ライフサイエンスコーパス: insufficiency

1 ergencies (Sim2-pump failure and Sim3-access insufficiency).

2 d low birthweight, an indicator of placental insufficiency.

3 atal anthropomorphic indicators of placental insufficiency.

4 peutic optimisation, she died of respiratory insufficiency.

5 osomal gene mutations that lead to ribosomal insufficiency.

6 erapy to restore hormonal balance in ovarian insufficiency.

7 ms, and three patients had suspected adrenal insufficiency.

8 regulatory-associated protein of mTOR haplo-insufficiency.

9 r size was observed, supportive of placental insufficiency.

10 growth restriction and attenuated placental insufficiency.

11 ronary disease, diabetes mellitus, and renal insufficiency.

12 city, corrected QT prolongation, and adrenal insufficiency.

13 formance in a rat model of peripheral artery insufficiency.

14 to computed tomography contrast due to renal insufficiency.

15 synonymous with the diagnosis of convergence insufficiency.

16 examined, especially in the setting of renal insufficiency.

17 rats and in a rat model of peripheral artery insufficiency.

18 f the SHH pathway secondary to Ptch1 or Sufu insufficiency.

19 normal tonsillar pillars, and velopharyngeal insufficiency.

20 ene or transcript, or ameliorating a genetic insufficiency.

21 3mug/dL or less is considered to be adrenal insufficiency.

22 d tau pathology, independent of haemodynamic insufficiency.

23 sociated with all comorbidities except renal insufficiency.

24 cularly in these patients who may have renal insufficiency.

25 type of lymphoma and/ or presence of adrenal insufficiency.

26 llenged with stressors such as growth factor insufficiency.

27 eading to pain, maldigestion, and pancreatic insufficiency.

28 sitive to stress induced by simulated energy insufficiency.

29 exhibiting mild to moderate or severe renal insufficiency.

30 No participants experienced renal insufficiency.

31 onatal pups died at birth due to respiratory insufficiency.

32 ome (PTMS), cardiovascular events, and renal insufficiency.

33 is a better indicator of chronic respiratory insufficiency.

34 ith pre-operative mild, moderate, and severe insufficiency.

35 rized by neutropenia and exocrine pancreatic insufficiency.

36 he chromosomal deletion causes CSNK1A1 haplo-insufficiency.

37 mprovement in cardiovascular features of ELN insufficiency.

38 cute liver decompensation seemed to be heart insufficiency.

39 causing DAD and leading to acute respiratory insufficiency.

40 g ultimately to beta-cell demise and insulin insufficiency.

41 o identify protein biomarkers of respiratory insufficiency.

42 that is fatal in infancy due to respiratory insufficiency.

43 llent method of inducing exocrine pancreatic insufficiency.

44 e (Adipoq (-/-) ) develop GDM due to insulin insufficiency.

45 d to sense conditions of potential metabolic insufficiency.

46 iagnosis, even in pregnancy and severe renal insufficiency.

47 ave vitamin D deficiency and 7.5 million its insufficiency.

48 omy for the induction of exocrine pancreatic insufficiency.

49 he exocrine pancreas, resulting in digestive insufficiencies.

50 leak, we found minimal evidence of oxidative insufficiencies.

51 [54.8%]; P = .02), especially cardiovascular insufficiencies (10 [43.5%] vs 4 [12.9%]; P = .02) and i

52 n cardiac (50%-60%), followed by respiratory insufficiency (15%-40%).

53 e classification of deficiency (< 20 ng/ml), insufficiency (20-30 ng/ml) and sufficiency (> 30 ng/ml)

54 ever, in Nordic-born mothers, maternal 25OHD insufficiency (25 - <50 nmol/L) at ~11 weeks gestation w

55 luded coronary artery disease (51.5%), renal insufficiency (27.4%), diabetes mellitus (29.5%), chroni

56 TPVR indications were pulmonary insufficiency (28%), stenosis (23%), and mixed (49%).

57 (23% vs 53%; P = .004), pancreatic exocrine insufficiency (29% vs 56%; P = .03), or endocrine insuff

58 l site infection (1.3%), velopharyngeal (VP) insufficiency (3.9%), and VP stenosis (1.6%).

59 ficiency (29% vs 56%; P = .03), or endocrine insufficiency (40% vs 64%; P = .05).

60 nts subjected to sclerotherapy due to venous insufficiency (45 women and 15 men, mean age 51.2 +/- 14

61 47.7%-69.4%]) and patients with severe renal insufficiency (62.5% [99% CI, 59.9-65.0]).

62 to a frequently lethal triad of respiratory insufficiency, acute cardiovascular failure, and coagulo

63 tochondrial dynamics, but how chronic energy insufficiency affects mitochondrial trafficking and qual

64 isk of prolonged fetal exposure to placental insufficiency against the risks of preterm delivery.

65 4); hemorrhage (aHR 4.92, 1.28-18.94); renal insufficiency (aHR 4.85, 1.20 - 19.61); inadequate perio

66 morphology, aortic stenosis (AS), and aortic insufficiency (AI) have been proposed as potential risk

67 In relation to Accommodative Insufficiency (AI), a frequency rate of 10% was recorded

68 ing "chronic kidney disease", "chronic renal insufficiency", "albuminuria", "proteinuria", and "rando

69 a convergence deficit other than convergence insufficiency (all with concurrent accommodative disorde

70 Notably, tRNA insufficiency also activated the reporter, independent o

71 idence of residual moderate or severe aortic insufficiency among patients with bicuspid AV (2.7% vers

72 tudy on human structural variation indicates insufficiencies and errors in the human reference genome

73 accommodation only; 21 (28%) had convergence insufficiency and accommodation deficits; and 6 (8%) had

74 ed high fever, progressed rapidly to hepatic insufficiency and coma, and died 8 days later.

75 with a statistically significant rate of VP insufficiency and globus sensation; however, studies lac

76 lepsy and the absence of exocrine pancreatic insufficiency and hypoplasia of nasal alae.

77 ltered phenotypes, possibly due to placental insufficiency and impaired fetal growth.

78 Placental insufficiency and intrauterine growth restriction (IUGR)

79 eferoxamine (DFO) to model chronic energetic insufficiency and its effects on mitochondrial dynamics

80 Using a sheep model of placental insufficiency and IUGR, we have previously demonstrated

81 ons with VKORC1v2, GlucII, or UGGT1, and the insufficiency and lack of direct contributory effects of

82 circulating SPINT1 is a marker of placental insufficiency and may identify pregnancies with an eleva

83 cluding degenerative changes, infection, and insufficiency and pathologic fractures.

84 ay, confinement to a wheelchair, respiratory insufficiency and premature death.

85 The ability to sense proteasome insufficiency and respond by directing the transcription

86 r mechanism of HSF2BP-S167L in human ovarian insufficiency and sub(in)fertility.

87 that strongly increase the risk of vitamin D insufficiency and tested their effect on risk of multipl

88 sociated with clinical measures of pulmonary insufficiency and the risk of mortality.(C) RSNA, 2019On

89 >=35 to be the threshold value for vitamin D insufficiency, and >=51 to be predictive of vitamin D de

90 peroxidation under conditions of glutathione insufficiency, and a reduced capacity to repair lipid da

91 natriuretic peptides, mild-to-moderate renal insufficiency, and a systolic blood pressure of at least

92 -KHDC3L autoantibodies and premature ovarian insufficiency, and between anti-RFX6 autoantibodies and

93 ted, history of heart failure (HF), or renal insufficiency, and by CART analysis were those with >=2

94 re, hypercholesterolemia, dysglycemia, renal insufficiency, and cognitive impairment.

95 ns such as malnutrition, pancreatic exocrine insufficiency, and diabetes; refer to a specialised cent

96 thesized that PE, maternal asthma, vitamin D insufficiency, and excess body mass index (BMI) might sh

97 een established as the cause of mitral valve insufficiency, and four different missense mutations in

98 poxia resulted in asymmetric IUGR, placental insufficiency, and reduced placental PPAR-gamma expressi

99 helicase complex mutated in primary ovarian insufficiency, and RPA1.

100 ronounced generalized hypotonia, respiratory insufficiency, and variable neurodegeneration and diffus

101 topia, Dandy-Walker malformation), pituitary insufficiency, and/or synpolydactyly.

102 observations in sheep models with placental insufficiency are consistent with cases of human IUGR.

103 ment, a determination of exocrine pancreatic insufficiency are scarce in the literature.

104 by which the ER defends against cholesterol insufficiency are well described, it remains unclear how

105 itamin D deficiency as 25-(OH)D < 50 nmol/L, insufficiency as 50-75 nmol/L, and sufficiency as >75nmo

106 eficiency was defined as PLP <20 nmol/L, and insufficiency as PLP 20-30 nmol/L.

107 reased risk factors for posttransplant renal insufficiency at study start, relative to patients from

108 y analyzed 3,328 CF patients with pancreatic insufficiency born after 1985 and recruited into the Fre

109 , similar to human mutation carriers, had TR insufficiency but an otherwise preserved transcriptome.

110 oximal dominance, hypotonia, and respiratory insufficiency but typically not cardiac dysfunction.

111 y limiting deaths resulting from respiratory insufficiency, but there is currently no effective thera

112 Placental insufficiency can cause fetal growth restriction and sti

113 ns, and either overexposure or (more rarely) insufficiency can cause neurological dysfunction.

114 Cellular stress responses to energy insufficiency can impact virus reproduction.

115 remains unclear whether persistent metabolic insufficiency can imprint permanent T cell dysfunction.

116 Renal insufficiency can occur in patients with congenital lowe

117 Placental insufficiency causes intrauterine growth restriction (IU

118 Elastin insufficiency causes recurrent vascular stenoses.

119 lity in a porcine model of chronic pulmonary insufficiency causing right ventricular volume overload.

120 The prevalence of definite Convergence Insufficiency (CI) in the children assessed was 2%.

121 t of critical illness-related corticosteroid insufficiency (CIRCI) in adult and pediatric patients.

122 9 participants enrolled in the Chronic Renal Insufficiency Cohort (CRIC) Study between 2003 and 2008

123 I) study and the subset of the Chronic Renal Insufficiency Cohort (CRIC) study enrollees recruited fr

124 lowed 1502 participants of the Chronic Renal Insufficiency Cohort (CRIC) Study for a mean of 6.72 yea

125 ty with CKD progression in 431 Chronic Renal Insufficiency Cohort (CRIC) Study participants, of whom

126 rticipating in the multicenter Chronic Renal Insufficiency Cohort (CRIC) Study.

127 participants with CKD from the Chronic Renal Insufficiency Cohort (CRIC) study.

128 500 subjects with DKD from the Chronic Renal Insufficiency Cohort for DKD phenotypes, including glyce

129 Using 2003-2013 data from the Chronic Renal Insufficiency Cohort Study (3,597 participants with CKD)

130 Using 2003-2013 data from the Chronic Renal Insufficiency Cohort Study (3,597 participants with CKD)

131 n our case-cohort study of 894 Chronic Renal Insufficiency Cohort Study participants with diabetes an

132 and 2161 men) enrolled in the Chronic Renal Insufficiency Cohort Study, a large, diverse CKD cohort.

133 In 3655 participants of the Chronic Renal Insufficiency Cohort study, we evaluated the association

134 tudy among participants in the Chronic Renal Insufficiency Cohort Study.

135 The prospective CRIC (Chronic Renal Insufficiency Cohort) study measured the estimated glome

136 ctive case-cohort study of the Chronic Renal Insufficiency Cohort, we measured FGF23 at two to five a

137 in an independent cohort, the Chronic Renal Insufficiency Cohort.

138 iency-induced OA is largely rescued by Runx2 insufficiency, confirming the miR-204/-211-Runx2 axis.

139 developing CNS; however, whether cholesterol insufficiency contributes to remyelination failure in MS

140 GnRH neurons and indicate that AMH signaling insufficiency contributes to the pathogenesis of CHH in

141 conditions, particularly given in pancreatic insufficiency, could threaten digestibility.

142 6.3; P=0.001) and the incidence of 2+ aortic insufficiency declined (14.0% versus 2.7%; P<0.001) in c

143 The proportions of VD insufficiency/deficiency were 72.5%, 91.3%, and 86.5% at

144 5(OH)VD levels < 30 ng/mL were defined as VD insufficiency/deficiency.

145 ataxia, spinal deformities, and respiratory insufficiency due to neuromuscular incoordination.

146 reviously well before developing respiratory insufficiency due to severe COVID-19, requiring mechanic

147 ase of micronutrient deficiency and vascular insufficiency, due to detectable structural changes in t

148 Placental insufficiency during early-mid gestation may have implic

149 ing surges in energy demand, and chronic ATP insufficiency endangers cell survival.

150 a minipig model leads to exocrine pancreatic insufficiency (EPI).

151 ous disease-specific quality of life (Venous Insufficiency Epidemiological and Economic Study Quality

152 re no reliable screening tests for placental insufficiency, especially near-term gestation when the r

153 tic shock (4.0-fold, 95% CI 3.58-4.43) renal insufficiency/failure (3.3-fold, 95% CI 2.91-3.65), any

154 atrophy, osteoporosis, Addison-like adrenal insufficiency, fatty liver, and type 2 diabetes syndrome

155 ons: accelerated OA progression, subchondral insufficiency fracture, complications of osteonecrosis,

156 ding accelerated OA progression, subchondral insufficiency fracture, complications of pre-existing os

157 Only globus and VP insufficiency had a significant incidence compared with

158 Subjects with serum vitamin D deficiency or insufficiency had higher odds for MS than those with suf

159 In times of vitamin K insufficiency, hepatic procoagulant factors are preferen

160 1.88; 95% CI, 1.12-3.16; P=0.02), and renal insufficiency (HR, 2.00; 95% CI, 1.33-3.01; P<0.001) as

161 th constitutional symptoms, colitis, adrenal insufficiency, hyperglycaemia, and hypokalaemia.

162 essive cholestatic liver disease, pancreatic insufficiency, hypoglycemia, anemia, intermittent protei

163 to the tissues is impaired leading to oxygen insufficiency (hypoxia).

164 ith SRNS and facultative ichthyosis, adrenal insufficiency, immunodeficiency, and neurological defect

165 ical correlations showed inconsistencies and insufficiencies in these patients.

166 Due to insufficiencies in transcriptome to proteome correlation

167 eplication, as well as current therapies and insufficiencies in treating HIV-infected individuals.

168 include diabetes in 38% to 40% and exocrine insufficiency in 30% to 48%.

169 to fibrotic injury and ultimately pancreatic insufficiency in 85% of the CF population.

170 obal regulatory mechanism in which ribosomal insufficiency in a subset of cells triggers organism-wid

171 Despite mitochondrial insufficiency in all DaNs, exclusively SNc neurons showe

172 validated against the outcome of respiratory insufficiency in an external cohort.

173 ceptor cell (PRC) death and identified early insufficiency in biosynthetic pathways of prohomeostatic

174 e to sunlight increase the risk of vitamin D insufficiency in children.The aim of the study was to ev

175 oidal vasculature and RPE cells, implicating insufficiency in choroidal macrophage function as a fact

176 enin deficiency in these cells indeed causes insufficiency in cortical bone regeneration.

177 viability includes the fact that defects or insufficiency in DNA ligase are casually linked to genom

178 ly contributes to the hypoargininemia and NO insufficiency in falciparum malaria in children.

179 e pathological elevation could lead to MMP-2 insufficiency in humans.

180 ifferentiation in asthma, overrepresented by insufficiency in insulin and Notch signaling, but with t

181 rial respiration and contribute to placental insufficiency in IUGR pregnancies.

182 ther directly underly or predict respiratory insufficiency in mutator mice.

183 ditions; consequently, proteasome functional insufficiency in return exacerbates IPTS.

184 nction causes hypoproliferation and cellular insufficiency in ribosomopathies and RP-mutant cancer.

185 ogical function of PGRN and the role of PGRN insufficiency in the pathogenesis of neurodegenerative d

186 ected group membership predicted respiratory insufficiency in the PRO-ACT cohort (concordance statist

187 ients with type 1 or type 2 diabetes have an insufficiency in their functional beta cell mass.

188 between low circulating SPINT1 and placental insufficiency in two other cohorts.

189 OX), are candidate genes for primary ovarian insufficiency in women.

190 .70 [95% CI, 1.16-2.48]; P=0.006), and renal insufficiency (incident rate ratio, 1.71 [95% CI, 1.03-2

191 1(S25A) mice exhibit hallmark features of HR insufficiency, including premature aging and hypersensit

192 vehicle-treated mutants displayed inotropic insufficiency, increased diastolic tension, and prematur

193 t smokers were more likely to have vitamin D insufficiency, independent of each other and other relev

194 d CVD death, myocardial infarction, coronary insufficiency, index admission for heart failure, and st

195 tic viruses were incomplete due to metabolic insufficiencies induced by the tumor microenvironment.

196 es mellitus, sleep apnea, cardiopathy, renal insufficiency, inflammatory bowel disease, immunosuppres

197 Non-genetic MMP-2 insufficiency is a relatively unexplored condition which

198 ovide a solid basis to explain why placental insufficiency is associated with disorders of sex develo

199 e fetal circulation resulting from placental insufficiency is associated with more favourable haemody

200 Chronic venous insufficiency is characterized by inadequate venous retu

201 e of the important causes of primary ovarian insufficiency is chemotherapy-induced primordial follicl

202 Vitamin D insufficiency is common in older people and may lead to

203 Vitamin D insufficiency is common, correctable, and influenced by

204 Primary adrenal insufficiency is life threatening and can present alone

205 fficacy of exercises for primary convergence insufficiency is plausible and supported by data, there

206 If placental insufficiency is present, the physician must trade off t

207 trast to nebulin-based severe NM where haplo-insufficiency is the disease driver, Compound-Het mice e

208 cluding retinal macular dystrophy and kidney insufficiency leading to transplantation, associated wit

209 Using mouse models, we show that stromal insufficiency limits the number of donor-derived HSCs an

210 of growth delay, immunodeficiency, endocrine insufficiencies, lipodystrophy, and cancer predispositio

211 ystems (proteinuria, thrombocytopenia, renal insufficiency, liver involvement, cerebral symptoms or p

212 Noncardiac diagnoses, preexisting renal insufficiency, longer time from onset of the cardiopulmo

213 ce of vitamin D deficiency (< 30 nmol/L) and insufficiency (< 50 nmol/L) were 1.7% (95% CI 1.0, 2.8)

214 Beside respiratory insufficiency, many hospitalized patients exhibit neurol

215 y or extra monitoring for possible placental insufficiency may be advisable.

216 its of using MAR in CABG patients with renal insufficiency may be offset by competing health risks.

217 l complex, and disease-associated complex II insufficiency may result from incorrect function of the

218 lso seen in the frequency of jaundice, renal insufficiency, mechanical ventilation, high-level cytome

219 Two minipigs failed to develop exocrine insufficiency most likely due to undetected accessory pa

220 Given the potential risk of liver insufficiency, most centres will only accept candidates

221 When fetal growth is compromised, placental insufficiency must be distinguished from modest genetic

222 y injury, colitis, hypokalaemia, and adrenal insufficiency (n=1 each).

223 1), prolonged QT interval (n=2), and adrenal insufficiency (n=1).

224 variant have an increased risk of vitamin D insufficiency (odds ratio [OR] = 2.2, 95% confidence int

225 ne status monitoring may in part explain the insufficiencies of current prognostic stratification met

226 We investigate the insufficiency of commonly used models for disease genome

227 and at the population level, because of the insufficiency of current region-of-interest-based approa

228 ions encompassing DLL1, we hypothesized that insufficiency of DLL1 causes a human neurodevelopmental

229 The present findings indicate the insufficiency of giant panda conservation for protecting

230 However, there is insufficiency of information in regard to these in Sudan

231 this was not the case in ACF, suggesting the insufficiency of methylation changes to modulate gene ex

232 Down-regulation or insufficiency of PGRN led to the increased lysosomal gen

233 g multiple organs that is caused by systemic insufficiency of the mitochondrial protein frataxin.

234 the phylogeny of CHKB-CPT1B synteny, and the insufficiency of UCP1 to account for eutherian BAT, thes

235 is (two [5%] and one [3%] patients), adrenal insufficiency (one [3%] and two [5%] patients), and coli

236 odation deficits; and 6 (8%) had convergence insufficiency only.

237 Adults presenting with aortic insufficiency or a dilated aortic annulus or ascending a

238 isms of channel dysfunction, including haplo-insufficiency or a dominant-negative effect caused by th

239 for the treatment of posterior tibial tendon insufficiency or chronic Achilles tendinopathy.

240 nadequate reduction of hydroperoxy-PE due to insufficiency or dysfunction of a selenoperoxidase, GPX4

241 accident or transient ischemic attack, renal insufficiency or failure, venous thromboembolism, pulmon

242 Conditional Myc haplo-insufficiency or overexpression combined with cell divis

243 persistent symptoms, signs of chronic venous insufficiency, or both, occurs in 25% to 50% of patients

244 ients (>=2 vascular beds affected, HF, renal insufficiency, or diabetes).

245 ion, pain control, replacement of pancreatic insufficiency, or mechanical drainage of obstructed panc

246 Premature ovarian insufficiency (POI) affects approximately 1% of women.

247 Premature ovarian insufficiency (POI) is a frequent long-term complication

248 Primary Ovarian Insufficiency (POI) is a major cause of infertility, but

249 Premature ovarian insufficiency (POI) is a significant complication of cyt

250 of obesity, type II diabetes mellitus, renal insufficiency, portal vein thrombosis, and poor performa

251 The folate insufficiency prevalence (RBC folate <748 nmol/L; NTD ri

252 We also measured exocrine insufficiency, quality of life (using the Short Form-36

253 omen have not decreased recently, and folate insufficiency rates are ~20%.

254 alies are partially due to right ventricular insufficiency, recent data support a mechanistic link to

255 onic pain, exocrine and endocrine pancreatic insufficiency, reduced quality of life, and a shorter li

256 ents with pre or posttransplant severe renal insufficiency remain more difficult to treat, due to mec

257 Convergence and accommodative insufficiency represent the main cause of complaints dur

258 microglial dysfunction caused by progranulin insufficiency represents a potential therapeutic strateg

259 tilation in 2 of 5 patients (40%), and renal insufficiency requiring hemodialysis in 2 of 5 patients

260 the specific mechanism by which maternal TH insufficiency results in this birth defect remains unkno

261 linked to KCNQ3 truncations arise from haplo-insufficiency should be reconsidered in some cases.

262 hemodynamic vulnerability indicators, renal insufficiency, single-ventricle physiology, and coagulat

263 tumor and can lead to exocrine and endocrine insufficiency such as malnutrition and diabetes mellitus

264 et diabetes, with subclinical or no exocrine insufficiency, suggesting an important role for GATA6 in

265 neuroplasticity in disorders of respiratory insufficiency suggests that membrane estrogen receptors

266 of Borsting et al's 2003 Revised Convergence-Insufficiency Symptom Survey (CISS) into a general schoo

267 Portuguese version of the CISS (Convergence Insufficiency Symptom Survey).

268 gional dynamic thoracic function in thoracic insufficiency syndrome (TIS) are limited.

269 Here, we have described a primary adrenal insufficiency syndrome and steroid-resistant nephrotic s

270 peration in pediatric patients with thoracic insufficiency syndrome, especially right lung volume wit

271 ed oxygen and nutrient supply with placental insufficiency that develop to slow hindlimb growth and m

272 iency anemia (IDA), a state of systemic iron insufficiency that limits the production of red blood ce

273 entae during pregnancy to identify placental insufficiency that may be indicative of local ischemia o

274 ystic fibrosis (CF) have pancreatic exocrine insufficiency that results in nutrient malabsorption and

275 ts dysfunction causes the plasma bicarbonate insufficiency that underlies acidemia.

276 ned mTORC1 signaling during simulated energy insufficiency that would otherwise restrict protein synt

277 inical and radiological PPCs and respiratory insufficiency therapies in a high-risk surgical populati

278 mong mothers with early and late prenatal VD insufficiency, those with early or late VD sufficiency (

279 ial fibrillation, hypophosphataemia, adrenal insufficiency, transaminitis, and infections.

280 With aging, retromer insufficiency triggers progressive endolysosomal dysfunc

281 une-related SAEs, including two with adrenal insufficiency, two with pneumonitis, and one with nephri

282 ly ascertained CHD events including coronary insufficiency/unstable angina, recognized myocardial inf

283 the hypothesis that maternal uteroplacental insufficiency (UPI) increases severity of retinopathy of

284 Ecological evidence suggests vitamin D insufficiency (VDI) due to lower ambient ultraviolet rad

285 oach to the induction of exocrine pancreatic insufficiency via pancreatic duct ligation in minipigs a

286 r type 2 diabetes not selected for vitamin D insufficiency, vitamin D(3) supplementation at a dose of

287 R) =1.860 (95% CI = 1.234-2.804), P = 0.003; insufficiency vs. sufficiency, aOR = 1.669 (95% CI = 1.2

288 Renal insufficiency was apparent at P21 when proteinuria prese

289 Exocrine pancreatic insufficiency was successfully induced in 12 Goettingen

290 When anastomotic insufficiency was suspected but no extravasation was pre

291 A noncardiac diagnosis and preexisting renal insufficiency were associated with increased odds of dea

292 Aortic bicuspidy, valvular stenosis, and insufficiency were considered as underlying valvulopathi

293 d had more often diabetes mellitus and renal insufficiency, whereas they had less often history of pr

294 poorly controlled in a host with global ATM insufficiency, whether the host is a mouse or a human.

295 humans, and is commonly caused by placental insufficiency, which results in fetal hypoxia.

296 patients, indicating extrahepatic vitamin K insufficiency, which was related to poor outcome while h

297 erforation of a hollow viscus or anastomotic insufficiency who had undergone OAT were included, and 1

298 and safety among patients with severe renal insufficiency, will continue to expand options for cure

299 ndomized Recompensation of Exacerbated Liver Insufficiency with Hyperbilirubinemia and/or Encephalopa

300 To determine how placental insufficiency with reduced oxygen and nutrient supply to