by natural killer (NK) cells and CD8alpha(+
105 n of resident mononuclear phagocytes, tissue infiltration by non-resident inflammatory cells, and the
106 reted CXCL10, in turn, stimulated the T-cell infiltration by serving as the ligand and chemoattractan
108 ha-smooth muscle actin, and mononuclear cell infiltration (CD11b(+) Ly-6c(hi) monocytes and CD11b(+)
109 2, RANTES, and TNF-alpha), inflammatory cell infiltration (CD3 + T cells, macrophages, and neutrophil
111 results suggest that increased myeloid cell infiltration contributes to autoreactive CD4 T cell-medi
115 -8040 increased CD8(+) effector T cell tumor infiltration, decreased myeloid-derived suppressor cells
117 ging methods for the detection of myocardial infiltration, device infection, and cardiovascular infla
119 analyzed histologically to characterize EpAT infiltration, fibrosis, and gap junction localization.
120 usion captured the spatiotemporal dynamic of infiltration for each molecule into the brain parenchyma
122 nds showed significantly greater lymphocytic infiltration, higher levels of MHC II, IFN-gamma, IL-1be
123 ll types to estimate relative proportions of infiltration in a panCNS tumor cohort spanning 80 subtyp
128 with FOLR1-TCB induced strong CD8(+) T-cell infiltration in HeLa tumors, where (89)Zr-Df-IAB22M2C ag
130 TH-MYCN tumors were found to resemble immune infiltration in human tumors more closely than the subcu
131 ment (defined as the composite of neutrophil infiltration in less than 5% of crypts and no crypt dest
132 eficiency causes pro-inflammatory macrophage infiltration in metabolic tissues and is linked to renin
133 the intraocular inflammation and reduced PMN infiltration in mouse eyes, but, increased the bacterial
135 (BBB) disruption and peripheral immune cell infiltration in the cerebellum following blast exposure.
137 ibrin depositions, macrophage and neutrophil infiltration in the placenta did not differ between heal
138 evel, abundance of IgG4 enhanced plasma cell infiltration in the portal region of the liver, and sati
139 ace hepatitis with IgG4 positive plasma cell infiltration in the portal region, without evidence of b
141 SA-IL-4 fusion protein prevents immune-cell infiltration in the spinal cord, decreases integrin expr
146 enotype in mice by inducing immune cell lung infiltration, including eosinophils, increasing cytokine
147 er levels of Th1 cytokines, decreased T cell infiltration, increased B cell numbers, and decreased ma
148 etroperitoneal lymph nodes, and immune cells infiltration, indicating that blocking VEGF-C signaling
149 mas were accompanied by increased macrophage infiltration, inflammatory activation and oxidative stre
150 w)Ly6g(high) cells, but only minor leukocyte infiltration into acutely ischemic-reperfused cortex and
151 evere toxicities, restricted trafficking to, infiltration into and activation within tumours, subopti
152 he factors that can play a role in bacterial infiltration into leafy greens by keeping stomata open a
155 n mice, TFH cells accordingly promote B cell infiltration into the CNS and the severity of MS animal
157 leakage, microglial activation, and antibody infiltration into the CNS, and have their olfactory func
158 eased leukocytosis in the CSF and blood, but infiltration into the cortex remained low over time.
159 mice fed a high-fat diet exhibit macrophage infiltration into the hypothalamus, whereas females were
160 duced more C. jejuni invasion and neutrophil infiltration into the Il10(-/-) mouse colon tissue compa
162 lower F4/80- and CX3CR1-positive macrophage infiltration into the liver in parallel with lower Ly6C(
164 myopathy, characterized by inflammatory cell infiltration into the myocardium and a high risk of dete
167 th chronic EAE, SA-IL-4 inhibits immune-cell infiltration into the spinal cord and completely abrogat
168 egulatory polarization of myeloid cells upon infiltration into tumors remain largely unexplored.
169 ithin previous continuum models of microbead infiltration into tumour spheroids as they rely on resol
173 n early hypoxia response and high lymphocyte infiltration levels exhibiting significantly improved LR
174 k mapped the distribution of neck muscle fat infiltration (MFI) in the deep cervical extensor muscles
175 in myeloid cells is essential for leukocyte infiltration, neuroinflammation, and demyelination in ex
177 vo approaches showed that increased cerebral infiltration of ACE10 as compared to wild-type monocytes
179 e of protumoral M2 macrophages and increased infiltration of antitumor CD8+ effector and memory T-cel
183 oma (GBM) is characterized by rapid cellular infiltration of brain tissue, raising the possibility th
184 of serum IL-6, which activates STAT3 and the infiltration of CD11b+ myeloid cells into the lung.
185 ibitor reduced skin phenotypes and decreased infiltration of CD4 T cells, macrophages, and neutrophil
186 nockout (KO) in breast cancer cells promoted infiltration of CD4(+) and CD8(+) T cells, leading to tu
190 ed that AA strikingly increased intratumoral infiltration of CD8+ T cells and macrophages, suggesting
194 promoted cell cycle progression, suppressed infiltration of cytotoxic T cells, and accelerated SCLC.
195 e-anchored systemic immunotherapy led to the infiltration of effector immune cells into the lungs, in
196 is suggested by the presence of IL-13, with infiltration of eosinophils and IgE-coated mast cells in
197 fect bone marrow-derived expansion and local infiltration of eosinophils, but markedly decreased M2 m
198 n inflammation along with a reduction in the infiltration of gammadelta T cells and the expression of
199 sequelae of eosinophil-dependent downstream infiltration of IL33-producing M2 macrophages leading to
200 clones, accumulation of somatic aberrations, infiltration of immune and stromal cells in proportions
202 e dysfunction in large part by eliciting the infiltration of immune-inhibitory cells, such as regulat
203 f BBB tight junction proteins, reduced brain infiltration of immunoglobulin, and attenuated neuroinfl
204 anoscale materials can improve targeting and infiltration of immunomodulatory payloads into tissues a
206 model, we show that JAK3 inhibition enhances infiltration of inflammatory cells, reduces expression o
209 ic activation of trypsinogen and more edema, infiltration of lung and pancreas by inflammatory cells,
210 ression inversely correlates with the immune infiltration of lung squamous tumors, while tumors with
211 an 11.5-fold increase of dendritic cells and infiltration of lymphocytes throughout the pancreatic tu
213 patocytes (Shp (Hep-/-)) resulted in massive infiltration of macrophages and CD4(+) T cells in the li
214 ion treatment markedly enhanced intratumoral infiltration of macrophages and CD8+ T lymphocytes, gran
215 ry gland and caused severe inflammation with infiltration of macrophages and neutrophils and upregula
216 Thus, our findings reveal that the combined infiltration of macrophages and neutrophils is required
219 prognostically favorable association between infiltration of mast cells and less aggressive tumor cel
220 inflammatory condition characterized by the infiltration of maternal CD8(+) T cells into the placent
221 cervical LNs had a greater tumor burden and infiltration of MDSC and M2 macrophages compared with LN
223 status, and attenuates proliferation and/or infiltration of microglia to the region thereby curtaili
224 s after peak viral titer and correlates with infiltration of monocytes, neutrophils and activated T c
226 Glp1r(-/-) mice displayed increased renal infiltration of neutrophils and T cells after induction
230 ses, and cholesterol biosynthesis, increased infiltration of neutrophils, inflammatory monocytes, and
232 immediately before and during the eruption, infiltration of rainfall into Kilauea Volcano's subsurfa
234 mors blunted the immune response by inducing infiltration of regulatory T cells and expression of imm
239 at the epidermis promotes the activation and infiltration of T cells into the skin, and provides a di
241 t elicited antitumour immunity and increased infiltration of T lymphocytes, resulting in highly poten
242 duced intratumor CCR2 expression and altered infiltration of TAMs and CTLs as evidenced by immunohist
244 educed cyst burden, it resulted in a massive infiltration of the cystic kidneys by macrophages and T
245 howed a big mass of soft tissue with diffuse infiltration of the gallbladder, displacement of the tra
246 ound that acute DSS colitis-induced enhanced infiltration of the hippocampus with macrophages and inf
247 d significant loss of BMAT with myeloma cell infiltration of the marrow, whereas BMAT was restored af
248 and pre-eclampsia are associated with T-cell infiltration of the placenta and placental pathological
250 mucus layer of the gut provides a barrier to infiltration of the underlying epithelia by both the nor
251 nin-dependent hypertension due to macrophage infiltration of the vasculature and direct activation of
252 hester disease (ECD) is characterized by the infiltration of tissues by foamy CD68+CD1a- histiocytes,
253 tasis, and immunosuppression, which inhibits infiltration of tumor-specific cytotoxic CD8+ T cells.
255 ure of mice to thermoneutrality promotes the infiltration of white adipose tissue with mast cells tha
257 alysis, grading G2 (OR 6.98), lymphovascular infiltration (OR 8.63), and size >15.5 mm (OR 35.28) wer
258 type and promoted intratumoral CD8(+) T-cell infiltration, overcoming the exclusion effect; TGFbeta1
259 gnificant levels of oesophageal eosinophilic infiltration (P < .05) in 4/6 of these animals, with two
260 YT/ICGscore had significantly increased CD8+ infiltration (p < 0.001), as expected, and worse surviva
261 se aggressiveness in GBM, particularly tumor infiltration (P = .0044) and hyperplastic blood vessels
265 MRI of the leg muscles showed fibrofatty infiltration predominating in the posterior thigh and th
266 signature analysis confirmed the lymphocyte infiltration profile consistent with the histomorphology
267 ause it regulates the dynamics of macrophage infiltration, proinflammatory and anti-inflammatory cyto
274 then used in Soil Water Balance groundwater infiltration simulations to understand the state-of-the-
275 l analysis revealed microglial or macrophage infiltration, suggesting activation in brain regions con
276 d inversely correlated with cytotoxic T cell infiltration, suggesting that HE4 may cause deregulated
278 revealed significantly decreased immune cell infiltration, synthesis of reactive oxygen species, infl
280 MCC950 significantly reduced neutrophil infiltration, T-cell activation, and disease severity in
281 xpression pattern that indicated immune cell infiltration; this tumor type was associated with the sh
282 within the aquifer are, in part, created by infiltration through the vadose zone, illustrating the c
283 -2 immune activation, inflammation, cellular infiltration, tissue repair enzymes, pathways of oxidati
285 make the catalyst synthesized by successive infiltration to have higher catalytic activity for soot
288 rophils (e.g. CXCL5, CXCL8) and neutrophilic infiltration was followed with much delay by reduction i
290 +/- 0.1 vs 7.8 +/- 0.2 um; p < 0.0001), cell infiltration was lower (20 +/- 2 vs 32 +/- 2 n/field; p
291 d pre-eclampsia, whereas CD79alpha(+) B-cell infiltration was only apparent with reduced fetal growth
292 ntigen load) and the degree of CD8(+) T cell infiltration were not associated with clinical response,
293 gh reliability and exhibit rapid, aggressive infiltration when transplanted into adult rodent brains.
294 infiltration system in hemp utilizing vacuum infiltration, which is an alternative method to stable t
295 E is driven predominantly by maternal T cell infiltration, which is significantly different from cont
296 dermal disorganization and inflammatory cell infiltration, which were improved in the 3 treated group
297 ivo treatments modifying apoplastic pH (stem infiltration with a pH buffer) or reducing stem metaboli
298 levels of viral genomes in blood and tissue infiltration with Epstein-Barr virus (EBV)-positive lymp
299 itored cortical epileptogenesis during tumor infiltration with in vivo electrophysiology and GCAMP7 c
301 buffer) or reducing stem metabolic activity (infiltration with sodium vanadate and sodium cyanide; pl