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1 gulation of the immune response to commensal intestinal bacteria.
2 tor-kappaB, are involved in the detection of intestinal bacteria.
3 s of oligosaccharides fermented by different intestinal bacteria.
4      Mucolytic potential is widespread among intestinal bacteria.
5 on of small-molecule metabolites produced by intestinal bacteria.
6 the host immune system in the gut and in the intestinal bacteria.
7 ding changes the composition and behavior of intestinal bacteria.
8 ngle epitope, which act together to regulate intestinal bacteria.
9 motherapy and immunotherapy is influenced by intestinal bacteria.
10 to the anaerobic growth requirements of most intestinal bacteria.
11 W and PflD, which are widespread among human intestinal bacteria.
12 al stimulation that can functionally replace intestinal bacteria.
13 atory agent cyclophosphamide (CTX) relies on intestinal bacteria.
14 enteropathy and the prevalence of pathogenic intestinal bacteria.
15 s.IMPORTANCE Butyrate is a lipid produced by intestinal bacteria.
16 utualistic relationship between the host and intestinal bacteria.
17 uld be attenuated in the complete absence of intestinal bacteria.
18 s an exaggerated immune response directed at intestinal bacteria.
19  promotes colonic neurogenesis, regulated by intestinal bacteria.
20 fense that protects against dissemination of intestinal bacteria.
21 lin or to different commensal and pathogenic intestinal bacteria.
22 on of IgM and complement-mediated control of intestinal bacteria.
23 e suggests a connection between diet and the intestinal bacteria.
24 tory IgA (SIgA) is found in association with intestinal bacteria.
25                              IgT coated most intestinal bacteria.
26 ce points to the various mechanisms by which intestinal bacteria act on distal tumors and how to harn
27                                              Intestinal bacteria aid host health and limit bacterial
28                             The reduction of intestinal bacteria also significantly improved the grow
29 y be due to excessive hydrogen production by intestinal bacteria altering the partial pressure of nit
30 ently, the O-protease OpeRATOR, derived from intestinal bacteria and expressed in Escherichia coli, h
31          Vancomycin altered the abundance of intestinal bacteria and fungi, measured by 16S and 18S r
32 n part, by the interaction between commensal intestinal bacteria and gut-associated lymphoid tissue (
33 cells, and the interrelationship between the intestinal bacteria and Tfh cell responses in the gut.
34 ng the complex interactions between resident intestinal bacteria and the immune system could improve
35        Intestinal barrier derangement allows intestinal bacteria and their products to translocate to
36 at host innate immune signaling can modulate intestinal bacteria and ultimately the host's susceptibi
37 ulting in tissue infiltration of 'symbiotic' intestinal bacteria and viruses that essentially become
38 tyrate, a short-chain fatty acid produced by intestinal bacteria, and lipoteichoic acid, a cell wall
39  of principles governing the localization of intestinal bacteria, and spatial relationships between b
40            Interestingly, RA was produced by intestinal bacteria, and the loss of bacterial-intrinsic
41                Our findings demonstrate that intestinal bacteria are critical for regulating allergic
42 strates that a substantial proportion of the intestinal bacteria are culturable.
43 inant daidzein metabolites produced by human intestinal bacteria are equol and O-desmethylangolensin.
44                                        Since intestinal bacteria are important for metabolism of nitr
45                                      Because intestinal bacteria are potent modulators of systemic im
46                                              Intestinal bacteria are required for development of gut-
47           Protein toxins released by certain intestinal bacteria are the cause of many diarrhoeal dis
48                                              Intestinal bacteria are thought to play a role in the pa
49 bacteriophage (phages) to eliminate targeted intestinal bacteria as an alternative to a broad-spectru
50   Enterococcus faecalis strains are resident intestinal bacteria associated with invasive infections,
51 ealed that neomycin altered the abundance of intestinal bacteria bearing gamma-d-glutamyl-meso-diamin
52 resent a major nutrient source, not just for intestinal bacteria but also for microorganisms that occ
53                            RegIIIgamma binds intestinal bacteria but lacks the complement recruitment
54 matic activity leads to growth inhibition of intestinal bacteria by causing local tryptophan depletio
55 hat commensal fungi can functionally replace intestinal bacteria by conferring protection against inj
56  is a short-chain fatty acid produced by the intestinal bacteria by the fermentation of nondigestible
57  how a marked proportion of oxygen-sensitive intestinal bacteria can be transmitted between individua
58          Our study highlights how protective intestinal bacteria can modify microbial-associated mole
59                                   Given that intestinal bacteria can produce ethanol, the aim of this
60                               Interestingly, intestinal bacteria can promote infection of several mam
61            Thus, introduction of immunogenic intestinal bacteria can promote Tfh-associated anti-tumo
62 is or defective host defense toward invasive intestinal bacteria can result in chronic inflammation.
63         A recent study in mice suggests that intestinal bacteria can trigger mammary carcinoma.
64             An improved understanding of how intestinal bacteria cause disease has become increasingl
65            Finally, new understanding of how intestinal bacteria cause disease is revealing that ente
66       Reviewed here are the varied ways that intestinal bacteria cause disease, which provide fundame
67                                    The Human intestinal Bacteria Collection (HiBC) contains 340 strai
68                    Importantly, we found the intestinal bacteria contain glycolipids which can be pre
69                   Our findings indicate that intestinal bacteria contribute to the development of NSA
70 t of progenitor cell responses to pathogenic intestinal bacteria could provide a measure of predispos
71    TMAO is produced by hepatic processing of intestinal bacteria-derived trimethylamine (TMA) followi
72                               The absence of intestinal bacteria did not affect the incidence of alph
73                            In animal models, intestinal bacteria drive colitis and in humans certain
74                                              Intestinal bacteria drive the formation of lymphoid tiss
75                      Abnormal composition of intestinal bacteria--"dysbiosis"-is characteristic of Cr
76                                              Intestinal bacteria employ microbial metabolites from th
77 tively, these data demonstrate that resident intestinal bacteria enhance the stability of beta-cateni
78 unication system where compounds produced by intestinal bacteria, especially short-chain fatty acids,
79                                        Small intestinal bacteria exhibit distinct gluten metabolic pa
80 rmfree or colonized with a limited number of intestinal bacteria failed to develop inflammatory bowel
81                                              Intestinal bacteria ferment dietary fibers into short-ch
82 d to a considerable interest in manipulating intestinal bacteria for therapeutic benefit, and trials
83                                              Intestinal bacteria form a resident community that has c
84 rier in Rd8 mice led to the translocation of intestinal bacteria from the lower gastrointestinal (GI)
85                                  Analysis of intestinal bacteria from the terminal ilea of Nod2-defic
86 , and to microbial products derived from the intestinal bacteria, has resulted in a distinctive local
87                            In summary, these intestinal bacteria have been highlighted as novel tools
88                                              Intestinal bacteria have been implicated in the pathogen
89                                        Human intestinal bacteria have many roles in human health, mos
90                         We hypothesized that intestinal bacteria impact the pathogenesis of multiple
91  IgA coating uniquely identifies colitogenic intestinal bacteria in a mouse model of microbiota-drive
92 to equol and O-desmethylangolensin (ODMA) by intestinal bacteria in approximately 30-50% and 80-90% o
93              Bifidobacteria are the dominant intestinal bacteria in breastfed infants.
94 bacillus plantarum and its interactions with intestinal bacteria in mice undergoing switches between
95             Animal models suggest a role for intestinal bacteria in supporting the systemic immune re
96 microbiota, focusing on the role of resident intestinal bacteria in the development of immune respons
97 scuss the roles of known and novel commensal intestinal bacteria in the pathogenesis of inflammatory
98   Dysregulated immune responses to commensal intestinal bacteria, including Escherichia coli, contrib
99 gated whether oral inoculation with specific intestinal bacteria increased colon inflammation in the
100 sion, these results support the concept that intestinal bacteria induce endogenous signals that play
101 ring the fermentation of fiber by endogenous intestinal bacteria, induces mitochondrial function-depe
102                                              Intestinal bacteria influence mammalian physiology, but
103    Our results suggest that translocation of intestinal bacteria into liver may be involved as a one
104                                    Escape of intestinal bacteria into the ascites is involved in the
105                   Increased translocation of intestinal bacteria is a hallmark of chronic liver disea
106 ses vs controls, we found that reactivity to intestinal bacteria is a normal property of the human CD
107                           In this review the intestinal bacteria is discussed in the context of devel
108  innate immune inflammatory response against intestinal bacteria is sufficient to induce colon cancer
109            Although the inability to control intestinal bacteria is thought to underlie IBD, the role
110                                     Resident intestinal bacteria likely play an important role in the
111 tyrate, a short-chain fatty acid produced by intestinal bacteria, lipoteichoic acid, a cell wall comp
112                                     Although intestinal bacteria live deep within the body, they are
113                              Thus, symbiotic intestinal bacteria modulate antiviral immunity and leve
114  accumulating to support the hypothesis that intestinal bacteria not only exchange resistance genes a
115 nce elicitor of systemic immune responses to intestinal bacteria observed in a loss-of-tolerance mode
116 al reservoir of particular importance is the intestinal bacteria of food-producing animals.
117 ing of the impact of translocating commensal intestinal bacteria on mucosal-associated T cell respons
118 eview examines the influence of signals from intestinal bacteria on the homeostasis of the mammalian
119 impact of less-virulent antibiotic-resistant intestinal bacteria on the outcome of CDI.
120                      We investigated whether intestinal bacteria or their products affect the adult E
121 ed that stress promotes the translocation of intestinal bacteria or their toxins into the systemic co
122            Our observations suggest that the intestinal bacteria, or the factors they produce, can af
123  rapidly respond to commensal and pathogenic intestinal bacteria, parasites and food components by po
124  a selective force in the evolution of their intestinal bacteria, particularly by increasing the prev
125                        Here, we analyzed the intestinal bacteria presented in normal and infected wor
126 ation after antibiotic-mediated depletion of intestinal bacteria prevents colitis and influenza, thus
127                                        Thus, intestinal bacteria, prior to detectable shifts in commu
128                                         Host intestinal bacteria produce its precursor trimethylamine
129          Accumulating evidence suggests that intestinal bacteria promote enteric virus infection in m
130 robiota.IMPORTANCE Recent data indicate that intestinal bacteria promote intestinal infection of seve
131                                        Which intestinal bacteria provide resistance to C. difficile i
132 ead to exaggerated inflammatory responses to intestinal bacteria, raising the possibility that defect
133 verse IS elements insert into the genomes of intestinal bacteria regardless of human host lifestyle.
134 ggravate chronic inflammation via effects on intestinal bacteria regulating gut permeability to visce
135 diarrheal disease, but their effect on other intestinal bacteria remains unexplored.
136                Furthermore, the reduction in intestinal bacteria resulted in the elimination of the e
137  chain fatty acids (SCFA) are metabolites of intestinal bacteria resulting from fermentation of dieta
138       Using these collections, we found that intestinal bacteria selected on the basis of high coatin
139 tem targets non-self rather than self, these intestinal bacteria should be considered foreign and the
140 n probiotics, nutritional components and the intestinal bacteria should be considered when examining
141 ses against a panel of taxonomically diverse intestinal bacteria species in sera from TN-10 participa
142 ponses influence host health, yet only a few intestinal bacteria species that induce cognate adaptive
143 entage of them become colonized by resistant intestinal bacteria such as extended-spectrum beta-lacta
144 ns that are highly specific for target Ag on intestinal bacteria, suggesting that an aberrant T cell
145 ry bowel disease, is attributed, in part, to intestinal bacteria that may initiate and perpetuate muc
146  overactive immunity to a subset of resident intestinal bacteria that mediate multiple inflammatory c
147 MIA phenotypes in offspring require maternal intestinal bacteria that promote TH17 cell differentiati
148 e contact at birth with maternal vaginal and intestinal bacteria, the authors proposed that this coul
149 s review, we explore a more sinister side of intestinal bacteria; their role as traffickers in antibi
150              However, the enzymes that allow intestinal bacteria to access sulfite from taurine have
151      New evidence has implicated the lack of intestinal bacteria to be responsible for the degradatio
152           To determine if fenugreek utilizes intestinal bacteria to offset the adverse effects of hig
153 ta indicate that enteric viruses can utilize intestinal bacteria to promote viral replication and pat
154 ve identified molecular cues propagated from intestinal bacteria to the brain that can affect neurolo
155 a, which may contribute to the adaptation of intestinal bacteria to the mucosal environment in health
156 ot clear how LACC1 balances defenses against intestinal bacteria vs intestinal inflammation or what c
157            Furthermore, supplement of killed intestinal bacteria was able to restore ConA-mediated NK
158 at colonization of GF mice with a variety of intestinal bacteria was capable of reducing T1D in MyD88
159  oxidative derivative of choline produced by intestinal bacteria, was positively associated with rect
160 erplay between the mucosal immune system and intestinal bacteria, we investigated the role of MIF in
161                                 The expanded intestinal bacteria were required for the parasite-induc
162          These antibody responses link human intestinal bacteria with T1D progression, adding predict
163 n of SAAs from meat to sulfide production by intestinal bacteria with use of both a model culture sys
164 utrient release acts as a source of fuel for intestinal bacteria, with implications for gut inflammat

 
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