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1 the gene, NXF1 binds to BR mRNPs only in the interchromatin.
2 nger RNA protein complexes (mRNPs) enter the interchromatin.
3 teady state, a subset of the BR mRNPs in the interchromatin binds NXF1, UPF2, and UPF3.
4 restricts viral capsid diffusion, but due to interchromatin channels capsids are able to reach the nu
5 leus with chromosome territories pervaded by interchromatin channels.
6 ovement of large macromolecular complexes to interchromatin corrals.
7 rent herpesviruses induced an enlargement of interchromatin domains and allowed particles to diffuse
8 pre-mRNA splicing machinery are localized in interchromatin granule clusters (IGCs) and perichromatin
9                To examine the involvement of interchromatin granule clusters (IGCs) in transcription
10 n on nuclear structures known as speckles or interchromatin granule clusters (IGCs) revealed by immun
11 SK RNA to be enriched in nuclear speckles or interchromatin granule clusters (IGCs), a subnuclear dom
12 lized to the nucleus and was concentrated in interchromatin granule clusters (IGCs), sites where spli
13 bition results in accumulation of DEK within interchromatin granule clusters (IGCs), sub-nuclear stru
14 stribution that predominantly corresponds to interchromatin granule clusters (IGCs).
15 roviral cyclin (RV-cyclin) that localizes to interchromatin granule clusters and binds CDK8.
16       Furthermore, Dfa(A) was present in the interchromatin granule clusters and was also found to bi
17 d, most of which are accumulated in enlarged interchromatin granule clusters in the nucleoplasm of RC
18 rs, P-TEFb and 7SK snRNA, assembled in large interchromatin granule clusters near the provirus within
19 ctural level, perispeckles are distinct from interchromatin granule clusters that may function as sto
20                         Nuclear speckles, or interchromatin granule clusters, are enriched in factors
21 d concentration of the IL-1alpha propiece in interchromatin granule clusters, concentrations of prote
22 s revealed that these speckles correspond to interchromatin granule clusters.
23 splicing factors and poly(A)+ RNA were fused interchromatin granule clusters.
24 sembles, but is functionally different from, interchromatin granule clusters.
25 wn structures such as the coiled body or the interchromatin granule.
26 rol genes between Polycomb bodies (PcGs) and interchromatin granules (ICGs) in response to growth sig
27 th endogenous cytoplasmic assemblies-mitotic interchromatin granules and cytoplasmic speckles (CSs)-w
28 teractions responsible for the clustering of interchromatin granules are disrupted when SR proteins a
29  30-nm particles, which closely resemble the interchromatin granules described from sections of somat
30 lap with structures previously identified as interchromatin granules or spliceosomal speckles.
31 lizes to punctate structures consistent with interchromatin granules that show a striking adjacent-lo
32 nterchromosomal interaction loci to distinct interchromatin granules, long thought to be "storage" si
33                ACBs are derived from mitotic interchromatin granules, transient mitotic structures wh
34 ch we have shown previously to coincide with interchromatin granules.
35 r, nascent RNA synthesis was observed in the interchromatin regions of the cells studied and spatiall
36                                          The interchromatin space is composed of nuclear subcompartme
37   Poly(A) RNA can move freely throughout the interchromatin space of the nucleus with properties char
38 oelastic properties and accessibility of the interchromatin space remain constant.
39 tion of DSB-flanking chromatin, reduction in interchromatin space, aberrant spreading of DNA repair p
40  Our ability to measure large strains in the interchromatin space, which reveals that movement of chr
41 aments coursing dendritically throughout the interchromatin space?
42 mplexes is restricted to the extranucleolar, interchromatin spaces.
43 ions, and their transcripts frequently share interchromatin trafficking channels.