ライフサイエンスコーパス: intestinal mucosa

1 and protection of epithelial barriers in the intestinal mucosa.

2 a transit from the intestinal lumen into the intestinal mucosa.

3 fferent colonization factors that target the intestinal mucosa.

4 omoted the translocation of Stx2a across the intestinal mucosa.

5 cally decreased the levels of ATG16L1 in the intestinal mucosa.

6 specific inflammatory conditions within the intestinal mucosa.

7 on other radiosensitive tissues, such as the intestinal mucosa.

8 ed (reduced and methylated) to 5-MTHF in the intestinal mucosa.

9 tervillous spaces, and increased IELs in the intestinal mucosa.

10 ct of viral infection on miRNA expression in intestinal mucosa.

11 eron-gamma and interleukin-17A in the murine intestinal mucosa.

12 LFABP; FABP1) is expressed both in liver and intestinal mucosa.

13 3 and T helper 17 signature cytokines in the intestinal mucosa.

14 rative colitis patients compared with normal intestinal mucosa.

15 e with hallmarks of adaptive immunity in the intestinal mucosa.

16 h are substantially enriched at the inflamed intestinal mucosa.

17 and are critical for barrier function of the intestinal mucosa.

18 -22, in response to bacterial entry into the intestinal mucosa.

19 mediators of innate immune signaling in the intestinal mucosa.

20 inducible regulatory T cells (iTreg) in the intestinal mucosa.

21 rption and buildup of DGAT substrates in the intestinal mucosa.

22 accumulate in the pulmonary compartment and intestinal mucosa.

23 uced LPS-mediated signaling within the fetal intestinal mucosa.

24 hogenic microbes direct host defenses at the intestinal mucosa.

25 (+)CD4(+)cells in lymphoid organs and in the intestinal mucosa.

26 iary and pancreatic secretions to the distal intestinal mucosa.

27 ncountered systemically or delivered via the intestinal mucosa.

28 on of active immunity or inflammation in the intestinal mucosa.

29 re with normal tolerogenic mechanisms in the intestinal mucosa.

30 ex regulation of barrier protection with the intestinal mucosa.

31 ic mechanisms and induce inflammation in the intestinal mucosa.

32 ruminant hosts by translocation through the intestinal mucosa.

33 mediate induction of homing potential to the intestinal mucosa.

34 D4 T cell priming in lymphoid tissue and the intestinal mucosa.

35 maintaining the Treg/Th17 balance within the intestinal mucosa.

36 ceptor superfamily, is expressed in inflamed intestinal mucosa.

37 ed inhibition of TLR4 signaling in the small intestinal mucosa.

38 ved in all VCMsh2 strains and limited to the intestinal mucosa.

39 its preferred colonization site at the host intestinal mucosa.

40 stemic circulation after passing through the intestinal mucosa.

41 ithelial migration of neutrophils across the intestinal mucosa.

42 nt was maintained in the upper crypts of the intestinal mucosa.

43 is a complex colloidal system that coats the intestinal mucosa.

44 on of the variety of these structures in the intestinal mucosa.

45 or crypt cell regions of healthy human small intestinal mucosa.

46 causes systemic infection by traversing the intestinal mucosa.

47 infection of cattle takes place through the intestinal mucosa.

48 wn to be associated with inflammation of the intestinal mucosa.

49 loss in lymphoid tissues and necrosis of the intestinal mucosa.

50 . enterica serovar Typhimurium with the host intestinal mucosa.

51 man intestinal Caco-2 cells and rodent small intestinal mucosa.

52 systemic spread of pathogens that infect the intestinal mucosa.

53 s or villous motility may be trophic for the intestinal mucosa.

54 or mediating the ITF healing response of the intestinal mucosa.

55 mucus depends on its spatial location in the intestinal mucosa.

56 ce of effector and regulatory T cells in the intestinal mucosa.

57 cant numbers in their tissues, including the intestinal mucosa.

58 ry signals and inflammatory mediators in the intestinal mucosa.

59 dence for mosaicism in APC in non-neoplastic intestinal mucosa.

60 afficking of Ly6C(+) monocytes from blood to intestinal mucosa.

61 sely mirrored the transcriptome of the small intestinal mucosa.

62 actors participate in antiviral responses in intestinal mucosa.

63 uced numbers of endogenous Th17 cells in the intestinal mucosa.

64 -derived CRC samples and adjacent uninvolved intestinal mucosa.

65 e of Listeria monocytogenes infection in the intestinal mucosa.

66 nic organisms, focusing in particular on the intestinal mucosa.

67 ponses both systemically and at the oral and intestinal mucosa.

68 ost-environment interfaces, such as skin and intestinal mucosa.

69 role in fluid and water transport across the intestinal mucosa.

70 MT1 was found to be markedly up-regulated in intestinal mucosa.

71 nt, and is integral to the repair of damaged intestinal mucosa(1-3).

72 Among the innate defense mechanisms of intestinal mucosa, a defective tight junction (TJ) barri

73 Microbial invasion into the intestinal mucosa after allogeneic hematopoietic cell tr

74 tion of structural/functional changes in the intestinal mucosa after food challenge.

75 e c, and the cleaved caspase 9 expression in intestinal mucosa after intestinal I/R injury (P<0.05).

76 ed epithelial conditioning that protects the intestinal mucosa against bacterial invasion by inducing

77 st to confer antiapoptotic protection of the intestinal mucosa against inflammatory stress-induced da

78 TI may provide a novel method of stabilizing intestinal mucosa against noxious agents and stimulating

79 AMT-101 was taken up by inflamed intestinal mucosa and activated pSTAT3 in the lamina pro

80 g of T cells by dendritic cells (DCs) in the intestinal mucosa and associated lymphoid tissues helps

81 ells whose presence had been reported in the intestinal mucosa and blood.

82 ed these data to RNA-Seq from both the small intestinal mucosa and colonic mucosa of healthy control

83 ients with DE exhibit abnormalities in their intestinal mucosa and CoSCs, which fail to generate in v

84 nd that embryonic precursor cells seeded the intestinal mucosa and demonstrated extensive in situ pro

85 hat classical monocytes constantly enter the intestinal mucosa and how the environment dictates their

86 s involved in lymphocyte localization to the intestinal mucosa and how they can be applied to therapy

87 therapeutic antibodies are lost through the intestinal mucosa and how this process affects treatment

88 tion of Toll-like receptors (TLRs) in murine intestinal mucosa and human intestinal epithelial cells

89 nflammatory macrophages and T cells into the intestinal mucosa and increased expression of inflammato

90 rched for T-UCRs that regulate growth of the intestinal mucosa and investigated the mechanism by whic

91 h a novel mechanism that is localized to the intestinal mucosa and is associated with significant cha

92 induction of the murine Hbeta D2 ortholog in intestinal mucosa and is dependent upon both TLR4 and CD

93 ne quantitative polymerase chain reaction of intestinal mucosa and mesenteric lymph nodes of Duoxa(-/

94 epithelium, as well as CD8(+) T cells in the intestinal mucosa and mesenteric lymph nodes, express th

95 he difficulty in directly sampling the small intestinal mucosa and microbial community (microbiota).

96 and migrate toward netrins expressed by the intestinal mucosa and pancreas; this attraction is requi

97 posaccharide level and bacteria loads in the intestinal mucosa and peripheral organs were elevated in

98 of tissue-resident memory in organs such as intestinal mucosa and skin.

99 3 noncoding RNA that regulates growth of the intestinal mucosa and stimulates intestinal epithelial r

100 The intestinal mucosa and submucosa contain three types of l

101 with increased aquaporin 4 expression in the intestinal mucosa and submucosa.

102 t on the structure and function of the small intestinal mucosa and suggest signaling through this pat

103 ted robust T cell responses primarily in the intestinal mucosa and that MNV-specific CD8 T cells dyna

104 ted with increased expression of TLR4 in the intestinal mucosa and that physiological stressors assoc

105 ed in the differentiation compartment of the intestinal mucosa and that the expression of JNK1 was si

106 flux transporters in Caco-2 cells and in the intestinal mucosa and the BBB in vivo are most likely du

107 OH) exhibited low permeation across both the intestinal mucosa and the blood-brain barrier (BBB).

108 licate infiltration of gut microbes into the intestinal mucosa and the corresponding inflammatory res

109 facilitation of bacterial invasion into the intestinal mucosa and the development of acute colitis.

110 nderstanding of the interactions between the intestinal mucosa and the enteric microbiota.

111 s that infiltration of gut microbes into the intestinal mucosa and the resulting inflammation are cau

112 Infiltration of gut microbes into the intestinal mucosa and the resulting inflammation are cau

113 he gut microbiome communicates with both the intestinal mucosa and the systemic immune system, given

114 ivation of islet-specific T cells within the intestinal mucosa and to autoimmune diabetes and provide

115 te miRNA expression profile of the mammalian intestinal mucosa and to determine the contribution of m

116 y vitamin folates, are biotransformed in the intestinal mucosa and transferred to the portal vein as

117 cyclase C (GUCY2C), a receptor expressed by intestinal mucosa and universally expressed by metastati

118 feration and stimulation of apoptosis in the intestinal mucosa and was associated with decreased acti

119 ntain a healthy inflammatory tone within the intestinal mucosa and, thus, enhances resistance to infe

120 nergy absorption and in the integrity of the intestinal mucosa, and a GLP-2R agonist, teduglutide, is

121 key regulator for the normal turnover of the intestinal mucosa, and abnormalities associated with thi

122 lycans present in our diets, secreted by our intestinal mucosa, and displayed on the surfaces of othe

123 gens, lymphoid follicular hyperplasia in the intestinal mucosa, and elevated host-defence ability aga

124 ed from animal organs, predominantly porcine intestinal mucosa, and goes through an extensive process

125 nt, colitis-attenuating SodA to the inflamed intestinal mucosa, and host antimicrobials may play a cr

126 ity, reduces subclinical inflammation of the intestinal mucosa, and improves gut barrier function to

127 ion, parasite colonization and damage to the intestinal mucosa, and peak diarrheal symptoms, compared

128 essential signals to cells in the underlying intestinal mucosa, and that intestinal epithelial cells,

129 highly expressed in both the small and large intestinal mucosa, and there is a 53% overlap in the top

130 d for successful treatment is a healed small intestinal mucosa, and therefore, the outcome measures i

131 etics, enhancement of permeation through the intestinal mucosa, and triggering drug precipitation upo

132 adherin, the natural ligand of KLRG1, in the intestinal mucosa; and have elevated levels of systemic

133 The major fuels for the small intestinal mucosa are amino acids (glutamine, glutamate,

134 zation and homeostasis of glial cells in the intestinal mucosa are regulated by the indigenous gut mi

135 and proinflammatory cytokine response in the intestinal mucosa are significantly higher in AW-recipie

136 ociated with a type 2 immune response in the intestinal mucosa are up-regulated in treatment-naive pe

137 ust reach the circulatory system through the intestinal mucosa as a sufficiently large fragment with

138 eractions between the gut microbiome and the intestinal mucosa-associated immune system.

139 rat, a model in which the adaptation of the intestinal mucosa, at least to fasting, is quite differe

140 in mice depleted constitutive Th17 cells in intestinal mucosa, blocked Th17 cell generation in the l

141 (+) and IgA(+) cells were seen in the normal intestinal mucosa, but they were significantly more freq

142 m subsp. paratuberculosis interacts with the intestinal mucosa by crossing both Peyer's patches and n

143 Growth inhibition of the small intestinal mucosa by fasting in mice was associated with

144 Analysis of CD3(+) lymphocytes in the intestinal mucosa by flow cytometry revealed that alphab

145 dies prevents commensal association with the intestinal mucosa by limiting bacterial motility.

146 The invasion of the intestinal mucosa by M. avium subsp. paratuberculosis an

147 n, we investigated expression of DMT1 in the intestinal mucosa by quantitative real-time polymerase c

148 The intestinal mucosa comprises the inner lining of the inte

149 Normal intestinal mucosa contains abundant immunoglobulin A (Ig

150 Intestinal mucosa contains leptin receptors, and leptin

151 oid-mediated ST2 production was evaluated in intestinal mucosa cultures.

152 al ligation and puncture, Chk2 levels in the intestinal mucosa decreased, associated with an inhibiti

153 e function and structural maintenance of the intestinal mucosa depend on a constant process of prolif

154 late intestinal epithelium to produce IDENs (intestinal mucosa-derived exosome-like nanoparticles) co

155 arget in the study of innate immunity in the intestinal mucosa due to their involvement in the regula

156 ze recent findings related to aspects of the intestinal mucosa during acute GVHD.

157 d IL-17A in response to curli fibrils in the intestinal mucosa during S. Typhimurium infection.

158 plification of inflammatory responses in the intestinal mucosa during serotype Typhimurium infection.

159 ed for in vitro dissolution, mucoadhesion to intestinal mucosa, enhancement of drug absorption in vit

160 The mammalian intestinal mucosa exhibits a spectrum of responses after

161 The intestinal mucosa exists in dynamic balance with trillio

162 Mice overexpressing progastrin (PG) in intestinal mucosa (fatty acid-binding protein (Fabp)-PG

163 ve immune responses that are elicited in the intestinal mucosa following ricin exposure and will prov

164 intestinal stem cells maintains a functional intestinal mucosa for a lifetime.

165 (-) cotransporter 1 might be a target in the intestinal mucosa for treatment of secretory diarrheas.

166 The intestinal mucosa forms the first line of defense agains

167 Dysregulated energy homeostasis in the intestinal mucosa frequently is observed in patients wit

168 d cells (ILCs) of the ILC22 type protect the intestinal mucosa from infection by secreting interleuki

169 Intestinal mucosa from patients with IBD exhibited reduc

170 ceptor and can be produced in the vertebrate intestinal mucosa from the oxidation of thiosulphate (S2

171 miRNA abundance varies dramatically in the intestinal mucosa, from 1 read per million to 250,000.

172 The intestinal mucosa functions is an immunologic organ that

173 key roles for the CX3CR1/CX3CL1 axis in the intestinal mucosa; further clarification of CX3CR1 funct

174 microbial activation and populate the human intestinal mucosa, generating functionally distinct CD10

175 treated patient does not guarantee that the intestinal mucosa has healed.

176 ripheral blood, the cellular response at the intestinal mucosa has never been directly assessed.

177 lly in critical tissue compartments like the intestinal mucosa, has not been completed.

178 ut, the physiological roles of MFG-E8 in the intestinal mucosa have not been explored.

179 ts of constitutively activated PI3K upon the intestinal mucosa have not been previously studied in an

180 xamining tolerogenic cell populations of the intestinal mucosa highlight the progress in understandin

181 In the intestinal mucosa, IL-17A was produced by three distinct

182 es in the short-circuit current (ISC) of the intestinal mucosa, impaired cAMP generation in acutely i

183 acterized fungi directly associated with the intestinal mucosa in healthy people and Crohn's disease

184 e production, and consequences for the large intestinal mucosa in humans.A randomized, double-blind,

185 r 5-HT affects growth and maintenance of the intestinal mucosa in mice.

186 e mesenteric lymph nodes (mLNs) and inflamed intestinal mucosa in patients with Crohn's disease (CD).

187 The intestinal mucosa in rats with cirrhosis showed a proinf

188 eated T84 cell monolayers and inflamed human intestinal mucosa in vivo.

189 duced endogenous miR-29b levels in the small intestinal mucosa increases cyclin-dependent kinase 2 (C

190 a virus that persistently replicates in the intestinal mucosa increases epithelial barrier permeabil

191 volved histology and immunohistochemistry of intestinal mucosa, indirect calorimetric measurements, w

192 CD8(+) T cells in the intestinal mucosa influence the HIV-associated pathogene

193 affects the development and function of the intestinal mucosa, influencing inflammatory responses in

194 play an important role in the maintenance of intestinal mucosa integrity.

195 Since immune activation at the level of intestinal mucosa is a hallmark of C. difficile-induced

196 Neutrophil (PMN) infiltration of the intestinal mucosa is a hallmark of tissue injury associa

197 The epithelium of the intestinal mucosa is a rapidly self-renewing tissue in t

198 s reveal that growth inhibition of the small intestinal mucosa is associated with increased expressio

199 into the structure and function of the small intestinal mucosa is becoming increasingly focused on th

200 The fetal intestinal mucosa is characterized by elevated Toll-like

201 Although the small intestinal mucosa is designed to transport large quantit

202 ever, it is assumed that crossing the bovine intestinal mucosa is important in order for M. avium sub

203 uridarum mouse infection model, we show that intestinal mucosa is infected via intranasal (i.n.) or p

204 teric glial cells (EGCs) residing within the intestinal mucosa is integrated into the dynamic microen

205 The intestinal mucosa is lined by a single layer of epitheli

206 e process by which the toxin transits across intestinal mucosa is not completely understood.

207 exaggerated inflammation in the skin and the intestinal mucosa leading to electrolyte disturbance, hy

208 molecular basis of gliadin interaction with intestinal mucosa leading to intestinal barrier impairme

209 nstrate that decreased expression of DMT1 in intestinal mucosa leads to compromised absorption and tr

210 Anti-microbial factors produced by the intestinal mucosa limit the translocation of both commen

211 , elevated copper levels are observed in the intestinal mucosa, liver, and blood.

212 terial to mice, GFP was observed in the mice intestinal mucosa, liver, and spleen in fluorescence and

213 ssembly, or "organoid," similar to the human intestinal mucosa, making it an ideal model for enteric

214 pathogenesis: infiltrated macrophages in the intestinal mucosa may promote local inflammation and tis

215 te lymphoid cells (ILC3s) that reside in the intestinal mucosa must function within a highly dynamic

216 along this axis in the histologically normal intestinal mucosa of Apc(1638N/+) mice before tumor deve

217 2-mediated antigen presentation in the small intestinal mucosa of Celiac Sprue patients therefore rep

218 be activated by dietary gluten in the small intestinal mucosa of celiac sprue patients, our findings

219 up isolates are a group of isolates from the intestinal mucosa of Crohn's disease patients that can i

220 arkedly fewer TH17 cells were present in the intestinal mucosa of Crtam(-/-) mice.

221 , that were differentially expressed between intestinal mucosa of fasted vs non-fasted mice.

222 lls toward deamidated gluten peptides in the intestinal mucosa of individuals with specific HLA-DQ ha

223 Thus, we analyzed the intestinal mucosa of JAM-A-deficient (JAM-A(-/-)) mice f

224 in vitro AMG uptake measurements into small intestinal mucosa of mice and human.

225 )-tagged gammadelta T cells within the small intestinal mucosa of mice infected with DsRed-labeled S

226 posed novel mechanism was operational in the intestinal mucosa of mice treated with dexamethasone or

227 r aim was to compare 5-HT disposition in the intestinal mucosa of neonatal and adult guinea pigs.

228 rowth factor-beta, which is increased in the intestinal mucosa of patients with active Crohn's diseas

229 The inflammatory environment in the intestinal mucosa of patients with CD contributes to the

230 In vivo, more CD14(+) macrophages from the intestinal mucosa of patients with CD than from controls

231 ammadelta TCR are more abundant in the small intestinal mucosa of patients with celiac disease (CD) c

232 FoxP3(+) T cells were identified in inflamed intestinal mucosa of patients with Crohn disease (CD), b

233 The intestinal mucosa of patients with inflammatory bowel di

234 Mediators released by the intestinal mucosa of patients with irritable bowel syndr

235 Conclusion: The intestinal mucosa of rats with cirrhosis acquires a proi

236 evealed that zonulin is overexpressed in the intestinal mucosa of subjects with celiac disease.

237 ected in leukocyte DNA and/or non-neoplastic intestinal mucosa of these patients.

238 nse to FOLFOX was partially sustained in the intestinal mucosa of VCMsh2(LoxP/G674D) animals.

239 Neutrophil (PMN) infiltration of the intestinal mucosa often leads to severe epithelial injur

240 nflammatory process specifically targets the intestinal mucosa, patients may present with gastrointes

241 vation play a key modulatory roles in normal intestinal mucosa permeability and in inflammatory and h

242 risingly, we detected donor-derived HSPCs in intestinal mucosa, Peyer's patches, mesenteric lymph nod

243 CD103(+) dendritic cells (DCs) in the intestinal mucosa play a crucial role in tolerance to co

244 Vaccination through large intestinal mucosa, previously proven protective for both

245 The intestinal mucosa promotes T cell responses that might b

246 ger RNA targeting relationships in the small intestinal mucosa provides insight into the molecular me

247 Migration to intestinal mucosa putatively depends on local activation

248 er loss of anti-TNF agents through ulcerated intestinal mucosa reduces the efficacy of these drugs in

249 Vagal afferents innervating the small intestinal mucosa regulate feeding, gastrointestinal (GI

250 However, the effects of LPS on the fish intestinal mucosa remain unknown.

251 ognition of bacterial amyloid fibrils in the intestinal mucosa represents a novel mechanism of immuno

252 Retention of lymphocytes in the intestinal mucosa requires specialized chemokine recepto

253 rs from Apc(Min/+) mice compared with normal intestinal mucosa, respectively.

254 Complete deletion of Klf5 from the intestinal mucosa resulted in neonatal lethality that co

255 Long-term increase in 5-HT in the intestinal mucosa results in increased 5-HT(3)R internal

256 Damage to the intestinal mucosa results in the translocation of microb

257 ixizumab, which induces immune tolerance, in intestinal mucosa samples from patients.

258 The animal and human intestinal mucosa secretes an assortment of compounds to

259 viremia; tonsil, mesentery lymph nodes, and intestinal mucosa served as major target sites of viral

260 The intestinal mucosa serves as a highly selective barrier t

261 The intestinal mucosa serves both as a conduit for the uptak

262 resence of one or more substances within the intestinal mucosa that directly modulate renal phosphate

263 wn about less typical endocrine cells in the intestinal mucosa that do not contain secretory granules

264 eased expression of Th2 cytokines within the intestinal mucosa that was significantly reduced in CCR6

265 on and differentiation of these cells in the intestinal mucosa, the functional consequences of cross-

266 ecause of their low permeability through the intestinal mucosa, the released protein would be soon de

267 The induction of protective immunity in the intestinal mucosa therefore represents a potentially des

268 odeoxycholate has a beneficial effect on the intestinal mucosa through an increase in resistance to a

269 M. avium subsp. paratuberculosis enters the intestinal mucosa through enterocytes in the absence of

270 e interaction between the microbiota and the intestinal mucosa through Toll-like receptors (TLRs) is

271 are key players in antimicrobial defense in intestinal mucosa, through interleukin 17 and interleuki

272 poptotic response of epithelial cells in the intestinal mucosa to cisplatin, which was defective in M

273 es to the permissiveness of both gastric and intestinal mucosa to colonization by persistent resident

274 tudy we report that villin is cleaved in the intestinal mucosa to generate a pro-apoptotic fragment t

275 /extracellular matrix from fresh gastric and intestinal mucosa to generate stroma-conditioned media.

276 and OT2 T cells reduced the capacity of the intestinal mucosa to make IFN-gamma and IL-17 after eith

277 activate sensory reflex mechanisms from the intestinal mucosa to stimulate or inhibit exocrine pancr

278 ns remain about how bacteria travel from the intestinal mucosa to the mesenteric lymph nodes (MLN), a

279 ur study provides a reference dataset of the intestinal mucosa transcriptional responses to chronic E

280 In the intestinal mucosa trefoil factors (TFF) and mucins (Muc)

281 Epithelial cells of the intestinal mucosa undergo a continual process of prolife

282 The intestinal mucosa undergoes a continual process of proli

283 ermined the miRNA transcriptome of the mouse intestinal mucosa using ultrahigh throughput sequencing.

284 ucose, hSGLT1-mediated AMG uptake into small intestinal mucosa was decreased by 40% to 50%.

285 TLR4 in the intestinal mucosa was inhibited with adenoviruses expres

286 Human intestinal mucosa was modeled using Caco-2 cells.

287 interleukin 2 and 4 mRNA within the inflamed intestinal mucosa was quantified by real-time PCR.

288 lous height/crypt depth (Vh/Cd) in the small-intestinal mucosa was significantly lower and the intrae

289 Since OPN is protective for the intestinal mucosa, we postulated that enhancing OPN expr

290 induction of p47 GTPase IGTP (Irgm3) in the intestinal mucosa were dependent upon functional MyD88.

291 erminals and their distribution in the small intestinal mucosa were examined by quantitatively compar

292 need to stimulate protective immunity in the intestinal mucosa, where HIV-1 infection causes severe C

293 nclude that B-cell development occurs in the intestinal mucosa, where it is regulated by extracellula

294 linase (NPP7) is an ecto-enzyme expressed in intestinal mucosa, which hydrolyses sphingomyelin (SM) t

295 e is a chronic inflammatory condition of the intestinal mucosa whose etiology is unclear but is likel

296 atic, and reversible remodeling of the small intestinal mucosa with significant villus shortening.

297 reduction of immune activation in blood and intestinal mucosa, with the latter maintained through 8

298 elicits acute neutrophil influx in the human intestinal mucosa within 1 or 2 days after infection, re

299 K974, drives rapid tumour clearance from the intestinal mucosa without effects on normal intestinal c

300 is critical for self-rejuvenation of normal intestinal mucosa, wound repair, and cancer metastasis.