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1 hN-P-Man-Man-(EthN-P)Man-GlcN attached to an inositol phospholipid.
2 naceous matrix, cytoskeletal components, and inositol phospholipids.
3 y removing phosphate from the D3 position of inositol phospholipids.
4 ore protein with a cationic motif that binds inositol phospholipids.
5 all implicated in the remodeling of membrane inositol phospholipids.
6 anding of the regulation of such proteins by inositol phospholipids.
7 ar signaling molecules derived from membrane inositol phospholipids.
8 e p47phox is unable to bind 3-phosphorylated inositol phospholipids.
9 nstream of PI 3-kinase associated changes in inositol phospholipids.
10 s demonstrate that chemical stress activates inositol phospholipid 3-kinase and glucose transport in
11  In this study, we have investigated whether inositol phospholipid 3-kinase is activated by chemical
12 l responses of these cells the activation of inositol phospholipid 3-kinase is not responsible for th
13 ving a PtdIns-specific, wortmannin-sensitive inositol phospholipid 3-kinase may be required to suppor
14  have attempted to assess the basis by which inositol phospholipid 3-kinase may participate in the re
15           In skeletal muscle and fat tissue, inositol phospholipid 3-kinase plays an integral role in
16 senite induced rapid activation (< 2 min) of inositol phospholipid 3-kinase with an approximately fou
17 hatase with specificity for 3-phosphorylated inositol phospholipids, accompany progression of brain t
18 tested the kinase in the presence of several inositol phospholipids and found that only low micromola
19 his interaction was direct and required both inositol phospholipids and GTP.
20 orm an "intermolecular" PH domain that binds inositol phospholipids and is required for ion channel l
21 in to generate a series of 3' phosphorylated inositol phospholipids and tested whether any of these l
22 ed intracellular levels of 3' phosphorylated inositol phospholipids and the induction of signaling re
23                                              Inositol phospholipids are a focus of renewed interest w
24  in Dictyostelium, the hydrocarbon chains of inositol phospholipids are a highly selected subset of t
25                                              Inositol phospholipids are critical regulators of membra
26 hatidylinositols, suggesting that endogenous inositol phospholipids are required for the activation o
27                                              Inositol phospholipids are thought to play an important
28 lly ablates the ability of PTEN to recognize inositol phospholipids as a substrate, suggesting that l
29 ma(2)-mediated inositol phosphate formation, inositol phospholipid assessments, fluorescence recovery
30 n fluorescent protein, which translocates to inositol phospholipids at the leading edge of cells duri
31 re are several recently reported examples of inositol phospholipids binding to pleckstrin homology (P
32 were needed for virus infection, whereas the inositol phospholipid-binding and F-actin-binding domain
33 4p or a ten-residue peptide derived from the inositol-phospholipid-binding site of gelsolin.
34 ents have established that the metabolism of inositol phospholipids by phosphoinositide 3-kinases (PI
35 in 2A to mitochondria and that modulation of inositol phospholipids by synaptojanin 2A may play a rol
36 auses phospholipase C-mediated hydrolysis of inositol phospholipids, calcium mobilization, and activa
37                                              Inositol phospholipids have long been known to have an i
38          Tachykinin peptides stimulated both inositol phospholipid hydrolysis and arachidonic acid re
39 tide receptors (P2UR or P2Y2R) that activate inositol phospholipid hydrolysis and Ca24 mobilization i
40 adioligand binding and by agonist-stimulated inositol phospholipid hydrolysis and intracellular Ca2+
41 educed M1-muscarinic receptor stimulation of inositol phospholipid hydrolysis in 1321N1 astrocytoma c
42 orylation that paralleled its stimulation of inositol phospholipid hydrolysis.
43 or the production of inositol phosphates and inositol phospholipids in all eukaryotes.
44 Our data indicate the involvement of IP3 and inositol phospholipids in both short- and long-term resp
45  protein phosphotyrosyl and 3-phosphorylated inositol phospholipids in vitro.
46  phogrin is able to dephosphorylate specific inositol phospholipids, including phosphatidylinositol (
47  of this PH domain to interact with membrane inositol phospholipids inside living cells.
48           The production of 3-phosphorylated inositol phospholipids is implicated in regulation of ce
49 gest that the production of 3-phosphorylated inositol phospholipids is involved in the activation of
50 dSer by choline, ethanolamine, glycerol, and inositol phospholipids markedly inhibits PtdSer transfer
51 This effect is accompanied by alterations in inositol phospholipid metabolism and inositol auxotrophy
52 binding proteins, heterotrimeric G proteins, inositol phospholipid metabolism, and protein serine/thr
53 gest that PI(20:4/20:4) is a novel bioactive inositol phospholipid molecule that regulates innate imm
54                       We identify an unusual inositol phospholipid molecule, PI(20:4/20:4), the level
55                        We show here that the inositol phospholipids of Dictyostelium are different, b
56  that only partially inhibited hydrolysis of inositol phospholipids or release of secretory granules.
57                                              Inositol phospholipids play multiple roles in cell signa
58                           Phosphorylation of inositol phospholipids plays a key role in cellular regu
59 up of protein tyrosine phosphatases that use inositol phospholipids, rather than phosphoproteins, as
60 ns have been found to interact in vitro with inositol phospholipids; recent experiments show that the
61                                              Inositol phospholipids regulate a variety of cellular pr
62 gest that alpha-synuclein may play a role in inositol phospholipid signaling.
63 c stimulation triggers activation of Src and inositol-phospholipid signalling, which polarizes in the
64 r molecules, including adenylyl cyclases and inositol-phospholipid-specific phospholipase C (PLC)1-3.
65                  These data demonstrate that inositol phospholipid synthesis is essential for the str
66                                  None of the inositol phospholipids tested activated or inhibited PKB
67 hospholipases of the C type, which hydrolyze inositol phospholipids to yield inositol trisphosphate a
68 n mutant syk gene failed to elicit increased inositol phospholipid turnover after EMF exposure, where
69 tion of the lyn gene did not elicit enhanced inositol phospholipid turnover after EMF exposure.
70 alanin and other galanin analogues increased inositol phospholipid turnover and intracellular calcium
71  normal EMF response, whereas no increase in inositol phospholipid turnover in response to EMF was no
72 s responsible for EMF-induced stimulation of inositol phospholipid turnover, and its SH2 domains are
73 C-gamma 2 (PLC-gamma2), leading to increased inositol phospholipid turnover.
74 se Cgamma2 (PLC-gamma2) leading to increased inositol phospholipid turnover.
75 elated to changes in both ubiquitination and inositol phospholipids, we have examined whether GLUT4 t
76 r the interaction of inositol phosphates and inositol phospholipids with syndecan-4 could regulate PK