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1 oblastoma if administered locally via direct intra-arterial infusion.
2 man protein (rhVEGF-C; 500 microg) by direct intra-arterial infusion.
3 peripheral blood flow in CHF patients after intra-arterial infusion and after oral administration in
4 ar implantation appeared more effective than intra-arterial infusion, and mobilized peripheral blood
5 ycemia (300 mg/dL) created by dextrose (50%) intra-arterial infusion, and with coadministration of vi
6 ncept for CBL0137 and its administration via intra-arterial infusion as a superior treatment compared
7 5.0 mg/kg) were administered as a 10-minute intra-arterial infusion; control animals received vehicl
8 titute CBL0137 in the ILP protocol, using an intra-arterial infusion method, to safely achieve effect
11 responses (strain-gauge plethysmography) to intra-arterial infusion of a selective blocker of ETA re
12 studied the forearm blood flow responses to intra-arterial infusion of acetylcholine (7.5 to 30 micr
13 on plethysmography and blood sampling during intra-arterial infusion of acetylcholine, bradykinin, so
14 lusion plethysmography in response to serial intra-arterial infusion of acetylcholine, sodium nitropr
15 anges in vascular conductance (FVC) to local intra-arterial infusion of ACh (endothelium-dependent di
18 ar conductance (CalfVC) were measured during intra-arterial infusion of an alpha-adrenoreceptor agoni
20 the nanomolar range and were not reduced by intra-arterial infusion of an angiotensin-converting enz
22 alation study provides evidence of safety of intra-arterial infusion of autologous MSCs in patients w
24 is trial was to determine whether repetitive intra-arterial infusion of bone marrow mononuclear cells
25 (FBF) and net t-PA release before and during intra-arterial infusion of bradykinin (25 to 400 ng/min)
30 found in stroke animals that received local intra-arterial infusion of human PRP lysate (p's<0.05).
32 was evaluated by measuring the effects of an intra-arterial infusion of incremental doses of carbacho
33 on of K(IR) channels in quiescent muscle via intra-arterial infusion of KCl independently amplified t
34 elium-dependent vasodilation was assessed by intra-arterial infusion of methacholine (0.3 to 10 micro
36 with and without NO synthase inhibition via intra-arterial infusion of N(G) -monomethyl-L -arginine
37 t nitric oxide synthase (NOS) inhibition via intra-arterial infusion of N(G)-monomethyl-L-arginine (L
39 ium-independent vasodilation was measured by intra-arterial infusion of nitroprusside (0.3 to 10 micr
41 low (LBF, Doppler ultrasound) and leg VO2 to intra-arterial infusion of phentolamine (PHEN, alpha-adr
42 anges in vascular conductance (FVC) to local intra-arterial infusion of phenylephrine (PE; alpha(1) -
43 tra-arterial pressure via catheter) to local intra-arterial infusion of phenylephrine (PE; alpha1 -ad
44 ted changes in vascular conductance (FVC) to intra-arterial infusion of phenylephrine (PE; alpha1 -ag
46 1)-agonist) or NPY (Y1R-agonist) during: (1) intra-arterial infusion of sodium nitroprusside (SNP; ni
47 occlusion plethysmography before and during intra-arterial infusion of the arginase inhibitor N(omeg
48 resistance vessels (blood flow responses to intra-arterial infusion of the endothelium-dependent dil
49 hours after enalapril (20 mg) or placebo, an intra-arterial infusion of the NEP inhibitor thiorphan (
50 acologically enhanced with Mch or reduced by intra-arterial infusion of the nitric oxide inhibitor N(
51 y measuring the effects on venous tone of an intra-arterial infusion of the NO synthase inhibitor N-m
52 ed by use of forearm blood flow responses to intra-arterial infusions of acetylcholine (a vasodilator
53 e above parameters were also assessed during intra-arterial infusions of acetylcholine and bradykinin
54 Agonist-induced vasodilation to incremental intra-arterial infusions of acetylcholine and bradykinin
55 Forearm venous occlusion plethysmography and intra-arterial infusions of acetylcholine and N(G)-monom
58 d 18 black control subjects before and after intra-arterial infusions of acetylcholine, nitroprusside
59 ascular conductance (FVC) responses to local intra-arterial infusions of ACh, ATP, and sodium nitropr
60 ation, and forearm blood flow in response to intra-arterial infusions of endothelial-dependent and -i
61 w (Q, ultrasound Doppler) at rest and during intra-arterial infusions of endothelium-dependent (acety
63 s occlusion plethysmography before and after intra-arterial infusions of N(G)-monomethyl-l-arginine (
64 otocols, measuring leg vascular responses to intra-arterial infusions of NG-monomethyl-l-arginine (l-
65 sures were also recorded in seven men during intra-arterial infusions of normal saline, phentolamine
66 alpha- and beta-adrenoreceptor blockade via intra-arterial infusions of phentolamine and propranolol
67 and calculated vascular conductance (FVC) to intra-arterial infusions of phenylephrine (alpha(1)-agon
68 ascular conductance (FVC) responses to local intra-arterial infusions of phenylephrine (alpha(1)-agon
69 forearm vascular conductance (FVC) to local intra-arterial infusions of phenylephrine (PE; alpha(1)-
70 sis were randomly assigned (1:1:1) bilateral intra-arterial infusions of placebo on days 1 and 30 (n=
72 in-gauge plethysmography) responses to local intra-arterial infusions of tyramine (which evokes endog
73 d, first before start of, and then during an intra-arterial infusion to, the muscle preparation of th