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1 n parallel with fibronectin and its receptor Integrin alpha5.
2 r kinases as potential downstream targets of integrin alpha5.
3 ase, becoming anti-correlated with levels of Integrin alpha5.
4 he ECM protein Fibronectin, and its receptor Integrin alpha5.
5 d disrupted the interaction between Cx43 and integrin alpha5.
6 pose its otherwise hidden FN synergy site to integrin alpha5.
7 s cell mobility through its interaction with integrin alpha5.
8 owed that IGFBP2 activates the expression of integrin alpha5.
9  we confirmed that IGFBP2 does interact with integrin alpha5.
10  D306E-IGFBP2 had no detectable binding with integrin alpha5.
11                     Inducible elimination of integrin alpha5 abrogates the epithelial-organizing effe
12  leucine rich repeat domain mutants restored integrin alpha5 abundance and directional cell migration
13 oper expression of focal adhesion kinase and integrin alpha5 adhesion receptor.
14 ut endothelial cells and markedly reduced in integrin alpha5/alphav double-knockout endothelial cell
15 for genetic interaction between mutations in integrin alpha5 and alphav and for overlapping functions
16  In vitro studies demonstrated that blocking integrin alpha5 and alphav during macrophage differentia
17  in 4T1 mammary tumors revealed differential integrin alpha5 and alphav expression and their associat
18 formation was inhibited by function-blocking integrin alpha5 and beta1 antibodies, suggesting the inv
19 ox D3 binds directly to the promoters of the integrin alpha5 and beta3 subunits, inducing subunit exp
20  uncoated or fibronectin-coated plastic, the integrin alpha5 and control (vector only) transfectants
21 on is promoted by cadherins in parallel with Integrin alpha5 and fibronectin, whereas negative feedba
22 ell mobility is through its interaction with integrin alpha5 and this interaction is specifically med
23                                              Integrin-alpha5 and integrin-beta1 subunits gave a simil
24                        Complexes formed with integrin-alpha5 and KCNB1 potassium channel wild type or
25 5), and 1.5-fold (P<0.05), respectively, but integrins alpha5 and beta5 increased 2.3-fold (P<0.01) a
26 EGF, IL-6, VEGF-C, HB-EGF, CTGF, tenascin C, integrin alpha5, and Eph receptor A2.
27 ted by FFSS directly phosphorylates Cx43 and integrin alpha5, and Ser-373 of Cx43 plays a predominant
28                                Both Cx43 and integrin alpha5 are directly phosphorylated by AKT.
29 transferase (GST) pulldown assays identified integrin alpha5 as a novel Scrib interacting protein.
30 the N-cadherin-p120 catenin complex excludes integrin alpha5 at the junctions to suppress local phosp
31  most prominent rat basophilic leukemia cell integrin (alpha5) avoids the patterned regions occupied
32 -Ala (RRETAWA) is a novel ligand peptide for integrin alpha5 beta1, which blocks alpha5 beta1-mediate
33 n is initiated by fibronectin binding to the integrins alpha5 beta1 and alphav beta3, and is complete
34         Neurosphere cells express five major integrins, alpha5 beta1, alpha 6Abeta1, alphav beta1, al
35                                Expression of integrins (alpha5, beta1, beta3, beta4 and beta5) was de
36 ng represents a potential mechanism by which integrin alpha5/beta1 exerts its tumor suppressor-like a
37                                              Integrin alpha5/beta1 expression is frequently lost in c
38 l epithelium, and colon cancer cells lacking integrin alpha5/beta1 expression utilize HER-2 signaling
39                             Re-expression of integrin alpha5/beta1 in colon cancer cells abrogated th
40                         Stable expression of integrin alpha5/beta1 in colon cancer cells with little
41 sults also suggest that a novel function for integrin alpha5/beta1 is the control of HER-2 expression
42                   These results suggest that integrin alpha5/beta1 mediates fibronectin-induced epith
43 on cancer cells with little or no detectable integrin alpha5/beta1 protein expression resulted in the
44 line Caco-2 to study EGF receptor (EGFR) and integrin alpha5/beta1 signaling interactions involved in
45                                              Integrin alpha5/beta1 was found to interact with HER-2,
46 ct with HER-2, and the cytoplasmic domain of integrin alpha5/beta1 was sufficient to mediate HER-2 do
47 d secretion of fibronectin and expression of integrin alpha5/beta1, the principal fibronectin recepto
48 y was necessary for both MAPK activation and integrin alpha5/beta1-mediated cell proliferation.
49                                              Integrin alpha5/beta1-mediated down-regulation of HER-2
50 man integrin alpha5 was transfected into the integrin alpha5/beta1-negative intestinal epithelial cel
51                      Decreased expression of integrin alpha5 by small interference RNA in IGFBP2-over
52  as well as endothelial-specific deletion of integrin alpha5, causes barrier leakage in mice.
53 ar endothelial growth factor receptor-2, and integrin alpha5 (CD49e).
54                              Simultaneously, Integrin alpha5 clusters and adopts the active conformat
55 on fluorescence (TIRF) microscopy, Scrib and integrin alpha5 colocalize at the basal plasma membrane
56 ave similar phenotypes, although, defects in integrin alpha5-deficient mice are milder.
57 r, in contrast to fibronectin, the ligand of integrin alpha5, directional migration on collagen media
58    Moreover, AKT phosphorylation on Cx43 and integrin alpha5 enhanced their interaction.
59 ling, visceral endoderm survival, as well as integrin alpha5 expression and its downstream signaling
60                                Concurrently, integrin alpha5 expression was enhanced but not integrin
61         VSMC adhesion force to FN (+33%) and integrin alpha5 expression were enhanced but COL1 adhesi
62                              We generated an integrin alpha5-floxed mouse line and ablated alpha5 int
63   Analysis of the intracellular mediators of integrin alpha5/Fn1 activity focal adhesion kinase (FAK)
64                                      Loss of integrin alpha5/Fn1 does not affect the fate or viabilit
65 nnel subfamily B member 1 (KCNB1, Kv2.1) and integrin-alpha5 form macromolecular complexes-named inte
66 expectedly, endothelial-specific knockout of integrin alpha5 has no obvious effect on developmental a
67                    The angiogenic effects of integrin alpha5 in macrophages involve upregulation of V
68 Mesp1(Cre) knock-in strain of mice to ablate integrin alpha5 in the anterior mesoderm, which gives ri
69 atin 8 expression and reduced Twist1-induced integrin alpha5, integrin beta1 and MMP9 expression.
70         We have recently shown that Cx43 and integrin alpha5 interact directly with each other, and a
71 ay results by showing that the expression of integrin alpha5 is indeed up-regulated at the protein le
72                          Here, we found that integrin alpha5 (ITGA5) was highly expressed in bone met
73 d encapsulated it into our recently-reported integrin alpha5(ITGA5) active targeting nanoparticles (u
74                                              Integrin alpha5 (Itgalpha5) and alphaV (ItgalphaV) are t
75 Cdh2 acts with the fibronectin (FN) receptor integrin alpha5 (Itgalpha5) to promote somite boundary f
76 at fibronectin1 (Fn1), an ECM component, and integrin alpha5, its cellular binding partner, are requi
77 n-alpha5 form macromolecular complexes-named integrin-alpha5-KCNB1 complexes (IKCs)-in the human brai
78 fibronectin assembly is somewhat affected in integrin alpha5 knockout endothelial cells and markedly
79 e that myeloid cell- and macrophage-specific integrin alpha5 knockout mice have accentuated adverse p
80 ly relevant role for TbetaRIII in regulating integrin alpha5 localization, reveal a novel crosstalk m
81      In the normal human colon, we show that integrin alpha5 localizes to the lateral membrane of ter
82 nd migrate into the heart in fibronectin- or integrin alpha5-mutant embryos, however, the hearts in t
83      Myoblasts ectopically expressing alpha5 integrin (alpha5 myoblasts) move normally when not in co
84                                              Integrin alpha5-null embryos die in mid-gestation from s
85 chyme reveals a physical association between Integrin alpha5 on adjacent cell membranes.
86                    Ectopic expression of the integrin alpha5 or alpha6A subunit in primary quail myob
87 e its ligand, cells were plated on mAbs anti-integrin alpha5 or anti-integrin alpha3.
88                 Here we show that macrophage integrin alpha5 protects the infarcted heart from advers
89  D3 expression in endothelial cells enhances integrin alpha5 protein and message expression, whereas
90        Whereas eCRT strongly induces ECM and integrin alpha5 proteins in K41 wild-type mouse embryo f
91            Thus, concurrent re-expression of integrin-alpha5, radixin, and RhoA abrogates miR-31-impo
92  explained by miR-31-mediated suppression of integrin-alpha5, radixin, and RhoA.
93 are abrogated by concurrent re-expression of integrin-alpha5, radixin, and RhoA.
94             Mechanistically, Scrib supported integrin alpha5 recycling and protein stability by block
95                     Furthermore, vitronectin-integrin alpha5 signaling maintains barrier integrity by
96 rminally differentiated colonocytes and that integrin alpha5 staining may be reduced in colorectal ca
97 s demonstrate that the angiogenic actions of integrin alpha5-stimulated macrophages involve activatio
98 tegrin through this motif, and silencing the integrin alpha5 subunit abolishes the Vi-mediated inhibi
99                        Overexpression of the integrin alpha5 subunit in RIE1 cells conferred protecti
100 ferentially to the cytoplasmic domain of the integrin alpha5 subunit, inhibits cell motility, and alt
101 in the presence of an inhibitory antibody to integrin alpha5 subunit.
102 eport that deletion of the gene encoding the integrin-alpha5 subunit (Itga5) using the Pdgfrb-Cre tra
103  neutralizing antibody to either TGF-beta or integrin alpha5, this increased basal promoter activity
104  confirm that IGFBP2 interacts directly with integrin alpha5 through the RGD domain, we created an RG
105 upport a model in which lens fiber cells use integrin alpha5 to migrate along a Fn-containing substra
106 esence of the EGFR antagonistic mAb 225, the integrin alpha5 transfectants and controls were signific
107 ted growth inhibition on fibronectin for the integrin alpha5 transfectants correlated with activation
108   However, when cultured on fibronectin, the integrin alpha5 transfectants were not growth inhibited
109 R with SHC could be demonstrated only in the integrin alpha5-transfected cells.
110 ugh EGFR activation occurred when either the integrin alpha5-transfected or control cells were cultur
111                Scrib is a novel regulator of integrin alpha5 turnover and sorting, which is required
112                                        Human integrin alpha5 was transfected into the integrin alpha5
113 ed, and discovered that both fibronectin and integrin alpha5 were required for cardiac morphogenesis,
114 the protein amount and surface expression of integrin alpha5 whereas surface expression of integrin a
115 e mediated in part by fibronectin binding to integrin alpha5, which recruits and activates phosphodie
116 egulation of TGF-beta1, fibronectin (Fn) and integrin alpha5, which was associated with decreased vis

 
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