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1 ion of long-lived plasma cells, reduction of interferon type I activity, and down-regulation of T-cel
3 wing of TCR repertoires were associated with interferon type I and III responses, early CD4(+) and CD
4 an increase in the constitutive activity of interferon type I and NF-kappaB pathways and an elevated
5 triggering Interferon Regulating Factor -3, interferon type I and proinflammatory cascades previousl
7 influenza A virus but are also inducible by interferons type I and II and are critical for interfero
8 us resistance A) versus SIN3A, a promoter of interferon type I-based proinflammatory signaling, were
9 espectively), and differential expression of interferon type I-driven antiviral protein MxA (myxoviru
10 own-regulate the transcriptional activity of interferon type I gene promoters in infected cells by in
12 G) pathway activation, which produces Type I Interferons (Type I IFN) critical for innate and adaptiv
13 g microglial phenotype is largely imposed by interferon type I (IFN-I) chronically present in aged br
15 ion of endogenous transposons and subsequent interferon type-I (IFN-I) response, show limited clinica
16 Signal transduction through receptors for interferons Type I (IFN-alpha, IFN-beta, IFN-omega) and
17 formed in susceptible cells that were either interferon type I/III (IFN-I/-III) nonresponsive or IFN-
18 not inhibited by blocking antibodies against interferon type I or type II, which, however, induced ID
19 at mice with a targeted null mutation in the interferon type I receptor (IFN-RI), which cannot respon
21 oxide synthase (NOS), inducible NOS, and the interferon type I receptor were similar in KO and hetero
23 priming depend not only on TLR9, but also on interferon type I signaling, and both mechanisms can be