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1  selective kinetic resolution of a protected inositol monophosphate.
2 in valproate leads to decreased synthesis of inositol monophosphate.
3 erase that cleaves the cyclic bond of cyclic inositol monophosphate.
4                        In addition, using an inositol monophosphate 1 (IP1) accumulation assay, we sh
5  coupling of the probe to substance P, while inositol monophosphate accumulation assays demonstrated
6  and LTB(4) induced Ca(2+) and intracellular inositol monophosphate accumulation, respectively, highl
7 es that, by trapping inositol in the form of inositol monophosphates and certain inositol polyphospha
8 al phosphatase that has dual activity toward inositol monophosphates and fructose 1,6-bisphosphate.
9 yme possessing the ability to hydrolyze both inositol monophosphates and Gal-1-P with equal efficienc
10 ch demonstrated similar ability to hydrolyze inositol monophosphates and galactose 1-phosphate.
11 ing bombesin receptors and the intracellular inositol monophosphate assay, it was possible to determi
12 s capacity to release free myo-inositol from inositol monophosphates generated from receptor-linked a
13 d for hormone binding and induction of cAMP, inositol monophosphate, inositol bisphosphate, and inosi
14  valproate does not increase accumulation of inositol monophosphates, inositol bisphosphates, or inos
15 rylates myo-inositol to produce multiple myo-inositol monophosphates, Ins1/3P, Ins4/6P and possibly I
16 cid resulted in a marked accumulation of [3H]inositol monophosphate (InsP1) and inositol-1,4,5-trisph
17 acellular calcium (iCa(2+)) mobilization and inositol monophosphate (IP(1)) accumulation assays.
18 -induced signaling with Ca(2+) mobilization, inositol monophosphate (IP(1)) accumulation, extracellul
19 xpressing mGlu(5) using Ca(2+) mobilization, inositol monophosphate (IP(1)) accumulation, extracellul
20 activation by measuring accumulation of [3H] inositol monophosphates (IP) in rats pre-labeled with [3
21 nvestigated their effects on KISS1R-mediated inositol monophosphate (IP1) and Ca2+ signaling in cell
22                              In a FRET-based inositol monophosphate (IP1) assay we identified compoun
23 y-EM293 cells with TSH and measuring cAMP or inositol monophosphate (IP1) production, a measure of ph
24 e more gradual and sustained accumulation of inositol monophosphate (IP1).
25 cAMP, KB = 4.9 and 5.9 nM, respectively) and inositol monophosphate (IP1, KB = 0.6 and 16 nM, respect
26 ol-1,4,5-trisphosphate, which is degraded to inositol monophosphate (IP1; phosphoinositide signaling)
27  the synthesis of free inositol from various inositol monophosphates, is encoded by a small multigene
28 ar to genes encoding bacterial and mammalian inositol monophosphate phosphatases.
29 lso effective in stimulating basal tritiated inositol monophosphate production in the neonatal rat ce
30 y myo-inositol monophosphatase (IMPase) from inositol monophosphate species.
31 overlay assays suggested the PH domain binds inositol monophosphates, surface plasmon resonance demon
32 phatase (IMPase) catalyzes the hydrolysis of inositol monophosphate to inorganic phosphate and inosit