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1                                              Integrin alpha4 (alpha4) mediates stromal adhesion of no
2                   The same cells coexpressed integrin alpha4 and beta1, further supporting that macro
3                We propose that antibodies to integrin alpha4 and CD44 prevent clinical disease by par
4 ressed by scleroderma fibroblasts, including integrin alpha4 and integrin beta1.
5 n macrophages and adipocytes mediated by the integrin alpha4 and its counter-receptor VCAM-1, respect
6 oietic cells depends on the adhesion factors Integrin alpha4 and Vcam1 but is at least partly indepen
7 s study examined the role of leukocyte beta1 integrin (alpha4) and its endothelial ligand VCAM-1 in C
8 ytokine receptors, including integrin beta1, integrin alpha4, and CXC chemokine receptor 4 (CXCR4).
9       Two are shared among multiple tissues (integrin alpha4/annexin A4 and cathepsin B/apolipoprotei
10  of neutrophils to mindin is blocked by anti-integrin alpha4, anti-integrin alpha(M), and anti-integr
11                 Additional studies implicate integrins alpha4, beta3, and alphavbeta3 in the mechanis
12 ted with synthetic peptides that contain the integrin alpha4 binding site in fibronectin displayed ab
13 d T cell lines and clones expressed CD44 and integrin alpha4, but not L-selectin, and entered the CNS
14  surprisingly, Rh-alpha4beta7 did not impact integrin alpha4(+) cells, but decreased the frequencies
15  antibodies to MAdCAM-1, alpha4beta7, or the integrin alpha4 chain indicating that MAdCAM-1 in inflam
16    It was previously shown that mutations of integrin alpha4 chain sites, within putative EF-hand-typ
17  open new questions on the roles of sOpn and integrin alpha4 during homeostasis and inflammation.
18                            Thus, mutation of integrin alpha4 "EF-hand-like" sites may impair 1) stati
19            In HSPCs, BMP4 directly regulates Integrin-alpha4 expression through SMAD-independent p38
20 netic or pharmacological inactivation of the integrin alpha4 in mice resulted in elevated expression
21 show that association of integrin beta1 with integrin alpha4 is essential for this process.
22                        The neutralization of integrin alpha4 (Itga4) is currently used as treatment i
23  vitro prevented the culture-induced loss of Integrin-alpha4 (ITGA4) expression as well as homing.
24 ether, these data suggest that CXCR3 and the integrin alpha4 mediate T cell recruitment to uninfected
25 n situ hybridization demonstrates that chick integrin alpha4 mRNA is expressed at high levels by migr
26 served after integrin beta1 blockade but not integrin alpha4 or CXCR4 blockade.
27 togenic T cells in vitro and did not deplete integrin alpha4- or CD44-positive cells in vivo.
28               mAbs directed against CD44 and integrin alpha4 prevented the transfer of experimental a
29 livary glands depended on chemokines and the integrin alpha4 Several chemokines were expressed in the
30        To examine the functional role of the integrin alpha4 subunit in neural crest cell migration,
31                 Our results suggest that the integrin alpha4 subunit is important for normal neural c
32                             Furthermore, the integrin alpha4 subunit represents a useful neural crest
33 ers, we obtained a PCR fragment of the chick integrin alpha4 subunit that was subsequently used to cl
34       In addition to neural crest cells, the integrin alpha4 subunit was later observed on the muscle
35 lpha9 subunit is structurally similar to the integrin alpha4 subunit, and alpha9beta1 and alpha4beta1
36 ublished beta-propeller folding model of the integrin alpha4 subunit, in which seven four-stranded be
37 nce of a blocking antibody against the mouse integrin alpha4 subunit, there was a profound abrogation
38                        We now show here that integrin alpha4 tail deletion markedly impairs static ce
39 nce was truncated, replaced with that of the integrin alpha4, the integrin alpha2, or maintained inta
40               Mechanistically, Th1 cells use integrin alpha4 to adhere to and induce TGF-beta in CFB
41 oid cells deficient in the adhesion molecule integrin alpha4 were protected from necrotic core expans
42 ized mice bearing a Y991A mutation in alpha4 integrin [alpha4(Y991A) mice], which blocks paxillin bin