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1 of 113 patients with isolated burn injury or inhalation injury.
2 episodes correlate with injury severity and inhalation injury.
3 - and anti-inflammation early after burn and inhalation injury.
4 ore TBSA burn similar in age, TBSA burn, and inhalation injury.
5 sfunction in sheep subjected to severe smoke inhalation injury.
6 ion in an established, large animal model of inhalation injury.
7 injury secondary to combined burn and smoke inhalation injury.
8 acious therapy for burn patients with severe inhalation injury.
9 ry, acute respiratory distress syndrome, and inhalation injury.
10 lmonary pathology in burn victims with smoke inhalation injury.
11 tion of burn patients with concomitant smoke inhalation injury.
12 cal studies of patients with burns and smoke inhalation injury.
13 lmonary pathology in burn victims with smoke inhalation injury.
14 ding to TBSA burn and presence or absence of inhalation injury.
15 nstriction following combined burn and smoke inhalation injury.
16 ARDS resulting from combined burn and smoke inhalation injury.
17 in sheep with severe combined burn and smoke inhalation injury.
18 in previous studies to reduce the degree of inhalation injury.
19 eaths administered (24, 36, 48) for a graded inhalation injury.
20 hat neutrophils play a pivotal role in smoke inhalation injury.
21 40 percent of body-surface area burned, and inhalation injury.
22 at are used commonly are age, burn size, and inhalation injury.
23 excision, wound healing, scar formation, and inhalation injury.
24 re shrapnel injuries, and 533 (64%) were gas inhalation injuries.
25 = nonsignificant), and clinical diagnosis of inhalation injury (39% vs. 35%, p = nonsignificant).
27 urn size, incidence in sepsis (20% vs. 26%), inhalation injury (46% vs. 27%), or mortality (8% vs. 7%
32 ctive and lumenal obstructive response after inhalation injury and identifies low-dose nebulization o
33 s been placed on the early identification of inhalation injury and its impact on fluid resuscitation,
34 ry response is enhanced with worse grades of inhalation injury and that those who die of injuries hav
39 BSA) burned, age > or =40 years, presence of inhalation injury, and ventilator days were found to be
41 idate models, a 3-variable model with %TBSA, inhalation injury, and von Willebrand factor-A2 had comp
42 fering from a 60% TBSA burn with concomitant inhalation injury are more likely to develop sepsis if t
44 were then compared to the graded severity of inhalation injury as determined by Abbreviated Injury Sc
45 in sheep with severe combined burn and smoke inhalation injury by preventing the formation of airway
50 l, regardless of age, burn size, presence of inhalation injury, delay in resuscitation, or laboratory
51 Lower age, larger burn size, presence of inhalation injury, delayed intravenous access, lower adm
53 e aimed to determine whether the severity of inhalation injury evokes an immune response measurable a
54 ep in the sepsis group received cotton smoke inhalation injury followed by instillation of Pseudomona
57 sma IL-1RA also correlated with % TBSA burn, inhalation injury grade, fluid resuscitation, Baux score
58 ing for the effects of age, % TBSA burn, and inhalation injury grade, plasma IL-1RA remained signific
60 low inhalation injury (grades 1-2) vs. high inhalation injury (grades 3-4), we found significant dif
61 Those who presented with worse grades of inhalation injury had higher plasma levels of carboxyhem
66 found that patients with >60% TBSA burn with inhalation injury have an 8% risk of developing sepsis i
67 hstanding significant % TBSA and presence of inhalation injury, have significantly declined compared
68 gender, total body surface area burned, and inhalation injury (hazard ratio, 1.73; 95% CI, 1.18-2.54
69 57.8 +/- 18.2 for children) and presence of inhalation injury in 38% of the adults and 54.8% of the
71 the neonatal and pediatric population, treat inhalation injury in pediatric and adult patients, and a
72 D and ADHD had higher rates of ingestion and inhalation injuries (IRR, 1.57 [95% CI, 1.06-2.25] and 1
75 e patient demographics, incidence of sepsis, inhalation injury, mortality, serum constitutive protein
76 o patient age, total body surface area burn, inhalation injury, number of units of blood transfused o
78 tient age, total body surface area burn, and inhalation injury on the probability of discharge and de
79 teristics of age, burn size, and presence of inhalation injury, outcome was correctly predicted in on
81 ere at their highest in those with the worst inhalation injury scores (grades 3 and 4), the greatest
82 a immune mediators were increased with worse inhalation injury severity, even after adjusting for age
86 nderstanding of the pathophysiology of smoke inhalation injury, the best evidence-based treatments, a
87 ose with limited donor sites, those who have inhalation injury, those with delays in resuscitation, a
89 than 55 years with severe burn injuries and inhalation injury to survive these devastating condition
90 ace area involvement of 54% +/- 4%, 63% with inhalation injury) underwent tracheostomy a mean of 3.9
91 catheters and underwent an LD50 cotton smoke inhalation injury via a tracheostomy under halothane ane
94 Age, total body surface area burned, and inhalation injury were also significantly associated wit