ライフサイエンスコーパス: intestinal tract

1 elease of these therapeutic cells across the intestinal tract.

2 orce the functional specificity of the adult intestinal tract.

3 tion consistent with their origins along the intestinal tract.

4 seen in nerve fibres innervating the gastro-intestinal tract.

5 r force similar to peristaltic forces in the intestinal tract.

6 ronic relapsing inflammatory disorder of the intestinal tract.

7 thogens and initiate inflammation within the intestinal tract.

8 of antibiotic-mediated perturbations in the intestinal tract.

9 the microbial breakdown of tryptophan in the intestinal tract.

10 ucosal and epithelial tissues, including the intestinal tract.

11 ifferent bacterial species cohabit the human intestinal tract.

12 efect in ABCG2 abundance and function in the intestinal tract.

13 r jejuni is a natural commensal of the avian intestinal tract.

14 y VRE-colonized mice eliminates VRE from the intestinal tract.

15 liferative cell loss and inflammation in the intestinal tract.

16 lions of bacteria, most of which live in the intestinal tract.

17 hly complex bacterial assembly in the canine intestinal tract.

18 d a complete loss of ability to colonize the intestinal tract.

19 mplex microbial communities of the mammalian intestinal tract.

20 hey exit their usual reservoir in the host's intestinal tract.

21 both viable and sufficiently numerous in the intestinal tract.

22 n clearance of Trichinella spiralis from the intestinal tract.

23 role in bacterial colonization of the murine intestinal tract.

24 ghout the body with dense innervation of the intestinal tract.

25 against helminth parasitic infections in the intestinal tract.

26 to control release of these cells across the intestinal tract.

27 ohn's disease, an inflammatory malady of the intestinal tract.

28 ay facilitate the shedding of prions via the intestinal tract.

29 colonization, and pathogenicity in the human intestinal tract.

30 salivary gland, urinary bladder, and distal intestinal tract.

31 e of the epithelial barrier integrity in the intestinal tract.

32 tivation and production, particularly in the intestinal tract.

33 s key modulators of viral persistence in the intestinal tract.

34 osity, and stenosis at various levels of the intestinal tract.

35 stasis and specific disease processes in the intestinal tract.

36 RNA and protein from epithelial cells of the intestinal tract.

37 treatments were effective at sterilizing the intestinal tract.

38 ications-especially in the brain, heart, and intestinal tract.

39 mal tumour is the most common sarcoma of the intestinal tract.

40 h bridge innate and adaptive immunity in the intestinal tract.

41 can regulate viral regionalization along the intestinal tract.

42 e their function as bacterial tethers in the intestinal tract.

43 riants were impaired for growth in the chick intestinal tract.

44 tenuates chronic inflammatory disease in the intestinal tract.

45 was provided to cells within the guinea pig intestinal tract.

46 am-positive commensal bacterium of the human intestinal tract.

47 me was stochastically activated in the mouse intestinal tract.

48 al pathogens that enter the host through the intestinal tract.

49 characterized by chronic inflammation of the intestinal tract.

50 ts ability to survive and proliferate in the intestinal tract.

51 ibute important to microbial survival in the intestinal tract.

52 mor (GIST) is the most common sarcoma of the intestinal tract.

53 s suspected to complex uranium in gonads and intestinal tract.

54 of bioactive sphingolipid metabolites in the intestinal tract.

55 that binds phosphorus and bile acids in the intestinal tract.

56 ve in eradicating some MRSA strains from the intestinal tract.

57 vival in the changing conditions of the fish intestinal tract.

58 udying the process of translocation from the intestinal tract.

59 g roles for one or more effector Yops in the intestinal tract.

60 effects of bile salts and fatty acids in the intestinal tract.

61 mRNA was expressed most predominantly in the intestinal tract.

62 at risk for reduced immune tolerance in the intestinal tract.

63 nerated mice lacking CDHR2 expression in the intestinal tract.

64 tors that can acquire human viruses in their intestinal tract.

65 community structure along the length of the intestinal tract.

66 of the gut microbiota and gene expression in intestinal tract.

67 selectively reduce lymphocyte homing to the intestinal tract.

68 l delivery or direct administration into the intestinal tract.

69 ssessment of epithelial cancers of the upper intestinal tract.

70 s may be revealed amid the complexity of the intestinal tract.

71 on in both the aquatic environment and human intestinal tract.

72 physiology of the innervation of the gastro-intestinal tract.

73 mmals in structures such as the limbs or the intestinal tract.

74 enewal of the epithelial cells that line the intestinal tract.

75 d this effect is especially important in the intestinal tract.

76 mber of AIDS patients have M. avium in their intestinal tracts.

77 al and beneficial microbe in the vaginal and intestinal tracts.

78 e linked to the MIA dimerization observed in intestinal tracts.

79 generalists with relatively undifferentiated intestinal tracts.

80 In the intestinal tract, AMPR genes were involved in early inte

81 ations of 5-HT (1-10 muM) present within the intestinal tract and a limit of detection of 540 nM.

82 es that are abundant in the biliary tree and intestinal tract and are sometimes elevated in the urine

83 influence the bacterial colonisation of the intestinal tract and can be visualised non-destructively

84 , differentiated epithelial cells lining the intestinal tract and exhibits a tumor suppressive effect

85 e and adaptive leukocyte localization to the intestinal tract and GALT, and discuss their relevance t

86 pathogen exploits a unique niche within the intestinal tract and has developed unique strategies to

87 s zinc loss by regulating excretion into the intestinal tract and hence influences the dietary zinc r

88 impact immune cell function both within the intestinal tract and in peripheral tissues.

89 rom cell membranes, is abundant in the human intestinal tract and in processed foods.

90 specific pathogens are able to colonize the intestinal tract and invade, despite the presence of an

91 , endothelial, and muscle cells of the human intestinal tract and is activated in inflamed and fibrot

92 ic bacterium that metabolizes oxalate in the intestinal tract and is present in a large proportion of

93 tinal epithelial cells that lines the host's intestinal tract and leads to mucosal damage and inflamm

94 nal mucosa comprises the inner lining of the intestinal tract and maintains close proximity with comm

95 olerae cycle between the nutrient-rich human intestinal tract and nutrient-poor aquatic environments

96 essed prominently in epithelial cells of the intestinal tract and other organs exposed to the environ

97 olerae challenge reduces colonization of the intestinal tract and prevents cholera-like diarrhea.

98 erfringens vegetative cells sporulate in the intestinal tract and produce an enterotoxin (CPE) that i

99 Due to the limited absorption from the intestinal tract and sensitivity to degradation, oral va

100 tes have abundant memory CD4+ T cells in the intestinal tract and spleen and that these are selective

101 t link between the microbes that inhabit the intestinal tract and the developing brain.

102 as a barrier to the luminal contents of the intestinal tract and to facilitate the bidirectional tra

103 of milk micellar casein in the porcine upper intestinal tract and to match the outcome with the gastr

104 ies of indole, which is widespread in animal intestinal tracts and in the rhizosphere.

105 the respiratory and, to a lesser extent, the intestinal tracts and internal organs; with limited hist

106 ncer, noncancer diseases of the upper gastro-intestinal tract, and a healthy cohort.

107 ncer, noncancer diseases of the upper gastro-intestinal tract, and a healthy cohort.

108 ncer, noncancer diseases of the upper gastro-intestinal tract, and a healthy upper gastrointestinal t

109 es insights into how the microbiota of skin, intestinal tract, and airways influence immune responses

110 A was isolated systematically throughout the intestinal tract, and expression of Paneth cell alpha-cr

111 ogenic dendritic cells and Treg cells in the intestinal tract, and increased intestinal permeability.

112 However, during in vivo infection of the intestinal tract, and likely in the tumor microenvironme

113 GISTs occur throughout the intestinal tract, and most harbor an activating mutation

114 ric (125)I-PrP(Sc) were transported from the intestinal tract, and protein misfolding cyclic amplific

115 urate excretion in both the human kidney and intestinal tract, and provide insight into the importanc

116 23A1 is a major ascorbate transporter in the intestinal tract, and some of its genetic variants have

117 nly found in bile duct epithelial cells, the intestinal tract, and the cerebellum and is activated by

118 es spp. are the predominant organisms in the intestinal tract, and they also are important opportunis

119 colon in the event that the contents of the intestinal tract are purged following exposure to a path

120 g epithelial cells, like those that line the intestinal tract, are specialized for solute processing

121 K-10 exhibited limited permeation across the intestinal tract as assessed via a Caco-2 bidirectional

122 that Cftr is a tumor suppressor gene in the intestinal tract as Cftr mutant mice developed significa

123 omplex, and the second one in the gonads and intestinal tract, as a protein complex.

124 icPEDV-mut4 replicated in the intestinal tract, as revealed by detection of viral RNA

125 strong epidemiologic evidence that the human intestinal tract, as well as household pets, may be a re

126 tic parameter both for findings in the upper intestinal tract (AUC 0.730, 0.66-0.79) and for the colo

127 survive the harsh environment of a churning intestinal tract, bacteria attach to the host epithelium

128 a novel inflammation-promoting action in the intestinal tract, because loss of p53 or the upstream ac

129 n orally because they are broken down in the intestinal tract before they are absorbed.

130 REVIEW: Bacterial colonization of the infant intestinal tract begins at birth.

131 Epithelial cells lining the intestinal tract build an apical array of microvilli kno

132 PPARdelta is highly expressed in the intestinal tract but its physiological function in this

133 d induced during in vivo growth in the chick intestinal tract, but an absence of these genes did not

134 t junctions (TJs); many are expressed in the intestinal tract, but little is known about their functi

135 wed that there was colonization of the avian intestinal tract by a Campylobacter strain having a know

136 ficantly associated with colonization of the intestinal tract by Citrobacter freundii, Clostridium sp

137 Selective protection of the intestinal tract by EGLN inhibition enables potentially

138 III) complexes, to colonization of the mouse intestinal tract by Escherichia coli.

139 pylobacter jejuni colonization of the animal intestinal tract by mediating the efflux of bile acids.

140 concept for the selective protection of the intestinal tract by the EGLN inhibition to enable ablati

141 ged intestinal colonization along the entire intestinal tract by the streptomycin-resistant V. paraha

142 ged, which is able to translocate across the intestinal tract, causing systemic infection and abortio

143 was performed in all groups using the Human Intestinal Tract Chip.

144 nstrated an enhanced ability to colonize the intestinal tract compared to the ure mutant strain.

145 The mammalian intestinal tract contains a complex microbial ecosystem

146 causative bacteria can be found in patients' intestinal tracts days before dissemination, and cohort

147 expression of murine MGAT2 protein along the intestinal tract, determined its subcellular localizatio

148 The brain, heart and gastro-intestinal tract develop distinct left-right (LR) asymme

149 In type I mucosa (such as the intestinal tract), dimeric IgA secreted by local plasma

150 ability of E. faecium to colonize the murine intestinal tract during antibiotic treatment.

151 ence regulation and colonization of the host intestinal tract during infection.

152 ts the polarized epithelial cells lining the intestinal tract early in infection.

153 cause acute gastroenteritis and colonize the intestinal tract for prolonged periods.

154 in all six subspecies persisted in the mouse intestinal tract for several weeks in multiple repeat ex

155 shedding was detected in the respiratory and intestinal tract for up to 9 days.

156 E. faecalis colonizes the nematode intestinal tract, forming a persistent lethal infection.

157 itor FG-4592, which selectively protects the intestinal tract from radiation toxicity without protect

158 barrier separating the commensal-containing intestinal tract from the sterile interior.

159 r gram of compartment, the following rating: intestinal tract > gonads >> test, was obtained.

160 The endodermal lining of the adult gastro-intestinal tract harbours stem cells that are responsibl

161 mechanisms of T. foetus pathogenicity in the intestinal tract have not been examined.

162 In the intestinal tract, IL-22 activates STAT3 to promote intes

163 emia initiated from translocation across the intestinal tract in an immunocompromised host is substan

164 ribution of these populations throughout the intestinal tract in healthy individuals remains unclear.

165 ent, and can originate from flagellin in the intestinal tract in inflammatory conditions in the intes

166 en and RNA are detected throughout the small intestinal tract in jejunal and ileal tissue from one pe

167 um suggests that regional differences in the intestinal tract in the frequency and nature of secondar

168 microbial communities that reside within the intestinal tract in vertebrates are complex and dynamic.

169 Hence, different to the intestinal tract, in the spleen, Nkrp1g is selectively e

170 that Wnt11 is expressed throughout the mouse intestinal tract including the epithelial cells.

171 All components of the Drosophila intestinal tract, including the endodermal midgut and ec

172 In the intestinal tract, individual cells build thousands of mi

173 using a variety of diseases, both within the intestinal tract (intestinal pathogenic strains) and out

174 Early-life colonization of the intestinal tract is a dynamic process influenced by nume

175 The mammalian intestinal tract is a highly complex and compartmentaliz

176 cedure for reconstruction of the oesophageal-intestinal tract is a highly debated topic.

177 The intestinal tract is a lymphocyte-rich site that undergoe

178 Since the intestinal tract is a major site of early viral replicat

179 The intestinal tract is a primary barrier to invading pathog

180 The intestinal tract is a site of intense immune cell activi

181 The human intestinal tract is colonised by a complex community of

182 The mammalian intestinal tract is colonized by trillions of beneficial

183 The intestinal tract is in intimate contact with the commens

184 The intestinal tract is inhabited by a large and diverse com

185 The intestinal tract is inhabited by a large and diverse com

186 We show that mCRAMP expression in the intestinal tract is largely restricted to surface epithe

187 The intestinal tract is lined by a single layer of columnar

188 ormally harmless behavior of bacteria in the intestinal tract is maintained by community structure an

189 Maintenance of the epithelial barrier in the intestinal tract is necessary to protect the host from t

190 As occupation of the intestinal tract is often a prerequisite for ExPEC-media

191 gh concentrations of bile salts in the human intestinal tract is vital for the survival of enteric ba

192 this pathotype infects tissues distal to the intestinal tract, is a frequent cause of such infections

193 oderm, which predominantly gives rise to the intestinal tract, is competent to respond to FGFs by ind

194 uc5ac, a mucin not normally expressed in the intestinal tract, is induced in the cecum of mice resist

195 richia coli in its primary niche, the animal intestinal tract, is remarkably unexplored.

196 by mediating resistance to bile salts in the intestinal tract, is required for successful colonizatio

197 ng neovascularization in the murine inflamed intestinal tract (IT) during GvHD.

198 netically heterogeneous protist found in the intestinal tract (IT) of many vertebrates, and although

199 rains most of the substances coming from the intestinal tract, it may also play a role in the pathoge

200 t from host-pathogen interactions within the intestinal tract itself.

201 estinal microbiota by the premature infant's intestinal tract, leading to inflammation and injury.

202 The intestinal tract maintains proper function by replacing

203 ced samples of the Metagenomics of the Human Intestinal Tract (MetaHit) project with 1,018 previously

204 de, it has become clear that respiratory and intestinal tract microbiota are related to pathogenesis

205 sized to be initiated and perpetuated by the intestinal tract microflora.

206 , leflunomide restores gut motility, reduces intestinal tract narrowing, and increases intestinal cel

207 -) zebrafish increased gut motility, reduced intestinal tract narrowing, increased intestinal cell su

208 athogens that replicate possibly only in the intestinal tract, noroviruses have developed unique stra

209 le in transepithelial HCO3- secretion in the intestinal tract, null mutant (NBC1-/-) mice were prepar

210 The inoculation of ATCV-1 into the intestinal tract of 9-11-wk-old mice resulted in a subse

211 Its ability to persistently colonize the intestinal tract of a broad range of hosts, including fo

212 that C. albicans strains can persist in the intestinal tract of a healthy individual over a 4-year p

213 exist at the site of bacterial invasion, the intestinal tract of a host animal.

214 to function as a surrogate mucin within the intestinal tract of a stressed host by inhibiting key in

215 nd depletion of CD4(+) T cells occurs in the intestinal tract of acutely infected macaques.

216 differentiated tissues, including the gastro-intestinal tract of adult rats.

217 sting at the site of bacterial invasion, the intestinal tract of an animal host.

218 essential for commensal colonization of the intestinal tract of avian species and infection of human

219 Surprisingly, subspecies IIIb colonizes the intestinal tract of BALB/c mice normally yet does not sp

220 crobiota communities that inhabit the gastro intestinal tract of free-range, broiler and feral chicke

221 Examination of the intestinal tract of Glut5(-/-) mice fed a high fructose

222 Trillions of beneficial bacteria inhabit the intestinal tract of healthy mammals from birth.

223 ore-forming bacterium that infects the lower intestinal tract of humans and is the most common known

224 re Gram-negative anaerobes indigenous to the intestinal tract of humans, and they are important oppor

225 lar environments, aquatic reservoirs and the intestinal tract of humans.

226 nt members of the microbial community in the intestinal tract of infants, and studies have shown that

227 Xenorhabdus nematophila colonizes the intestinal tract of infective-juvenile (IJ) stage Steine

228 The preferential expression of DPP4 in the intestinal tract of insectivorous bats, suggests that tr

229 in lung tissue, and were recovered from the intestinal tract of intranasally inoculated ferrets.

230 ce microscopy (LSFM) to visualize H2S in the intestinal tract of live zebrafish.

231 The intestinal tract of mammals is colonized by a large numb

232 h factor (EGF) of maternal origin within the intestinal tract of mice correlated to the translocation

233 ed to lower levels of myeloperoxidase in the intestinal tract of mice developing GvHD and a reduced m

234 analogous populations of macrophages in the intestinal tract of rhesus macaques (Macaca mulatta) wit

235 he adaptation of Campylobacter jejuni in the intestinal tract of the chicken, a natural host and a ma

236 As part of its survival mechanism in the intestinal tract of the host, the worm produces a number

237 m its periplasm into the lumen of the gastro-intestinal tract of the host.

238 ajor virulence factor cholera toxin into the intestinal tract of the human host.

239 Ambergris, a waxy substance excreted by the intestinal tract of the sperm whale, has been a highly p

240 ransfer (HGT) between bacteria occurs in the intestinal tract of their animal hosts and facilitates b

241 x N-glycans are prominently expressed on the intestinal tract of various avian species.

242 of eight distinctive bacterial phylotypes in intestinal tracts of adult worker bees.

243 compounds, which are normally present in the intestinal tracts of animals.

244 gned to quantify SPI-1 expression within the intestinal tracts of animals.

245 s in humans and a commensal bacterium of the intestinal tracts of many wild and agriculturally signif

246 f 1,000 species of bacteria that inhabit the intestinal tracts of poultry and livestock.

247 ed Deltaproteobacteria widely distributed in intestinal tracts of termites and cockroaches.

248 against invading bacterial pathogens in the intestinal tract, on the skin or on the vaginal mucosa.

249 promote commensal colonization of the avian intestinal tract or invasion of human intestinal cells r

250 ong B cells than occurred within the spleen, intestinal tract, or mesenteric lymph nodes and were pre

251 cteroides fragilis, is a highly aerotolerant intestinal tract organism that has evolved a complex oxi

252 her living on exposed surfaces or within our intestinal tract, our microbial inhabitants produce a re

253 sed changes in the microbiota throughout the intestinal tract over the time course of infection.

254 e collection of all bacterial members in the intestinal tract, plays a key role in health.

255 of parenteral piperacillin excreted into the intestinal tract, preserving colonization resistance of

256 Gal-4) and Gal-8, which are expressed in the intestinal tract, recognize and kill human blood group a

257 symptoms of VM, such as abnormal dilation of intestinal tracts, reduced gut motility, feeding defects

258 glial cells residing within the walls of the intestinal tract, regulate intestinal motility, a well-c

259 ues, including the enterocytes that line the intestinal tract, remodel their apical surface during di

260 The surface epithelium lining the intestinal tract renews itself rapidly by a coordinated

261 (CD), a chronic inflammatory disease of the intestinal tract, report tertiary lymphoid organs presen

262 The mucosa of the intestinal tract represents a finely tuned system where

263 The intestinal tract represents a portal of entry for many i

264 ued to express a lethal phenotype within the intestinal tract reservoir-a hostile, nutrient scarce en

265 represented in nonhematopoietic cells of the intestinal tract, responds to microbial stimuli once bar

266 The infusion of feces into the intestinal tract shows great promise for treatment and m

267 helial cells of both the respiratory and the intestinal tract, similar to what has been reported for

268 In the intestinal tract, such derepression of toxin synthesis w

269 s intact and functional forms throughout the intestinal tract, suggesting that the peptides may media

270 gen dispersed more widely through the gastro-intestinal tract than intranasally delivered antigen and

271 or remitting, inflammatory disorders of the intestinal tract that although somewhat similar clinical

272 e of TSC that presented tumors of the gastro intestinal tract that are commonly unrelated to the dise

273 gan system with distinct functions along the intestinal tract that are critical for health.

274 ntitatively selective, radioprotector of the intestinal tract that is capable of enabling clinically

275 s an enteric bacterial pathogen of the mouse intestinal tract that triggers inflammatory responses re

276 spleens) of live mice, and levels within the intestinal tract (the presumed origin of the gas) were f

277 jejuni invade the cells that line the human intestinal tract, the bacterial proteins that enable thi

278 ed at high levels in epithelial cells of the intestinal tract, the lung, and in cells of the immune s

279 Like the human intestinal tract, the mosquito midgut contains a diverse

280 In the intestinal tract, these protrusions play central roles i

281 EHEC colonizes the intestinal tract through a range of virulence factors en

282 dicate that p53 promotes inflammation in the intestinal tract through suppression of epithelium-prote

283 to transport pathogenic Salmonella from the intestinal tract to the mesenteric lymph nodes.

284 t of uropathogenic Escherichia coli from the intestinal tract to the urinary tract.

285 dependent microvascular beds (such as gastro-intestinal tract) to plasma volume regulation.

286 ctly quantitate sPLA2 activity in the murine intestinal tract utilizing a fluorescent BODIPY-labeled

287 cs of dust particles in the lungs and gastro-intestinal tract via X-ray fluorescence (XRF) microscopy

288 s foetus but having a unique tropism for the intestinal tract was recognized as a significant cause o

289 the physicochemical conditions of the gastro-intestinal tract was used in association with a mathemat

290 differences in gene transcription along the intestinal tract were accompanied by major alterations i

291 for this receptor are largely unknown in the intestinal tract, where epithelial cells are normally ex

292 coli to thrive in the gallbladder and upper intestinal tract, where high bile concentrations are pro

293 , CaR has been identified in the stomach and intestinal tract, where it has been proposed to function

294 es suggest that in barrier sites such as the intestinal tract, where pathogen-associated molecular pa

295 ILCs were first identified in the intestinal tract, where they contribute to epithelial ba

296 eins leads to the release of peptides in the intestinal tract, where they may exert a variety of func

297 and that the released bacteria move into the intestinal tract, where they pass into the environment a

298 s, and the increasing differentiation of the intestinal tract, which also creates new niches for micr

299 s) are chronic inflammatory disorders of the intestinal tract with unknown multifactorial etiology th

300 mary reservoir for O157, which colonizes the intestinal tract without inducing any overt clinical sym