ライフサイエンスコーパス: interferon-gamma

1 es (interleukin-17A, TNF, interleukin-6, and interferon-gamma).

2 eins, which are upregulated upon exposure to interferon gamma.

3 cells that express tumor necrosis factor and interferon gamma.

4 odeling, the JAK-STAT signaling pathway, and interferon gamma.

5 wing stimulation with lipopolysaccharide and interferon gamma.

6 iated with higher baseline concentrations of interferon gamma.

7 pha, interleukin 1beta (IL-1beta), IL-6, and interferon gamma.

8 pression of IRF8 that was further induced by interferon-gamma.

9 rophages by limiting the effects of systemic interferon-gamma.

10 n of interleukin-4 and reduced production of interferon-gamma.

11 ic islets treated with interleukin-1beta and interferon-gamma.

12 o-cultures and in vivo, in part by secreting interferon-gamma.

13 y-stimulating factor and monokine induced by interferon-gamma.

14 and 1 and IL-10, and decreased expression of interferon-gamma.

15 on factor and its expression is augmented by interferon-gamma.

16 filtrating T-cells expressing granzyme B and interferon gamma-1.

17 [3.1%-18.2%]; P < .05), reduced CD4+ T-cell interferon-gamma (21% [0.5%-28%]; P < .05) and granzyme

18 al epithelial cells (HCE-T cells) exposed to interferon-gamma, a cytokine elevated in SS, showed up t

19 Rather, elimination of type I cytokine interferon-gamma activity enhanced insulin sensitivity i

20 CD8(+) T-cell production of interferon gamma after stimulation with HDV peptides cor

21 in (IL)-2 (P = .004), but similar amounts of interferon-gamma, after stimulation with tetanus toxoid.

22 gamma receptor 1, which mediates response to interferon gamma, an essential molecule for T-cell-media

23 programmed death-1 expression and defects in interferon gamma and antigen presentation pathway compon

24 differentiation capacity and expressed less interferon gamma and interleukin 17 after polyclonal sti

25 Bs levels and cells producing HBsAg-specific interferon gamma and interleukin 2 (T-helper 1-type cyto

26 th acute infection and reduced the levels of interferon gamma and transforming growth factor beta (TG

27 reduced production of the OL toxic molecules interferon-gamma and chemokine (C-X-C motif) ligand, CXC

28 s normal skin showed increased expression of interferon-gamma and chemokines that attract T cells and

29 mory phenotype and were capable of producing interferon-gamma and destroying target cells ex vivo or

30 -1beta (IL-1beta), IL-6, IL-8, IL-10, IL-17, interferon-gamma and differential T and B cell subset ly

31 n of interleukin (IL)-12 depends strictly on interferon-gamma and is controlled negatively by IL-4 si

32 or Cxcr3 and produce both the T(H)1 cytokine interferon-gamma and the T(FH)-associated cytokine inter

33 on of a Th1-biased response characterized by interferon-gamma and tumor necrosis factor-alpha cytokin

34 Resident splenocytes show higher interferon-gamma and tumor necrosis factor-alpha secreti

35 cells was assessed by release of cytokines (interferon-gamma and tumor necrosis factor-alpha), upreg

36 g CEnCs against effector T cell-mediated and interferon-gamma and tumor necrosis factor-alpha-induced

37 levels of interleukin-10, interleukin-12p40, interferon-gamma and tumour necrosis factor-alpha.

38 ]-21, IL-1beta, tumor necrosis factor-alpha, interferon-gamma) and CCR5, HLA-DR, CD38, and Th17 frequ

39 of antigen-specific T-helper 1 (IL-2, IL-12, interferon-gamma) and T-helper 2 (IL-4, IL-5) cytokines

40 d decreased cytokine (ie, interleukin 1beta, interferon gamma, and interleukin 17) responses when med

41 o production of tumor necrosis factor alpha, interferon gamma, and interleukin 1beta but increased ph

42 significantly elevated levels of eotaxin-1, interferon-gamma, and IL-12p70 relative to children with

43 T cells in old brains also express interferon-gamma, and the subset of neural stem cells th

44 antigen-specific B-cell, and interleukin 2-, interferon gamma-, and tumor necrosis factor alpha-secre

45 icacy and safety of emapalumab (a human anti-interferon-gamma antibody), administered with dexamethas

46 vere disease) and 16 unexposed donors, using interferon-gamma-based assays with peptides spanning SAR

47 Infusion of maternal VSTs, isolated using interferon-gamma capture, associated with clinical impro

48 Incubation of BE cells with interferon gamma caused them to increase expression of I

49 Frequencies of interferon-gamma +CD4+T cells expressing CD38, HLADR, an

50 Three Tax(+)/interferon-gamma(-/-) cell lines were generated for in v

51 increased motility, and higher production of interferon gamma, compared with T cells from unirradiate

52 s infection causes intestinal pathology, and interferon-gamma controls early infection while T cells

53 yte-macrophage colony-stimulating factor and interferon-gamma) critical to the cytokine storm that am

54 This engraftment led to interferon-gamma-dependent functional changes in the poo

55 hils were essential for the activation of an interferon-gamma-dependent pathway of immune resistance,

56 Serum interferon-gamma elevation (P < .001) and possibly inter

57 lular and humoral responses were measured by interferon-gamma ELISPOT and virus neutralization assay

58 after transplantation was assessed using an interferon gamma enzyme-linked immunosorbent spot assay.

59 We used interferon-gamma enzyme-linked immune absorbent spot ana

60 lar responses were assessed using an ex-vivo interferon-gamma enzyme-linked immunospot assay.

61 D11c(+) macrophage) M1-like polarization and interferon-gamma-expressing T-helper type 1 (Th1) cells

62 afin improved leptin sensitivity via reduced interferon-gamma expression and induced adipose leptin e

63 lis lipopolysaccharide (PgLPS), Pam3CSK4, or interferon-gamma for 48 hours.

64 hrough the rapid synthesis of a glycosylated interferon-gamma fragment and the chemokine-binding prot

65 ssion of PD-L1, total mutation burden, or an interferon-gamma gene expression signature.

66 NMP also reduced the number of interferon gamma (IFN-gamma) and interleukin (IL)-17-pro

67 ccinated infants had increased production of interferon gamma (IFN-gamma) and monokine induced by gam

68 T helper 1 (Th1) cells producing interferon gamma (IFN-gamma) and Th17 cells producing in

69 the absence of miR-155, there was decreased interferon gamma (IFN-gamma) and tumor necrosis factor a

70 CD4(+) T helper 1 (Th1) cells producing interferon gamma (IFN-gamma) are critical for the resolu

71 nters underwent ELISPOT testing to enumerate interferon gamma (IFN-gamma) binding spot-forming units

72 higher levels of interleukin-12 (IL-12) and interferon gamma (IFN-gamma) by as early as 4 hpi, but t

73 The mechanisms by which interferon gamma (IFN-gamma) controls the replication of

74 t viruses expressing interleukin-4 (IL-4) or interferon gamma (IFN-gamma) did not.

75 Inborn errors of human interferon gamma (IFN-gamma) immunity underlie mycobacte

76 Interferon gamma (IFN-gamma) is a potent cytokine produc

77 and evaluated CAR-T cell activation through interferon gamma (IFN-gamma) production and CD107a membr

78 g superantigen-induced T cell activation and interferon gamma (IFN-gamma) production during infection

79 ity (CMV-CMI) can be determined by levels of interferon gamma (IFN-gamma) production using an enzyme-

80 Interferon gamma (IFN-gamma), critical for host defense

81 at influenza diagnosis and 28 days later for interferon gamma (IFN-gamma), IL-4, IL-13, and IL-10.

82 Furthermore, we quantified the number of interferon gamma (IFN-gamma)-producing CD4 T cells speci

83 Adhesin domain immunization also elicited interferon gamma (IFN-gamma)-producing CD8-positive (CD8

84 ound that early intratumoral accumulation of interferon gamma (IFN-gamma)-producing natural killer (N

85 generation of anti-viral cytokines including interferon gamma (IFN-gamma).

86 nsforming growth factor beta (TGF-beta), and interferon gamma (IFN-gamma).

87 h reduced liver MDSC accumulation, increased interferon-gamma (IFN-gamma) and granzyme B production i

88 Although viral infections elicit robust interferon-gamma (IFN-gamma) and long-lived antibody-sec

89 Activated ILC1s produced interferon-gamma (IFN-gamma) and protected mice from CCl

90 vels of interleukin (IL)-1beta, IL-6, IL-17, interferon-gamma (IFN-gamma) and superoxide dismutase 1

91 -22), whereas ILC1s produced proinflammatory interferon-gamma (IFN-gamma) and tumor necrosis factor-a

92 Multiple type I interferons and interferon-gamma (IFN-gamma) are expressed under physiol

93 The natural history of anti-interferon-gamma (IFN-gamma) autoantibody-associated imm

94 While clinical manifestations of anti-interferon-gamma (IFN-gamma) autoantibody-associated imm

95 was needed to prevent premature induction of interferon-gamma (IFN-gamma) expression in T cells and t

96 The proinflammatory cytokine interferon-gamma (IFN-gamma) has been implicated in huma

97 In contrast to the importance of type II interferon-gamma (IFN-gamma) in control of toxoplasmosis

98 in vitro infectivity, and are infectious to interferon-gamma (IFN-gamma) knockout mice.

99 In this work, we designed a conjugated anti-interferon-gamma (IFN-gamma) molecular aptamer beacon (M

100 Interferon-gamma (IFN-gamma) plays an important role in

101 decreases interleukin (IL)-12/IL-18-induced interferon-gamma (IFN-gamma) production versus controls.

102 While T-bet ablation restricted interferon-gamma (IFN-gamma) production, loss of Blimp-1

103 cell lymphoma-bearing mice displayed reduced interferon-gamma (IFN-gamma) production.

104 a key antitumor function of CD4(+) T cells, interferon-gamma (IFN-gamma) production.

105 dity NK cells possessed greater capacity for interferon-gamma (IFN-gamma) production.

106 specific T cells expressed higher amounts of interferon-gamma (IFN-gamma) receptor and were more susc

107 study of long-term T-cell responses using an interferon-gamma (IFN-gamma) release assay and a flow cy

108 dentified using the tuberculin skin test and interferon-gamma (IFN-gamma) release assay IGRA, and a p

109 el vaccines against tuberculosis (TB) and in interferon-gamma (IFN-gamma) release assays (IGRAs).

110 and death receptor ligand TRAIL, as well as interferon-gamma (IFN-gamma) secretion, was more pronoun

111 d from WNV or ZIKV infection, T cell-derived interferon-gamma (IFN-gamma) signaling in microglia unde

112 We find that T cell infiltration and interferon-gamma (IFN-gamma) signaling signatures corres

113 actor Elf5 in TNBC cells activates intrinsic interferon-gamma (IFN-gamma) signalling, promoting tumou

114 cebo in the median fold increase in secreted interferon-gamma (IFN-gamma) was 38.2-fold (95% CI, 4.7-

115 ference was observed regarding the number of interferon-gamma (IFN-gamma)+ cells in lesions from preg

116 A polymerase II pause release in response to interferon-gamma (IFN-gamma), a universal cytokine invol

117 -alpha), interleukin-6 (IL-6), IL-1beta, and interferon-gamma (IFN-gamma), as well as reduces cell ap

118 d with a significant increase in gB-elicited interferon-gamma (IFN-gamma), granzyme B, and CD107a and

119 t study evaluated the in vitro production of interferon-gamma (IFN-gamma), interleukin (IL)-6, and IL

120 that increased expression of factors such as interferon-gamma (IFN-gamma), vascular endothelial growt

121 ulin G1 monoclonal antibody directed against interferon-gamma (IFN-gamma), which in November 2018 rec

122 onstrate that Ct growth inhibition occurs by interferon-gamma (IFN-gamma)-mediated depletion of intra

123 produce proinflammatory cytokines, including interferon-gamma (IFN-gamma).

124 in-4 (IL-4), but not the T helper 1 cytokine interferon-gamma (IFN-gamma).

125 8R2-deficient mice contained more IL-17A and interferon-gamma (IFN-gamma).

126 ation, we find inflammatory cytokines (e.g., interferon gamma [IFN-gamma] and tumor necrosis factor a

127 med macrophage polarization by suppressing M(interferon-gamma [IFN-gamma]) yet promoting M(interleuki

128 major resident retinal cell types respond to interferon gamma (IFNG) by changing their patterns of ge

129 Interferon-gamma (IFNG) augments immune function yet pro

130 logy was largely rescued by CD8 depletion or interferon-gamma (IFNg) blockade.

131 tors and activated by stress signals such as interferon gamma (IFNgamma) and hypoxia.

132 nockdown of FTO sensitizes melanoma cells to interferon gamma (IFNgamma) and sensitizes melanoma to a

133 is vital, inflammatory cytokines, including interferon gamma (IFNgamma) and TNFalpha, also facilitat

134 Interferon gamma (IFNgamma) elicits a variety of anti-To

135 The mice were treated with interferon gamma (IFNgamma) intraperitoneally for 3 d or

136 nes involved in antigen presentation and the interferon gamma (IFNgamma) pathway play an important ro

137 Mechanistically, interferon gamma (IFNgamma) released from CD8(+) T cells

138 The frequency of PPD-specific interferon gamma (IFNgamma) secreting cells (SFU) were c

139 ns (P < 0.01) were observed between elevated interferon gamma (IFNgamma)-expressing T cytotoxic LPLs

140 ture of the establishment and maintenance of interferon gamma (IFNgamma)-induced priming of human cel

141 t manner, which was enhanced by priming with interferon gamma (IFNgamma).

142 We also demonstrate a role for the cytokine interferon-gamma (IFNgamma) and the enzyme transglutamin

143 tors for the lymphotoxin-alpha (LTalpha) and interferon-gamma (IFNgamma) genes.

144 led to a reduction in interleukin (IL)-4 and interferon-gamma (IFNgamma) in the diencephalon, as well

145 The cytokine interferon-gamma (IFNgamma) is a central coordinator of

146 Blocking of interferon-gamma (IFNgamma) reduced the transmigration p

147 d by high levels of infiltrating T cells and interferon-gamma (IFNgamma) signalling, improves the res

148 hrough the antiviral effects of antibody and interferon-gamma (IFNgamma), but RNA persists.

149 The expression of interferon-gamma (IFNgamma), indoleamine 2,3 dioxygenase

150 increases in CSF white blood cells, protein, interferon-gamma (IFNgamma), interleukin (IL)-6, IL-10,

151 reduced secretion of interleukin-2 (IL2) and interferon-gamma (IFNgamma), two factors critical for T

152 cells suppressed CD8(+) T cell secretion of interferon-gamma (IFNgamma), which would otherwise block

153 r maintaining cell fitness after exposure to interferon-gamma (IFNgamma).

154 y tumor necrosis factor alpha (TNFalpha) and interferon-gamma (IFNgamma).

155 ased STAT activation in vitro in response to interferon-gamma, IL-2 and IL-4 that is reverted by the

156 d with PLX5622, whereas other cytokines (eg, interferon-gamma, IL-4, and IL-10) were reduced.

157 PSGL-1 is induced by interferon-gamma in activated CD4(+) T cells to inhibit

158 ction of higher (p < 0.01) concentrations of interferon-gamma in plasma of low responders compared to

159 gnificantly abolish the anti-HIV activity of interferon-gamma in primary CD4(+) T cells.

160 22 convert into ILC1-like cells that produce interferon-gamma in vitro, but whether this conversion o

161 Furthermore, interferon-gamma increased the transcytosis of dimeric I

162 l as significantly reduced the expression of interferon-gamma induced by F. nucleatum.

163 Furthermore, ex vivo stimulation with interferon-gamma induced endogenous PD-L1 expression and

164 a, monocyte chemoattractant protein (MCP)-1, interferon gamma-induced protein (IP)-10, monokine induc

165 L-2Ralpha), sCD27, B-cell activating factor, interferon gamma-induced protein 10 (IP-10), soluble int

166 very high CSF levels of the pro-inflammatory interferon gamma-induced protein 10 (IP-10/CXCL10) in HI

167 L)-6, IL-8, tumor necrosis factor alpha, and interferon gamma-induced protein 10 were measured under

168 , interleukin-10, interleukin-15, eotaxin-3, interferon gamma-induced protein 10, macrophage-derived

169 -1beta]/C-C motif chemokine ligand 4 [CCL4], interferon gamma-induced protein-10 [IP-10]/C-X-C motif

170 Interferon-gamma induces FLT3L expression in UC-MSCs thr

171 (GM-CSF) (6.6-fold), RANTES (14.8-fold), and interferon gamma inducible protein 10 kDa (IP-10) (53-fo

172 entified polymorphisms in inflammasome genes interferon gamma inducible protein 16 (IFI16) and absent

173 s associated with HIV acquisition, including interferon-gamma inducible protein (IP)-10, macrophage i

174 -reactive protein (CRP), interleukin (IL)-6, interferon-gamma inducible protein (IP)-10, soluble CD14

175 The interferon gamma-inducible protein 16 (IFI16) is known a

176 and HIN domain family member (PYHIN) protein interferon-gamma-inducible protein 16 (IFI16) detect DNA

177 responses, especially T-cell cytokines (ie, interferon gamma, interleukin 17, and interleukin 22).

178 After dMNP delivery of AFV, interferon gamma, interleukin 2, and interleukin 4 produ

179 00 cells/muL had higher median CSF levels of interferon-gamma, interleukin (IL)-6, IL-8, and IL-13, a

180 nd neutralizing) and CD4+ T-cell (expressing interferon-gamma, interleukin-2, and CD40 ligand) respon

181 osporin A (CSA), corticosteroids, dupilumab, interferon-gamma, intravenous immunoglobulins (IVIG), me

182 response characterized by the production of interferon-gamma is needed.

183 opeptidome response to CDK4/6 inhibition and interferon-gamma - known modulators of antigen presentat

184 unced disease, and correlates with increased interferon-gamma levels in the lungs and spleens before

185 Interferon-gamma levels of endotoxin-stimulated PBMCs fr

186 ector memory T cells and diminished systemic interferon-gamma levels only in young recipients.

187 Furthermore, plasma interferon-gamma levels were reduced in smokers compared

188 Interleukin-1b and interferon-gamma levels were upregulated in skin of allo

189 2-NBDG(lo)) and decreased effector function (interferon gamma(lo)).

190 partments and polarize naive T cells into an interferon-gamma(low), interleukin-4(high) and FoxP3(+)

191 Tax(+) and Tax(+)/interferon-gamma(-/-) malignancies of the ear, tail, and

192 We demonstrate that DUX4 expression blocks interferon-gamma-mediated induction of MHC class I, impl

193 malignancies, wild-type, Tax(+), and Tax(+)/interferon-gamma(-/-) mice were assessed using necropsy,

194 ir wild-type littermates and lower levels of interferon-gamma mRNA were detected in the infected muco

195 elated with soluble inflammatory biomarkers (interferon-gamma, myeloperoxidase, tumor necrosis factor

196 ion and are abolished by T cell depletion or interferon-gamma neutralization.

197 -derived T cells, and can be counteracted by interferon-gamma or checkpoint blockade, respectively.

198 including IL-12, eotaxin, RANTES, IL-10 and interferon-gamma (p < 0.05).

199 r levels of IL-6 (P = .02), IL-10 (P = .02), interferon-gamma (P = .003), TNF-alpha (P = .006), IL-1b

200 via the DNA damage signalling and neoantigen-interferon-gamma pathway under oxidative stress.

201 AK-STAT, immune deficiency and cross-species interferon-gamma pathways, have advanced our understandi

202 ) T cell and CD68(+) macrophages), cytokine (interferon gamma-positive [IFN-gamma(+)] and tumor necro

203 8 [5%, 1.9-10.7]) and T-cell responses (CD4+ interferon gamma-positive and/or CD4+ interleukin 2-posi

204 otection was associated with a strong CD8(+) interferon gamma-positive recall response against NS4.

205 reduced in smokers compared with nonsmokers (interferon-gamma-positive rate, 14.9% versus 28.7%; P <

206 IFN-gamma (interferon-gamma) primes EC responsiveness to MAC by inc

207 Interferon gamma produced by activated ILC1 is critical

208 t increased the number of interleukin 4- and interferon gamma-producing natural killer T cells in the

209 inally identified as a receptor expressed on interferon-gamma-producing CD4(+) and CD8(+) T cells.

210 s and a modest reduction in the frequency of interferon-gamma-producing CD4(+) and CD8(+) T cells.

211 faeces that is capable of robustly inducing interferon-gamma-producing CD8 T cells in the intestine.

212 Last, frequencies of interferon-gamma-producing EBV-specific CD4 T cells were

213 with a reduction of CD11c cells, CD4 TN, and interferon-gamma-producing EBV-specific CD4 T cells, sug

214 he transcription factor TCF1, which promotes interferon-gamma-producing gammadelta T cells (Tgammadel

215 HEV-specific interferon-gamma-producing T cells were 2-fold higher in

216 panied by the expansion of gluten-sensitive, interferon-gamma-producing Vdelta1(+) IELs bearing T cel

217 ysis and intracellular cytokine staining for interferon gamma production at the single-peptide level.

218 regulation of CCL20 expression and increases interferon gamma production by Th1 cells.

219 reserved HSPC-NK killing, proliferation, and interferon gamma production capacity, whereas AZA dimini

220 phenotype profile associated with decreased interferon gamma production seems similar to those of ot

221 CCR5 expression), and decreased capacity of interferon gamma production were observed; mean perforin

222 ML), which displayed significantly increased interferon gamma production, degranulation, and specific

223 T cell (iTreg) differentiation and inhibited interferon-gamma production after allo-BMT.

224 accompanied by profound local suppression of interferon-gamma production and cytotoxic function of lu

225 CD8(+) T cells showed reduced interferon-gamma production and reduced expression of pe

226 NY-eso-1-SLL-specific T-cell clone provoked interferon-gamma production and/or cytolysis upon stimul

227 (VL) caused by Leishmania donovani requires interferon-gamma production by CD4+ T cells.

228 pinal cord injury infected mice and enhanced interferon-gamma production by natural killer T cells as

229 a normal adaptive NK phenotype and enhances interferon-gamma production by this cell subset.

230 the compounds to stimulate proliferation and interferon-gamma production of Vgamma9Vdelta2 T cells.

231 ence (CMV-CD8+), as measured by enumeration, interferon-gamma production, and CD107a/b degranulation.

232 e, spike, and nucleocapsid peptides elicited interferon-gamma production, in 27 (59%), 12 (26%), and

233 (+) T cells as measured by interleukin-2 and interferon-gamma production, respectively.

234 KO-CAR-T cells with strong activation (CD25, interferon gamma), proliferation, and specific killing u

235 tumor demonstrated a frameshift deletion in interferon gamma receptor 1, which mediates response to

236 colitis and lethal hemorrhagic pneumonia in interferon gamma receptor-deficient (IFNgammaR(-/-)) mic

237 ding ICAM-1, VCAM-1, HLA class I and II, and interferon gamma receptor.

238 mice lacking the pro-inflammatory caspase 1, interferon gamma-receptor, and nitric oxide synthase (Ca

239 associated with upregulation of T cell- and interferon-gamma-related gene expression, but downregula

240 itative polymerase chain reaction (PCR), and interferon gamma release (IGRA) assays.

241 .Methods People with a positive skin test or interferon gamma release assay (IGRA) result from 1985 t

242 TBI) testing (tuberculin skin test [TST] and interferon gamma release assay [IGRA]), and human immuno

243 computed tomography [CT calcification], and interferon gamma release assay for tuberculosis [TB-IGRA

244 of tuberculosis infection, as assessed by an interferon gamma release assay in the fourth year, among

245 Studies comparing the performance of interferon gamma release assays (IGRAs) and the tubercul

246 Plus (QFT-Plus) is the latest generation of interferon gamma release assays (IGRAs) to receive appro

247 By comparison, peripheral blood interferon gamma release assays in the same cohort achie

248 c TB comparable to that of Xpert MTB/RIF and interferon gamma release assays.

249 umbers of individuals who could be tested by interferon-gamma release assay (IGRA) and treated for LT

250 with smear-positive tuberculosis (TB) had an interferon-gamma release assay (IGRA) at baseline and 14

251 usehold contacts of TB cases were tested for interferon-gamma release assay (IGRA) conversion between

252 investigated feasibility and accuracy of an interferon-gamma release assay (IGRA) for detection of T

253 pared the results to the use of the existing interferon-gamma release assay (IGRA).

254 ecific cell-mediated immunity (CMI) using an interferon-gamma release assay to predict CMV infection

255 ed TB screening (tuberculin skin test and/or interferon-gamma release assay) was performed in 15 pati

256 ed TB screening (tuberculin skin test and/or interferon-gamma release assay) was performed in 15 pati

257 is infection (defined by positive results on interferon-gamma release assay) without evidence of acti

258 M. tuberculosis sensitization determined by interferon-gamma release assay, 12/23 participants had n

259 tly negative tuberculin skin tests (TST) and interferon-gamma release assays (IGRA), after close cont

260 The clinical utility of interferon-gamma release assays (IGRAs) for diagnosis of

261 ulosis infection in children, measured using interferon-gamma release assays (IGRAs).

262 tment did not demonstrate genetic defects in interferon gamma response or antigen presentation.

263 HIV restriction factor and a key mediator of interferon-gamma's anti-HIV activity.

264 titers and median frequencies of F-specific interferon gamma-secreting T cells also increased substa

265 MVA-NP+M1 activated a marked interferon gamma-secreting T-cell response to M1 peptide

266 The median frequency of RSV-F-specific interferon gamma-secreting T-cells after a ChAd155-RSV h

267 The rapid induction of interferon-gamma-secreting cells in ferrets previously i

268 antibodies were detected, but cross-reactive interferon-gamma-secreting T cells were detected in the

269 T cell effector functions (interferon gamma secretion, proliferation, and CD8-speci

270 ddition, low responder pigs with high plasma interferon-gamma showed lower (p < 0.01) birth weights t

271 by polarization with lipopolysaccharide and interferon-gamma, showed significantly higher expression

272 d a positive-feedback loop between PPARD and interferon gamma signaling that sustained gastric inflam

273 that promote inflammation and activation of interferon gamma signaling.

274 nd highlighted the importance of cancer cell interferon-gamma signaling in modulating NK activity.

275 (e.g. the Cadherin pathway for COPD and the interferon-gamma signaling pathway for breast cancer) as

276 higher levels of interleukin (IL) 6, IL-10, interferon-gamma, soluble tumor necrosis factor-related

277 Here we show that in interferon-gamma-stimulated cells guanylate-binding prot

278 12 induced sustained intratumoural levels of interferon-gamma, substantially reduced its systemic lev

279 In ex vivo analyses, we showed that interferon gamma, the main cytokine produced by Th1 and

280 ecretion of pathogenic cytokines (eg, IL-17, interferon-gamma, tissue necrosis factor, granulocyte-ma

281 In addition, the elevated number of interferon-gamma(+) TNF-alpha(+) CD8(+) T cells at D7 wa

282 roinflammatory T helper 1-type features with interferon-gamma, TNFalpha, and TNFR1 expression, loss o

283 osition of proteasome catalytic subunits via interferon-gamma treatment or siRNA knockdown results in

284 Anti-interferon-gamma treatment rescued neurons by preventing

285 Supplementation of antifungal agents with interferon-gamma treatment slowed disease progression, a

286 easured CSF concentrations of the following: interferon gamma, tumor necrosis factor (TNF) alpha, gra

287 significant increases in interleukin 1beta, interferon gamma, tumor necrosis factor alpha, and ioniz

288 th the increased production of the cytokines interferon gamma, tumor necrosis factor alpha, interleuk

289 y subjects, and that NK cells expressed more interferon-gamma, tumor necrosis factor (TNF), granzyme

290 using intracellular cytokine (interleukin-2, interferon-gamma, tumor necrosis factor-alpha) staining

291 CD8+ T cells demonstrated impaired interferon-gamma/tumor necrosis factor-alpha production

292 Adenosine deaminase (ADA), IRISA-TB (interferon gamma ultrasensitive rapid immunosuspension a

293 Interferon gamma was shown to contribute to the host inf

294 Interferon-gamma was produced by a large fraction of RBD

295 f proinflammatory cytokines (interleukin-12, interferon-gamma) was observed.

296 m levels of tumor necrosis factor -alpha and interferon-gamma were significantly increased in the CSD

297 (tumor necrosis factor-alpha) and IFN-gamma (interferon-gamma) were upregulated in the DC ACT renal C

298 Activated mutant SOD1 CD8(+) T cells produce interferon-gamma, which elicits the expression of the MH

299 CD4 tissue-resident memory T cells secrete interferon-gamma, which induces expression of chemokines

300 lls were significantly more prone to produce interferon-gamma, while secretion of the cytotoxicity mo