ライフサイエンスコーパス: intestinal flora

1 ly Enterococci species, are present in human intestinal flora.

2 -operated mice in the presence or absence of intestinal flora.

3 ut did not affect Escherichia coli of normal intestinal flora.

4 base, queuine, obtained from the diet or the intestinal flora.

5 titutive TLR signals are driven by commensal intestinal flora.

6 nt host immunity or the emergence of harmful intestinal flora.

7 inflammation requires the presence of viable intestinal flora.

8 art by a dysregulated immune response to the intestinal flora.

9 stinal epithelium serves as a barrier to the intestinal flora.

10 tion) on antibiotic resistance in the normal intestinal flora.

11 a short chain fatty acid produced by normal intestinal flora.

12 duced by nonpathogenic E. coli of the normal intestinal flora.

13 evelopment occurring independently of viable intestinal flora.

14 egulate the pathogenic role of H. pylori and intestinal flora.

15 ich can skew immune responses against normal intestinal flora.

16 lial MHC class II expression is regulated by intestinal flora.

17 min K2 and lithocholic acid, a by-product of intestinal flora, activate pregnane X receptor (PXR) and

18 metabolite of cholate produced by the normal intestinal flora, also induced germination of C. diffici

19 low skatole deposition in wild boars such as intestinal flora and anatomy, dietary composition, housi

20 s and chronic non-infectious diseases, where intestinal flora and endotoxemia play a role.

21 cognized gut-brain axis and an impact of the intestinal flora and meningeal IL-17(+) gammadelta T cel

22 the abundance of a group of bacteria in the intestinal flora and relapse/progression of disease afte

23 Recently, the bacterial diversity of the intestinal flora and the diversity of various environmen

24 utrophil counts, depending on the prevailing intestinal flora, and do not produce IL-17A.

25 reported previously that alterations in the intestinal flora are associated with GVHD, bacteremia, a

26 llo-HSCT, the diversity and stability of the intestinal flora are disrupted, resulting in domination

27 Enterococci are members of the healthy human intestinal flora, but are also leading causes of highly

28 Restoring the balance of intestinal flora by introducing probiotics for disease p

29 The potential benefits of modifying the intestinal flora composition of certain high-risk groups

30 ects, we hypothesized that components of the intestinal flora could be associated with relapse after

31 lude that specific members of the commensal, intestinal flora drive GALT development through a specif

32 ial therapy, which alters the balance of the intestinal flora, enabling C. difficile to proliferate i

33 ted in maintenance of the composition of the intestinal flora, even after a moderate gluten challenge

34 for risk assessment and manipulation of the intestinal flora for prevention and treatment of radiati

35 to be functionally hyporesponsive to normal intestinal flora, human intestinal epithelial cells can

36 Conventional (complex-intestinal flora) IL-10 KO mice developed IBD within 10

37 Low protein digestibility causes intestinal flora imbalance, wet litter, and nitrogen pol

38 here is a difference in response towards the intestinal flora in dectin-1 deficient macrophages, duri

39 rs triggering a loss of tolerance for normal intestinal flora in genetically susceptible individuals.

40 onal significance, since manipulation of the intestinal flora in individuals with congenital biopteri

41 Increasing evidence links altered intestinal flora in infancy to eczema and asthma.

42 ther, these results support a vital role for intestinal flora in the maturation of intestinal barrier

43 n germfree conditions, implicating undefined intestinal flora in the pathogenesis of lesions.

44 a support the hypothesis that the indigenous intestinal flora inhibit growth of VRE and suggest that

45 Maintaining balance with intestinal flora is an important activity of the immune

46 omised hosts IL-10 stimulated in response to intestinal flora is important in preventing IBD.

47 Intestinal flora is required to develop a normal adaptiv

48 teroides fragilis, a component of the normal intestinal flora, is an obligate anaerobe capable of lon

49 Maternal intestinal flora may be an important environmental expos

50 The intestinal flora may promote colon tumor formation.

51 Toxin production by intestinal flora may represent a novel strategy of patho

52 erococcus species combined with conventional intestinal flora obtained from the gastrointestinal trac

53 In summary, the intestinal flora of healthy girls is complex, with frequ

54 Our findings suggest that the intestinal flora of healthy girls is multiclonal with fr

55 have investigated the influence of maternal intestinal flora on wheezing and eczema in early childho

56 When the GALT receives signals from the intestinal flora or food Ags, it must induce a state of

57 s, treatment with a probiotic (to modify the intestinal flora) or anti-TNF antibodies (to inhibit TNF

58 The interaction between polyphenols and intestinal flora plays a significant role in gastrointes

59 It is now well established that the intestinal flora plays an important role in the pathogen

60 w that antibiotic-induced alterations in the intestinal flora reduce ischemic brain injury in mice, a

61 Intestinal flora regulation is an effective method to in

62 as the advances in practical application of intestinal flora regulation.

63 been demonstrated to alter the pre-existing intestinal flora so as to provide an advantage to the ho

64 athogenic strains from within the host's own intestinal flora, strain sharing between household membe

65 estion or to certain metabolites produced by intestinal flora that degraded alcohol was investigated.

66 We then identified specific members of the intestinal flora that promote GALT development.

67 absorption and metabolism by regulating the intestinal flora, thereby enhancing bioavailability, pro

68 ragilis and antibiotic-mediated depletion of intestinal flora to initiate colitis.

69 ragilis and antibiotic-mediated depletion of intestinal flora to initiate colitis.

70 in integrating signals from the diet and the intestinal flora to modulate ongoing inflammation in the

71 sults suggest that the susceptibility of the intestinal flora to the Shiga toxin phage could exert ei

72 , these junctions become loose, enabling the intestinal flora to trigger a constitutive activation of

73 n vivo effect of diosgenin on the density of intestinal flora was examined in a murine model of food

74 ce an aberrant immune response to indigenous intestinal flora, which might favor IBD development.

75 ates the decolonization of oxalate degrading intestinal flora with a higher risk of calcium oxalate s

76 nsplantation (FMT) re-establishes a balanced intestinal flora with resultant cure of recurrent Clostr