ライフサイエンスコーパス: intestinal infection

1 e for this NADPH oxidase in both colitis and intestinal infection.

2 17 and CD8(+) T(c)17) in vitro and following intestinal infection.

3 ivo, and persistent strains exhibit lifelong intestinal infection.

4 ential for V. vulnificus pathogenesis during intestinal infection.

5 ost frequently identified protozoan cause of intestinal infection.

6 x mutual regulation of both cytokines during intestinal infection.

7 al MPs in ILC3-mediated host defence against intestinal infection.

8 s were neutropenia, febrile neutropenia, and intestinal infection.

9 ction with IL-13, which may underpin chronic intestinal infection.

10 of intranasally infected animals in clearing intestinal infection.

11 efficacy against asymptomatic E. histolytica intestinal infection.

12 e is known about the mechanisms that control intestinal infection.

13 ed in vitro and in the infant mouse model of intestinal infection.

14 for the mucosal influx of neutrophils during intestinal infection.

15 s is a critical function of the toxin during intestinal infection.

16 a coli often develop septicemia secondary to intestinal infection.

17 ntified by a screen for genes induced during intestinal infection.

18 is a bacterium that causes life-threatening intestinal infections.

19 ineage that causes multidrug-resistant extra-intestinal infections.

20 t to play a major role in protection against intestinal infections.

21 gastroenteritis, septicemia, and other extra-intestinal infections.

22 encounters for bacterial pneumonia or viral intestinal infections.

23 promotes health and reduces the incidence of intestinal infections.

24 istance is key to the prevention and cure of intestinal infections.

25 Four MAbs given therapeutically each reduced intestinal infection 34.4 to 42.2% compared to isotype-m

26 E. coli strains causing extra-intestinal infections accumulate hotspot mutations at th

27 t public health concerns by causing an acute intestinal infection afflicting millions of people each

28 Instead, intestinal infection and bloating of the lumen, which de

29 ere found in models of acute colitis, type 2 intestinal infection and colitis-associated cancer.

30 udy indicate an association between prenatal intestinal infection and genitourinary tract infection i

31 1.09; 95% CI: 1.02-1.17), and ED visits for intestinal infection and heat waves defined by average t

32 mportant role for TLR11 in preventing murine intestinal infection and modulating antigen transportati

33 NS1 secretion is essential for intestinal infection and resistance to IFN-lambda in viv

34 Intestinal infection and the associated diarrhea are sig

35 be elucidated, the likelihood of TV causing intestinal infection and the availability of a tissue cu

36 enteric viruses utilize bacteria to promote intestinal infection and viral stability.

37 is essential for understanding and treating intestinal infections and inflammatory diseases.

38 Intestinal infections and non-COVID-19 respiratory illne

39 tic toxin interactions during C. perfringens intestinal infections and support a possible role for CP

40 e treatment of inflammatory bowl disease and intestinal infections and to new immunization strategies

41 me an antiparasite effect of IL-22 during an intestinal infection, and they suggest that IL-17A and I

42 RPOSE OF REVIEW: Clostridium difficile is an intestinal infection associated with antibiotic use, com

43 amebiasis can develop in some children after intestinal infection, but protective immunity may be tra

44 MAbs or combinations of two MAbs and reduced intestinal infection by 86 to 93%.

45 CD98 in the innate host defense response to intestinal infection by attaching and effacing (A/E) pat

46 effects of leptin receptor polymorphisms on intestinal infection by E. histolytica.

47 tic uremic syndrome (HUS) is associated with intestinal infection by enterohemorrhagic Escherichia co

48 Primary intestinal infection by T. spiralis induces mastocytosis

49 We found increased susceptibility to intestinal infection by this parasite associated with an

50 Intestinal infections by attaching and effacing (A/E) ba

51 rome (HUS) is the life-threatenig sequela of intestinal infections by Shiga toxin (Stx)-producing Esc

52 Cholera is an acute intestinal infection caused by the bacterium Vibrio chol

53 ential for IFN-based treatment of SARS-CoV-2 intestinal infection compared to type I IFNs.

54 reased lethality in response to C. difficile intestinal infection despite comparable levels of intest

55 which is sufficient to protect mice against intestinal infections from S. Typhimurium.

56 ase; however, the importance of EutR to EHEC intestinal infection has not been examined.

57 ta unveil a role for P.copri in exacerbating intestinal infection, highlighting that pathogens such a

58 nd inflammatory functions, during natural RV intestinal infection.IMPORTANCE Rotavirus is a highly in

59 ns to pathogenesis, we utilized the model of intestinal infection in adult mice sensitive to the acti

60 fector domain region is essential for lethal intestinal infection in mice, but the contribution of ea

61 in the liver and spleen following C. parvum intestinal infection in neonatal mice.

62 rs that induce defense against intracellular intestinal infection in the model nematode Caenorhabditi

63 ed settings, with high prevalence of chronic intestinal infections in children and other factors disc

64 membranes, has been used to treat protozoal intestinal infections in human patients.

65 cile is an important pathogen causing severe intestinal infections in humans and animals.

66 a common cause of gastroenteritis and extra-intestinal infections in humans.

67 utic tools for the treatment of pathological intestinal infections in infants.

68 host susceptibility to intestinal and extra-intestinal infections, including those caused by viruses

69 strains known to cause intestinal and extra intestinal infections, induce reorganization of the acti

70 However, these intestinal infection-induced plasmablasts lack the CLA h

71 Prenatal maternal infections, including intestinal infection, influenza, upper airway infection,

72 Intra-amniotic C.albicans caused intestinal infection, injury and inflammation.

73 mansoni (and rarely Schistosoma haematobium) intestinal infection is also not very common and is foun

74 Immunity to Entamoeba species intestinal infection is associated with the presence of

75 ortality in Africa, but host defense against intestinal infection is poorly understood and may depend

76 biological significance of this event during intestinal infection is unclear.

77 ver, the function of macrophages in reovirus intestinal infection is unknown.

78 stressors, including antibiotic therapy and intestinal infections, is associated with multiple healt

79 ium subsp. paratuberculosis causes a chronic intestinal infection leading to a chronic wasting diseas

80 aerophilic bacterium associated with chronic intestinal infection leading to hepatitis and colonic an

81 ntibody-secreting cells in response to acute intestinal infection, likely helping target these cells

82 models, including diabetes mellitus, anemia, intestinal infection, liver diseases, gastrointestinal h

83 n inside macrophages, resulting in increased intestinal infection loads in DBA/2J mice.

84 Our data also suggest that intestinal infection might be initiated from the basolat

85 as further tested in vivo in an experimental intestinal infection model using antibody-free newborn p

86 rulent than the wild-type strain in a murine intestinal infection model, suggesting that survival and

87 Using an adult mouse intestinal infection model, this study examines the cont

88 Using an adult mouse intestinal infection model, this study examines the cont

89 dine KDU731 results in a potent reduction in intestinal infection of immunocompromised mice.

90 racellular growth of L. monocytogenes during intestinal infection of mice.

91 sed by Eimeria spp. presents a self-limiting intestinal infection of poultry.

92 ta indicate that intestinal bacteria promote intestinal infection of several enteric viruses.

93 Cryptosporidium is a common intestinal infection of vertebrates and a significant th

94 In contrast, responses to intestinal infection or flagellin administration were un

95 or without concurrent Entamoeba histolytica intestinal infection or were infection free 1 year after

96 ulsions (OR, 0.39; 95% CI, 0.21-0.67), viral intestinal infections (OR, 0.56; 95% CI, 0.43-0.71), or

97 Their prolonged intestinal infections persist despite intact immune syst

98 anisms by which they protect the host during intestinal infection remains poorly understood.

99 dulation of these neuroendocrine pathways by intestinal infection serves as a systemic feedback that

100 phocytes were important for the clearance of intestinal infection, since severe combined immunodefici

101 nferring protection against subsequent extra-intestinal infections, such as urinary tract infections.

102 odes (gdLNs) in mediating this effect, as an intestinal infection that drains to the proximal gdLNs p

103 N-beta, and Mx-1 in PPs and caused localized intestinal infection that was cleared in 10 d.

104 y of the antigen-specific T-cell response to intestinal infection, the prominence of microbial mechan

105 We found that resistance to intestinal infection was independent of lymphocyte activ

106 To better understand intestinal infections, we performed a genetic screen for

107 Offspring exposed to prenatal intestinal infection (weighted OR, 1.46; 95% CI, 1.17-1.

108 Furthermore, maternal intestinal infection (weighted OR, 6.05; 95% CI, 3.80-9.

109 neutrophils, and T lymphocytes during human intestinal infection with Campylobacter.

110 ion from TH17 but not ILCs in the context of intestinal infection with Citrobacter rodentium, resulti

111 IL-22 and resulted in crippled responses to intestinal infection with Citrobacter rodentium.

112 tomycin-fed C57BL/6 mice were susceptible to intestinal infection with El Tor strains, which caused r

113 Establishment of intestinal infection with Entamoeba histolytica depends

114 titers of antibody and is protective against intestinal infection with ETEC.

115 In an independent IgE-inducing model, i.e., intestinal infection with H. polygyrus, we validated the

116 id and 17% triglycerides), as well as distal intestinal infection with Helicobacter hepaticus, influe

117 atf3 deficiency converted a normally chronic intestinal infection with Heligmosomoides polygyrus into

118 (KO) mice following sensitization to OVA or intestinal infection with Heligmosomoides polygyrus Spec

119 the infecting serotype, or who had previous intestinal infection with homologous poliovirus.

120 of increased fluid secretion in response to intestinal infection with invasive bacteria.

121 bolization with eggs of Schistosoma mansoni, intestinal infection with Nippostrongylus brasiliensis,

122 hila model of gut pathogenesis, we show that intestinal infection with Pseudomonas aeruginosa, a huma

123 y manifestation of COVID-19, we investigated intestinal infection with SARS-CoV-2, its effect on path

124 Most cases are caused by an intestinal infection with Shiga toxin-producing strains

125 ve as a model for renal disease secondary to intestinal infection with STEC.

126 pecific IgG2c failed to protect rats against intestinal infection with T. spiralis.

127 We have examined whether intestinal infection with the natural murine helminth He

128 Intestinal infection with the parasitic nematode, Trichi

129 During intestinal infection with the pathogen Salmonella Typhim

130 eless, T-bet(-/-) mice respond vigorously to intestinal infection with Trichinella spiralis, eliminat

131 naerobic Gram-positive bacterium that causes intestinal infections with symptoms ranging from mild di

132 th spread to respiratory tissues, or through intestinal infection, with spread to the liver and pancr

133 r, unlike wild-type mice, which resolved the intestinal infection within 10 days, LTalpha(-/-) mice s