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1  treated with antibiotics partially restores intestinal motility.
2 sponses contributing to neural regulation of intestinal motility.
3 or potential ankyrin A1 (Trpa1) and increase intestinal motility.
4 eurons, thereby enabling the coordination of intestinal motility.
5 cts in ascl1a-/- embryos also contributes to intestinal motility.
6 ition of PAK in edematous intestine improved intestinal motility.
7 e enteric nervous system (ENS), and abnormal intestinal motility.
8 oendocrine products in maintenance of normal intestinal motility.
9 te primarily to ischaemia-induced changes in intestinal motility.
10  reported in adult human gut, and OT affects intestinal motility.
11 ting novel and relevant information on small intestinal motility.
12 ting novel and relevant information on small intestinal motility.
13 iety of gastrointestinal functions including intestinal motility.
14 et for modulating immune-mediated effects on intestinal motility.
15 esponsive eczema and a marked disturbance of intestinal motility.
16 hlights the importance of these compounds in intestinal motility.
17 nterstitial cells of Cajal in the control of intestinal motility.
18  in the regulation of smooth muscle tone and intestinal motility.
19  is associated with a subsequent decrease in intestinal motility.
20 n of the integrity of the epithelium and the intestinal motility.
21 ilation of the urinary bladder and defective intestinal motility.
22 ic neurons as key interneurons that regulate intestinal motility.
23 h is composed of neurons and glia, regulates intestinal motility.
24 the myenteric plexus circuitry that controls intestinal motility.
25  the walls of the intestinal tract, regulate intestinal motility, a well-characterized reflex control
26                                      Loss of intestinal motility after surgery is associated with acu
27 l resection and to follow-up the recovery of intestinal motility after surgical manipulation.
28 t reduced intestine diameters, but increased intestinal motilities along with thickened muscle fibers
29 include intestinal mucositis associated with intestinal motility alterations maybe due to an effect o
30 associated with reduction in BO, increase in intestinal motility and amelioration in intestinal perme
31 erotonergic neurons contribute to control of intestinal motility and anxiety-like behavior and sugges
32 lude the ion channels involved in regulating intestinal motility and bile viscosity.
33 ug absorption may be attributed to decreased intestinal motility and compromised intestinal mucosal f
34 optimize physiological gut processes such as intestinal motility and epithelial barrier integrity thr
35 a subset IPANs and that these cells regulate intestinal motility and epithelial cell proliferation in
36 ups with exenatide, but suppression of small intestinal motility and flow was observed even in subjec
37                           Interestingly, mdx intestinal motility and gene expressions of tight juncti
38                                         Both intestinal motility and inflammation are governed by the
39 mportant molecular mechanism for controlling intestinal motility and secretion by regulating the exci
40 , as well as in the physiological control of intestinal motility and secretion.
41 onment and controls the detailed patterns of intestinal motility and secretion.
42    The mechanism is believed to be decreased intestinal motility and stool frequency resulting in mor
43 ent in mature B and T lymphocytes, to assess intestinal motility and the role of the adaptive immune
44 roepithelial signaling, these cells modulate intestinal motility and transit of digestive products.
45 lial cells and fluid shear that results from intestinal motility and transit.
46 important gastrointestinal functions such as intestinal motility and transport, intestinal permeabili
47 ntestinal tract and plays important roles in intestinal motility and visceral hypersensitivity.
48 ease of intestinal neuropeptides, control of intestinal motility, and/or modulation of intestinal flu
49 h intestinal barrier dysfunction and reduced intestinal motility as determined by in vivo intestinal
50 ew added to the basic understanding of small-intestinal motility as well as its alteration in disease
51  model and moreover, the FD mice had reduced intestinal motility, as did patients.
52 togen have the potential to influence gastro-intestinal motility; both are key components of hormone
53 s are also postulated to result from altered intestinal motility, but rotavirus infection had no effe
54                       We assessed changes in intestinal motility by assessing stool frequency, bead e
55 kely peptide candidate because inhibition of intestinal motility by fat in the distal gut correlated
56 inct subpopulation, and glutamate can affect intestinal motility by modulating enteric neuron activit
57 ion in the colon during colitis, paralleling intestinal motility changes.
58 ocus, which in humans is associated with the intestinal motility disorder known as Hirschsprung disea
59           For decades, IBS was considered an intestinal motility disorder, and more recently a gut di
60 y be relevant for understanding and treating intestinal motility disorders and also suggest that enha
61 ailability or metabolism could contribute to intestinal motility disorders.
62 n antibiotic treatment or chemically induced intestinal motility disturbance, and C. difficile exploi
63 n administered orally, were able to increase intestinal motility during morphine-induced constipation
64              Previous studies indicated that intestinal motility dysfunction after edema development
65 ion between bacterial overgrowth and altered intestinal motility gathers support in a variety of cond
66                       However, they regulate intestinal motility in a sex-dependent manner.
67            Currently, preclinical imaging of intestinal motility in animal models is performed either
68 ithelium, but are required for regulation of intestinal motility in females.
69 ting novel and relevant information on small intestinal motility in humans and animals.
70 aimed to evaluate the effect of SNS on small intestinal motility in IBS patients.
71 ls and this activity is necessary for normal intestinal motility in male mice.
72 ng mainly at peripheral while increasing the intestinal motility in morphine-pelleted mice (ED(50) =
73  monitor constrained respiratory airflow and intestinal motility in neonates in the neonatal intensiv
74                                              Intestinal motility is modulated by mechanical forces ac
75                                We found that intestinal motility is reduced early in NEC in mice, pre
76                    Glial-mediated control of intestinal motility is region and pathway specific.
77                 IBS patients underwent small intestinal motility (manometry; fasted and fed contracti
78 mation, whereas pharmacologic restoration of intestinal motility reduced NEC severity.
79                                        Small intestinal motility remains an understudied area.
80 intestine, luminal acidification and altered intestinal motility, resulting in blockage.
81                                              Intestinal motility, secretion, and blood flow are contr
82 rstanding and treatment of injury-associated intestinal motility/sensory disorders.
83 c smooth muscle cells, resulting in abnormal intestinal motility, severe abdominal pain, malnutrition
84          5-HT(3) receptor agonists stimulate intestinal motility, shorten transit times and in a pilo
85          The gastric emptying time and small intestinal motility were evaluated by MTS-1, and the tot
86 iated with significant functional effects on intestinal motility which can be reversed by GDNF supple